20-2200 Search Results


trypticase soy broth tsb  (ATCC)
99
ATCC trypticase soy broth tsb
Trypticase Soy Broth Tsb, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/trypticase soy broth tsb/product/ATCC
Average 99 stars, based on 1 article reviews
trypticase soy broth tsb - by Bioz Stars, 2025-03
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methyl behenate  (Larodan)
92
Larodan methyl behenate
Methyl Behenate, supplied by Larodan, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 92 stars, based on 1 article reviews
methyl behenate - by Bioz Stars, 2025-03
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astrocyte toxin α aminoadipic acid  (Santa Cruz Biotechnology)
88
Santa Cruz Biotechnology astrocyte toxin α aminoadipic acid
Control studies supporting a complement bystander mechanism for neuron injury. a Astrocyte-neuron cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% C1q-depleted serum (i) or with 20 μg/ml AQP4-IgG and 2% C6-depleted serum (ii); cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% human complement that was pre-exposed for 1 h to 1 μg/ml Fc hexamer (complement inhibitor) (iii); AQP4 −/− astrocyte-neuron cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% human complement (iv). GFAP, MAP2, and C5b-9 immunofluorescence as indicated, with dead cells stained red. b Direct astrocyte injury caused by <t>α-aminoadipic</t> acid. Cocultures were exposed to 2 mM α-aminoadipic acid for 75 min, with AQP4 and MAP2 immunofluorescence and dead cell stain shown. Filled arrowheads show dead astrocytes. Micrographs representative of three sets of studies done on different cocultures
Astrocyte Toxin α Aminoadipic Acid, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 88/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/astrocyte toxin α aminoadipic acid/product/Santa Cruz Biotechnology
Average 88 stars, based on 1 article reviews
astrocyte toxin α aminoadipic acid - by Bioz Stars, 2025-03
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spectrum aeromed 20 2200 stretcher base module  (AeroMed Inc)
86
AeroMed Inc spectrum aeromed 20 2200 stretcher base module
Control studies supporting a complement bystander mechanism for neuron injury. a Astrocyte-neuron cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% C1q-depleted serum (i) or with 20 μg/ml AQP4-IgG and 2% C6-depleted serum (ii); cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% human complement that was pre-exposed for 1 h to 1 μg/ml Fc hexamer (complement inhibitor) (iii); AQP4 −/− astrocyte-neuron cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% human complement (iv). GFAP, MAP2, and C5b-9 immunofluorescence as indicated, with dead cells stained red. b Direct astrocyte injury caused by <t>α-aminoadipic</t> acid. Cocultures were exposed to 2 mM α-aminoadipic acid for 75 min, with AQP4 and MAP2 immunofluorescence and dead cell stain shown. Filled arrowheads show dead astrocytes. Micrographs representative of three sets of studies done on different cocultures
Spectrum Aeromed 20 2200 Stretcher Base Module, supplied by AeroMed Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/spectrum aeromed 20 2200 stretcher base module/product/AeroMed Inc
Average 86 stars, based on 1 article reviews
spectrum aeromed 20 2200 stretcher base module - by Bioz Stars, 2025-03
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Image Search Results


Control studies supporting a complement bystander mechanism for neuron injury. a Astrocyte-neuron cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% C1q-depleted serum (i) or with 20 μg/ml AQP4-IgG and 2% C6-depleted serum (ii); cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% human complement that was pre-exposed for 1 h to 1 μg/ml Fc hexamer (complement inhibitor) (iii); AQP4 −/− astrocyte-neuron cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% human complement (iv). GFAP, MAP2, and C5b-9 immunofluorescence as indicated, with dead cells stained red. b Direct astrocyte injury caused by α-aminoadipic acid. Cocultures were exposed to 2 mM α-aminoadipic acid for 75 min, with AQP4 and MAP2 immunofluorescence and dead cell stain shown. Filled arrowheads show dead astrocytes. Micrographs representative of three sets of studies done on different cocultures

Journal: Journal of Neuroinflammation

Article Title: Complement-dependent bystander injury to neurons in AQP4-IgG seropositive neuromyelitis optica

doi: 10.1186/s12974-018-1333-z

Figure Lengend Snippet: Control studies supporting a complement bystander mechanism for neuron injury. a Astrocyte-neuron cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% C1q-depleted serum (i) or with 20 μg/ml AQP4-IgG and 2% C6-depleted serum (ii); cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% human complement that was pre-exposed for 1 h to 1 μg/ml Fc hexamer (complement inhibitor) (iii); AQP4 −/− astrocyte-neuron cocultures were incubated for 2 h with 20 μg/ml AQP4-IgG and 2% human complement (iv). GFAP, MAP2, and C5b-9 immunofluorescence as indicated, with dead cells stained red. b Direct astrocyte injury caused by α-aminoadipic acid. Cocultures were exposed to 2 mM α-aminoadipic acid for 75 min, with AQP4 and MAP2 immunofluorescence and dead cell stain shown. Filled arrowheads show dead astrocytes. Micrographs representative of three sets of studies done on different cocultures

Article Snippet: In some experiments, the astrocyte toxin α-aminoadipic acid (Santa Cruz Biotechnology, Dallas, TX) at 2 mM was added to astrocyte-neuron cocultures for 75 min. For live-cell real-time imaging, astrocyte-neuron cocultures were grown on 6-well plates and imaged by phase-contrast optics using a 20×, 0.45 NA objective lens on a Nikon Eclipse Ti microscope equipped with an environmental chamber at 37 °C and 5% CO 2 .

Techniques: Incubation, Immunofluorescence, Staining