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Tokyo Chemical Industry tmp
Tmp, supplied by Tokyo Chemical Industry, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/tmps/tmp/pm41892445-178-0-7
Average 86 stars, based on 1 article reviews
tmp - by Bioz Stars, 2026-10
86/100 stars

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High Performance Liquid Chromatography:

Article Title: Impact of vacuum ultraviolet (VUV) intensity on the performance of VUV irradiation for the mineralization of trimethoprim in neutral media: Kinetics, mechanisms, and by-products formation.
Article Snippet: A comparative study on the mineralization of antibiotic trimethoprim (TMP) in neutral medium was investigated by applying irradiation with five types of ultraviolet lamps.. Among these lamps, the whole envelope of one lamp contained ordinary quartz, which could only transmit ultraviolet-C (UVC) light.. For the other four lamps, approximately one tenth, a quarter, a half, and full of envelopes were comprised of high-purity synthetic quartz, which can transmit both vacuum ultraviolet (VUV) and UVC light.

Article Title: Insights into the efficient mineralization of antibiotic trimethoprim in aqueous media by Fe 2+ catalytically enhanced vacuum-UV irradiation: Kinetics, mechanisms, and toxicity evaluation.
Article Snippet: The widespread existence of antibiotics in the environment has attracted growing concerns regarding the potential adverse effects on aquatic organisms, ecosystems, and human health even at low concentrations.. Extensive efforts have been devoted to developing new methods for effective elimination of antibiotics from wastewater.. Herein, a novel process of Fe2+ catalytically enhanced vacuum ultraviolet (VUV) irradiation was proposed as a promising approach for the removal of antibiotic trimethoprim (TMP) in water.

Article Title: Insights into enhanced degradation of antibiotic trimethoprim in water using a novel K doped g-C3N4 photocatalyst under vacuum-UV irradiation: Performance and mechanisms
Article Snippet: In this work, an innovative strategy combining vacuum ultraviolet (VUV) and K doping graphite carbon nitride (K-g-C3N4) was proposed for antibiotic removal in aqueous.. The properties of K-g-C3N4 were systematically characterized by XRD, FT-IR, BET, SEM, UV-vis DRS, and XPS, showing that K-g-C3N4 possessed a larger specific surface and richer functional groups than g-C3N4.. The catalytic efficiency of K-g-C3N4 was investigated for the trimethoprim (TMP) removal under VUV irradiation.

Article Title: Revitalizing Trimethoprim/Sulfamethoxazole via Nanotechnology for Improved Pharmacokinetics and Antibacterial Efficacy
Article Snippet: TMP (CAS: 738-70-5, >98.0%) was purchased from Tokyo Chemical Industry Co., Ltd. (TCI, Tokyo, Japan), while Ormetoprim (OMP) (CAS: 6981-18-6, >99.0%) was obtained from SCR-BIOTECH (Shanghai, China).

Article Title: Trimesic Acid as a Building Block for Ternary and Quaternary Salts and Salt Cocrystals
Article Snippet: Pyr, tmp, H 3 tma, bipy, ebipy, pbipy, and phpy were purchased from Tokyo Chemical Industry Europe.

Article Title: Revitalizing Trimethoprim/Sulfamethoxazole via Nanotechnology for Improved Pharmacokinetics and Antibacterial Efficacy.
Article Snippet: TMP (CAS: 738-70-5, >98.0%) was purchased from Tokyo Chemical Industry Co., Ltd. (TCI, Tokyo, Japan), while Ormetoprim (OMP) (CAS: 6981-18-6, >99.0%) was obtained from SCR-BIOTECH (Shanghai, China).

Article Title: Transesterification of methyl oleate for biolubricant production over K2CO3/activated carbon catalyst under conventional and microwave heating
Article Snippet: Trimethylolpropane fatty acid triester (TFATE) as an environmentally friendly biolubricant is a promising alternative to mineral-based lubricants.. However, the synthesis of TFATE by transesterification reaction has the issues of difficult catalyst recovery and slow reaction rate.. In this work, K2CO3/activated carbon (AC) catalyst was prepared and used in the transesterification of methyl oleate (MO) with trimethylolpropane (TMP) for TFATE production.

Refractive Index:

Article Title: Impact of vacuum ultraviolet (VUV) intensity on the performance of VUV irradiation for the mineralization of trimethoprim in neutral media: Kinetics, mechanisms, and by-products formation.
Article Snippet: A comparative study on the mineralization of antibiotic trimethoprim (TMP) in neutral medium was investigated by applying irradiation with five types of ultraviolet lamps.. Among these lamps, the whole envelope of one lamp contained ordinary quartz, which could only transmit ultraviolet-C (UVC) light.. For the other four lamps, approximately one tenth, a quarter, a half, and full of envelopes were comprised of high-purity synthetic quartz, which can transmit both vacuum ultraviolet (VUV) and UVC light.

Article Title: Insights into the efficient mineralization of antibiotic trimethoprim in aqueous media by Fe 2+ catalytically enhanced vacuum-UV irradiation: Kinetics, mechanisms, and toxicity evaluation.
Article Snippet: The widespread existence of antibiotics in the environment has attracted growing concerns regarding the potential adverse effects on aquatic organisms, ecosystems, and human health even at low concentrations.. Extensive efforts have been devoted to developing new methods for effective elimination of antibiotics from wastewater.. Herein, a novel process of Fe2+ catalytically enhanced vacuum ultraviolet (VUV) irradiation was proposed as a promising approach for the removal of antibiotic trimethoprim (TMP) in water.

Article Title: Insights into enhanced degradation of antibiotic trimethoprim in water using a novel K doped g-C3N4 photocatalyst under vacuum-UV irradiation: Performance and mechanisms
Article Snippet: In this work, an innovative strategy combining vacuum ultraviolet (VUV) and K doping graphite carbon nitride (K-g-C3N4) was proposed for antibiotic removal in aqueous.. The properties of K-g-C3N4 were systematically characterized by XRD, FT-IR, BET, SEM, UV-vis DRS, and XPS, showing that K-g-C3N4 possessed a larger specific surface and richer functional groups than g-C3N4.. The catalytic efficiency of K-g-C3N4 was investigated for the trimethoprim (TMP) removal under VUV irradiation.

Article Title: Revitalizing Trimethoprim/Sulfamethoxazole via Nanotechnology for Improved Pharmacokinetics and Antibacterial Efficacy
Article Snippet: TMP (CAS: 738-70-5, >98.0%) was purchased from Tokyo Chemical Industry Co., Ltd. (TCI, Tokyo, Japan), while Ormetoprim (OMP) (CAS: 6981-18-6, >99.0%) was obtained from SCR-BIOTECH (Shanghai, China).

Article Title: Trimesic Acid as a Building Block for Ternary and Quaternary Salts and Salt Cocrystals
Article Snippet: Pyr, tmp, H 3 tma, bipy, ebipy, pbipy, and phpy were purchased from Tokyo Chemical Industry Europe.

Article Title: Revitalizing Trimethoprim/Sulfamethoxazole via Nanotechnology for Improved Pharmacokinetics and Antibacterial Efficacy.
Article Snippet: TMP (CAS: 738-70-5, >98.0%) was purchased from Tokyo Chemical Industry Co., Ltd. (TCI, Tokyo, Japan), while Ormetoprim (OMP) (CAS: 6981-18-6, >99.0%) was obtained from SCR-BIOTECH (Shanghai, China).

Article Title: Transesterification of methyl oleate for biolubricant production over K2CO3/activated carbon catalyst under conventional and microwave heating
Article Snippet: Trimethylolpropane fatty acid triester (TFATE) as an environmentally friendly biolubricant is a promising alternative to mineral-based lubricants.. However, the synthesis of TFATE by transesterification reaction has the issues of difficult catalyst recovery and slow reaction rate.. In this work, K2CO3/activated carbon (AC) catalyst was prepared and used in the transesterification of methyl oleate (MO) with trimethylolpropane (TMP) for TFATE production.

Solvent:

Article Title: Impact of vacuum ultraviolet (VUV) intensity on the performance of VUV irradiation for the mineralization of trimethoprim in neutral media: Kinetics, mechanisms, and by-products formation.
Article Snippet: A comparative study on the mineralization of antibiotic trimethoprim (TMP) in neutral medium was investigated by applying irradiation with five types of ultraviolet lamps.. Among these lamps, the whole envelope of one lamp contained ordinary quartz, which could only transmit ultraviolet-C (UVC) light.. For the other four lamps, approximately one tenth, a quarter, a half, and full of envelopes were comprised of high-purity synthetic quartz, which can transmit both vacuum ultraviolet (VUV) and UVC light.

Article Title: Insights into the efficient mineralization of antibiotic trimethoprim in aqueous media by Fe 2+ catalytically enhanced vacuum-UV irradiation: Kinetics, mechanisms, and toxicity evaluation.
Article Snippet: The widespread existence of antibiotics in the environment has attracted growing concerns regarding the potential adverse effects on aquatic organisms, ecosystems, and human health even at low concentrations.. Extensive efforts have been devoted to developing new methods for effective elimination of antibiotics from wastewater.. Herein, a novel process of Fe2+ catalytically enhanced vacuum ultraviolet (VUV) irradiation was proposed as a promising approach for the removal of antibiotic trimethoprim (TMP) in water.

Article Title: Insights into enhanced degradation of antibiotic trimethoprim in water using a novel K doped g-C3N4 photocatalyst under vacuum-UV irradiation: Performance and mechanisms
Article Snippet: In this work, an innovative strategy combining vacuum ultraviolet (VUV) and K doping graphite carbon nitride (K-g-C3N4) was proposed for antibiotic removal in aqueous.. The properties of K-g-C3N4 were systematically characterized by XRD, FT-IR, BET, SEM, UV-vis DRS, and XPS, showing that K-g-C3N4 possessed a larger specific surface and richer functional groups than g-C3N4.. The catalytic efficiency of K-g-C3N4 was investigated for the trimethoprim (TMP) removal under VUV irradiation.

Article Title: Revitalizing Trimethoprim/Sulfamethoxazole via Nanotechnology for Improved Pharmacokinetics and Antibacterial Efficacy
Article Snippet: TMP (CAS: 738-70-5, >98.0%) was purchased from Tokyo Chemical Industry Co., Ltd. (TCI, Tokyo, Japan), while Ormetoprim (OMP) (CAS: 6981-18-6, >99.0%) was obtained from SCR-BIOTECH (Shanghai, China).

Article Title: Trimesic Acid as a Building Block for Ternary and Quaternary Salts and Salt Cocrystals
Article Snippet: Pyr, tmp, H 3 tma, bipy, ebipy, pbipy, and phpy were purchased from Tokyo Chemical Industry Europe.

Article Title: Revitalizing Trimethoprim/Sulfamethoxazole via Nanotechnology for Improved Pharmacokinetics and Antibacterial Efficacy.
Article Snippet: TMP (CAS: 738-70-5, >98.0%) was purchased from Tokyo Chemical Industry Co., Ltd. (TCI, Tokyo, Japan), while Ormetoprim (OMP) (CAS: 6981-18-6, >99.0%) was obtained from SCR-BIOTECH (Shanghai, China).

Article Title: Transesterification of methyl oleate for biolubricant production over K2CO3/activated carbon catalyst under conventional and microwave heating
Article Snippet: Trimethylolpropane fatty acid triester (TFATE) as an environmentally friendly biolubricant is a promising alternative to mineral-based lubricants.. However, the synthesis of TFATE by transesterification reaction has the issues of difficult catalyst recovery and slow reaction rate.. In this work, K2CO3/activated carbon (AC) catalyst was prepared and used in the transesterification of methyl oleate (MO) with trimethylolpropane (TMP) for TFATE production.



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( A ) 4 dpci <t>DMSO-treated</t> <t>and</t> <t>TGF-β</t> inhibitor-treated ventricular sections stained for MyHC (CMs, green), mCherry (macrophages, magenta), and DAPI (DNA, blue). Arrowheads point to mpeg1.1 :mCherryF + cells. ( B ) Number of mpeg1.1 :mCherryF + cells per mm 2 injured area in DMSO-treated ( n = 6) and TGFβ inhibitor-treated ( n = 5) ventricles at 4 dpci. ( C ) RT-qPCR analysis of tnfa, il6 and il1b mRNA levels in control (Ctrl) ( n = 4) and TGF-β inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( D ) RT-qPCR analysis of ccl20a.3 and cdaa mRNA levels in control (Ctrl) ( n = 5) and TGFβ inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( E ) 4 dpci sulf1 +/+ , sulf1 +/+ dexamethasone-treated (DEX), sulf1 −/− and sulf1 −/− dexamethasone-treated ventricular sections stained for pSmad3 (magenta) and DAPI (DNA, blue). Arrowheads point to pSmad3 + cells. ( F ) Number of pSmad3 + cells per mm 2 injured area in sulf1 +/+ ( n = 4), sulf1 +/+ dexamethasone-treated ( n = 4), sulf1 −/− ( n = 5) and sulf1 −/− dexamethasone-treated ( n = 6) ventricles at 4 dpci. ( G ) 30 dpci sulf1 −/− DMSO-treated and sulf1 −/− dexamethasone-treated ventricular sections stained with AFOG for muscle (yellow), collagen (blue), and fibrin (red). ( H ) Percentage representation of groups of different fibrotic area sizes at 30 dpci for cryoinjured sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles. Black dotted lines delineate the area with collagen and fibrin deposits. ( I ) Percentage of collagen/fibrin in fibrotic area in sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles at 30 dpci. Scale bars: 100 µm. White dashed lines delineate the injured area. Statistical tests: Nonparametric Mann–Whitney test ( C , D ), Student’s t test ( B , F , I ). For the boxplots, the center line indicates the median; the box bounds represent the 25th and 75th percentiles; the whiskers extend to the most extreme data points; and each point represents an independent measurement. .
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MedChemExpress tmp
( A ) 4 dpci <t>DMSO-treated</t> <t>and</t> <t>TGF-β</t> inhibitor-treated ventricular sections stained for MyHC (CMs, green), mCherry (macrophages, magenta), and DAPI (DNA, blue). Arrowheads point to mpeg1.1 :mCherryF + cells. ( B ) Number of mpeg1.1 :mCherryF + cells per mm 2 injured area in DMSO-treated ( n = 6) and TGFβ inhibitor-treated ( n = 5) ventricles at 4 dpci. ( C ) RT-qPCR analysis of tnfa, il6 and il1b mRNA levels in control (Ctrl) ( n = 4) and TGF-β inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( D ) RT-qPCR analysis of ccl20a.3 and cdaa mRNA levels in control (Ctrl) ( n = 5) and TGFβ inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( E ) 4 dpci sulf1 +/+ , sulf1 +/+ dexamethasone-treated (DEX), sulf1 −/− and sulf1 −/− dexamethasone-treated ventricular sections stained for pSmad3 (magenta) and DAPI (DNA, blue). Arrowheads point to pSmad3 + cells. ( F ) Number of pSmad3 + cells per mm 2 injured area in sulf1 +/+ ( n = 4), sulf1 +/+ dexamethasone-treated ( n = 4), sulf1 −/− ( n = 5) and sulf1 −/− dexamethasone-treated ( n = 6) ventricles at 4 dpci. ( G ) 30 dpci sulf1 −/− DMSO-treated and sulf1 −/− dexamethasone-treated ventricular sections stained with AFOG for muscle (yellow), collagen (blue), and fibrin (red). ( H ) Percentage representation of groups of different fibrotic area sizes at 30 dpci for cryoinjured sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles. Black dotted lines delineate the area with collagen and fibrin deposits. ( I ) Percentage of collagen/fibrin in fibrotic area in sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles at 30 dpci. Scale bars: 100 µm. White dashed lines delineate the injured area. Statistical tests: Nonparametric Mann–Whitney test ( C , D ), Student’s t test ( B , F , I ). For the boxplots, the center line indicates the median; the box bounds represent the 25th and 75th percentiles; the whiskers extend to the most extreme data points; and each point represents an independent measurement. .
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TargetMol recombinant human vascular endothelial growth factor 165
( A ) 4 dpci <t>DMSO-treated</t> <t>and</t> <t>TGF-β</t> inhibitor-treated ventricular sections stained for MyHC (CMs, green), mCherry (macrophages, magenta), and DAPI (DNA, blue). Arrowheads point to mpeg1.1 :mCherryF + cells. ( B ) Number of mpeg1.1 :mCherryF + cells per mm 2 injured area in DMSO-treated ( n = 6) and TGFβ inhibitor-treated ( n = 5) ventricles at 4 dpci. ( C ) RT-qPCR analysis of tnfa, il6 and il1b mRNA levels in control (Ctrl) ( n = 4) and TGF-β inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( D ) RT-qPCR analysis of ccl20a.3 and cdaa mRNA levels in control (Ctrl) ( n = 5) and TGFβ inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( E ) 4 dpci sulf1 +/+ , sulf1 +/+ dexamethasone-treated (DEX), sulf1 −/− and sulf1 −/− dexamethasone-treated ventricular sections stained for pSmad3 (magenta) and DAPI (DNA, blue). Arrowheads point to pSmad3 + cells. ( F ) Number of pSmad3 + cells per mm 2 injured area in sulf1 +/+ ( n = 4), sulf1 +/+ dexamethasone-treated ( n = 4), sulf1 −/− ( n = 5) and sulf1 −/− dexamethasone-treated ( n = 6) ventricles at 4 dpci. ( G ) 30 dpci sulf1 −/− DMSO-treated and sulf1 −/− dexamethasone-treated ventricular sections stained with AFOG for muscle (yellow), collagen (blue), and fibrin (red). ( H ) Percentage representation of groups of different fibrotic area sizes at 30 dpci for cryoinjured sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles. Black dotted lines delineate the area with collagen and fibrin deposits. ( I ) Percentage of collagen/fibrin in fibrotic area in sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles at 30 dpci. Scale bars: 100 µm. White dashed lines delineate the injured area. Statistical tests: Nonparametric Mann–Whitney test ( C , D ), Student’s t test ( B , F , I ). For the boxplots, the center line indicates the median; the box bounds represent the 25th and 75th percentiles; the whiskers extend to the most extreme data points; and each point represents an independent measurement. .
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( A ) 4 dpci <t>DMSO-treated</t> <t>and</t> <t>TGF-β</t> inhibitor-treated ventricular sections stained for MyHC (CMs, green), mCherry (macrophages, magenta), and DAPI (DNA, blue). Arrowheads point to mpeg1.1 :mCherryF + cells. ( B ) Number of mpeg1.1 :mCherryF + cells per mm 2 injured area in DMSO-treated ( n = 6) and TGFβ inhibitor-treated ( n = 5) ventricles at 4 dpci. ( C ) RT-qPCR analysis of tnfa, il6 and il1b mRNA levels in control (Ctrl) ( n = 4) and TGF-β inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( D ) RT-qPCR analysis of ccl20a.3 and cdaa mRNA levels in control (Ctrl) ( n = 5) and TGFβ inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( E ) 4 dpci sulf1 +/+ , sulf1 +/+ dexamethasone-treated (DEX), sulf1 −/− and sulf1 −/− dexamethasone-treated ventricular sections stained for pSmad3 (magenta) and DAPI (DNA, blue). Arrowheads point to pSmad3 + cells. ( F ) Number of pSmad3 + cells per mm 2 injured area in sulf1 +/+ ( n = 4), sulf1 +/+ dexamethasone-treated ( n = 4), sulf1 −/− ( n = 5) and sulf1 −/− dexamethasone-treated ( n = 6) ventricles at 4 dpci. ( G ) 30 dpci sulf1 −/− DMSO-treated and sulf1 −/− dexamethasone-treated ventricular sections stained with AFOG for muscle (yellow), collagen (blue), and fibrin (red). ( H ) Percentage representation of groups of different fibrotic area sizes at 30 dpci for cryoinjured sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles. Black dotted lines delineate the area with collagen and fibrin deposits. ( I ) Percentage of collagen/fibrin in fibrotic area in sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles at 30 dpci. Scale bars: 100 µm. White dashed lines delineate the injured area. Statistical tests: Nonparametric Mann–Whitney test ( C , D ), Student’s t test ( B , F , I ). For the boxplots, the center line indicates the median; the box bounds represent the 25th and 75th percentiles; the whiskers extend to the most extreme data points; and each point represents an independent measurement. .
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Fisher Scientific trimethoprim tmp
( A ) 4 dpci <t>DMSO-treated</t> <t>and</t> <t>TGF-β</t> inhibitor-treated ventricular sections stained for MyHC (CMs, green), mCherry (macrophages, magenta), and DAPI (DNA, blue). Arrowheads point to mpeg1.1 :mCherryF + cells. ( B ) Number of mpeg1.1 :mCherryF + cells per mm 2 injured area in DMSO-treated ( n = 6) and TGFβ inhibitor-treated ( n = 5) ventricles at 4 dpci. ( C ) RT-qPCR analysis of tnfa, il6 and il1b mRNA levels in control (Ctrl) ( n = 4) and TGF-β inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( D ) RT-qPCR analysis of ccl20a.3 and cdaa mRNA levels in control (Ctrl) ( n = 5) and TGFβ inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( E ) 4 dpci sulf1 +/+ , sulf1 +/+ dexamethasone-treated (DEX), sulf1 −/− and sulf1 −/− dexamethasone-treated ventricular sections stained for pSmad3 (magenta) and DAPI (DNA, blue). Arrowheads point to pSmad3 + cells. ( F ) Number of pSmad3 + cells per mm 2 injured area in sulf1 +/+ ( n = 4), sulf1 +/+ dexamethasone-treated ( n = 4), sulf1 −/− ( n = 5) and sulf1 −/− dexamethasone-treated ( n = 6) ventricles at 4 dpci. ( G ) 30 dpci sulf1 −/− DMSO-treated and sulf1 −/− dexamethasone-treated ventricular sections stained with AFOG for muscle (yellow), collagen (blue), and fibrin (red). ( H ) Percentage representation of groups of different fibrotic area sizes at 30 dpci for cryoinjured sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles. Black dotted lines delineate the area with collagen and fibrin deposits. ( I ) Percentage of collagen/fibrin in fibrotic area in sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles at 30 dpci. Scale bars: 100 µm. White dashed lines delineate the injured area. Statistical tests: Nonparametric Mann–Whitney test ( C , D ), Student’s t test ( B , F , I ). For the boxplots, the center line indicates the median; the box bounds represent the 25th and 75th percentiles; the whiskers extend to the most extreme data points; and each point represents an independent measurement. .
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( A ) 4 dpci <t>DMSO-treated</t> <t>and</t> <t>TGF-β</t> inhibitor-treated ventricular sections stained for MyHC (CMs, green), mCherry (macrophages, magenta), and DAPI (DNA, blue). Arrowheads point to mpeg1.1 :mCherryF + cells. ( B ) Number of mpeg1.1 :mCherryF + cells per mm 2 injured area in DMSO-treated ( n = 6) and TGFβ inhibitor-treated ( n = 5) ventricles at 4 dpci. ( C ) RT-qPCR analysis of tnfa, il6 and il1b mRNA levels in control (Ctrl) ( n = 4) and TGF-β inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( D ) RT-qPCR analysis of ccl20a.3 and cdaa mRNA levels in control (Ctrl) ( n = 5) and TGFβ inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( E ) 4 dpci sulf1 +/+ , sulf1 +/+ dexamethasone-treated (DEX), sulf1 −/− and sulf1 −/− dexamethasone-treated ventricular sections stained for pSmad3 (magenta) and DAPI (DNA, blue). Arrowheads point to pSmad3 + cells. ( F ) Number of pSmad3 + cells per mm 2 injured area in sulf1 +/+ ( n = 4), sulf1 +/+ dexamethasone-treated ( n = 4), sulf1 −/− ( n = 5) and sulf1 −/− dexamethasone-treated ( n = 6) ventricles at 4 dpci. ( G ) 30 dpci sulf1 −/− DMSO-treated and sulf1 −/− dexamethasone-treated ventricular sections stained with AFOG for muscle (yellow), collagen (blue), and fibrin (red). ( H ) Percentage representation of groups of different fibrotic area sizes at 30 dpci for cryoinjured sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles. Black dotted lines delineate the area with collagen and fibrin deposits. ( I ) Percentage of collagen/fibrin in fibrotic area in sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles at 30 dpci. Scale bars: 100 µm. White dashed lines delineate the injured area. Statistical tests: Nonparametric Mann–Whitney test ( C , D ), Student’s t test ( B , F , I ). For the boxplots, the center line indicates the median; the box bounds represent the 25th and 75th percentiles; the whiskers extend to the most extreme data points; and each point represents an independent measurement. .
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( A ) 4 dpci <t>DMSO-treated</t> <t>and</t> <t>TGF-β</t> inhibitor-treated ventricular sections stained for MyHC (CMs, green), mCherry (macrophages, magenta), and DAPI (DNA, blue). Arrowheads point to mpeg1.1 :mCherryF + cells. ( B ) Number of mpeg1.1 :mCherryF + cells per mm 2 injured area in DMSO-treated ( n = 6) and TGFβ inhibitor-treated ( n = 5) ventricles at 4 dpci. ( C ) RT-qPCR analysis of tnfa, il6 and il1b mRNA levels in control (Ctrl) ( n = 4) and TGF-β inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( D ) RT-qPCR analysis of ccl20a.3 and cdaa mRNA levels in control (Ctrl) ( n = 5) and TGFβ inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( E ) 4 dpci sulf1 +/+ , sulf1 +/+ dexamethasone-treated (DEX), sulf1 −/− and sulf1 −/− dexamethasone-treated ventricular sections stained for pSmad3 (magenta) and DAPI (DNA, blue). Arrowheads point to pSmad3 + cells. ( F ) Number of pSmad3 + cells per mm 2 injured area in sulf1 +/+ ( n = 4), sulf1 +/+ dexamethasone-treated ( n = 4), sulf1 −/− ( n = 5) and sulf1 −/− dexamethasone-treated ( n = 6) ventricles at 4 dpci. ( G ) 30 dpci sulf1 −/− DMSO-treated and sulf1 −/− dexamethasone-treated ventricular sections stained with AFOG for muscle (yellow), collagen (blue), and fibrin (red). ( H ) Percentage representation of groups of different fibrotic area sizes at 30 dpci for cryoinjured sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles. Black dotted lines delineate the area with collagen and fibrin deposits. ( I ) Percentage of collagen/fibrin in fibrotic area in sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles at 30 dpci. Scale bars: 100 µm. White dashed lines delineate the injured area. Statistical tests: Nonparametric Mann–Whitney test ( C , D ), Student’s t test ( B , F , I ). For the boxplots, the center line indicates the median; the box bounds represent the 25th and 75th percentiles; the whiskers extend to the most extreme data points; and each point represents an independent measurement. .
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86
Tokyo Chemical Industry tmp
( A ) 4 dpci <t>DMSO-treated</t> <t>and</t> <t>TGF-β</t> inhibitor-treated ventricular sections stained for MyHC (CMs, green), mCherry (macrophages, magenta), and DAPI (DNA, blue). Arrowheads point to mpeg1.1 :mCherryF + cells. ( B ) Number of mpeg1.1 :mCherryF + cells per mm 2 injured area in DMSO-treated ( n = 6) and TGFβ inhibitor-treated ( n = 5) ventricles at 4 dpci. ( C ) RT-qPCR analysis of tnfa, il6 and il1b mRNA levels in control (Ctrl) ( n = 4) and TGF-β inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( D ) RT-qPCR analysis of ccl20a.3 and cdaa mRNA levels in control (Ctrl) ( n = 5) and TGFβ inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( E ) 4 dpci sulf1 +/+ , sulf1 +/+ dexamethasone-treated (DEX), sulf1 −/− and sulf1 −/− dexamethasone-treated ventricular sections stained for pSmad3 (magenta) and DAPI (DNA, blue). Arrowheads point to pSmad3 + cells. ( F ) Number of pSmad3 + cells per mm 2 injured area in sulf1 +/+ ( n = 4), sulf1 +/+ dexamethasone-treated ( n = 4), sulf1 −/− ( n = 5) and sulf1 −/− dexamethasone-treated ( n = 6) ventricles at 4 dpci. ( G ) 30 dpci sulf1 −/− DMSO-treated and sulf1 −/− dexamethasone-treated ventricular sections stained with AFOG for muscle (yellow), collagen (blue), and fibrin (red). ( H ) Percentage representation of groups of different fibrotic area sizes at 30 dpci for cryoinjured sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles. Black dotted lines delineate the area with collagen and fibrin deposits. ( I ) Percentage of collagen/fibrin in fibrotic area in sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles at 30 dpci. Scale bars: 100 µm. White dashed lines delineate the injured area. Statistical tests: Nonparametric Mann–Whitney test ( C , D ), Student’s t test ( B , F , I ). For the boxplots, the center line indicates the median; the box bounds represent the 25th and 75th percentiles; the whiskers extend to the most extreme data points; and each point represents an independent measurement. .
Tmp, supplied by Tokyo Chemical Industry, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/tmps/tmp/pm41892445-178-0-7
Average 86 stars, based on 1 article reviews
tmp - by Bioz Stars, 2026-10
86/100 stars
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86
Inari Medical Inc o procedimento indicado foi tmp com o sistema clottriever
Radioscopia demonstra o <t>procedimento</t> de remoção de trombo da veia femoral profunda esquerda. (A) Inserido pela veia femoral comum direita, o introdutor longo 6Fr x 55cm foi posicionado na origem da veia femoral profunda esquerda. Pelo introdutor longo, o cateter Fogarty n° 4 foi inserido, posicionado no segmento distal da veia femoral comum esquerda e insuflado com contraste diluído em solução fisiológica; (B) O <t>sistema</t> <t>ClotTriever®</t> foi posicionado aberto no segmento ilíaco esquerdo e o conjunto introdutor-Fogarty foi tracionado até a veia femoral comum.
O Procedimento Indicado Foi Tmp Com O Sistema Clottriever, supplied by Inari Medical Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/tmps/clottriever+system/pmc13021007-78-0-9
Average 86 stars, based on 1 article reviews
o procedimento indicado foi tmp com o sistema clottriever - by Bioz Stars, 2026-10
86/100 stars
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Image Search Results


( A ) 4 dpci DMSO-treated and TGF-β inhibitor-treated ventricular sections stained for MyHC (CMs, green), mCherry (macrophages, magenta), and DAPI (DNA, blue). Arrowheads point to mpeg1.1 :mCherryF + cells. ( B ) Number of mpeg1.1 :mCherryF + cells per mm 2 injured area in DMSO-treated ( n = 6) and TGFβ inhibitor-treated ( n = 5) ventricles at 4 dpci. ( C ) RT-qPCR analysis of tnfa, il6 and il1b mRNA levels in control (Ctrl) ( n = 4) and TGF-β inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( D ) RT-qPCR analysis of ccl20a.3 and cdaa mRNA levels in control (Ctrl) ( n = 5) and TGFβ inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( E ) 4 dpci sulf1 +/+ , sulf1 +/+ dexamethasone-treated (DEX), sulf1 −/− and sulf1 −/− dexamethasone-treated ventricular sections stained for pSmad3 (magenta) and DAPI (DNA, blue). Arrowheads point to pSmad3 + cells. ( F ) Number of pSmad3 + cells per mm 2 injured area in sulf1 +/+ ( n = 4), sulf1 +/+ dexamethasone-treated ( n = 4), sulf1 −/− ( n = 5) and sulf1 −/− dexamethasone-treated ( n = 6) ventricles at 4 dpci. ( G ) 30 dpci sulf1 −/− DMSO-treated and sulf1 −/− dexamethasone-treated ventricular sections stained with AFOG for muscle (yellow), collagen (blue), and fibrin (red). ( H ) Percentage representation of groups of different fibrotic area sizes at 30 dpci for cryoinjured sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles. Black dotted lines delineate the area with collagen and fibrin deposits. ( I ) Percentage of collagen/fibrin in fibrotic area in sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles at 30 dpci. Scale bars: 100 µm. White dashed lines delineate the injured area. Statistical tests: Nonparametric Mann–Whitney test ( C , D ), Student’s t test ( B , F , I ). For the boxplots, the center line indicates the median; the box bounds represent the 25th and 75th percentiles; the whiskers extend to the most extreme data points; and each point represents an independent measurement. .

Journal: EMBO Reports

Article Title: Sulf1 regulates inflammation via modulation of TGF-β dependent epicardial activation

doi: 10.1038/s44319-026-00910-5

Figure Lengend Snippet: ( A ) 4 dpci DMSO-treated and TGF-β inhibitor-treated ventricular sections stained for MyHC (CMs, green), mCherry (macrophages, magenta), and DAPI (DNA, blue). Arrowheads point to mpeg1.1 :mCherryF + cells. ( B ) Number of mpeg1.1 :mCherryF + cells per mm 2 injured area in DMSO-treated ( n = 6) and TGFβ inhibitor-treated ( n = 5) ventricles at 4 dpci. ( C ) RT-qPCR analysis of tnfa, il6 and il1b mRNA levels in control (Ctrl) ( n = 4) and TGF-β inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( D ) RT-qPCR analysis of ccl20a.3 and cdaa mRNA levels in control (Ctrl) ( n = 5) and TGFβ inhibitor-treated ( n = 5) cryoinjured ventricles at 4 dpci. ( E ) 4 dpci sulf1 +/+ , sulf1 +/+ dexamethasone-treated (DEX), sulf1 −/− and sulf1 −/− dexamethasone-treated ventricular sections stained for pSmad3 (magenta) and DAPI (DNA, blue). Arrowheads point to pSmad3 + cells. ( F ) Number of pSmad3 + cells per mm 2 injured area in sulf1 +/+ ( n = 4), sulf1 +/+ dexamethasone-treated ( n = 4), sulf1 −/− ( n = 5) and sulf1 −/− dexamethasone-treated ( n = 6) ventricles at 4 dpci. ( G ) 30 dpci sulf1 −/− DMSO-treated and sulf1 −/− dexamethasone-treated ventricular sections stained with AFOG for muscle (yellow), collagen (blue), and fibrin (red). ( H ) Percentage representation of groups of different fibrotic area sizes at 30 dpci for cryoinjured sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles. Black dotted lines delineate the area with collagen and fibrin deposits. ( I ) Percentage of collagen/fibrin in fibrotic area in sulf1 −/− DMSO-treated ( n = 6) and sulf1 −/− dexamethasone-treated ( n = 5) ventricles at 30 dpci. Scale bars: 100 µm. White dashed lines delineate the injured area. Statistical tests: Nonparametric Mann–Whitney test ( C , D ), Student’s t test ( B , F , I ). For the boxplots, the center line indicates the median; the box bounds represent the 25th and 75th percentiles; the whiskers extend to the most extreme data points; and each point represents an independent measurement. .

Article Snippet: For the TGF-β inhibitor experiment, fish were incubated in system water supplemented with TGF-β inhibitor (Cat. no. SB431542, 50 mg BOC Sciences) or DMSO at a concentration of 10 μM per fish starting 1 day prior to cryoinjuries until 4 dpci when the hearts were extracted.

Techniques: Staining, Quantitative RT-PCR, Analysis, Control, MANN-WHITNEY

Radioscopia demonstra o procedimento de remoção de trombo da veia femoral profunda esquerda. (A) Inserido pela veia femoral comum direita, o introdutor longo 6Fr x 55cm foi posicionado na origem da veia femoral profunda esquerda. Pelo introdutor longo, o cateter Fogarty n° 4 foi inserido, posicionado no segmento distal da veia femoral comum esquerda e insuflado com contraste diluído em solução fisiológica; (B) O sistema ClotTriever® foi posicionado aberto no segmento ilíaco esquerdo e o conjunto introdutor-Fogarty foi tracionado até a veia femoral comum.

Journal: Jornal Vascular Brasileiro

Article Title: Percutaneous mechanical thrombectomy in a patient with extensive deep vein thrombosis: pioneering surgery in the northern region

doi: 10.1590/1677-5449.202501162

Figure Lengend Snippet: Radioscopia demonstra o procedimento de remoção de trombo da veia femoral profunda esquerda. (A) Inserido pela veia femoral comum direita, o introdutor longo 6Fr x 55cm foi posicionado na origem da veia femoral profunda esquerda. Pelo introdutor longo, o cateter Fogarty n° 4 foi inserido, posicionado no segmento distal da veia femoral comum esquerda e insuflado com contraste diluído em solução fisiológica; (B) O sistema ClotTriever® foi posicionado aberto no segmento ilíaco esquerdo e o conjunto introdutor-Fogarty foi tracionado até a veia femoral comum.

Article Snippet: O procedimento indicado foi TMP com o sistema ClotTriever® (Inari Medical, Inc., EUA), abrangendo os segmentos cavo-ilíaco e fêmoro-poplíteo do MIE.

Techniques: