anticd206 (Proteintech)
96
Structured Review
Proteintech
anticd206
Anticd206, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 817 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anticd206/MMP13+Antibody/pm41702086-68-62-65
Average 96 stars, based on 817 article reviews
Anticd206, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 817 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anticd206/MMP13+Antibody/pm41702086-68-62-65
Average 96 stars, based on 817 article reviews
anticd206 - by Bioz Stars,
2026-10
96/100 stars
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Blocking Assay:Article Title: Acupuncture as a modulator of microglial polarization and pyroptosis in depression: evidence from a chronic unpredictable mild stress rat model. Article Snippet: A 10% SDS-PAGE gel was used to separate the protein samples, which were then transferred to PVDF membranes (IPVH00010, Merck Millipore) by electrotransfer. .. After blocking with 5% skimmed milk powder for 1 h at room temperature, the membranes were incubated overnight at 4 °C with the corresponding primary antibodies: anti-NF-κB-p65 (1:5000, 80979-1-RR, RRID: AB_2918923, Proteintech); anti-NLRP3 (1:2000, 68102-1-Ig, RRID: AB_2923634, Proteintech); anti-ASC (1:5000, 67494-1-Ig, RRID:AB_2882718, Proteintech); anti-pro-caspase-1 (1:1000, ab179515, RRID:AB_2884954, Abcam); anti-caspase-1 (1:2000, 22915-1- AP, RRID:AB_2876874, Proteintech); anti-GSDMD(1:2 000, 20770-1-AP, RRID:AB_10696319, Proteintech); anti-iNOS (1:2000, 80517-1-RR, RRID:AB_291 8898, Proteintech); anti-CD16 (1:500, 16559-1- AP, RRID:AB_2878279, Proteintech);anti- Arg-1 (1:50 00, 16001-1-AP, RRID:AB_2289842, Proteintech); and Incubation:Article Title: Acupuncture as a modulator of microglial polarization and pyroptosis in depression: evidence from a chronic unpredictable mild stress rat model. Article Snippet: A 10% SDS-PAGE gel was used to separate the protein samples, which were then transferred to PVDF membranes (IPVH00010, Merck Millipore) by electrotransfer. .. After blocking with 5% skimmed milk powder for 1 h at room temperature, the membranes were incubated overnight at 4 °C with the corresponding primary antibodies: anti-NF-κB-p65 (1:5000, 80979-1-RR, RRID: AB_2918923, Proteintech); anti-NLRP3 (1:2000, 68102-1-Ig, RRID: AB_2923634, Proteintech); anti-ASC (1:5000, 67494-1-Ig, RRID:AB_2882718, Proteintech); anti-pro-caspase-1 (1:1000, ab179515, RRID:AB_2884954, Abcam); anti-caspase-1 (1:2000, 22915-1- AP, RRID:AB_2876874, Proteintech); anti-GSDMD(1:2 000, 20770-1-AP, RRID:AB_10696319, Proteintech); anti-iNOS (1:2000, 80517-1-RR, RRID:AB_291 8898, Proteintech); anti-CD16 (1:500, 16559-1- AP, RRID:AB_2878279, Proteintech);anti- Arg-1 (1:50 00, 16001-1-AP, RRID:AB_2289842, Proteintech); and Article Title: ANAPC5 mitigates acute lung injury through regulating macrophage M1/M2 polarization via the EGFR/CD24 axis. Article Snippet: Acute lung injury (ALI) is a respiratory disease induced by uncontrolled inflammatory responses in the lungs.. The pathological features of ALI include alveolar structural damage and pulmonary edema, which ultimately leads to pulmonary dysfunction.. ANAPC5 (Anaphase-promoting complex subunit 5) is an E3 ubiquitin ligase known for its anti-inflammatory properties. Article Title: HDAC6 exacerbates osteoarthritis by promoting macrophage inflammation-mediated chondrocyte degeneration. Article Snippet: Osteoarthritis (OA) is a degenerative joint disorder characterized by cartilage degradation and synovial inflammation, with its pathogenesis involving intricate interactions between cellular metabolic processes and inflammatory pathways.. In this study, we established OA models in both wild-type and histone deacetylase 6 (HDAC6) knockout mice via anterior cruciate ligament transection (ACLT) to investigate the role of HDAC6 in OA progression.. Histological and immunohistochemical analyses revealed that the absence of HDAC6 significantly mitigated ACLT-induced cartilage damage and synovitis in mice, while also restoring imbalances in cartilage metabolic markers. Article Title: Microenvironment responsive nanoplatform for targeted removal of cholesterol and reshaping inflammatory microenvironment in atherosclerotic plaques. Article Snippet: The microenvironment of atherosclerotic plaques features abundant cholesterol crystals (CCs), reactive oxygen species (ROS), and pro-inflammatory M1 macrophages, leading to the progression and mortality of atherosclerotic cardiovascular disease (ASCVD).. Targeted removal of CCs and scavenging of ROS are crucial for treatment of ASCVD.. In this study, an intelligent nanoformulation consisting of epigallocatechin gallate (EGCG), cysteine (Cys), ursodeoxycholic acid (UDCA) and VHPK(Valine-Histidine-Proline-Lysine)-Lipo, was designed to precisely target and remodel the inflammatory microenvironment of atherosclerotic plaques. Article Title: Glioblastoma- derived exosomes (GBM-Exo) regulate microglial M2 polarization via the RAC1/AKT/NRF2 pathway. Article Snippet: 1 Lanzhou University Second Hospital, Lanzhou University, Lanzhou, Gansu Province 730000, China 2 Department of Neurosurgery, Liangzhou Hospital of Wuwei, Wuwei, Gansu Province 733000, China 3 Institute of Genetics, School of Basic Medical Sciences, Lanzhou University, Lanzhou, Gansu Province 730000, China Abstract Purpose The impact of exosome-mediated communication between glioblastoma and microglia on the formation of an immunosuppressive microenvironment remains to be explored.. Tumor-associated macrophages are more likely to adopt an M2-like phenotype within the immunosuppressive environment.. Here, we investigate the molecular mechanisms by which glioblastoma-derived exosomes promote microglial M2 polarization through RAC1. Immunohistochemistry:Article Title: PTGDS deficiency as a driver of M2 macrophage polarization and immunosuppressive microenvironment in esophageal squamous cell carcinoma: an experimental study Article Snippet: .. All retrieved tumor specimens were analyzed by IHC using indicated antibodies as anti-PTGDS (10754-2-AP, proteintech), Membrane:Article Title: Microenvironment responsive nanoplatform for targeted removal of cholesterol and reshaping inflammatory microenvironment in atherosclerotic plaques. Article Snippet: The microenvironment of atherosclerotic plaques features abundant cholesterol crystals (CCs), reactive oxygen species (ROS), and pro-inflammatory M1 macrophages, leading to the progression and mortality of atherosclerotic cardiovascular disease (ASCVD).. Targeted removal of CCs and scavenging of ROS are crucial for treatment of ASCVD.. In this study, an intelligent nanoformulation consisting of epigallocatechin gallate (EGCG), cysteine (Cys), ursodeoxycholic acid (UDCA) and VHPK(Valine-Histidine-Proline-Lysine)-Lipo, was designed to precisely target and remodel the inflammatory microenvironment of atherosclerotic plaques. |