[4298 Search Results


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    • total akt  (Cell Signaling Technology Inc)
    96
    Cell Signaling Technology Inc total akt
    Effect of TF3 on <t>Akt/MDM2</t> pathway in A2780/CP70 cells. Protein lysates were prepared from A2780/CP70 cells after treatment with various concentrations (0, 5, 10 and 20 μM) of TF3 for 24 h. <t>phospho-Akt,</t> <t>total-Akt</t> and MDM2 protein expression levels were analyzed by western blotting. The quantification histograms are shown with error bars. Data represent means ± SD from three independent experiments. Significant differences among the treatments are indicated by different letters (p<0.05).
    Total Akt, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/total akt/product/Cell Signaling Technology Inc
    Average 96 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    total akt - by Bioz Stars, 2023-03
    96/100 stars
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    mirna hsa mir 4298 465290 mat  (Thermo Fisher)
    85
    Thermo Fisher mirna hsa mir 4298 465290 mat
    Correlation of serum levels of three miRNAs with clinicopathological factors in GBM patients.
    Mirna Hsa Mir 4298 465290 Mat, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 85/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/mirna hsa mir 4298 465290 mat/product/Thermo Fisher
    Average 85 stars, based on 1 article reviews
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    pamoic acid  (Tocris)
    86
    Tocris pamoic acid
    Lodoxamide induced the activation of GPR35, and this activation was inhibited by CID2745687 (a GPR35 selective antagonist). (A) Concentration–response curves for lodoxamide, <t>pamoic</t> acid <t>and</t> <t>zaprinast</t> as determined by the AP‐TGFα shedding assay. Agonism of GPR35 agonists was examined in GPR35 coexpressed with a mixture of eight Gα proteins in HEK‐293 cells. Results are presented as the means ± SEM of three individual experiments. (B) Inhibition of the lodoxamide‐induced GPR35 activation by CID2745687. Antagonism by CID2745687 was tested in the presence of 10 nM of lodoxamide. Results are presented as the means ± SEM of three individual experiments.
    Pamoic Acid, supplied by Tocris, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/pamoic acid/product/Tocris
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    pamoic acid - by Bioz Stars, 2023-03
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    recombinant mouse nephronectin  (R&D Systems)
    93
    R&D Systems recombinant mouse nephronectin
    Lodoxamide induced the activation of GPR35, and this activation was inhibited by CID2745687 (a GPR35 selective antagonist). (A) Concentration–response curves for lodoxamide, <t>pamoic</t> acid <t>and</t> <t>zaprinast</t> as determined by the AP‐TGFα shedding assay. Agonism of GPR35 agonists was examined in GPR35 coexpressed with a mixture of eight Gα proteins in HEK‐293 cells. Results are presented as the means ± SEM of three individual experiments. (B) Inhibition of the lodoxamide‐induced GPR35 activation by CID2745687. Antagonism by CID2745687 was tested in the presence of 10 nM of lodoxamide. Results are presented as the means ± SEM of three individual experiments.
    Recombinant Mouse Nephronectin, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/recombinant mouse nephronectin/product/R&D Systems
    Average 93 stars, based on 1 article reviews
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    z rouxii atcc 4298 strain  (ATCC)
    86
    ATCC z rouxii atcc 4298 strain
    Lodoxamide induced the activation of GPR35, and this activation was inhibited by CID2745687 (a GPR35 selective antagonist). (A) Concentration–response curves for lodoxamide, <t>pamoic</t> acid <t>and</t> <t>zaprinast</t> as determined by the AP‐TGFα shedding assay. Agonism of GPR35 agonists was examined in GPR35 coexpressed with a mixture of eight Gα proteins in HEK‐293 cells. Results are presented as the means ± SEM of three individual experiments. (B) Inhibition of the lodoxamide‐induced GPR35 activation by CID2745687. Antagonism by CID2745687 was tested in the presence of 10 nM of lodoxamide. Results are presented as the means ± SEM of three individual experiments.
    Z Rouxii Atcc 4298 Strain, supplied by ATCC, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/z rouxii atcc 4298 strain/product/ATCC
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    z rouxii atcc 4298 strain - by Bioz Stars, 2023-03
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    Image Search Results


    Effect of TF3 on Akt/MDM2 pathway in A2780/CP70 cells. Protein lysates were prepared from A2780/CP70 cells after treatment with various concentrations (0, 5, 10 and 20 μM) of TF3 for 24 h. phospho-Akt, total-Akt and MDM2 protein expression levels were analyzed by western blotting. The quantification histograms are shown with error bars. Data represent means ± SD from three independent experiments. Significant differences among the treatments are indicated by different letters (p<0.05).

    Journal: International Journal of Oncology

    Article Title: Theaflavin-3, 3′-digallate induces apoptosis and G2 cell cycle arrest through the Akt/MDM2/p53 pathway in cisplatin-resistant ovarian cancer A2780/CP70 cells

    doi: 10.3892/ijo.2016.3472

    Figure Lengend Snippet: Effect of TF3 on Akt/MDM2 pathway in A2780/CP70 cells. Protein lysates were prepared from A2780/CP70 cells after treatment with various concentrations (0, 5, 10 and 20 μM) of TF3 for 24 h. phospho-Akt, total-Akt and MDM2 protein expression levels were analyzed by western blotting. The quantification histograms are shown with error bars. Data represent means ± SD from three independent experiments. Significant differences among the treatments are indicated by different letters (p<0.05).

    Article Snippet: The primary antibodies against caspase-8 and -9, Puma, Bax, cyclin B1, phospho-cdc2 (Tyr15), cdc2, DR5, FADD, phospho-Akt (Thr180/Tyr182) and total-Akt were purchased from Cell Signaling Technology, Inc. (Danvers, MA, USA).

    Techniques: Expressing, Western Blot

    Correlation of serum levels of three miRNAs with clinicopathological factors in GBM patients.

    Journal: PLoS ONE

    Article Title: Low serum level of miR-485-3p predicts poor survival in patients with glioblastoma

    doi: 10.1371/journal.pone.0184969

    Figure Lengend Snippet: Correlation of serum levels of three miRNAs with clinicopathological factors in GBM patients.

    Article Snippet: The assay ID number is 001141 for mir-451a, 001277 for mir-485-3p, 465290_mat for mir-4298, 000391 for mir-16.

    Techniques: Expressing

    The average serum levels of the 3 miRNAs in GBM patients (n = 24) were compared with those in healthy volunteer controls (n = 12). (A) serum level of miR-451a in GBM patients is significantly higher than that in healthy controls. (B) serum level of miR-485-3p in GBM patients is significantly lower than that in healthy controls. (C) serum level of miR-4298 in GBM patients is significantly lower than that in healthy controls. The serum levels of the 3 miRNAs in GBM patients are in concordance with the results of microarray analysis. miRNA levels in each sample were normalized to miR-16. ΔCt = mean value of Ct (reference miRNA16)–mean value of Ct (target miRNA).

    Journal: PLoS ONE

    Article Title: Low serum level of miR-485-3p predicts poor survival in patients with glioblastoma

    doi: 10.1371/journal.pone.0184969

    Figure Lengend Snippet: The average serum levels of the 3 miRNAs in GBM patients (n = 24) were compared with those in healthy volunteer controls (n = 12). (A) serum level of miR-451a in GBM patients is significantly higher than that in healthy controls. (B) serum level of miR-485-3p in GBM patients is significantly lower than that in healthy controls. (C) serum level of miR-4298 in GBM patients is significantly lower than that in healthy controls. The serum levels of the 3 miRNAs in GBM patients are in concordance with the results of microarray analysis. miRNA levels in each sample were normalized to miR-16. ΔCt = mean value of Ct (reference miRNA16)–mean value of Ct (target miRNA).

    Article Snippet: The assay ID number is 001141 for mir-451a, 001277 for mir-485-3p, 465290_mat for mir-4298, 000391 for mir-16.

    Techniques: Microarray

    (A) There is no difference of survivals (OS and PFS) between patients with a low and high level of serum miR-451a (B) Patients with a low level of serum miR-485-3p had significantly shorter OS (P = 0.023) and PFS (P = 0.004) than those with a high level. (C) There is no difference of survivals (OS and PFS) between patients with a low and high level of serum miR-4298.

    Journal: PLoS ONE

    Article Title: Low serum level of miR-485-3p predicts poor survival in patients with glioblastoma

    doi: 10.1371/journal.pone.0184969

    Figure Lengend Snippet: (A) There is no difference of survivals (OS and PFS) between patients with a low and high level of serum miR-451a (B) Patients with a low level of serum miR-485-3p had significantly shorter OS (P = 0.023) and PFS (P = 0.004) than those with a high level. (C) There is no difference of survivals (OS and PFS) between patients with a low and high level of serum miR-4298.

    Article Snippet: The assay ID number is 001141 for mir-451a, 001277 for mir-485-3p, 465290_mat for mir-4298, 000391 for mir-16.

    Techniques:

    Univariate and multivariate COX proportional hazard analyses of prognostic factors for progression-free survival in GBM patients.

    Journal: PLoS ONE

    Article Title: Low serum level of miR-485-3p predicts poor survival in patients with glioblastoma

    doi: 10.1371/journal.pone.0184969

    Figure Lengend Snippet: Univariate and multivariate COX proportional hazard analyses of prognostic factors for progression-free survival in GBM patients.

    Article Snippet: The assay ID number is 001141 for mir-451a, 001277 for mir-485-3p, 465290_mat for mir-4298, 000391 for mir-16.

    Techniques: Expressing

    Univariate and multivariate COX proportional hazard analyses of prognostic factors for overall survival in GBM patients.

    Journal: PLoS ONE

    Article Title: Low serum level of miR-485-3p predicts poor survival in patients with glioblastoma

    doi: 10.1371/journal.pone.0184969

    Figure Lengend Snippet: Univariate and multivariate COX proportional hazard analyses of prognostic factors for overall survival in GBM patients.

    Article Snippet: The assay ID number is 001141 for mir-451a, 001277 for mir-485-3p, 465290_mat for mir-4298, 000391 for mir-16.

    Techniques: Expressing

    Lodoxamide induced the activation of GPR35, and this activation was inhibited by CID2745687 (a GPR35 selective antagonist). (A) Concentration–response curves for lodoxamide, pamoic acid and zaprinast as determined by the AP‐TGFα shedding assay. Agonism of GPR35 agonists was examined in GPR35 coexpressed with a mixture of eight Gα proteins in HEK‐293 cells. Results are presented as the means ± SEM of three individual experiments. (B) Inhibition of the lodoxamide‐induced GPR35 activation by CID2745687. Antagonism by CID2745687 was tested in the presence of 10 nM of lodoxamide. Results are presented as the means ± SEM of three individual experiments.

    Journal: British Journal of Pharmacology

    Article Title: GPR35 mediates lodoxamide‐induced migration inhibitory response but not CXCL17‐induced migration stimulatory response in THP‐1 cells; is GPR35 a receptor for CXCL17?

    doi: 10.1111/bph.14082

    Figure Lengend Snippet: Lodoxamide induced the activation of GPR35, and this activation was inhibited by CID2745687 (a GPR35 selective antagonist). (A) Concentration–response curves for lodoxamide, pamoic acid and zaprinast as determined by the AP‐TGFα shedding assay. Agonism of GPR35 agonists was examined in GPR35 coexpressed with a mixture of eight Gα proteins in HEK‐293 cells. Results are presented as the means ± SEM of three individual experiments. (B) Inhibition of the lodoxamide‐induced GPR35 activation by CID2745687. Antagonism by CID2745687 was tested in the presence of 10 nM of lodoxamide. Results are presented as the means ± SEM of three individual experiments.

    Article Snippet: Recombinant mouse CXCL17 and human CXCL17 were purchased from R&D system (Minneapolis, MN, USA), and zaprinast, CID‐2745687 and pamoic acid were from Tocris (Ellisville, Missouri, USA).

    Techniques: Activation Assay, Concentration Assay, Inhibition