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mouse p selectin  (Sino Biological)


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    Structured Review

    Sino Biological mouse p selectin
    Mouse P Selectin, supplied by Sino Biological, used in various techniques. Bioz Stars score: 93/100, based on 4 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/50737/Mouse+SELP+%2F+selectin+P+%2F+P-selectin+Protein/pm39468361-166-16-22
    Average 93 stars, based on 4 article reviews
    mouse p selectin - by Bioz Stars, 2026-09
    93/100 stars

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    Related Articles

    other:

    Article Title: NIR-II scattering gold superclusters for intravascular optical coherence tomography molecular imaging.
    Article Snippet: Currently, intravascular optical coherence tomography (IV-OCT) is limited to anatomical imaging, providing structural information about atherosclerotic plaque morphology, thrombus and dissection.. Earlier detection and risk stratification would be possible through molecular characterization of endothelium but necessitates a purpose-engineered IV-OCT contrast agent.. Here we developed gold superclusters (AuSCs) tailored to clinical instrumentation and integrated into clinically relevant workflows.

    Recombinant:

    Article Title: Real-time cancer monitoring via leukocyte adhesion in a biomimetic microfluidic assay.
    Article Snippet: Cancer progression involves cell adhesion molecules (CAMs), which facilitate the recruitment of leukocytes and metastatic tumor cells to distant organs by mediating adhesion with endothelial cells.. While CAM-mediated tumor dissemination is well studied, the diagnostic potential of CAM ligand-expressing activated leukocytes as biomarkers reflecting the host’s inflammatory response to cancer remains relatively unexplored.. This study presents a microfluidic device that assesses cancer-driven leukocyte activation in a 4T1 breast cancer mouse model by quantifying leukocyte adhesion to CAM-coated microchannels under physiological flow conditions.

    Sterility:

    Article Title: Real-time cancer monitoring via leukocyte adhesion in a biomimetic microfluidic assay.
    Article Snippet: Cancer progression involves cell adhesion molecules (CAMs), which facilitate the recruitment of leukocytes and metastatic tumor cells to distant organs by mediating adhesion with endothelial cells.. While CAM-mediated tumor dissemination is well studied, the diagnostic potential of CAM ligand-expressing activated leukocytes as biomarkers reflecting the host’s inflammatory response to cancer remains relatively unexplored.. This study presents a microfluidic device that assesses cancer-driven leukocyte activation in a 4T1 breast cancer mouse model by quantifying leukocyte adhesion to CAM-coated microchannels under physiological flow conditions.



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    Image Search Results


    DUB-IN-3 inhibits ATC progression by activating ferroptosis. (A-C) The CCK8 assay (A), colony formation assay (B), and transwell assay (C) was applied to present the effect of DUB-IN-3 on the proliferation, colony formation, and migration capability of BHT101 cells. (D) The CCK8 assay was used to assess the responses of BHT101 cells with DUB-IN-3 treatment to RSL-3 ± Fer-1. (E, F) Lipid ROS (E) and GSH (F) levels in DUB-IN-3 treating ATC cells. (G) The parental BHT101 cells were injected into the right underarm of nude mouse, then DUB-IN-3 was intraperitoneally injected every 3 days ( n = 3 mouse per group). The tumors were removed and weighted after 17 days. (H) Representative images of IHC staining for Ki-67. (I) GPR34 stabilized by deubiquitinase USP8 promotes ATC progression via inhibiting ferroptosis. Ns, nonsignificant. p > 0.05, ⁣ ∗ p < 0.05, ⁣ ∗∗ p < 0.01, ⁣ ∗∗∗ p < 0.001.

    Journal: Mediators of Inflammation

    Article Title: GPR34 Stabilized by Deubiquitinase USP8 Suppresses Ferroptosis of ATC

    doi: 10.1155/mi/5576056

    Figure Lengend Snippet: DUB-IN-3 inhibits ATC progression by activating ferroptosis. (A-C) The CCK8 assay (A), colony formation assay (B), and transwell assay (C) was applied to present the effect of DUB-IN-3 on the proliferation, colony formation, and migration capability of BHT101 cells. (D) The CCK8 assay was used to assess the responses of BHT101 cells with DUB-IN-3 treatment to RSL-3 ± Fer-1. (E, F) Lipid ROS (E) and GSH (F) levels in DUB-IN-3 treating ATC cells. (G) The parental BHT101 cells were injected into the right underarm of nude mouse, then DUB-IN-3 was intraperitoneally injected every 3 days ( n = 3 mouse per group). The tumors were removed and weighted after 17 days. (H) Representative images of IHC staining for Ki-67. (I) GPR34 stabilized by deubiquitinase USP8 promotes ATC progression via inhibiting ferroptosis. Ns, nonsignificant. p > 0.05, ⁣ ∗ p < 0.05, ⁣ ∗∗ p < 0.01, ⁣ ∗∗∗ p < 0.001.

    Article Snippet: DUB-IN-3 (HY-50737-11866) were obtained from MedChemExpress.

    Techniques: CCK-8 Assay, Colony Assay, Transwell Assay, Migration, Injection, Immunohistochemistry