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dmt1  (Proteintech)


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    Structured Review

    Proteintech dmt1
    Dmt1, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 144 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/dmt1/DMT1+Antibody/pmc12995813-217-24-25
    Average 96 stars, based on 144 article reviews
    dmt1 - by Bioz Stars, 2026-09
    96/100 stars

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    Related Articles

    Ubiquitin Proteomics:

    Article Title: CDC26 facilitates ferroptosis through SLC7A11 degradation and cell cycle arrest
    Article Snippet: .. The antibodies to CDC26 (14125-1-AP), CDK1 (19532-1-AP), CDK2 (10122-1-AP), CKD4 (11026-1-AP), CDK6 (14052-1-AP), C-Myc (10828-1-AP), Cyclin D1 (60186-1-Ig), P53 (10442-1-AP), PARP1 (13371-1-AP), CYP51A1 (13431-1-AP), LIAS (11577-1-AP), Flag (80801-2-RR), Ubiquitin (10201-2-AP), DMT1 (81609-1-RR) and TFRC (10084-2-AP) were obtained from Proteintech Biotechnology. .. The antibodies to GPX4 (F1580) and SLC7A11 (A5912) were purchased from Selleck Chemicals.

    other:

    Article Title: Aloperine Induces Ferroptosis of Colorectal Cancer Cells via the Nrf2 Pathway
    Article Snippet: DMT1 , 60-70 , 1:1000 , Rabbit , 20507-1-AP , Proteintech.

    Western Blot:

    Article Title: BDH2-driven lysosome-to-mitochondria iron transfer shapes ferroptosis vulnerability of the melanoma cell states.
    Article Snippet: .. Primary antibodies to MITF (1:1,000, MA5-14154) and OxPhos Human WB Antibody Cocktail (1:1,000, 45-8199) were purchased from Thermo Fisher Scientific; BDH2 (1:1,000, 27207-1-AP) and DMT1 (1:1,000, 20507-1-AP) were purchased from Proteintech; Sox10 (1:1,000, sc-365692), v-ATPase H (1:1,000, sc-166227) and v-ATPase A (1:1,000, sc-374475) were purchased from Santa Cruz Biotechnology; AXL (1:1,000, 8661), SOX9 (1:1,000, 82630), IRP2 (1:1,000, 37135), RAB7 (1:1,000, 2094), HA-Tag (1:1,000, 14904), EGFR (1:1,000, 4267), ZEB1 (1:1,000, 3396), MLKL (1:1,000, 14993) and VDAC (1:1,000, 4866) were purchased from Cell Signaling Technology; TRPML1 (1:1,000, ab28508) and TCF4 (1:1,000, ab217668) were purchased from Abcam; MCU (1:1000, HPA016480) and β-actin (1:2,000, A5441) were purchased from Sigma-Aldrich. .. Appropriate secondary antibodies for chemical (HRP-based) detection were purchased from Cell Signalling Technologies (7074S, 7076S), and those used for infrared detection were purchased from Invitrogen (35568, 5A535521).

    Article Title: BDH2-driven lysosome-to-mitochondria iron transfer shapes ferroptosis vulnerability of the melanoma cell states
    Article Snippet: .. Primary antibodies to MITF (1:1,000, MA5-14154) and OxPhos Human WB Antibody Cocktail (1:1,000, 45-8199) were purchased from Thermo Fisher Scientific; BDH2 (1:1,000, 27207-1-AP) and DMT1 (1:1,000, 20507-1-AP) were purchased from Proteintech; Sox10 (1:1,000, sc-365692), v-ATPase H (1:1,000, sc-166227) and v-ATPase A (1:1,000, sc-374475) were purchased from Santa Cruz Biotechnology; AXL (1:1,000, 8661), SOX9 (1:1,000, 82630), IRP2 (1:1,000, 37135), RAB7 (1:1,000, 2094), HA-Tag (1:1,000, 14904), EGFR (1:1,000, 4267), ZEB1 (1:1,000, 3396), MLKL (1:1,000, 14993) and VDAC (1:1,000, 4866) were purchased from Cell Signaling Technology; TRPML1 (1:1,000, ab28508) and TCF4 (1:1,000, ab217668) were purchased from Abcam; MCU (1:1000, HPA016480) and β-actin (1:2,000, A5441) were purchased from Sigma-Aldrich. .. Appropriate secondary antibodies for chemical (HRP-based) detection were purchased from Cell Signalling Technologies (7074S, 7076S), and those used for infrared detection were purchased from Invitrogen (35568, 5A535521).



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    Abnormal expression of antioxidant and iron metabolism-related proteins in liver samples of rats with chronic cholestasis. Control group was fed a chow diet, whereas the ANIT and DFO-treated groups were intragastrically administered ANIT olive oil solution with or without DFO (n=8/group). (A) Changes in ferroptosis antioxidant-related protein expression. The blots shown are representative of three independent experiments. A dotted line indicates that the lanes were non-adjacent on the original gel. All target protein bands and their corresponding loading control bands shown side-by-side were derived from the same membrane. Relative expression levels of (B) Nrf2, (C) Keap1, (D) XCT, (E) HO-1 and (F) GPX4. (G) Changes in iron metabolism-related protein expression. The blots shown are representative of three independent experiments. A dotted line indicates that the lanes were non-adjacent on the original gel. All target protein bands and their corresponding loading control bands shown side-by-side were derived from the same membrane. Relative expression levels of (H) TFR1, (I) FPN1, (J) <t>DMT1,</t> (K) Steap3 and (L) FTH1. Data are presented as the mean ± SD. P-values were determined by one-way ANOVA. *P<0.05, **P<0.01, ***P<0.001. ANIT, α-naphthyl isothiocyanate; DFO, deferoxamine; Nrf2, nuclear factor erythroid-2-related factor 2; Keap1, Kelch-like ECH-associated protein 1, XCT, cystine/glutamate transporter; HO-1, heme oxygenase 1; GPX4, glutathione peroxidase 4; TFR1, transferrin receptor 1; FPN1, ferroportin 1; DMT1, divalent metal transporter 1; Steap3, six-transmembrane epithelial antigen of the prostate 3; FTH1, ferritin heavy chain 1.
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    Image Search Results


    Abnormal expression of antioxidant and iron metabolism-related proteins in liver samples of rats with chronic cholestasis. Control group was fed a chow diet, whereas the ANIT and DFO-treated groups were intragastrically administered ANIT olive oil solution with or without DFO (n=8/group). (A) Changes in ferroptosis antioxidant-related protein expression. The blots shown are representative of three independent experiments. A dotted line indicates that the lanes were non-adjacent on the original gel. All target protein bands and their corresponding loading control bands shown side-by-side were derived from the same membrane. Relative expression levels of (B) Nrf2, (C) Keap1, (D) XCT, (E) HO-1 and (F) GPX4. (G) Changes in iron metabolism-related protein expression. The blots shown are representative of three independent experiments. A dotted line indicates that the lanes were non-adjacent on the original gel. All target protein bands and their corresponding loading control bands shown side-by-side were derived from the same membrane. Relative expression levels of (H) TFR1, (I) FPN1, (J) DMT1, (K) Steap3 and (L) FTH1. Data are presented as the mean ± SD. P-values were determined by one-way ANOVA. *P<0.05, **P<0.01, ***P<0.001. ANIT, α-naphthyl isothiocyanate; DFO, deferoxamine; Nrf2, nuclear factor erythroid-2-related factor 2; Keap1, Kelch-like ECH-associated protein 1, XCT, cystine/glutamate transporter; HO-1, heme oxygenase 1; GPX4, glutathione peroxidase 4; TFR1, transferrin receptor 1; FPN1, ferroportin 1; DMT1, divalent metal transporter 1; Steap3, six-transmembrane epithelial antigen of the prostate 3; FTH1, ferritin heavy chain 1.

    Journal: Molecular Medicine Reports

    Article Title: Pathological mechanism of ferroptosis in a rat model of α-naphthyl isothiocyanate-induced chronic cholestasis

    doi: 10.3892/mmr.2026.13802

    Figure Lengend Snippet: Abnormal expression of antioxidant and iron metabolism-related proteins in liver samples of rats with chronic cholestasis. Control group was fed a chow diet, whereas the ANIT and DFO-treated groups were intragastrically administered ANIT olive oil solution with or without DFO (n=8/group). (A) Changes in ferroptosis antioxidant-related protein expression. The blots shown are representative of three independent experiments. A dotted line indicates that the lanes were non-adjacent on the original gel. All target protein bands and their corresponding loading control bands shown side-by-side were derived from the same membrane. Relative expression levels of (B) Nrf2, (C) Keap1, (D) XCT, (E) HO-1 and (F) GPX4. (G) Changes in iron metabolism-related protein expression. The blots shown are representative of three independent experiments. A dotted line indicates that the lanes were non-adjacent on the original gel. All target protein bands and their corresponding loading control bands shown side-by-side were derived from the same membrane. Relative expression levels of (H) TFR1, (I) FPN1, (J) DMT1, (K) Steap3 and (L) FTH1. Data are presented as the mean ± SD. P-values were determined by one-way ANOVA. *P<0.05, **P<0.01, ***P<0.001. ANIT, α-naphthyl isothiocyanate; DFO, deferoxamine; Nrf2, nuclear factor erythroid-2-related factor 2; Keap1, Kelch-like ECH-associated protein 1, XCT, cystine/glutamate transporter; HO-1, heme oxygenase 1; GPX4, glutathione peroxidase 4; TFR1, transferrin receptor 1; FPN1, ferroportin 1; DMT1, divalent metal transporter 1; Steap3, six-transmembrane epithelial antigen of the prostate 3; FTH1, ferritin heavy chain 1.

    Article Snippet: Ferroportin 1 (FPN1; cat. no. 26601-1-AP) and divalent metal transporter 1 (DMT1; cat. no. 20507-1-AP) antibodies were purchased from Proteintech Group, Inc. Ferritin heavy chain 1 (FTH1; cat. no. PA4412) and cystine/glutamate transporter (XCT; cat. no. T57046 ) antibodies were purchased from Abmart Pharmaceutical Technology Co., Ltd.

    Techniques: Expressing, Control, Derivative Assay, Membrane

    Diagram of the mechanism of ferroptosis. ASCL4, acyl-CoA synthetase long-chain family member 4; COX2, cyclooxygenase 2; DMT1, divalent metal transporter 1; FPN, ferroportin; GSSG, oxidized glutathione; GPX4, glutathione peroxidase 4; GSH, glutathione; HO-1, heme oxygenase 1; Keap1, Kelch-like ECH-associated protein 1; LIP, labile iron pool; LPCAT3, lysophosphatidylcholine acyltransferase 3; NCOA4, nuclear receptor coactivator 4; NOX1, nicotinamide adenine dinucleotide phosphate oxidase 1; NQO1, NAD(P)H quinone dehydrogenase 1; Nrf2, nuclear factor erythroid-2-related factor 2; PL-PUFA-OOH, phospholipid-polyunsaturated fatty acid hydroperoxide; SLC7A11, solute carrier family 7 member 11; Steap3, six-transmembrane epithelial antigen of the prostate 3; TFR1, transferrin receptor 1.

    Journal: Molecular Medicine Reports

    Article Title: Pathological mechanism of ferroptosis in a rat model of α-naphthyl isothiocyanate-induced chronic cholestasis

    doi: 10.3892/mmr.2026.13802

    Figure Lengend Snippet: Diagram of the mechanism of ferroptosis. ASCL4, acyl-CoA synthetase long-chain family member 4; COX2, cyclooxygenase 2; DMT1, divalent metal transporter 1; FPN, ferroportin; GSSG, oxidized glutathione; GPX4, glutathione peroxidase 4; GSH, glutathione; HO-1, heme oxygenase 1; Keap1, Kelch-like ECH-associated protein 1; LIP, labile iron pool; LPCAT3, lysophosphatidylcholine acyltransferase 3; NCOA4, nuclear receptor coactivator 4; NOX1, nicotinamide adenine dinucleotide phosphate oxidase 1; NQO1, NAD(P)H quinone dehydrogenase 1; Nrf2, nuclear factor erythroid-2-related factor 2; PL-PUFA-OOH, phospholipid-polyunsaturated fatty acid hydroperoxide; SLC7A11, solute carrier family 7 member 11; Steap3, six-transmembrane epithelial antigen of the prostate 3; TFR1, transferrin receptor 1.

    Article Snippet: Ferroportin 1 (FPN1; cat. no. 26601-1-AP) and divalent metal transporter 1 (DMT1; cat. no. 20507-1-AP) antibodies were purchased from Proteintech Group, Inc. Ferritin heavy chain 1 (FTH1; cat. no. PA4412) and cystine/glutamate transporter (XCT; cat. no. T57046 ) antibodies were purchased from Abmart Pharmaceutical Technology Co., Ltd.

    Techniques: