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  • 79
    Thermo Fisher takara dna polymerase
    Takara Dna Polymerase, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 79/100, based on 8 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/takara dna polymerase/product/Thermo Fisher
    Average 79 stars, based on 8 article reviews
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    takara dna polymerase - by Bioz Stars, 2020-01
    79/100 stars
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    91
    TaKaRa deoxyribonucleic acid gdna eraser
    Deoxyribonucleic Acid Gdna Eraser, supplied by TaKaRa, used in various techniques. Bioz Stars score: 91/100, based on 62 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 91 stars, based on 62 article reviews
    Price from $9.99 to $1999.99
    deoxyribonucleic acid gdna eraser - by Bioz Stars, 2020-01
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    78
    TaKaRa pbr322 deoxyribonucleic acid dna
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Pbr322 Deoxyribonucleic Acid Dna, supplied by TaKaRa, used in various techniques. Bioz Stars score: 78/100, based on 4 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/pbr322 deoxyribonucleic acid dna/product/TaKaRa
    Average 78 stars, based on 4 article reviews
    Price from $9.99 to $1999.99
    pbr322 deoxyribonucleic acid dna - by Bioz Stars, 2020-01
    78/100 stars
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    78
    TaKaRa taq deoxyribonucleic acid dna polymerase
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Taq Deoxyribonucleic Acid Dna Polymerase, supplied by TaKaRa, used in various techniques. Bioz Stars score: 78/100, based on 6 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/taq deoxyribonucleic acid dna polymerase/product/TaKaRa
    Average 78 stars, based on 6 article reviews
    Price from $9.99 to $1999.99
    taq deoxyribonucleic acid dna polymerase - by Bioz Stars, 2020-01
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    99
    TaKaRa dna ligation kit
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Dna Ligation Kit, supplied by TaKaRa, used in various techniques. Bioz Stars score: 99/100, based on 2053 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/dna ligation kit/product/TaKaRa
    Average 99 stars, based on 2053 article reviews
    Price from $9.99 to $1999.99
    dna ligation kit - by Bioz Stars, 2020-01
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    87
    TaKaRa takara dexpat easy
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara Dexpat Easy, supplied by TaKaRa, used in various techniques. Bioz Stars score: 87/100, based on 18 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/takara dexpat easy/product/TaKaRa
    Average 87 stars, based on 18 article reviews
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    takara dexpat easy - by Bioz Stars, 2020-01
    87/100 stars
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    77
    TaKaRa takara dna blunting kit
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara Dna Blunting Kit, supplied by TaKaRa, used in various techniques. Bioz Stars score: 77/100, based on 4 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/takara dna blunting kit/product/TaKaRa
    Average 77 stars, based on 4 article reviews
    Price from $9.99 to $1999.99
    takara dna blunting kit - by Bioz Stars, 2020-01
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    99
    TaKaRa takara epitaq hs
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara Epitaq Hs, supplied by TaKaRa, used in various techniques. Bioz Stars score: 99/100, based on 334 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/takara epitaq hs/product/TaKaRa
    Average 99 stars, based on 334 article reviews
    Price from $9.99 to $1999.99
    takara epitaq hs - by Bioz Stars, 2020-01
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    79
    TaKaRa takara recochips
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara Recochips, supplied by TaKaRa, used in various techniques. Bioz Stars score: 79/100, based on 15 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/takara recochips/product/TaKaRa
    Average 79 stars, based on 15 article reviews
    Price from $9.99 to $1999.99
    takara recochips - by Bioz Stars, 2020-01
    79/100 stars
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    89
    TaKaRa takara taq kit
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara Taq Kit, supplied by TaKaRa, used in various techniques. Bioz Stars score: 89/100, based on 69 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/takara taq kit/product/TaKaRa
    Average 89 stars, based on 69 article reviews
    Price from $9.99 to $1999.99
    takara taq kit - by Bioz Stars, 2020-01
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    93
    TaKaRa takara hs taq
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara Hs Taq, supplied by TaKaRa, used in various techniques. Bioz Stars score: 93/100, based on 55 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/takara hs taq/product/TaKaRa
    Average 93 stars, based on 55 article reviews
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    takara hs taq - by Bioz Stars, 2020-01
    93/100 stars
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    92
    TaKaRa takara dexpat kit
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara Dexpat Kit, supplied by TaKaRa, used in various techniques. Bioz Stars score: 92/100, based on 123 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/takara dexpat kit/product/TaKaRa
    Average 92 stars, based on 123 article reviews
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    takara dexpat kit - by Bioz Stars, 2020-01
    92/100 stars
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    99
    TaKaRa takara la taq
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara La Taq, supplied by TaKaRa, used in various techniques. Bioz Stars score: 99/100, based on 2501 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/takara la taq/product/TaKaRa
    Average 99 stars, based on 2501 article reviews
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    takara la taq - by Bioz Stars, 2020-01
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    99
    TaKaRa takara ex taq
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara Ex Taq, supplied by TaKaRa, used in various techniques. Bioz Stars score: 99/100, based on 3018 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/takara ex taq/product/TaKaRa
    Average 99 stars, based on 3018 article reviews
    Price from $9.99 to $1999.99
    takara ex taq - by Bioz Stars, 2020-01
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    99
    TaKaRa takara ex taqtm
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara Ex Taqtm, supplied by TaKaRa, used in various techniques. Bioz Stars score: 99/100, based on 41 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 99 stars, based on 41 article reviews
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    takara ex taqtm - by Bioz Stars, 2020-01
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    89
    TaKaRa takara extaq kit
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Takara Extaq Kit, supplied by TaKaRa, used in various techniques. Bioz Stars score: 89/100, based on 56 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    TaKaRa cdna reference takara
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    TaKaRa takara transcriptase kit
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    Thermo Fisher takara taq polymerase
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    TaKaRa takara emeraldamp mix
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    TaKaRa taq polymerase hot start version dna
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    TaKaRa takara rr006a extaq
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    TaKaRa ztaq dna polymerase
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    Ztaq Dna Polymerase, supplied by TaKaRa, used in various techniques. Bioz Stars score: 78/100, based on 7 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    TaKaRa hot start takara taq dna polymerase
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    TaKaRa gc buffer
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    TaKaRa t4 dna polymerase takara dna blunting kit takara biotechnology kyoto japan
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
    T4 Dna Polymerase Takara Dna Blunting Kit Takara Biotechnology Kyoto Japan, supplied by TaKaRa, used in various techniques. Bioz Stars score: 79/100, based on 11 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    TaKaRa dna off
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    TaKaRa dl 2000 dna marker takara 3427a
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    TaKaRa dna manipulation
    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled <t>pBR322</t> <t>DNA.</t> As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P
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    TaKaRa dna eraser
    ( A ) Human VDR gene locus. Four overlapping cosmid clones were isolated from a human lymphocyte genomic library (Stratagene) and directly sequenced. Clone J5 extends from the 5′ flanking region to intron 2; AE, from intron 1b to intron 5; D2, from intron 3 to the 3′ UTR; WE, from intron 6 through the 3′ flanking region. Sequence upstream of exon 1f was obtained by anchored <t>PCR</t> from genomic <t>DNA.</t> ( B ). Transcripts 6–10 originate from exon 1d and transcripts 11–14 originate from exon 1f. Boxed numbers indicate the major transcript (based on the relative intensities of the multiple PCR products) within each exon-specific group of transcripts generated with a single primer set. While all transcripts have a translation initiation codon in exon 2, exon 1d transcripts have the potential to initiate translation upstream in exon 1d, with transcripts 6 and 9 encoding VDR proteins with extended N termini. ( C ) and encodes the 427-aa hVDR protein. Transcripts 6 and 9 code for a protein with an extra 50 aa or 23 aa, respectively, at the N-terminal. The 23 aa of the hVDR A/B domain are shown in bold.
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    TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled pBR322 DNA. As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P

    Journal: BioMed Research International

    Article Title: β-Elemene Inhibits Cell Proliferation by Regulating the Expression and Activity of Topoisomerases I and IIα in Human Hepatocarcinoma HepG-2 Cells

    doi: 10.1155/2015/153987

    Figure Lengend Snippet: TOPO I catalytic activity is inhibited by β -ELE. (a) The effect of β -ELE ( β -Elemene) on the relaxation of TOPO I-mediated, negative supercoiled pBR322 DNA. As shown in (a), lane 1 is negative supercoiled pBR322 DNA (supercoiled DNA, S); lane 2 is relaxed DNA that is the product of supercoiled DNA reacted with the enzyme (relaxed DNA, R); and lanes 3 and 4 are positive control groups. HCPT (a typical inhibitor of TOPO I) had no inhibitory effect on the DNA relaxation activity of TOPO I at the concentration of 5 μ g/mL, whereas it completely inhibited the relaxation of pBR322 DNA mediated by TOPO I at a concentration of 1 μ g/ μ L. Lane 5 shows the effect of 1 μ g/ μ L HCPT combined with 40 μ g/mL β -ELE on the relaxation of TOPO I-mediated negative supercoiled pBR322 DNA. As shown in (a), the combination inhibited the relaxation activity of TOPO I. Lanes 6 to 11 show the effects of different concentrations of β -ELE (40, 60, 80, and 100 μ g/mL) on the relaxation of negative supercoiled pBR322 DNA mediated by TOPO I. β -ELE has no inhibitory effect on the relaxation activity of TOPO I at concentrations of 10 and 20 μ g/mL; however, with increasing drug concentration, β -ELE showed an increasing inhibitory effect on the DNA relaxation activity of TOPO I at concentrations of 40, 60, 80, and 100 μ g/mL. The OD of MAX was 58 ± 3, 80 ± 6, 92 ± 10, and 134 ± 12. The statistical analysis showed significant differences between the 100 μ g/mL and 40 μ g/mL treatment groups and the 60 μ g/mL and 80 μ g/mL treatment groups ( P

    Article Snippet: PBR322 deoxyribonucleic acid (DNA) and DNA-TOPO I were purchased from Takara Biotechnology (Dalian) Co., Ltd. (Dalian, China); kDNA and DNA-TOPO IIα were purchased from Vaxron Corporation (Rockaway, NJ, USA).

    Techniques: Activity Assay, Positive Control, Concentration Assay

    ( A ) Human VDR gene locus. Four overlapping cosmid clones were isolated from a human lymphocyte genomic library (Stratagene) and directly sequenced. Clone J5 extends from the 5′ flanking region to intron 2; AE, from intron 1b to intron 5; D2, from intron 3 to the 3′ UTR; WE, from intron 6 through the 3′ flanking region. Sequence upstream of exon 1f was obtained by anchored PCR from genomic DNA. ( B ). Transcripts 6–10 originate from exon 1d and transcripts 11–14 originate from exon 1f. Boxed numbers indicate the major transcript (based on the relative intensities of the multiple PCR products) within each exon-specific group of transcripts generated with a single primer set. While all transcripts have a translation initiation codon in exon 2, exon 1d transcripts have the potential to initiate translation upstream in exon 1d, with transcripts 6 and 9 encoding VDR proteins with extended N termini. ( C ) and encodes the 427-aa hVDR protein. Transcripts 6 and 9 code for a protein with an extra 50 aa or 23 aa, respectively, at the N-terminal. The 23 aa of the hVDR A/B domain are shown in bold.

    Journal: Proceedings of the National Academy of Sciences of the United States of America

    Article Title: Multiple promoters direct the tissue-specific expression of novel N-terminal variant human vitamin D receptor gene transcripts

    doi:

    Figure Lengend Snippet: ( A ) Human VDR gene locus. Four overlapping cosmid clones were isolated from a human lymphocyte genomic library (Stratagene) and directly sequenced. Clone J5 extends from the 5′ flanking region to intron 2; AE, from intron 1b to intron 5; D2, from intron 3 to the 3′ UTR; WE, from intron 6 through the 3′ flanking region. Sequence upstream of exon 1f was obtained by anchored PCR from genomic DNA. ( B ). Transcripts 6–10 originate from exon 1d and transcripts 11–14 originate from exon 1f. Boxed numbers indicate the major transcript (based on the relative intensities of the multiple PCR products) within each exon-specific group of transcripts generated with a single primer set. While all transcripts have a translation initiation codon in exon 2, exon 1d transcripts have the potential to initiate translation upstream in exon 1d, with transcripts 6 and 9 encoding VDR proteins with extended N termini. ( C ) and encodes the 427-aa hVDR protein. Transcripts 6 and 9 code for a protein with an extra 50 aa or 23 aa, respectively, at the N-terminal. The 23 aa of the hVDR A/B domain are shown in bold.

    Article Snippet: Sequence upstream of exon 1f was obtained by anchored PCR from genomic DNA by using commercially available anchor-ligated DNA (CLONTECH).

    Techniques: Clone Assay, Isolation, Sequencing, Polymerase Chain Reaction, Generated