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polyclonal rabbit anti cmklr1  (Bioss)


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    Structured Review

    Bioss polyclonal rabbit anti cmklr1
    <t>Chemerin/CMKLR1</t> was increased in ovarian tissues in a rat model of polycystic ovary syndrome (PCOS) (n = 6). (a) Rats in the high-fat diet (F), testosterone propionate (T), model (F&T), and control groups (C) were weighed every 2 days during the modeling. (b) Oral glucose tolerance tests were performed in all the experimental groups to reveal impaired glucose tolerance. (c) Vaginal smears were examined after modeling in each group. (d and e) CMKLR1/chemerin mRNA and protein levels were upregulated in ovarian tissues in the model group. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.
    Polyclonal Rabbit Anti Cmklr1, supplied by Bioss, used in various techniques. Bioz Stars score: 91/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/polyclonal rabbit anti cmklr1/product/Bioss
    Average 91 stars, based on 2 article reviews
    polyclonal rabbit anti cmklr1 - by Bioz Stars, 2026-02
    91/100 stars

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    1) Product Images from "Chemerin regulates autophagy to participate in polycystic ovary syndrome"

    Article Title: Chemerin regulates autophagy to participate in polycystic ovary syndrome

    Journal: The Journal of International Medical Research

    doi: 10.1177/03000605211058376

    Chemerin/CMKLR1 was increased in ovarian tissues in a rat model of polycystic ovary syndrome (PCOS) (n = 6). (a) Rats in the high-fat diet (F), testosterone propionate (T), model (F&T), and control groups (C) were weighed every 2 days during the modeling. (b) Oral glucose tolerance tests were performed in all the experimental groups to reveal impaired glucose tolerance. (c) Vaginal smears were examined after modeling in each group. (d and e) CMKLR1/chemerin mRNA and protein levels were upregulated in ovarian tissues in the model group. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.
    Figure Legend Snippet: Chemerin/CMKLR1 was increased in ovarian tissues in a rat model of polycystic ovary syndrome (PCOS) (n = 6). (a) Rats in the high-fat diet (F), testosterone propionate (T), model (F&T), and control groups (C) were weighed every 2 days during the modeling. (b) Oral glucose tolerance tests were performed in all the experimental groups to reveal impaired glucose tolerance. (c) Vaginal smears were examined after modeling in each group. (d and e) CMKLR1/chemerin mRNA and protein levels were upregulated in ovarian tissues in the model group. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.

    Techniques Used: Standard Deviation

    Chemerin regulated cell proliferation and apoptosis in vitro . (a and b) CMKLR1 protein levels were upregulated by transfection with LV5-chemerin and downregulated by transfection with LV3-590 and LV3-1623. (c) CMKLR1 mRNA levels were upregulated by transfection with LV5-chemerin and downregulated by transfection with LV3-590 and LV3-1623. (d) COV434 cell proliferation was detected using an EdU fluorescence microscope. (e) Apoptotic cells were measured by flow cytometry. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.
    Figure Legend Snippet: Chemerin regulated cell proliferation and apoptosis in vitro . (a and b) CMKLR1 protein levels were upregulated by transfection with LV5-chemerin and downregulated by transfection with LV3-590 and LV3-1623. (c) CMKLR1 mRNA levels were upregulated by transfection with LV5-chemerin and downregulated by transfection with LV3-590 and LV3-1623. (d) COV434 cell proliferation was detected using an EdU fluorescence microscope. (e) Apoptotic cells were measured by flow cytometry. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.

    Techniques Used: In Vitro, Transfection, Fluorescence, Microscopy, Flow Cytometry, Standard Deviation



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    polyclonal rabbit anti cmklr1  (Bioss)
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    Bioss polyclonal rabbit anti cmklr1
    <t>Chemerin/CMKLR1</t> was increased in ovarian tissues in a rat model of polycystic ovary syndrome (PCOS) (n = 6). (a) Rats in the high-fat diet (F), testosterone propionate (T), model (F&T), and control groups (C) were weighed every 2 days during the modeling. (b) Oral glucose tolerance tests were performed in all the experimental groups to reveal impaired glucose tolerance. (c) Vaginal smears were examined after modeling in each group. (d and e) CMKLR1/chemerin mRNA and protein levels were upregulated in ovarian tissues in the model group. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.
    Polyclonal Rabbit Anti Cmklr1, supplied by Bioss, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    bs-10410r  (Bioss)
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    <t>Chemerin/CMKLR1</t> was increased in ovarian tissues in a rat model of polycystic ovary syndrome (PCOS) (n = 6). (a) Rats in the high-fat diet (F), testosterone propionate (T), model (F&T), and control groups (C) were weighed every 2 days during the modeling. (b) Oral glucose tolerance tests were performed in all the experimental groups to reveal impaired glucose tolerance. (c) Vaginal smears were examined after modeling in each group. (d and e) CMKLR1/chemerin mRNA and protein levels were upregulated in ovarian tissues in the model group. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.
    Bs 10410r, supplied by Bioss, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Image Search Results


    Chemerin/CMKLR1 was increased in ovarian tissues in a rat model of polycystic ovary syndrome (PCOS) (n = 6). (a) Rats in the high-fat diet (F), testosterone propionate (T), model (F&T), and control groups (C) were weighed every 2 days during the modeling. (b) Oral glucose tolerance tests were performed in all the experimental groups to reveal impaired glucose tolerance. (c) Vaginal smears were examined after modeling in each group. (d and e) CMKLR1/chemerin mRNA and protein levels were upregulated in ovarian tissues in the model group. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.

    Journal: The Journal of International Medical Research

    Article Title: Chemerin regulates autophagy to participate in polycystic ovary syndrome

    doi: 10.1177/03000605211058376

    Figure Lengend Snippet: Chemerin/CMKLR1 was increased in ovarian tissues in a rat model of polycystic ovary syndrome (PCOS) (n = 6). (a) Rats in the high-fat diet (F), testosterone propionate (T), model (F&T), and control groups (C) were weighed every 2 days during the modeling. (b) Oral glucose tolerance tests were performed in all the experimental groups to reveal impaired glucose tolerance. (c) Vaginal smears were examined after modeling in each group. (d and e) CMKLR1/chemerin mRNA and protein levels were upregulated in ovarian tissues in the model group. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.

    Article Snippet: We analyzed the protein expression levels of CMKLR1, phosphoinositide 3-kinase (PI3K), Akt, MAPK, ULK-1, extracellular signal-regulated kinase (ERK), phospho-mTOR (p-mTOR), and β-actin by western blot, using the following primary antibodies: polyclonal rabbit anti-chemerin (ab103153; Abcam, Shanghai, China); polyclonal rabbit anti-CMKLR1 (bs-10410R; Bioss Biotechnology Company, Beijing, China); rabbit monoclonal to ULK1 (D8H5) (#8054; Cell Signaling Technology, MA, USA); rabbit monoclonal to p-mTOR (Ser2448) (#5536; Cell Signaling Technology); rabbit monoclonal to MAPK (#8690; Cell Signaling Technology); rabbit monoclonal to Akt (#4691; Cell Signaling Technology); rabbit monoclonal to PI3K Class III (D9A5) (#4263; Cell Signaling Technology); rabbit monoclonal to LC3A/B (D3U4C) (#12741S; Cell Signaling Technology); and polyclonal rabbit anti-β-actin (#4970S; Cell Signaling Technology).

    Techniques: Standard Deviation

    Chemerin regulated cell proliferation and apoptosis in vitro . (a and b) CMKLR1 protein levels were upregulated by transfection with LV5-chemerin and downregulated by transfection with LV3-590 and LV3-1623. (c) CMKLR1 mRNA levels were upregulated by transfection with LV5-chemerin and downregulated by transfection with LV3-590 and LV3-1623. (d) COV434 cell proliferation was detected using an EdU fluorescence microscope. (e) Apoptotic cells were measured by flow cytometry. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.

    Journal: The Journal of International Medical Research

    Article Title: Chemerin regulates autophagy to participate in polycystic ovary syndrome

    doi: 10.1177/03000605211058376

    Figure Lengend Snippet: Chemerin regulated cell proliferation and apoptosis in vitro . (a and b) CMKLR1 protein levels were upregulated by transfection with LV5-chemerin and downregulated by transfection with LV3-590 and LV3-1623. (c) CMKLR1 mRNA levels were upregulated by transfection with LV5-chemerin and downregulated by transfection with LV3-590 and LV3-1623. (d) COV434 cell proliferation was detected using an EdU fluorescence microscope. (e) Apoptotic cells were measured by flow cytometry. Error bars represent standard error. Bars show mean ± standard deviation of three independent experiments. * P < 0.05, ** P < 0.01, *** P < 0.001 compared with control group.

    Article Snippet: We analyzed the protein expression levels of CMKLR1, phosphoinositide 3-kinase (PI3K), Akt, MAPK, ULK-1, extracellular signal-regulated kinase (ERK), phospho-mTOR (p-mTOR), and β-actin by western blot, using the following primary antibodies: polyclonal rabbit anti-chemerin (ab103153; Abcam, Shanghai, China); polyclonal rabbit anti-CMKLR1 (bs-10410R; Bioss Biotechnology Company, Beijing, China); rabbit monoclonal to ULK1 (D8H5) (#8054; Cell Signaling Technology, MA, USA); rabbit monoclonal to p-mTOR (Ser2448) (#5536; Cell Signaling Technology); rabbit monoclonal to MAPK (#8690; Cell Signaling Technology); rabbit monoclonal to Akt (#4691; Cell Signaling Technology); rabbit monoclonal to PI3K Class III (D9A5) (#4263; Cell Signaling Technology); rabbit monoclonal to LC3A/B (D3U4C) (#12741S; Cell Signaling Technology); and polyclonal rabbit anti-β-actin (#4970S; Cell Signaling Technology).

    Techniques: In Vitro, Transfection, Fluorescence, Microscopy, Flow Cytometry, Standard Deviation