anti rad1 (Proteintech)
91
Structured Review
Proteintech
anti rad1
Anti Rad1, supplied by Proteintech, used in various techniques. Bioz Stars score: 91/100, based on 6 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rad1/RAD1+Antibody/10__1016_slash_j__phymed__2026__157899-66-11-23
Average 91 stars, based on 6 article reviews
Anti Rad1, supplied by Proteintech, used in various techniques. Bioz Stars score: 91/100, based on 6 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rad1/RAD1+Antibody/10__1016_slash_j__phymed__2026__157899-66-11-23
Average 91 stars, based on 6 article reviews
anti rad1 - by Bioz Stars,
2026-10
91/100 stars
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In Vitro:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Binding Assay:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Purification:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Recombinant:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Incubation:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Western Blot:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Modification:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Irradiation:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Immunoprecipitation:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Control:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Negative Control:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Stable Transfection:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Expressing:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Mutagenesis:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. In Situ:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. Staining:Article Title: DNA damage-induced translocation of mitochondrial factor HIGD1A into the nucleus regulates homologous recombination and radio/chemo-sensitivity. Article Snippet: HIGD1A is an important mitochondrial protein recently shown to have a novel nuclear localization under severe stress.. However, whether this protein is also associated with the DNA damage response has rarely been studied.. Here, we reported that DSBsinduced the translocation of mitochondrial HIGD1A to the nucleus is dependent on nuclear pore complex (NPCs), which finally promotes HR and radio/chemo-resistance. |