pdh (Proteintech)
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Proteintech
pdh
Pdh, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 13 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/pdh/PPM2C+Antibody/pm41003915-76-85-86
Average 93 stars, based on 13 article reviews
Pdh, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 13 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/pdh/PPM2C+Antibody/pm41003915-76-85-86
Average 93 stars, based on 13 article reviews
pdh - by Bioz Stars,
2026-10
93/100 stars
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SDS Page:Article Title: FGF21 protects against HFpEF by improving cardiac mitochondrial bioenergetics in mice. Article Snippet: The supernatant was used to determine total protein concentration with the BCA assay (23227, Thermo Scientific, USA). .. Equal amounts (20μg) of complete protein were subjected to SDS-PAGE and transferred onto polyvinylidene fluoride (PVDF) membranes (IPVH00010, Millipore, Germany).Membraneswere blockedwith 5%milk and then Purification:Article Title: Formononetin attenuates high glucose-induced neurotoxicity by negatively regulating oxidative stress and mitochondrial dysfunction in Schwann cells via activation of SIRT3. Article Snippet: High glucose induces Schwann cells death and neurotoxicity.. Formononetin was originally found in Astragalus membranaceus and showed anti-tumor and anti-neuroinflammation properties.. The aim of this study is to explore the molecular mechanism underlying the neuroprotective effects of formononetin and identify its direct protein target. Generated:Article Title: Formononetin attenuates high glucose-induced neurotoxicity by negatively regulating oxidative stress and mitochondrial dysfunction in Schwann cells via activation of SIRT3. Article Snippet: High glucose induces Schwann cells death and neurotoxicity.. Formononetin was originally found in Astragalus membranaceus and showed anti-tumor and anti-neuroinflammation properties.. The aim of this study is to explore the molecular mechanism underlying the neuroprotective effects of formononetin and identify its direct protein target. Pyrolysis Gas Chromatography:Article Title: Formononetin attenuates high glucose-induced neurotoxicity by negatively regulating oxidative stress and mitochondrial dysfunction in Schwann cells via activation of SIRT3. Article Snippet: High glucose induces Schwann cells death and neurotoxicity.. Formononetin was originally found in Astragalus membranaceus and showed anti-tumor and anti-neuroinflammation properties.. The aim of this study is to explore the molecular mechanism underlying the neuroprotective effects of formononetin and identify its direct protein target. Incubation:Article Title: FGF21 protects against HFpEF by improving cardiac mitochondrial bioenergetics in mice Article Snippet: Equal amounts (20 μg) of complete protein were subjected to SDS-PAGE and transferred onto polyvinylidene fluoride (PVDF) membranes (IPVH00010, Millipore, Germany). .. Membranes were blocked with 5% milk and then incubated with primary Article Title: FTO-Mediated m6A Demethylation of MZF1 Regulates DECR1 to Promote Fatty Acid Oxidation and Exacerbate Myocardial Ischemia/Reperfusion Injury : FTO-Mediated m6A Demethylation of MZF1 Enhances Fatty Acid Oxidation and Aggravates Myocardial I/R Injury. Article Snippet: Myocardial ischemia-reperfusion injury (MIRI) is a major clinical challenge, marked by metabolic disruptions and cellular damage following the restoration of blood flow after ischemia.. During ischemia, the heart shifts from fatty acid metabolism to glucose metabolism, leading to metabolic abnormalities, including reduced intracellular pH, ion disturbances, cell swelling, and apoptosis.. Upon reperfusion, fatty acid β-oxidation resumes, becoming the dominant energy source, which induces excessive oxidative stress and aggravates myocardial injury. other:Article Title: PDK4 rescues high-glucose-induced senescent fibroblasts and promotes diabetic wound healing through enhancing glycolysis and regulating YAP and JNK pathway Article Snippet: Antibodies P53 (10442-1-AP), PAI-1 (13801-1-AP), PDK4 (12949-1-AP), p-PDH (29582-1-AP), PDH (18068-1-AP), P21 (10355-1-AP), P16 INK4a (10883-1-AP), MMP3 (17873-1-AP), β-actin (20536-1-AP), β-tubulin (10094-1-AP), Vimentin (10366-1-AP), IL-1β (16806-1-AP), IL-6 (21865-1-AP), CTGF (25474-1-AP), JNK (66210-1-Ig), and Article Title: PDK4 rescues high-glucose-induced senescent fibroblasts and promotes diabetic wound healing through enhancing glycolysis and regulating YAP and JNK pathway. Article Snippet: Antibodies P53 (10442-1-AP), PAI-1 (13801-1-AP), PDK4 (12949-1-AP), p-PDH (29582-1-AP), PDH (18068-1-AP), P21 (10355-1-AP), P16INK4a (10883-1-AP), MMP3 (17873-1-AP), β-actin (20536-1-AP), β-tubulin (10094- 1-AP), Vimentin (10366-1-AP), IL-1β (16806-1-AP), IL-6 (21865-1-AP), CTGF (25474-1-AP), JNK (66210-1-Ig), and Membrane:Article Title: FTO-Mediated m6A Demethylation of MZF1 Regulates DECR1 to Promote Fatty Acid Oxidation and Exacerbate Myocardial Ischemia/Reperfusion Injury : FTO-Mediated m6A Demethylation of MZF1 Enhances Fatty Acid Oxidation and Aggravates Myocardial I/R Injury. Article Snippet: Myocardial ischemia-reperfusion injury (MIRI) is a major clinical challenge, marked by metabolic disruptions and cellular damage following the restoration of blood flow after ischemia.. During ischemia, the heart shifts from fatty acid metabolism to glucose metabolism, leading to metabolic abnormalities, including reduced intracellular pH, ion disturbances, cell swelling, and apoptosis.. Upon reperfusion, fatty acid β-oxidation resumes, becoming the dominant energy source, which induces excessive oxidative stress and aggravates myocardial injury. |