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    Santa Cruz Biotechnology mouse hmga1 sirna
    a , b Protein expression of <t>HMGA1</t> in DCM mouse hearts ( n = 6 samples per group, a and b from the same sample. * P < 0.05 vs. the control (CON) group). c , d Western blot image and quantification of HMGA1 in high-glucose (HG)-stimulated neonatal rat cardiomyocytes (NRCMs) ( n = 6, * P < 0.05 vs. the CON group). e HMGA1 and α-actin staining in diabetic cardiomyopathy (DCM) mouse hearts ( n = 5 samples per group). f HMGA1 and α-actin staining in HG-stimulated NRCMs ( n = 5). All the in vitro experiments were performed independently for three times.
    Mouse Hmga1 Sirna, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/mouse hmga1 sirna/product/Santa Cruz Biotechnology
    Average 93 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    mouse hmga1 sirna - by Bioz Stars, 2024-04
    93/100 stars
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    92
    DSMZ name micromonospora ferruginea sp nov
    HPLC-ELSD chromatogram of the crude extract of <t>Micromonospora</t> sp. 28ISP2-46 T .
    Name Micromonospora Ferruginea Sp Nov, supplied by DSMZ, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/name micromonospora ferruginea sp nov/product/DSMZ
    Average 92 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    name micromonospora ferruginea sp nov - by Bioz Stars, 2024-04
    92/100 stars
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    Standard format Plasmid sent in bacteria as agar stab
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    Image Search Results


    a , b Protein expression of HMGA1 in DCM mouse hearts ( n = 6 samples per group, a and b from the same sample. * P < 0.05 vs. the control (CON) group). c , d Western blot image and quantification of HMGA1 in high-glucose (HG)-stimulated neonatal rat cardiomyocytes (NRCMs) ( n = 6, * P < 0.05 vs. the CON group). e HMGA1 and α-actin staining in diabetic cardiomyopathy (DCM) mouse hearts ( n = 5 samples per group). f HMGA1 and α-actin staining in HG-stimulated NRCMs ( n = 5). All the in vitro experiments were performed independently for three times.

    Journal: Cell Death & Disease

    Article Title: High-mobility group AT-hook 1 promotes cardiac dysfunction in diabetic cardiomyopathy via autophagy inhibition

    doi: 10.1038/s41419-020-2316-4

    Figure Lengend Snippet: a , b Protein expression of HMGA1 in DCM mouse hearts ( n = 6 samples per group, a and b from the same sample. * P < 0.05 vs. the control (CON) group). c , d Western blot image and quantification of HMGA1 in high-glucose (HG)-stimulated neonatal rat cardiomyocytes (NRCMs) ( n = 6, * P < 0.05 vs. the CON group). e HMGA1 and α-actin staining in diabetic cardiomyopathy (DCM) mouse hearts ( n = 5 samples per group). f HMGA1 and α-actin staining in HG-stimulated NRCMs ( n = 5). All the in vitro experiments were performed independently for three times.

    Article Snippet: AAV9-shHMGA1 was constructed with mouse HMGA1 siRNA from Santa Cruz (sc-44334).

    Techniques: Expressing, Western Blot, Staining, In Vitro

    a Western blot image and quantification of HMGA1 in NRCMs transfected with Ad-HMGA1 ( n = 6, * P < 0.05 vs. the Ad-NC group). b – e NRCMs were transfected with Ad-HMGA1 and then stimulated with HG for 48 h. b Cell viability detected by MTT assay in NRCMs in the indicated group ( n = 6). c mRNA expression of proinflammatory markers in NRCMs ( n = 6). d TUNEL staining and quantification results in NRCMs ( n = 5). e Western blot image and quantification of Bax, Bcl-2, and cytochrome C ( n = 6). * P < 0.05 vs. the Ad-NC-CON group; # P < 0.05 vs. the Ad-NC-HG group. f Western blot image and quantification of HMGA1 in NRCMs transfected with HMGA1 siRNA ( n = 6, * P < 0.05 vs. the ScRNA group). g – j NRCMs were transfected with HMGA1 siRNA and then stimulated with HG for 48 h. g Cell viability detected by MTT assay in NRCMs ( n = 6). h mRNA levels of proinflammatory markers in NRCMs ( n = 6). i TUNEL staining and quantification results in NRCMs ( n = 5). j Protein expression of Bax, Bcl-2, and cytochrome C ( n = 6). * P < 0.05 vs. the ScRNA-CON group; # P < 0.05 vs. the ScRNA-HG group. All the in vitro experiments were performed independently for three times.

    Journal: Cell Death & Disease

    Article Title: High-mobility group AT-hook 1 promotes cardiac dysfunction in diabetic cardiomyopathy via autophagy inhibition

    doi: 10.1038/s41419-020-2316-4

    Figure Lengend Snippet: a Western blot image and quantification of HMGA1 in NRCMs transfected with Ad-HMGA1 ( n = 6, * P < 0.05 vs. the Ad-NC group). b – e NRCMs were transfected with Ad-HMGA1 and then stimulated with HG for 48 h. b Cell viability detected by MTT assay in NRCMs in the indicated group ( n = 6). c mRNA expression of proinflammatory markers in NRCMs ( n = 6). d TUNEL staining and quantification results in NRCMs ( n = 5). e Western blot image and quantification of Bax, Bcl-2, and cytochrome C ( n = 6). * P < 0.05 vs. the Ad-NC-CON group; # P < 0.05 vs. the Ad-NC-HG group. f Western blot image and quantification of HMGA1 in NRCMs transfected with HMGA1 siRNA ( n = 6, * P < 0.05 vs. the ScRNA group). g – j NRCMs were transfected with HMGA1 siRNA and then stimulated with HG for 48 h. g Cell viability detected by MTT assay in NRCMs ( n = 6). h mRNA levels of proinflammatory markers in NRCMs ( n = 6). i TUNEL staining and quantification results in NRCMs ( n = 5). j Protein expression of Bax, Bcl-2, and cytochrome C ( n = 6). * P < 0.05 vs. the ScRNA-CON group; # P < 0.05 vs. the ScRNA-HG group. All the in vitro experiments were performed independently for three times.

    Article Snippet: AAV9-shHMGA1 was constructed with mouse HMGA1 siRNA from Santa Cruz (sc-44334).

    Techniques: Western Blot, Transfection, MTT Assay, Expressing, TUNEL Assay, Staining, In Vitro

    a , c – g Mice received an AAV9-HMGA1 injection at 10 weeks after the final STZ injection ( n = 12 per group). a Protein levels of HMGA1 in mouse hearts 6 weeks after AAV9-HMGA1 injection and with or without STZ injection ( n = 6 mice hearts). b Mice received an AAV9-HMGA1 injection for 6 weeks. The expression of HMGA1 in cardiomyocytes ( n = 6) and fibroblasts ( n = 6) isolated from heart tissues. c Body weights and blood glucose levels in DCM mice 0, 1, 2, 3, and 4 months after the final STZ injection ( n = 12 sample). d Immunohistochemical staining and quantification results for CD45, CD68, and TNFα in DCM mouse hearts ( n = 5). e mRNA levels of proinflammatory markers in DCM mouse hearts ( n = 6). f TUNEL staining and quantification results in DCM mouse hearts ( n = 5). g Protein levels of Bax, Bcl-2, and cytochrome C in DCM mouse hearts ( n = 6). * P < 0.05 vs. the AAV9-NC-CON group; # P < 0.05 vs. the AAV9-NC-DCM group.

    Journal: Cell Death & Disease

    Article Title: High-mobility group AT-hook 1 promotes cardiac dysfunction in diabetic cardiomyopathy via autophagy inhibition

    doi: 10.1038/s41419-020-2316-4

    Figure Lengend Snippet: a , c – g Mice received an AAV9-HMGA1 injection at 10 weeks after the final STZ injection ( n = 12 per group). a Protein levels of HMGA1 in mouse hearts 6 weeks after AAV9-HMGA1 injection and with or without STZ injection ( n = 6 mice hearts). b Mice received an AAV9-HMGA1 injection for 6 weeks. The expression of HMGA1 in cardiomyocytes ( n = 6) and fibroblasts ( n = 6) isolated from heart tissues. c Body weights and blood glucose levels in DCM mice 0, 1, 2, 3, and 4 months after the final STZ injection ( n = 12 sample). d Immunohistochemical staining and quantification results for CD45, CD68, and TNFα in DCM mouse hearts ( n = 5). e mRNA levels of proinflammatory markers in DCM mouse hearts ( n = 6). f TUNEL staining and quantification results in DCM mouse hearts ( n = 5). g Protein levels of Bax, Bcl-2, and cytochrome C in DCM mouse hearts ( n = 6). * P < 0.05 vs. the AAV9-NC-CON group; # P < 0.05 vs. the AAV9-NC-DCM group.

    Article Snippet: AAV9-shHMGA1 was constructed with mouse HMGA1 siRNA from Santa Cruz (sc-44334).

    Techniques: Injection, Expressing, Isolation, Immunohistochemical staining, Staining, TUNEL Assay

    a – c Protein levels of LC3, P62, and Atg7 in NRCMs transfected with Ad-HMGA1 ( n = 6). d NRCMs were transfected with mRFP-GFP-LC3-adenovirus and Ad-HMGA1 and then stimulated with HG for 48 h. mRFP and GFP fluorescence and quantitated yellow puncta number per cell ( n = 5). * P < 0.05 vs. the Ad-NC-CON group; # P < 0.05 vs. the Ad-NC-HG group. e – g Western blot image and quantification of LC3, P62, and Atg7 in NRCMs transfected with HMGA1 siRNA ( n = 6). h NRCMs were transfected with mRFP-GFP-LC3-adenovirus and HMGA1 siRNA and then stimulated with HG for 48 h. mRFP and GFP fluorescence and quantitated red, green, yellow puncta number per cell ( n = 5). * P < 0.05 vs. the ScRNA-CON group; # P < 0.05 vs. the ScRNA-HG group. All the in vitro experiments were performed independently for three times.

    Journal: Cell Death & Disease

    Article Title: High-mobility group AT-hook 1 promotes cardiac dysfunction in diabetic cardiomyopathy via autophagy inhibition

    doi: 10.1038/s41419-020-2316-4

    Figure Lengend Snippet: a – c Protein levels of LC3, P62, and Atg7 in NRCMs transfected with Ad-HMGA1 ( n = 6). d NRCMs were transfected with mRFP-GFP-LC3-adenovirus and Ad-HMGA1 and then stimulated with HG for 48 h. mRFP and GFP fluorescence and quantitated yellow puncta number per cell ( n = 5). * P < 0.05 vs. the Ad-NC-CON group; # P < 0.05 vs. the Ad-NC-HG group. e – g Western blot image and quantification of LC3, P62, and Atg7 in NRCMs transfected with HMGA1 siRNA ( n = 6). h NRCMs were transfected with mRFP-GFP-LC3-adenovirus and HMGA1 siRNA and then stimulated with HG for 48 h. mRFP and GFP fluorescence and quantitated red, green, yellow puncta number per cell ( n = 5). * P < 0.05 vs. the ScRNA-CON group; # P < 0.05 vs. the ScRNA-HG group. All the in vitro experiments were performed independently for three times.

    Article Snippet: AAV9-shHMGA1 was constructed with mouse HMGA1 siRNA from Santa Cruz (sc-44334).

    Techniques: Transfection, Fluorescence, Western Blot, In Vitro

    NRCMs were transfected with HMGA1 siRNA, Atg5 siRNA, treated with bafilomycin A1 (Baf) for 2 h and then stimulated with HG for 48 h. NRCMs were transfected with Ad-HMGA1, Ad-Atg5, and treated with rapamycin (RAP) for 2 h, and then stimulated with HG for 48 h. a The expression level of Atg5 when cells were transfected with Atg5 siRNA or Ad-Atg5 ( n = 6) . b NRCMs were transfected with mRFP-GFP-LC3-adenovirus. mRFP and GFP fluorescence and quantitated red, green, yellow puncta number per cell ( n = 5). c Transcription levels of proinflammatory markers in NRCMs in the indicated group ( n = 6). d , e TUNEL staining and quantification results in NRCMs in the indicated groups ( n = 5). * P < 0.05 vs. the CON group; # P < 0.05 vs. the HG group. All the in vitro experiments were performed independently for three times.

    Journal: Cell Death & Disease

    Article Title: High-mobility group AT-hook 1 promotes cardiac dysfunction in diabetic cardiomyopathy via autophagy inhibition

    doi: 10.1038/s41419-020-2316-4

    Figure Lengend Snippet: NRCMs were transfected with HMGA1 siRNA, Atg5 siRNA, treated with bafilomycin A1 (Baf) for 2 h and then stimulated with HG for 48 h. NRCMs were transfected with Ad-HMGA1, Ad-Atg5, and treated with rapamycin (RAP) for 2 h, and then stimulated with HG for 48 h. a The expression level of Atg5 when cells were transfected with Atg5 siRNA or Ad-Atg5 ( n = 6) . b NRCMs were transfected with mRFP-GFP-LC3-adenovirus. mRFP and GFP fluorescence and quantitated red, green, yellow puncta number per cell ( n = 5). c Transcription levels of proinflammatory markers in NRCMs in the indicated group ( n = 6). d , e TUNEL staining and quantification results in NRCMs in the indicated groups ( n = 5). * P < 0.05 vs. the CON group; # P < 0.05 vs. the HG group. All the in vitro experiments were performed independently for three times.

    Article Snippet: AAV9-shHMGA1 was constructed with mouse HMGA1 siRNA from Santa Cruz (sc-44334).

    Techniques: Transfection, Expressing, Fluorescence, TUNEL Assay, Staining, In Vitro

    a , b , e Western blot image and quantification of phosphorylated (P-) and total (T-) AMPKα, mTOR, 4EBP1, AKT, ERK1/2, P27, and CDK2 in NRCMs transfected with Ad-HMGA1 ( n = 6, * P < 0.05 vs. the Ad-NC-CON group; # P < 0.05 vs. the Ad-NC-HG group). c , d , f Western blot image and quantification of phosphorylated and total AMPKα, mTOR, 4EBP1, AKT, ERK1/2, P27, and CDK2 in NRCMs transfected with HMGA1 siRNA ( n = 6, * P < 0.05 vs. the ScRNA-CON group; # P < 0.05 vs. the ScRNA-HG group). All the in vitro experiments were performed independently for three times.

    Journal: Cell Death & Disease

    Article Title: High-mobility group AT-hook 1 promotes cardiac dysfunction in diabetic cardiomyopathy via autophagy inhibition

    doi: 10.1038/s41419-020-2316-4

    Figure Lengend Snippet: a , b , e Western blot image and quantification of phosphorylated (P-) and total (T-) AMPKα, mTOR, 4EBP1, AKT, ERK1/2, P27, and CDK2 in NRCMs transfected with Ad-HMGA1 ( n = 6, * P < 0.05 vs. the Ad-NC-CON group; # P < 0.05 vs. the Ad-NC-HG group). c , d , f Western blot image and quantification of phosphorylated and total AMPKα, mTOR, 4EBP1, AKT, ERK1/2, P27, and CDK2 in NRCMs transfected with HMGA1 siRNA ( n = 6, * P < 0.05 vs. the ScRNA-CON group; # P < 0.05 vs. the ScRNA-HG group). All the in vitro experiments were performed independently for three times.

    Article Snippet: AAV9-shHMGA1 was constructed with mouse HMGA1 siRNA from Santa Cruz (sc-44334).

    Techniques: Western Blot, Transfection, In Vitro

    Mice received AAV9-HMGA1 and AAV9-P27 injections at 10 weeks after the final STZ injection ( n = 12 per group). a Protein levels of P27 in mouse hearts 6 weeks after AAV9-P27 injection ( n = 6). b Protein levels of LC3 and P62 in DCM mouse hearts ( n = 6). c , d Immunohistochemical staining and quantification results for CD45, CD68 in DCM mouse hearts ( n = 5). e Transcription levels of proinflammatory markers in DCM mouse hearts ( n = 6). f TUNEL staining and quantification results in mouse hearts ( n = 5). * P < 0.05 vs. the AAV9-NC-CON group.

    Journal: Cell Death & Disease

    Article Title: High-mobility group AT-hook 1 promotes cardiac dysfunction in diabetic cardiomyopathy via autophagy inhibition

    doi: 10.1038/s41419-020-2316-4

    Figure Lengend Snippet: Mice received AAV9-HMGA1 and AAV9-P27 injections at 10 weeks after the final STZ injection ( n = 12 per group). a Protein levels of P27 in mouse hearts 6 weeks after AAV9-P27 injection ( n = 6). b Protein levels of LC3 and P62 in DCM mouse hearts ( n = 6). c , d Immunohistochemical staining and quantification results for CD45, CD68 in DCM mouse hearts ( n = 5). e Transcription levels of proinflammatory markers in DCM mouse hearts ( n = 6). f TUNEL staining and quantification results in mouse hearts ( n = 5). * P < 0.05 vs. the AAV9-NC-CON group.

    Article Snippet: AAV9-shHMGA1 was constructed with mouse HMGA1 siRNA from Santa Cruz (sc-44334).

    Techniques: Injection, Immunohistochemical staining, Staining, TUNEL Assay

    a miR-222 expression levels in NRCMs transfected with Ad-HMGA1 or HMGA1 siRNA ( n = 6, * P < 0.05 vs. the ScRNA/Ad-NC group) . b Relative luciferase activity of miR-222 in NRCMs transfected with Ad-HMGA1 or HMGA1 siRNA. c ULK1 mRNA levels in NRCMs transfected with Ad-HMGA1 or HMGA1 siRNA ( n = 6). d – f NRCMs were transfected with Ad-HMGA1, treated with miR-222 antagomir, and then stimulated with HG for 48 h. d Protein expression levels of P27 in the indicated group ( n = 6). e Transcription levels of proinflammatory markers in NRCMs ( n = 6). f TUNEL staining and quantification results in NRCMs ( n = 5). * P < 0.05 vs. the CON group; # P < 0.05 vs. the HG group. All the in vitro experiments were performed independently for three times.

    Journal: Cell Death & Disease

    Article Title: High-mobility group AT-hook 1 promotes cardiac dysfunction in diabetic cardiomyopathy via autophagy inhibition

    doi: 10.1038/s41419-020-2316-4

    Figure Lengend Snippet: a miR-222 expression levels in NRCMs transfected with Ad-HMGA1 or HMGA1 siRNA ( n = 6, * P < 0.05 vs. the ScRNA/Ad-NC group) . b Relative luciferase activity of miR-222 in NRCMs transfected with Ad-HMGA1 or HMGA1 siRNA. c ULK1 mRNA levels in NRCMs transfected with Ad-HMGA1 or HMGA1 siRNA ( n = 6). d – f NRCMs were transfected with Ad-HMGA1, treated with miR-222 antagomir, and then stimulated with HG for 48 h. d Protein expression levels of P27 in the indicated group ( n = 6). e Transcription levels of proinflammatory markers in NRCMs ( n = 6). f TUNEL staining and quantification results in NRCMs ( n = 5). * P < 0.05 vs. the CON group; # P < 0.05 vs. the HG group. All the in vitro experiments were performed independently for three times.

    Article Snippet: AAV9-shHMGA1 was constructed with mouse HMGA1 siRNA from Santa Cruz (sc-44334).

    Techniques: Expressing, Transfection, Luciferase, Activity Assay, TUNEL Assay, Staining, In Vitro

    HPLC-ELSD chromatogram of the crude extract of Micromonospora sp. 28ISP2-46 T .

    Journal: Marine Drugs

    Article Title: A New Micromonospora Strain with Antibiotic Activity Isolated from the Microbiome of a Mid-Atlantic Deep-Sea Sponge

    doi: 10.3390/md19020105

    Figure Lengend Snippet: HPLC-ELSD chromatogram of the crude extract of Micromonospora sp. 28ISP2-46 T .

    Article Snippet: 16S rRNA gene and whole-genome sequencing of strain 28ISP2-46 T was consistent with its classification as a novel species of the genus Micromonospora , for which the name Micromonospora ferruginea sp. nov. (NCTC number: 14469 T , DSMZ number: 111791 T ) is proposed.

    Techniques:

    Phylogenetic tree for strain 28ISP2-46 T and the other group IA Micromonospora species generated using the TYGS and drawn with iTOL. The tree was constructed using FastME 2.1.6.1 from GBDP distances calculated from genome sequences. The branch lengths are scaled in terms of GBDP distance formula d5. The numbers above branches are GBDP pseudo-bootstrap support values from 100 replications, with an average branch support of 98.4% and a delta statistic of 0.135. The tree was rooted using Micromonospora pallida as the outgroup.

    Journal: Marine Drugs

    Article Title: A New Micromonospora Strain with Antibiotic Activity Isolated from the Microbiome of a Mid-Atlantic Deep-Sea Sponge

    doi: 10.3390/md19020105

    Figure Lengend Snippet: Phylogenetic tree for strain 28ISP2-46 T and the other group IA Micromonospora species generated using the TYGS and drawn with iTOL. The tree was constructed using FastME 2.1.6.1 from GBDP distances calculated from genome sequences. The branch lengths are scaled in terms of GBDP distance formula d5. The numbers above branches are GBDP pseudo-bootstrap support values from 100 replications, with an average branch support of 98.4% and a delta statistic of 0.135. The tree was rooted using Micromonospora pallida as the outgroup.

    Article Snippet: 16S rRNA gene and whole-genome sequencing of strain 28ISP2-46 T was consistent with its classification as a novel species of the genus Micromonospora , for which the name Micromonospora ferruginea sp. nov. (NCTC number: 14469 T , DSMZ number: 111791 T ) is proposed.

    Techniques: Generated, Construct

    Biosynthetic gene clusters identified in the genome of strain 28ISP2-46 T using AntiSMASH.

    Journal: Marine Drugs

    Article Title: A New Micromonospora Strain with Antibiotic Activity Isolated from the Microbiome of a Mid-Atlantic Deep-Sea Sponge

    doi: 10.3390/md19020105

    Figure Lengend Snippet: Biosynthetic gene clusters identified in the genome of strain 28ISP2-46 T using AntiSMASH.

    Article Snippet: 16S rRNA gene and whole-genome sequencing of strain 28ISP2-46 T was consistent with its classification as a novel species of the genus Micromonospora , for which the name Micromonospora ferruginea sp. nov. (NCTC number: 14469 T , DSMZ number: 111791 T ) is proposed.

    Techniques:

    ( a ) ClusterBLAST hits for the kosinostatin cluster. Homologues of kst genes are coloured as follows: kstA genes red, kstB genes yellow, kstC genes green, kstD genes blue, kstRg genes black, kstRs genes white, and genes uninvolved in kosinostatin biosynthesis grey. A black line at the end of a cluster indicates that it is close to the end of a contig. Gene sizes not to scale. The NCBI protein IDs for first and last coloured genes of each cluster are as follows: Micromonospora sp. TP-A0468 AFJ52719.1 and AFJ52701.1; Micromonospora sp. 28ISP2-46 T QLQ36639.1 and QLQ36600.1; N. valliformis WP_017579213.1 and WP_017579258.1; N. alkaliphila WP_017604198.1 and WP_017604153.1; Streptomyces sp. SM8 PKA38649.1 and PKA38729.1; Streptomyces sp. ScaeMP-6W SCE31237.1 and SCE31955.1; A. bangkokensis OLR89622.1 and OLR89598.1; M. haikouensis SCF11188.1 and SCF11278.1. ( b ) The same gene clusters rearranged for ease of comparison. Coloured as 6a.

    Journal: Marine Drugs

    Article Title: A New Micromonospora Strain with Antibiotic Activity Isolated from the Microbiome of a Mid-Atlantic Deep-Sea Sponge

    doi: 10.3390/md19020105

    Figure Lengend Snippet: ( a ) ClusterBLAST hits for the kosinostatin cluster. Homologues of kst genes are coloured as follows: kstA genes red, kstB genes yellow, kstC genes green, kstD genes blue, kstRg genes black, kstRs genes white, and genes uninvolved in kosinostatin biosynthesis grey. A black line at the end of a cluster indicates that it is close to the end of a contig. Gene sizes not to scale. The NCBI protein IDs for first and last coloured genes of each cluster are as follows: Micromonospora sp. TP-A0468 AFJ52719.1 and AFJ52701.1; Micromonospora sp. 28ISP2-46 T QLQ36639.1 and QLQ36600.1; N. valliformis WP_017579213.1 and WP_017579258.1; N. alkaliphila WP_017604198.1 and WP_017604153.1; Streptomyces sp. SM8 PKA38649.1 and PKA38729.1; Streptomyces sp. ScaeMP-6W SCE31237.1 and SCE31955.1; A. bangkokensis OLR89622.1 and OLR89598.1; M. haikouensis SCF11188.1 and SCF11278.1. ( b ) The same gene clusters rearranged for ease of comparison. Coloured as 6a.

    Article Snippet: 16S rRNA gene and whole-genome sequencing of strain 28ISP2-46 T was consistent with its classification as a novel species of the genus Micromonospora , for which the name Micromonospora ferruginea sp. nov. (NCTC number: 14469 T , DSMZ number: 111791 T ) is proposed.

    Techniques: