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human foreskin fibroblast hff 1 cell line  (ATCC)


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    Structured Review

    ATCC human foreskin fibroblast hff 1 cell line
    Human Foreskin Fibroblast Hff 1 Cell Line, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1591 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/hff-1/HFF-1/10__2147_slash_ijn__s624901-85-0-9
    Average 99 stars, based on 1591 article reviews
    human foreskin fibroblast hff 1 cell line - by Bioz Stars, 2026-10
    99/100 stars

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    Related Articles

    Cell Culture:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Control:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Positive Control:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Negative Control:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Mutagenesis:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Expressing:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Generated:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Clone Assay:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Stable Transfection:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Transduction:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Mouse Assay:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Plasmid Preparation:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Transfection:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Irradiation:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma

    Modification:

    Article Title: Extracellular matrix abnormalities in Hutchinson–Gilford progeria fibroblasts: a specific defect in collagen IV and basement membrane architecture?
    Article Snippet: ance, a recognized feature of aged human skin [33] . Patient fibroblasts were established by outgrowth from skin biopsy as previously described [34] . Neonatal foreskin fibroblasts from Invitrogen and ATCC (HFF-1, SCRC-1041) were used as control line #1 and #2, respectively. Fibroblasts were cultured in DMEM/F12 supplemented with 10% FBS and 1% penicillin/streptomycin and controlled for mycoplasma



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    Image Search Results


    Journal: Archiv Der Pharmazie

    Article Title: Spirooxindole 5‐(Trifluoromethyl)benzothiazole Hybrids: Synthesis, Crystal Structure, and Antioxidant Activity

    doi: 10.1002/ardp.70343

    Figure Lengend Snippet: Cytotoxic effects of the selected compounds on HFF‐1 cells.

    Article Snippet: Human foreskin fibroblast cell line (HFF‐1, ATCC, SCRC‐1041) was used in the study.

    Techniques:

    (A) and (B) Cell viability was measured in FaDu, Cal27, HFF-1, and IMR90 cells. Cells were treated with different concentrations of PG3, cisplatin, or DMSO for 72 h. Luciferase activity was imaged using the IVIS Imaging System after treatment. Cell viability data were normalized to those of DMSO treatment control in each cell line, and data analyses were performed using Combenefit software. (C) Dose-response experiments: FaDu and Cal27 cells were treated with different doses of cisplatin for 24 hours, then western blot assays were performed using the indicated antibodies. (D) and (E) Time-course experiments: FaDu and Cal27 cells were treated with PG3 and cisplatin for the indicated periods, respectively, and then western blot assays were performed using the indicated antibodies. (F) and (G) FaDu and Cal27 cells were treated with PG3, cisplatin, and PG3+cisplatin for 16 hours, and then western blot assays were performed using the indicated antibodies.

    Journal: bioRxiv

    Article Title: Integrated stress response activation in combination PG3 and cisplatin-treated TP53 -mutated head and neck squamous carcinoma cells induces potent apoptotic response

    doi: 10.64898/2026.09.22.752907

    Figure Lengend Snippet: (A) and (B) Cell viability was measured in FaDu, Cal27, HFF-1, and IMR90 cells. Cells were treated with different concentrations of PG3, cisplatin, or DMSO for 72 h. Luciferase activity was imaged using the IVIS Imaging System after treatment. Cell viability data were normalized to those of DMSO treatment control in each cell line, and data analyses were performed using Combenefit software. (C) Dose-response experiments: FaDu and Cal27 cells were treated with different doses of cisplatin for 24 hours, then western blot assays were performed using the indicated antibodies. (D) and (E) Time-course experiments: FaDu and Cal27 cells were treated with PG3 and cisplatin for the indicated periods, respectively, and then western blot assays were performed using the indicated antibodies. (F) and (G) FaDu and Cal27 cells were treated with PG3, cisplatin, and PG3+cisplatin for 16 hours, and then western blot assays were performed using the indicated antibodies.

    Article Snippet: FaDu (R248L), Cal27 (H193L), PANC-1(R273H), HT29 (R273H), and normal fibroblast cell lines HFF-1, and IMR90 cells were purchased from ATCC.

    Techniques: Luciferase, Activity Assay, Imaging, Control, Software, Western Blot

    Cell viability was measured in FaDu, Cal27, HFF-1, and IMR90 cells. Cells were treated with different concentrations of PG3, cisplatin, or DMSO for 72 h. Luciferase activity was imaged using the IVIS Imaging System after treatment. Cell viability data were normalized to those of DMSO treatment control in each cell line, and data analyses were performed using Combenefit software.

    Journal: bioRxiv

    Article Title: Integrated stress response activation in combination PG3 and cisplatin-treated TP53 -mutated head and neck squamous carcinoma cells induces potent apoptotic response

    doi: 10.64898/2026.09.22.752907

    Figure Lengend Snippet: Cell viability was measured in FaDu, Cal27, HFF-1, and IMR90 cells. Cells were treated with different concentrations of PG3, cisplatin, or DMSO for 72 h. Luciferase activity was imaged using the IVIS Imaging System after treatment. Cell viability data were normalized to those of DMSO treatment control in each cell line, and data analyses were performed using Combenefit software.

    Article Snippet: FaDu (R248L), Cal27 (H193L), PANC-1(R273H), HT29 (R273H), and normal fibroblast cell lines HFF-1, and IMR90 cells were purchased from ATCC.

    Techniques: Luciferase, Activity Assay, Imaging, Control, Software