arginine vasopressin avp receptor 2 Search Results


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  • 91
    Alomone Labs arginine vasopressin avp receptor 2
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    PerkinElmer vasopressin linear v 1a antagonist phenylacetyl1 0 me d tyr2 125i arg6
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    Millipore arg8 vasopressin
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    Tocris arg8 vasopressin
    V1a receptor modulation directly alters LC noradrenergic neuronal activity. (A1) Representative traces of the spontaneous firing patterns of an LC neuron before and after the application of the V1a receptor antagonist [d(CH2)51, Tyr(Me)2, <t>Arg8]-Vasopressin,</t> 30 nM, from a cohort of neurons that responded with a significant increase in the frequency of spontaneous action potentials. (A2) Quantification of the spontaneous firing rates (Hz) of LC neurons before and after the application of the V1a receptor antagonist. (A3) Quantification of the afterhyperpolarization time constant (msec) of LC neurons before and after the application of the V1a receptor antagonist. (B1) Representative traces of the spontaneous firing patterns of an LC neuron before and after the application of the V1a receptor antagonist, from a cohort of neurons that responded with a significant decrease in the frequency of spontaneous action potentials. (B2) Quantification of the spontaneous firing rates (Hz) of LC neurons before and after the application of the V1a receptor antagonist. (B3) Quantification of the afterhyperpolarization time constant (msec) of LC neurons before and after the application of the V1a receptor antagonist. In the graphs, the dots represent the values for individual cells, the long red bars represents the mean for all cells within that group, and the short red bars represent the SEM. ∗ p
    Arg8 Vasopressin, supplied by Tocris, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    V1a receptor modulation directly alters LC noradrenergic neuronal activity. (A1) Representative traces of the spontaneous firing patterns of an LC neuron before and after the application of the V1a receptor antagonist [d(CH2)51, Tyr(Me)2, Arg8]-Vasopressin, 30 nM, from a cohort of neurons that responded with a significant increase in the frequency of spontaneous action potentials. (A2) Quantification of the spontaneous firing rates (Hz) of LC neurons before and after the application of the V1a receptor antagonist. (A3) Quantification of the afterhyperpolarization time constant (msec) of LC neurons before and after the application of the V1a receptor antagonist. (B1) Representative traces of the spontaneous firing patterns of an LC neuron before and after the application of the V1a receptor antagonist, from a cohort of neurons that responded with a significant decrease in the frequency of spontaneous action potentials. (B2) Quantification of the spontaneous firing rates (Hz) of LC neurons before and after the application of the V1a receptor antagonist. (B3) Quantification of the afterhyperpolarization time constant (msec) of LC neurons before and after the application of the V1a receptor antagonist. In the graphs, the dots represent the values for individual cells, the long red bars represents the mean for all cells within that group, and the short red bars represent the SEM. ∗ p

    Journal: Frontiers in Neuroscience

    Article Title: Dynamic Modulation of Mouse Locus Coeruleus Neurons by Vasopressin 1a and 1b Receptors

    doi: 10.3389/fnins.2018.00919

    Figure Lengend Snippet: V1a receptor modulation directly alters LC noradrenergic neuronal activity. (A1) Representative traces of the spontaneous firing patterns of an LC neuron before and after the application of the V1a receptor antagonist [d(CH2)51, Tyr(Me)2, Arg8]-Vasopressin, 30 nM, from a cohort of neurons that responded with a significant increase in the frequency of spontaneous action potentials. (A2) Quantification of the spontaneous firing rates (Hz) of LC neurons before and after the application of the V1a receptor antagonist. (A3) Quantification of the afterhyperpolarization time constant (msec) of LC neurons before and after the application of the V1a receptor antagonist. (B1) Representative traces of the spontaneous firing patterns of an LC neuron before and after the application of the V1a receptor antagonist, from a cohort of neurons that responded with a significant decrease in the frequency of spontaneous action potentials. (B2) Quantification of the spontaneous firing rates (Hz) of LC neurons before and after the application of the V1a receptor antagonist. (B3) Quantification of the afterhyperpolarization time constant (msec) of LC neurons before and after the application of the V1a receptor antagonist. In the graphs, the dots represent the values for individual cells, the long red bars represents the mean for all cells within that group, and the short red bars represent the SEM. ∗ p

    Article Snippet: Desmopressin, [d(CH2)51, Tyr(Me)2, Arg8]-Vasopressin and TASP 0390325 were obtained from Tocris, United Kingdom, and dissolved in ECS.

    Techniques: Activity Assay