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    Alomone Labs anti aqp4
    The synthesized GILZ-p inhibited LPS induced Müller cell gliosis. Western blot analysis was performed to determine the protein expression levels of glial fibrillary acidic protein (GFAP) (A,B) , and <t>AQP4</t> (C,D) in Müller cells treated with 1000 ng/ml LPS in combination with different concentrations of GILZ-p (0.01, 0.1, 1, and 10 μM) for 24 h. β-actin was used as the loading control. The results of quantitative analysis, as determined by densitometric analysis, were expressed as relative to β-actin. Data represent the mean ± SE; the Mann–Whitney U -test was used for comparisons between two groups. n = 3 for each group. ∗ P
    Anti Aqp4, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/anti aqp4/product/Alomone Labs
    Average 94 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    anti aqp4 - by Bioz Stars, 2022-09
    94/100 stars
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    The synthesized GILZ-p inhibited LPS induced Müller cell gliosis. Western blot analysis was performed to determine the protein expression levels of glial fibrillary acidic protein (GFAP) (A,B) , and AQP4 (C,D) in Müller cells treated with 1000 ng/ml LPS in combination with different concentrations of GILZ-p (0.01, 0.1, 1, and 10 μM) for 24 h. β-actin was used as the loading control. The results of quantitative analysis, as determined by densitometric analysis, were expressed as relative to β-actin. Data represent the mean ± SE; the Mann–Whitney U -test was used for comparisons between two groups. n = 3 for each group. ∗ P

    Journal: Frontiers in Pharmacology

    Article Title: A Synthesized Glucocorticoid- Induced Leucine Zipper Peptide Inhibits Retinal Müller Cell Gliosis

    doi: 10.3389/fphar.2018.00331

    Figure Lengend Snippet: The synthesized GILZ-p inhibited LPS induced Müller cell gliosis. Western blot analysis was performed to determine the protein expression levels of glial fibrillary acidic protein (GFAP) (A,B) , and AQP4 (C,D) in Müller cells treated with 1000 ng/ml LPS in combination with different concentrations of GILZ-p (0.01, 0.1, 1, and 10 μM) for 24 h. β-actin was used as the loading control. The results of quantitative analysis, as determined by densitometric analysis, were expressed as relative to β-actin. Data represent the mean ± SE; the Mann–Whitney U -test was used for comparisons between two groups. n = 3 for each group. ∗ P

    Article Snippet: The membranes were blocked in 5% non-fat milk at room temperature for 1 h and incubated with the following antibodies: anti-monocyte chemoattractant protein (MCP)-1 (ab25124; Abcam, Cambridge, United Kingdom), anti-intercellular adhesion molecule (ICAM)-1 (ab171123; Proteintech, Chicago, IL, United States), anti-IL1β (ab9787; Abcam), Anti-tumor necrosis factor (TNF)α (PB0270, Boster Biological Technology, Wuhan, China), rabbit anti-p65 polyclonal antibody (ab16502; Abcam), anti-phospho-NF-κB p65 (Ser536) rabbit monoclonal antibody (3033P; Cell Signaling Technology, Beverly, MA, United States), anti-AQP4 (300-314, Alomone Labs, Jerusalem, Israel), anti-glial fibrillary acidic protein (GFAP) (ab10062, Abcam), or rabbit anti β-actin antibody (ab69512; Abcam) overnight.

    Techniques: Synthesized, Western Blot, Expressing, MANN-WHITNEY

    CTBS improved AQP4 polarization in APP/PS1 mice. (a) Immunofluorescent staining of AQP4 in the cortex and hippocampus (200×, scale bar: 100 μ m). (b) Statistical analysis of relative AQP4 expression levels in the cortex. (c) Statistical analysis of relative AQP4 expression levels in the hippocampus. (d) Statistical analysis of relative perivascular polarization of AQP4 in the cortex. (e) Statistical analysis of relative perivascular polarization of AQP4 in the hippocampus. Data are presented as the mean ± SD ( n = 6 mice per group, unpaired t -test for comparing two individual groups; ∗ P

    Journal: Mediators of Inflammation

    Article Title: Continuous Theta-Burst Stimulation Promotes Paravascular CSF-Interstitial Fluid Exchange through Regulation of Aquaporin-4 Polarization in APP/PS1 Mice

    doi: 10.1155/2022/2140524

    Figure Lengend Snippet: CTBS improved AQP4 polarization in APP/PS1 mice. (a) Immunofluorescent staining of AQP4 in the cortex and hippocampus (200×, scale bar: 100 μ m). (b) Statistical analysis of relative AQP4 expression levels in the cortex. (c) Statistical analysis of relative AQP4 expression levels in the hippocampus. (d) Statistical analysis of relative perivascular polarization of AQP4 in the cortex. (e) Statistical analysis of relative perivascular polarization of AQP4 in the hippocampus. Data are presented as the mean ± SD ( n = 6 mice per group, unpaired t -test for comparing two individual groups; ∗ P

    Article Snippet: Antibodies for AQP4 (Alomone Labs, 300-314; Jerusalem, Israel) and glial fibrillary acidic protein (GFAP, Sigma, G3893, USA) were used to detect aquaporins and astrocytes.

    Techniques: Mouse Assay, Staining, Expressing