mitochondrial membrane 20 tomm20 (Proteintech)
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mitochondrial membrane 20 tomm20
Mitochondrial Membrane 20 Tomm20, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1337 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/tom20/TOM20+Antibody/pm41956485-102-47-52
Average 96 stars, based on 1337 article reviews
Mitochondrial Membrane 20 Tomm20, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1337 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/tom20/TOM20+Antibody/pm41956485-102-47-52
Average 96 stars, based on 1337 article reviews
mitochondrial membrane 20 tomm20 - by Bioz Stars,
2026-10
96/100 stars
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Immunofluorescence:Article Title: Pannexin1 promotes cellular senescence by triggering mtDNA release to activate the cGAS-STING pathway. Article Snippet: Cellular senescence is a pivotal driver of the transition from acute kidney injury (AKI) to chronic kidney disease (CKD).. We previously identified Pannexin 1 (Panx1) as an endoplasmic reticulum (ER)-resident calcium (Ca2+) leak channel that promotes renal tubular senescence; however, the precise downstream effectors remain elusive.. Here, we establish a novel mechanistic link between Panx1 and the cGAS-STING pathway. Staining:Article Title: Pannexin1 promotes cellular senescence by triggering mtDNA release to activate the cGAS-STING pathway. Article Snippet: Cellular senescence is a pivotal driver of the transition from acute kidney injury (AKI) to chronic kidney disease (CKD).. We previously identified Pannexin 1 (Panx1) as an endoplasmic reticulum (ER)-resident calcium (Ca2+) leak channel that promotes renal tubular senescence; however, the precise downstream effectors remain elusive.. Here, we establish a novel mechanistic link between Panx1 and the cGAS-STING pathway. other:Article Title: cGAS-STING/HMGB1-mediated senescence induced by LRRK2 accelerates cartilage degeneration in osteoarthritis. Article Snippet: LRRK2 antibody (#A17253) was obtained from Abclonal Antibody (Wuhan, China). Article Title: cGAS-STING/HMGB1-mediated senescence induced by LRRK2 accelerates cartilage degeneration in osteoarthritis Article Snippet: Anti-HMGB1 (#CPA7037) was obtained from Cohesion Biosciences (London, UK). Incubation:Article Title: Mitophagy Activation via the YAP/Parkin Pathway Underlies the Neuroprotective Action of Tetramethylpyrazine in Cerebral Ischemia/Reperfusion Injury. Article Snippet: .. Brain sections and cells were permeabilized with 0.3% Triton X-100, blocked with 5% bovine serum albumin, and incubated overnight at 4 ◦C with primary antibodies against NeuN (1:500; Huabio, Woburn, MA, USA; catalog no. HA601397), LC3 (1:500; Proteintech; catalog no. 14600-1-AP), Article Title: FABP5 attenuates pulmonary fibrosis by regulating lipid metabolism in type II alveolar epithelial cells. Article Snippet: Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive disease resulting from alveolar epithelial cell injury.. Senescence and dysfunction of alveolar epithelial type II (AT2) cells are key drivers of IPF pathogenesis.. Given that metabolism underpins cellular homeostasis and AT2 cell function critically depends on lipid metabolism, we investigated the role of fatty acid binding protein 5 (FABP5), a key regulator of intracellular fatty acid trafficking. Article Title: ADGRG1-targeted hypoxia preconditioned extracellular vesicles ameliorate intervertebral disc degeneration by delivering taurine to disrupt the oxidative stress feedback loop-driven ferroptosis in nucleus pulposus cells Article Snippet: .. After permeabilization and blocking with 10% goat serum containing 0.2% Triton X-100, the sections were incubated with primary antibodies against ADGRG1 (1:50; sc-390192, Santa Cruz Biotechnology), TOM20( Article Title: Progressive mitochondrial and autophagic dysfunction during RGC development driven by a LHON-associated mitochondrial tRNA Thr mutation. Article Snippet: Leber's hereditary optic neuropathy (LHON) is a genetically inherited disease of the eye triggered by mtDNA mutations, leading to degeneration of RGCs.. We previously reported that the mitochondrial tRNAThr (MT-TT) 15927G > A homoplasmic mutation disrupted the base pairing (28C-42G) conserved in the anticodon stem of tRNAThr, impairing t6A modification, aminoacylation, and steady-state tRNAThr levels, ultimately resulting in mitochondrial dysfunction.. However, the absence of suitable animal and cell models for LHON has delayed efforts to elucidate disease pathophysiology, particularly tissue-specific effects. Cell-Signaling:Article Title: FABP5 attenuates pulmonary fibrosis by regulating lipid metabolism in type II alveolar epithelial cells. Article Snippet: Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive disease resulting from alveolar epithelial cell injury.. Senescence and dysfunction of alveolar epithelial type II (AT2) cells are key drivers of IPF pathogenesis.. Given that metabolism underpins cellular homeostasis and AT2 cell function critically depends on lipid metabolism, we investigated the role of fatty acid binding protein 5 (FABP5), a key regulator of intracellular fatty acid trafficking. Article Title: Progressive mitochondrial and autophagic dysfunction during RGC development driven by a LHON-associated mitochondrial tRNA Thr mutation. Article Snippet: Leber's hereditary optic neuropathy (LHON) is a genetically inherited disease of the eye triggered by mtDNA mutations, leading to degeneration of RGCs.. We previously reported that the mitochondrial tRNAThr (MT-TT) 15927G > A homoplasmic mutation disrupted the base pairing (28C-42G) conserved in the anticodon stem of tRNAThr, impairing t6A modification, aminoacylation, and steady-state tRNAThr levels, ultimately resulting in mitochondrial dysfunction.. However, the absence of suitable animal and cell models for LHON has delayed efforts to elucidate disease pathophysiology, particularly tissue-specific effects. Blocking Assay:Article Title: ADGRG1-targeted hypoxia preconditioned extracellular vesicles ameliorate intervertebral disc degeneration by delivering taurine to disrupt the oxidative stress feedback loop-driven ferroptosis in nucleus pulposus cells Article Snippet: .. After permeabilization and blocking with 10% goat serum containing 0.2% Triton X-100, the sections were incubated with primary antibodies against ADGRG1 (1:50; sc-390192, Santa Cruz Biotechnology), TOM20( |