treatment human colorectal cancer cell lines  (ATCC)


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    ATCC treatment human colorectal cancer cell lines
    <t>Anti-colorectal</t> cancer effects of curcumin and/or irinotecan are dependent on ROS ( A , B ) The effects of NAC on cell growth inhibition induced by curcumin and/or irinotecan. After pretreatment with 5 mM NAC for 2 h, LoVo cells (A) or HT-29 cells (B) were treated with curcumin and/or irinotecan for 24 h, then cell viability was assessed by CCK-8 assay. ( C , D ) The effects of NAC on apoptosis induced by curcumin and/or irinotecan. After cells were treated as described above, cell apoptosis was measured by Annexin V-FITC/PI staining. Values are means ± SEM. *
    Treatment Human Colorectal Cancer Cell Lines, supplied by ATCC, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/treatment human colorectal cancer cell lines/product/ATCC
    Average 86 stars, based on 1 article reviews
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    treatment human colorectal cancer cell lines - by Bioz Stars, 2022-09
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    1) Product Images from "Curcumin enhances the effects of irinotecan on colorectal cancer cells through the generation of reactive oxygen species and activation of the endoplasmic reticulum stress pathway"

    Article Title: Curcumin enhances the effects of irinotecan on colorectal cancer cells through the generation of reactive oxygen species and activation of the endoplasmic reticulum stress pathway

    Journal: Oncotarget

    doi: 10.18632/oncotarget.16828

    Anti-colorectal cancer effects of curcumin and/or irinotecan are dependent on ROS ( A , B ) The effects of NAC on cell growth inhibition induced by curcumin and/or irinotecan. After pretreatment with 5 mM NAC for 2 h, LoVo cells (A) or HT-29 cells (B) were treated with curcumin and/or irinotecan for 24 h, then cell viability was assessed by CCK-8 assay. ( C , D ) The effects of NAC on apoptosis induced by curcumin and/or irinotecan. After cells were treated as described above, cell apoptosis was measured by Annexin V-FITC/PI staining. Values are means ± SEM. *
    Figure Legend Snippet: Anti-colorectal cancer effects of curcumin and/or irinotecan are dependent on ROS ( A , B ) The effects of NAC on cell growth inhibition induced by curcumin and/or irinotecan. After pretreatment with 5 mM NAC for 2 h, LoVo cells (A) or HT-29 cells (B) were treated with curcumin and/or irinotecan for 24 h, then cell viability was assessed by CCK-8 assay. ( C , D ) The effects of NAC on apoptosis induced by curcumin and/or irinotecan. After cells were treated as described above, cell apoptosis was measured by Annexin V-FITC/PI staining. Values are means ± SEM. *

    Techniques Used: Inhibition, CCK-8 Assay, Staining

    ER Stress is mediates the anti-colorectal cancer effects of curcumin alone or combined with irinotecan ( A , B ) The effects of an ER stress inhibitor on cell growth inhibition induced by curcumin alone or with irinotecan. After pretreatment with 0.1 μM mithramycin (MTM) for 30 min, LoVo cells (A) or HT-29 cells (B) were treated with curcumin alone or with irinotecan for 24 h, then cell viability was assessed by CCK-8 assay. ( C , D ) The effects of an ER stress inhibitor on apoptosis induced by curcumin alone or with irinotecan. After cells were treated as described above, cell apoptosis was measured by Annexin V-FITC/PI staining. Values are means ± SEM. *
    Figure Legend Snippet: ER Stress is mediates the anti-colorectal cancer effects of curcumin alone or combined with irinotecan ( A , B ) The effects of an ER stress inhibitor on cell growth inhibition induced by curcumin alone or with irinotecan. After pretreatment with 0.1 μM mithramycin (MTM) for 30 min, LoVo cells (A) or HT-29 cells (B) were treated with curcumin alone or with irinotecan for 24 h, then cell viability was assessed by CCK-8 assay. ( C , D ) The effects of an ER stress inhibitor on apoptosis induced by curcumin alone or with irinotecan. After cells were treated as described above, cell apoptosis was measured by Annexin V-FITC/PI staining. Values are means ± SEM. *

    Techniques Used: Inhibition, CCK-8 Assay, Staining

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    ATCC treatment human colorectal cancer cell lines
    <t>Anti-colorectal</t> cancer effects of curcumin and/or irinotecan are dependent on ROS ( A , B ) The effects of NAC on cell growth inhibition induced by curcumin and/or irinotecan. After pretreatment with 5 mM NAC for 2 h, LoVo cells (A) or HT-29 cells (B) were treated with curcumin and/or irinotecan for 24 h, then cell viability was assessed by CCK-8 assay. ( C , D ) The effects of NAC on apoptosis induced by curcumin and/or irinotecan. After cells were treated as described above, cell apoptosis was measured by Annexin V-FITC/PI staining. Values are means ± SEM. *
    Treatment Human Colorectal Cancer Cell Lines, supplied by ATCC, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/treatment human colorectal cancer cell lines/product/ATCC
    Average 90 stars, based on 1 article reviews
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    98
    ATCC chemical treatment human colon cancer cell line dld 1
    IL-8 expression level of COLO 205 and <t>DLD-1</t> cell during 3M002 treatment ( * p
    Chemical Treatment Human Colon Cancer Cell Line Dld 1, supplied by ATCC, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    99
    ATCC human crc cell lines hct116
    NVP-BEZ235 treatment results in decreased cell viability, transient PI3K blockade inhibition, and sustained mTORC1/mTORC2 blockade. (A) Cell viability of <t>HCT116,</t> DLD-1, and SW480 <t>CRC</t> cell lines was assessed by MTS assay after treatment with increasing concentrations (0–500 nM) of NVP-BEZ235 for 48 hours. Results shown are the mean of three independent experiments. (B) Western blot analysis for p-AKT Thr308 , p-AKT Ser473 , p-S6 Ser240/244 , p-S6 Ser235/236 , cleaved caspase 3, and cleaved PARP was performed after 2, 6, 24, and 48 hours incubation with (−) 0 or (+) 500 nM NVP-BEZ235.
    Human Crc Cell Lines Hct116, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    94
    ATCC human colorectal cancer cell lines
    Myricetin inhibited the viability of human <t>colorectal</t> cancer cells. HCT116 and SW620 cells were treated with 0–400 μmol/L myricetin for 24 h, 48 h, and 72 h. Cell viability was analysed by means of a resazurin cell viability assay. Three replicate wells were set up in 96-well plates for each experimental group, and the experiment was repeated three times
    Human Colorectal Cancer Cell Lines, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/human colorectal cancer cell lines/product/ATCC
    Average 94 stars, based on 1 article reviews
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    human colorectal cancer cell lines - by Bioz Stars, 2022-09
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    Anti-colorectal cancer effects of curcumin and/or irinotecan are dependent on ROS ( A , B ) The effects of NAC on cell growth inhibition induced by curcumin and/or irinotecan. After pretreatment with 5 mM NAC for 2 h, LoVo cells (A) or HT-29 cells (B) were treated with curcumin and/or irinotecan for 24 h, then cell viability was assessed by CCK-8 assay. ( C , D ) The effects of NAC on apoptosis induced by curcumin and/or irinotecan. After cells were treated as described above, cell apoptosis was measured by Annexin V-FITC/PI staining. Values are means ± SEM. *

    Journal: Oncotarget

    Article Title: Curcumin enhances the effects of irinotecan on colorectal cancer cells through the generation of reactive oxygen species and activation of the endoplasmic reticulum stress pathway

    doi: 10.18632/oncotarget.16828

    Figure Lengend Snippet: Anti-colorectal cancer effects of curcumin and/or irinotecan are dependent on ROS ( A , B ) The effects of NAC on cell growth inhibition induced by curcumin and/or irinotecan. After pretreatment with 5 mM NAC for 2 h, LoVo cells (A) or HT-29 cells (B) were treated with curcumin and/or irinotecan for 24 h, then cell viability was assessed by CCK-8 assay. ( C , D ) The effects of NAC on apoptosis induced by curcumin and/or irinotecan. After cells were treated as described above, cell apoptosis was measured by Annexin V-FITC/PI staining. Values are means ± SEM. *

    Article Snippet: Cell culture and treatment Human colorectal cancer cell lines, LoVo and HT-29, were obtained from the American Type Culture Collection (Manassas, VA, USA).

    Techniques: Inhibition, CCK-8 Assay, Staining

    ER Stress is mediates the anti-colorectal cancer effects of curcumin alone or combined with irinotecan ( A , B ) The effects of an ER stress inhibitor on cell growth inhibition induced by curcumin alone or with irinotecan. After pretreatment with 0.1 μM mithramycin (MTM) for 30 min, LoVo cells (A) or HT-29 cells (B) were treated with curcumin alone or with irinotecan for 24 h, then cell viability was assessed by CCK-8 assay. ( C , D ) The effects of an ER stress inhibitor on apoptosis induced by curcumin alone or with irinotecan. After cells were treated as described above, cell apoptosis was measured by Annexin V-FITC/PI staining. Values are means ± SEM. *

    Journal: Oncotarget

    Article Title: Curcumin enhances the effects of irinotecan on colorectal cancer cells through the generation of reactive oxygen species and activation of the endoplasmic reticulum stress pathway

    doi: 10.18632/oncotarget.16828

    Figure Lengend Snippet: ER Stress is mediates the anti-colorectal cancer effects of curcumin alone or combined with irinotecan ( A , B ) The effects of an ER stress inhibitor on cell growth inhibition induced by curcumin alone or with irinotecan. After pretreatment with 0.1 μM mithramycin (MTM) for 30 min, LoVo cells (A) or HT-29 cells (B) were treated with curcumin alone or with irinotecan for 24 h, then cell viability was assessed by CCK-8 assay. ( C , D ) The effects of an ER stress inhibitor on apoptosis induced by curcumin alone or with irinotecan. After cells were treated as described above, cell apoptosis was measured by Annexin V-FITC/PI staining. Values are means ± SEM. *

    Article Snippet: Cell culture and treatment Human colorectal cancer cell lines, LoVo and HT-29, were obtained from the American Type Culture Collection (Manassas, VA, USA).

    Techniques: Inhibition, CCK-8 Assay, Staining

    IL-8 expression level of COLO 205 and DLD-1 cell during 3M002 treatment ( * p

    Journal: International Journal of Molecular Sciences

    Article Title: Upregulation of TLRs and IL-6 as a Marker in Human Colorectal Cancer

    doi: 10.3390/ijms16010159

    Figure Lengend Snippet: IL-8 expression level of COLO 205 and DLD-1 cell during 3M002 treatment ( * p

    Article Snippet: Cell Line, Cell Culture and Chemical Treatment Human colon cancer cell line DLD-1 (CCL-221) and human colorectal carcinoma cell line COLO 205 (CCL-222) were purchased from American Type Culture Collection (ATCC).

    Techniques: Expressing

    NVP-BEZ235 treatment results in decreased cell viability, transient PI3K blockade inhibition, and sustained mTORC1/mTORC2 blockade. (A) Cell viability of HCT116, DLD-1, and SW480 CRC cell lines was assessed by MTS assay after treatment with increasing concentrations (0–500 nM) of NVP-BEZ235 for 48 hours. Results shown are the mean of three independent experiments. (B) Western blot analysis for p-AKT Thr308 , p-AKT Ser473 , p-S6 Ser240/244 , p-S6 Ser235/236 , cleaved caspase 3, and cleaved PARP was performed after 2, 6, 24, and 48 hours incubation with (−) 0 or (+) 500 nM NVP-BEZ235.

    Journal: PLoS ONE

    Article Title: The Dual PI3K/mTOR Inhibitor NVP-BEZ235 Induces Tumor Regression in a Genetically Engineered Mouse Model of PIK3CA Wild-Type Colorectal Cancer

    doi: 10.1371/journal.pone.0025132

    Figure Lengend Snippet: NVP-BEZ235 treatment results in decreased cell viability, transient PI3K blockade inhibition, and sustained mTORC1/mTORC2 blockade. (A) Cell viability of HCT116, DLD-1, and SW480 CRC cell lines was assessed by MTS assay after treatment with increasing concentrations (0–500 nM) of NVP-BEZ235 for 48 hours. Results shown are the mean of three independent experiments. (B) Western blot analysis for p-AKT Thr308 , p-AKT Ser473 , p-S6 Ser240/244 , p-S6 Ser235/236 , cleaved caspase 3, and cleaved PARP was performed after 2, 6, 24, and 48 hours incubation with (−) 0 or (+) 500 nM NVP-BEZ235.

    Article Snippet: In vitro treatment of human CRC cell lines HCT116 (PIK3CA mutant; kinase domain at H1047R), DLD-1 (PIK3CA mutant; helical domain at E545K), and SW480 (PIK3CA wild-type) human CRC cell lines (ATCC) and isogenic DLD-1 PIK3CA mutant and wild-type cells (obtained from B. Vogelstein) were maintained in DMEM (Invitrogen) with 10% FBS and 1× Penicillin/Streptomycin (Invitrogen).

    Techniques: Inhibition, MTS Assay, Western Blot, Incubation

    Myricetin inhibited the viability of human colorectal cancer cells. HCT116 and SW620 cells were treated with 0–400 μmol/L myricetin for 24 h, 48 h, and 72 h. Cell viability was analysed by means of a resazurin cell viability assay. Three replicate wells were set up in 96-well plates for each experimental group, and the experiment was repeated three times

    Journal: BMC Complementary Medicine and Therapies

    Article Title: Myricetin induces apoptosis and autophagy by inhibiting PI3K/Akt/mTOR signalling in human colon cancer cells

    doi: 10.1186/s12906-020-02965-w

    Figure Lengend Snippet: Myricetin inhibited the viability of human colorectal cancer cells. HCT116 and SW620 cells were treated with 0–400 μmol/L myricetin for 24 h, 48 h, and 72 h. Cell viability was analysed by means of a resazurin cell viability assay. Three replicate wells were set up in 96-well plates for each experimental group, and the experiment was repeated three times

    Article Snippet: Cell culture and treatment Four human colorectal cancer cell lines, HT-29, HCT116, SW480 and SW620, were obtained from American Type Culture Collection (ATCC, Manassas, VA, USA).

    Techniques: Viability Assay