rabbit anti angiopoietin 1 ang 1 polyclonal antibody  (Bioss)


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  • 94

    Structured Review

    Bioss rabbit anti angiopoietin 1 ang 1 polyclonal antibody
    SVEP1 expression is positively related to the expression <t>of</t> <t>Ang-1,</t> which plays an important role in the maintenance of vascular stability. (A) Representative IHC images of Ang-1 in ICC tissues. (B) IHC image quantification results. Relative proportions of high and low levels of Ang-1 in the group of tumor tissues with high and low expression of SVEP1, and the correlation analysis between Ang-1 and SVEP1. (C–D) Correlation of Ang-1 expression with the OS (C) and DFS (D) time of ICC patients, respectively.
    Rabbit Anti Angiopoietin 1 Ang 1 Polyclonal Antibody, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti angiopoietin 1 ang 1 polyclonal antibody/product/Bioss
    Average 94 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit anti angiopoietin 1 ang 1 polyclonal antibody - by Bioz Stars, 2024-07
    94/100 stars

    Images

    1) Product Images from "The impact of decreased expression of SVEP1 on abnormal neovascularization and poor prognosis in patients with intrahepatic cholangiocarcinoma"

    Article Title: The impact of decreased expression of SVEP1 on abnormal neovascularization and poor prognosis in patients with intrahepatic cholangiocarcinoma

    Journal: Frontiers in Genetics

    doi: 10.3389/fgene.2022.1127753

    SVEP1 expression is positively related to the expression of Ang-1, which plays an important role in the maintenance of vascular stability. (A) Representative IHC images of Ang-1 in ICC tissues. (B) IHC image quantification results. Relative proportions of high and low levels of Ang-1 in the group of tumor tissues with high and low expression of SVEP1, and the correlation analysis between Ang-1 and SVEP1. (C–D) Correlation of Ang-1 expression with the OS (C) and DFS (D) time of ICC patients, respectively.
    Figure Legend Snippet: SVEP1 expression is positively related to the expression of Ang-1, which plays an important role in the maintenance of vascular stability. (A) Representative IHC images of Ang-1 in ICC tissues. (B) IHC image quantification results. Relative proportions of high and low levels of Ang-1 in the group of tumor tissues with high and low expression of SVEP1, and the correlation analysis between Ang-1 and SVEP1. (C–D) Correlation of Ang-1 expression with the OS (C) and DFS (D) time of ICC patients, respectively.

    Techniques Used: Expressing


    Structured Review

    Millipore rabbit polyclonal anti ang ii type 1
    (A) Traces and (B) bar graphs showing <t>Ang</t> <t>II</t> (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Rabbit Polyclonal Anti Ang Ii Type 1, supplied by Millipore, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii type 1/product/Millipore
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit polyclonal anti ang ii type 1 - by Bioz Stars, 2024-07
    86/100 stars

    Images

    1) Product Images from "Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs"

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    Journal: Clinical science (London, England : 1979)

    doi: 10.1042/CS20180815

    (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Techniques Used: Control

    (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.
    Figure Legend Snippet: (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Techniques Used: Control

    (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Techniques Used: Control, Concentration Assay, Western Blot, Expressing, Isolation

    rabbit polyclonal anti ang ii type 1  (Danaher Inc)


    Bioz Verified Symbol Danaher Inc is a verified supplier
    Bioz Manufacturer Symbol Danaher Inc manufactures this product  
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  • 86

    Structured Review

    Danaher Inc rabbit polyclonal anti ang ii type 1
    (A) Traces and (B) bar graphs showing <t>Ang</t> <t>II</t> (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Rabbit Polyclonal Anti Ang Ii Type 1, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii type 1/product/Danaher Inc
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit polyclonal anti ang ii type 1 - by Bioz Stars, 2024-07
    86/100 stars

    Images

    1) Product Images from "Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs"

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    Journal: Clinical science (London, England : 1979)

    doi: 10.1042/CS20180815

    (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Techniques Used: Control

    (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.
    Figure Legend Snippet: (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Techniques Used: Control

    (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Techniques Used: Control, Concentration Assay, Western Blot, Expressing, Isolation

    rabbit polyclonal anti ang ii type 1  (Danaher Inc)


    Bioz Verified Symbol Danaher Inc is a verified supplier
    Bioz Manufacturer Symbol Danaher Inc manufactures this product  
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  • 86

    Structured Review

    Danaher Inc rabbit polyclonal anti ang ii type 1
    (A) Traces and (B) bar graphs showing <t>Ang</t> <t>II</t> (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Rabbit Polyclonal Anti Ang Ii Type 1, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii type 1/product/Danaher Inc
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit polyclonal anti ang ii type 1 - by Bioz Stars, 2024-07
    86/100 stars

    Images

    1) Product Images from "Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs"

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    Journal: Clinical science (London, England : 1979)

    doi: 10.1042/CS20180815

    (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Techniques Used: Control

    (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.
    Figure Legend Snippet: (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Techniques Used: Control

    (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Techniques Used: Control, Concentration Assay, Western Blot, Expressing, Isolation


    Structured Review

    Millipore rabbit polyclonal anti ang ii type 1
    (A) Traces and (B) bar graphs showing <t>Ang</t> <t>II</t> (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Rabbit Polyclonal Anti Ang Ii Type 1, supplied by Millipore, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii type 1/product/Millipore
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit polyclonal anti ang ii type 1 - by Bioz Stars, 2024-07
    86/100 stars

    Images

    1) Product Images from "Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs"

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    Journal: Clinical science (London, England : 1979)

    doi: 10.1042/CS20180815

    (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Techniques Used: Control

    (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.
    Figure Legend Snippet: (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Techniques Used: Control

    (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Techniques Used: Control, Concentration Assay, Western Blot, Expressing, Isolation

    rabbit polyclonal anti ang ii type 1  (WuXi AppTec)


    Bioz Manufacturer Symbol WuXi AppTec manufactures this product  
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    WuXi AppTec rabbit polyclonal anti ang ii type 1
    (A) Traces and (B) bar graphs showing <t>Ang</t> <t>II</t> (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Rabbit Polyclonal Anti Ang Ii Type 1, supplied by WuXi AppTec, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii type 1/product/WuXi AppTec
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit polyclonal anti ang ii type 1 - by Bioz Stars, 2024-07
    86/100 stars

    Images

    1) Product Images from "Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs"

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    Journal: Clinical science (London, England : 1979)

    doi: 10.1042/CS20180815

    (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Techniques Used: Control

    (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.
    Figure Legend Snippet: (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Techniques Used: Control

    (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Techniques Used: Control, Concentration Assay, Western Blot, Expressing, Isolation

    rabbit polyclonal anti ang ii type 1  (WuXi AppTec)


    Bioz Manufacturer Symbol WuXi AppTec manufactures this product  
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  • 86

    Structured Review

    WuXi AppTec rabbit polyclonal anti ang ii type 1
    (A) Traces and (B) bar graphs showing <t>Ang</t> <t>II</t> (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Rabbit Polyclonal Anti Ang Ii Type 1, supplied by WuXi AppTec, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii type 1/product/WuXi AppTec
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit polyclonal anti ang ii type 1 - by Bioz Stars, 2024-07
    86/100 stars

    Images

    1) Product Images from "Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs"

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    Journal: Clinical science (London, England : 1979)

    doi: 10.1042/CS20180815

    (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Techniques Used: Control

    (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.
    Figure Legend Snippet: (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Techniques Used: Control

    (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.
    Figure Legend Snippet: (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Techniques Used: Control, Concentration Assay, Western Blot, Expressing, Isolation

    rabbit anti human ang ii polyclonal antibody  (Thermo Fisher)


    Bioz Verified Symbol Thermo Fisher is a verified supplier
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    Thermo Fisher rabbit anti human ang ii polyclonal antibody
    <t>Ang</t> II, AT1R, and AT2R expression in endometrium between fertile controls, RM and RIF. (A) Representative images of immunohistochemical staining of Ang II, AT1R, and AT2R among groups. LE, luminal epithelium; GE, glandular epithelium; ST, stroma cells. Scale bar=100 μm. (B) Corresponding quantitative analysis. Data are presented as mean ± SEM for AngII, AT1R, and AT2R and median (range) for stromal AT1R/AT2R expression ratios in all compartments of endometrium. *p < 0.05, ***p<0.001, NS, not significant.
    Rabbit Anti Human Ang Ii Polyclonal Antibody, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti human ang ii polyclonal antibody/product/Thermo Fisher
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit anti human ang ii polyclonal antibody - by Bioz Stars, 2024-07
    86/100 stars

    Images

    1) Product Images from "Association of angiotensin II and receptors in peri-implantation endometrium with microvessel density and pregnancy outcomes of women with recurrent implantation failure after embryo transfer"

    Article Title: Association of angiotensin II and receptors in peri-implantation endometrium with microvessel density and pregnancy outcomes of women with recurrent implantation failure after embryo transfer

    Journal: Frontiers in Endocrinology

    doi: 10.3389/fendo.2023.1206326

    Ang II, AT1R, and AT2R expression in endometrium between fertile controls, RM and RIF. (A) Representative images of immunohistochemical staining of Ang II, AT1R, and AT2R among groups. LE, luminal epithelium; GE, glandular epithelium; ST, stroma cells. Scale bar=100 μm. (B) Corresponding quantitative analysis. Data are presented as mean ± SEM for AngII, AT1R, and AT2R and median (range) for stromal AT1R/AT2R expression ratios in all compartments of endometrium. *p < 0.05, ***p<0.001, NS, not significant.
    Figure Legend Snippet: Ang II, AT1R, and AT2R expression in endometrium between fertile controls, RM and RIF. (A) Representative images of immunohistochemical staining of Ang II, AT1R, and AT2R among groups. LE, luminal epithelium; GE, glandular epithelium; ST, stroma cells. Scale bar=100 μm. (B) Corresponding quantitative analysis. Data are presented as mean ± SEM for AngII, AT1R, and AT2R and median (range) for stromal AT1R/AT2R expression ratios in all compartments of endometrium. *p < 0.05, ***p<0.001, NS, not significant.

    Techniques Used: Expressing, Immunohistochemistry, Staining


    Structured Review

    Abbiotec Inc rabbit polyclonal anti ang ii
    Rabbit Polyclonal Anti Ang Ii, supplied by Abbiotec Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii/product/Abbiotec Inc
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit polyclonal anti ang ii - by Bioz Stars, 2024-07
    86/100 stars

    Images


    Structured Review

    Abbiotec Inc rabbit polyclonal anti ang ii
    Rabbit Polyclonal Anti Ang Ii, supplied by Abbiotec Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii/product/Abbiotec Inc
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit polyclonal anti ang ii - by Bioz Stars, 2024-07
    86/100 stars

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    rabbit anti angiopoietin 1 ang 1 polyclonal antibody  (Bioss)


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    Bioss rabbit anti angiopoietin 1 ang 1 polyclonal antibody
    SVEP1 expression is positively related to the expression <t>of</t> <t>Ang-1,</t> which plays an important role in the maintenance of vascular stability. (A) Representative IHC images of Ang-1 in ICC tissues. (B) IHC image quantification results. Relative proportions of high and low levels of Ang-1 in the group of tumor tissues with high and low expression of SVEP1, and the correlation analysis between Ang-1 and SVEP1. (C–D) Correlation of Ang-1 expression with the OS (C) and DFS (D) time of ICC patients, respectively.
    Rabbit Anti Angiopoietin 1 Ang 1 Polyclonal Antibody, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti angiopoietin 1 ang 1 polyclonal antibody/product/Bioss
    Average 94 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit anti angiopoietin 1 ang 1 polyclonal antibody - by Bioz Stars, 2024-07
    94/100 stars

    Images

    1) Product Images from "The impact of decreased expression of SVEP1 on abnormal neovascularization and poor prognosis in patients with intrahepatic cholangiocarcinoma"

    Article Title: The impact of decreased expression of SVEP1 on abnormal neovascularization and poor prognosis in patients with intrahepatic cholangiocarcinoma

    Journal: Frontiers in Genetics

    doi: 10.3389/fgene.2022.1127753

    SVEP1 expression is positively related to the expression of Ang-1, which plays an important role in the maintenance of vascular stability. (A) Representative IHC images of Ang-1 in ICC tissues. (B) IHC image quantification results. Relative proportions of high and low levels of Ang-1 in the group of tumor tissues with high and low expression of SVEP1, and the correlation analysis between Ang-1 and SVEP1. (C–D) Correlation of Ang-1 expression with the OS (C) and DFS (D) time of ICC patients, respectively.
    Figure Legend Snippet: SVEP1 expression is positively related to the expression of Ang-1, which plays an important role in the maintenance of vascular stability. (A) Representative IHC images of Ang-1 in ICC tissues. (B) IHC image quantification results. Relative proportions of high and low levels of Ang-1 in the group of tumor tissues with high and low expression of SVEP1, and the correlation analysis between Ang-1 and SVEP1. (C–D) Correlation of Ang-1 expression with the OS (C) and DFS (D) time of ICC patients, respectively.

    Techniques Used: Expressing

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    Bioss rabbit anti angiopoietin 1 ang 1 polyclonal antibody
    SVEP1 expression is positively related to the expression <t>of</t> <t>Ang-1,</t> which plays an important role in the maintenance of vascular stability. (A) Representative IHC images of Ang-1 in ICC tissues. (B) IHC image quantification results. Relative proportions of high and low levels of Ang-1 in the group of tumor tissues with high and low expression of SVEP1, and the correlation analysis between Ang-1 and SVEP1. (C–D) Correlation of Ang-1 expression with the OS (C) and DFS (D) time of ICC patients, respectively.
    Rabbit Anti Angiopoietin 1 Ang 1 Polyclonal Antibody, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti angiopoietin 1 ang 1 polyclonal antibody/product/Bioss
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    rabbit anti angiopoietin 1 ang 1 polyclonal antibody - by Bioz Stars, 2024-07
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    Millipore rabbit polyclonal anti ang ii type 1
    (A) Traces and (B) bar graphs showing <t>Ang</t> <t>II</t> (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Rabbit Polyclonal Anti Ang Ii Type 1, supplied by Millipore, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Danaher Inc rabbit polyclonal anti ang ii type 1
    (A) Traces and (B) bar graphs showing <t>Ang</t> <t>II</t> (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Rabbit Polyclonal Anti Ang Ii Type 1, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii type 1/product/Danaher Inc
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    WuXi AppTec rabbit polyclonal anti ang ii type 1
    (A) Traces and (B) bar graphs showing <t>Ang</t> <t>II</t> (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.
    Rabbit Polyclonal Anti Ang Ii Type 1, supplied by WuXi AppTec, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii type 1/product/WuXi AppTec
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
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    86
    Thermo Fisher rabbit anti human ang ii polyclonal antibody
    <t>Ang</t> II, AT1R, and AT2R expression in endometrium between fertile controls, RM and RIF. (A) Representative images of immunohistochemical staining of Ang II, AT1R, and AT2R among groups. LE, luminal epithelium; GE, glandular epithelium; ST, stroma cells. Scale bar=100 μm. (B) Corresponding quantitative analysis. Data are presented as mean ± SEM for AngII, AT1R, and AT2R and median (range) for stromal AT1R/AT2R expression ratios in all compartments of endometrium. *p < 0.05, ***p<0.001, NS, not significant.
    Rabbit Anti Human Ang Ii Polyclonal Antibody, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit anti human ang ii polyclonal antibody/product/Thermo Fisher
    Average 86 stars, based on 1 article reviews
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    rabbit anti human ang ii polyclonal antibody - by Bioz Stars, 2024-07
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    86
    Abbiotec Inc rabbit polyclonal anti ang ii
    <t>Ang</t> II, AT1R, and AT2R expression in endometrium between fertile controls, RM and RIF. (A) Representative images of immunohistochemical staining of Ang II, AT1R, and AT2R among groups. LE, luminal epithelium; GE, glandular epithelium; ST, stroma cells. Scale bar=100 μm. (B) Corresponding quantitative analysis. Data are presented as mean ± SEM for AngII, AT1R, and AT2R and median (range) for stromal AT1R/AT2R expression ratios in all compartments of endometrium. *p < 0.05, ***p<0.001, NS, not significant.
    Rabbit Polyclonal Anti Ang Ii, supplied by Abbiotec Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rabbit polyclonal anti ang ii/product/Abbiotec Inc
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rabbit polyclonal anti ang ii - by Bioz Stars, 2024-07
    86/100 stars
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    Image Search Results


    SVEP1 expression is positively related to the expression of Ang-1, which plays an important role in the maintenance of vascular stability. (A) Representative IHC images of Ang-1 in ICC tissues. (B) IHC image quantification results. Relative proportions of high and low levels of Ang-1 in the group of tumor tissues with high and low expression of SVEP1, and the correlation analysis between Ang-1 and SVEP1. (C–D) Correlation of Ang-1 expression with the OS (C) and DFS (D) time of ICC patients, respectively.

    Journal: Frontiers in Genetics

    Article Title: The impact of decreased expression of SVEP1 on abnormal neovascularization and poor prognosis in patients with intrahepatic cholangiocarcinoma

    doi: 10.3389/fgene.2022.1127753

    Figure Lengend Snippet: SVEP1 expression is positively related to the expression of Ang-1, which plays an important role in the maintenance of vascular stability. (A) Representative IHC images of Ang-1 in ICC tissues. (B) IHC image quantification results. Relative proportions of high and low levels of Ang-1 in the group of tumor tissues with high and low expression of SVEP1, and the correlation analysis between Ang-1 and SVEP1. (C–D) Correlation of Ang-1 expression with the OS (C) and DFS (D) time of ICC patients, respectively.

    Article Snippet: The primary antibody included rabbit anti-SVEP1 polyclonal antibody (Abcam, cat#ab121677), rabbit anti-Angiopoietin-1 (Ang-1) polyclonal antibody (Bioss, cat#bs-0800R), and mouse anti-Ki-67 monoclonal antibody (Zhongshan Goldbridge Biotechno-logy CO., Ltd., Beijing, China).

    Techniques: Expressing

    (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Journal: Clinical science (London, England : 1979)

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    doi: 10.1042/CS20180815

    Figure Lengend Snippet: (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Article Snippet: Antibodies and reagents Rabbit polyclonal anti-TRPV4 (AP18990a) [ 15 ], rabbit polyclonal anti-Ang II type 1 (AT 1 ) receptor (SAB3500209) [ 49 ], and mouse monoclonal anti-β-actin ( MA515739 ) [ 15 ] primary antibodies were purchased from Abgent Inc. (San Diego, CA), Sigma–Aldrich (St. Louis, MO), and Abcam (Cambridge, MA), respectively.

    Techniques: Control

    (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Journal: Clinical science (London, England : 1979)

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    doi: 10.1042/CS20180815

    Figure Lengend Snippet: (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Article Snippet: Antibodies and reagents Rabbit polyclonal anti-TRPV4 (AP18990a) [ 15 ], rabbit polyclonal anti-Ang II type 1 (AT 1 ) receptor (SAB3500209) [ 49 ], and mouse monoclonal anti-β-actin ( MA515739 ) [ 15 ] primary antibodies were purchased from Abgent Inc. (San Diego, CA), Sigma–Aldrich (St. Louis, MO), and Abcam (Cambridge, MA), respectively.

    Techniques: Control

    (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Journal: Clinical science (London, England : 1979)

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    doi: 10.1042/CS20180815

    Figure Lengend Snippet: (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Article Snippet: Antibodies and reagents Rabbit polyclonal anti-TRPV4 (AP18990a) [ 15 ], rabbit polyclonal anti-Ang II type 1 (AT 1 ) receptor (SAB3500209) [ 49 ], and mouse monoclonal anti-β-actin ( MA515739 ) [ 15 ] primary antibodies were purchased from Abgent Inc. (San Diego, CA), Sigma–Aldrich (St. Louis, MO), and Abcam (Cambridge, MA), respectively.

    Techniques: Control, Concentration Assay, Western Blot, Expressing, Isolation

    (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Journal: Clinical science (London, England : 1979)

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    doi: 10.1042/CS20180815

    Figure Lengend Snippet: (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Article Snippet: Antibodies and reagents Rabbit polyclonal anti-TRPV4 (AP18990a) [ 15 ], rabbit polyclonal anti-Ang II type 1 (AT 1 ) receptor (SAB3500209) [ 49 ], and mouse monoclonal anti-β-actin ( MA515739 ) [ 15 ] primary antibodies were purchased from Abgent Inc. (San Diego, CA), Sigma–Aldrich (St. Louis, MO), and Abcam (Cambridge, MA), respectively.

    Techniques: Control

    (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Journal: Clinical science (London, England : 1979)

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    doi: 10.1042/CS20180815

    Figure Lengend Snippet: (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Article Snippet: Antibodies and reagents Rabbit polyclonal anti-TRPV4 (AP18990a) [ 15 ], rabbit polyclonal anti-Ang II type 1 (AT 1 ) receptor (SAB3500209) [ 49 ], and mouse monoclonal anti-β-actin ( MA515739 ) [ 15 ] primary antibodies were purchased from Abgent Inc. (San Diego, CA), Sigma–Aldrich (St. Louis, MO), and Abcam (Cambridge, MA), respectively.

    Techniques: Control

    (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Journal: Clinical science (London, England : 1979)

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    doi: 10.1042/CS20180815

    Figure Lengend Snippet: (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Article Snippet: Antibodies and reagents Rabbit polyclonal anti-TRPV4 (AP18990a) [ 15 ], rabbit polyclonal anti-Ang II type 1 (AT 1 ) receptor (SAB3500209) [ 49 ], and mouse monoclonal anti-β-actin ( MA515739 ) [ 15 ] primary antibodies were purchased from Abgent Inc. (San Diego, CA), Sigma–Aldrich (St. Louis, MO), and Abcam (Cambridge, MA), respectively.

    Techniques: Control, Concentration Assay, Western Blot, Expressing, Isolation

    (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Journal: Clinical science (London, England : 1979)

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    doi: 10.1042/CS20180815

    Figure Lengend Snippet: (A) Traces and (B) bar graphs showing Ang II (100 nM)-induced reduction in luminal diameter of pressurized (100 mmHg) neonatal pig renal interlobular arteries in the absence and presence of HC (1 μM) pretreatment. (C–H) Traces and bar graphs demonstrating Ang II (10 μg/kg/min; 5 min)-induced changes in renal cortical perfusion (RCoP), total RBF, and MAP in the absence and presence of HC (20 μg/kg/min; 30 min) pretreatment. (I) Bar graphs summarizing Ang II-induced changes in RVR in the absence and presence of HC pretreatment. Both Ang II and HC were administered intrarenally. n=5 each; *P<0.05 vs. Control. Abbreviation: NS, not significant.

    Article Snippet: Antibodies and reagents Rabbit polyclonal anti-TRPV4 (AP18990a) [ 15 ], rabbit polyclonal anti-Ang II type 1 (AT 1 ) receptor (SAB3500209) [ 49 ], and mouse monoclonal anti-β-actin ( MA515739 ) [ 15 ] primary antibodies were purchased from Abgent Inc. (San Diego, CA), Sigma–Aldrich (St. Louis, MO), and Abcam (Cambridge, MA), respectively.

    Techniques: Control

    (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Journal: Clinical science (London, England : 1979)

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    doi: 10.1042/CS20180815

    Figure Lengend Snippet: (A) Traces and (B) bar graphs illustrating that HC (1 μM) did not alter thapsigargin (TG; 1 μM)-induced [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence or presence of extracellular Ca2+ [n=7 and 5 for Control (TG) and HC + TG, respectively]. (C) Traces and (D) bar graphs showing that Ang II (10 μM) triggered [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in the absence and presence of extracellular Ca2+ and that HC (1 μM) had no effect on Ang II-induced [Ca2+]i store release but partially reversed Ang II-induced [Ca2+]i elevation in the presence of extracellular Ca2+. (E) Traces and (F) bar graphs demonstrating the effect of HC (1 μM) on Ang II (10 μM)-evoked [Ca2+]i elevation in neonatal pig afferent arteriolar SMCs in which SR Ca2+ stores had been depleted by TG (1 μM) and l-type Ca2+ channels blocked by nimodipine (1 μM); n=7 each. The zero Ca2+ bath solution was supplemented with Ca2+ chelator EGTA (0.1 mM). *P<0.05, 0 vs. 2 mM Ca2+; #P<0.05 vs. control.

    Article Snippet: Antibodies and reagents Rabbit polyclonal anti-TRPV4 (AP18990a) [ 15 ], rabbit polyclonal anti-Ang II type 1 (AT 1 ) receptor (SAB3500209) [ 49 ], and mouse monoclonal anti-β-actin ( MA515739 ) [ 15 ] primary antibodies were purchased from Abgent Inc. (San Diego, CA), Sigma–Aldrich (St. Louis, MO), and Abcam (Cambridge, MA), respectively.

    Techniques: Control

    (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Journal: Clinical science (London, England : 1979)

    Article Title: Pharmacological inhibition of TRPV4 channels protects against ischemia–reperfusion-induced renal insufficiency in neonatal pigs

    doi: 10.1042/CS20180815

    Figure Lengend Snippet: (A) Bar graphs summarizing Ang II levels in the urine of neonatal pigs 3 h after sham and IR operations (n=5 each). To control for variations in urine flow rate and creatinine clearance, the urine concentration of Ang II was normalized to that of creatinine. (B and C) Western blot images and bar graphs showing AT1 protein expression levels in renal microvessels (pooled interlobular arteries and afferent arterioles) isolated from sham- and IR-operated neonatal pig kidneys (n=4 each). (D–H) Bar graphs summarizing changes in renal cortical perfusion (RCoP), total RBF, GFR, serum BUN, and serum creatinine in IR (n=3)- and losartan + IR (n=4)-operated neonatal pigs. Losartan (1 mg/kg/min) were infused intrarenally for 20 min starting at the time of reperfusion. *P<0.05 vs. IR. Abbreviation: NS, not significant.

    Article Snippet: Antibodies and reagents Rabbit polyclonal anti-TRPV4 (AP18990a) [ 15 ], rabbit polyclonal anti-Ang II type 1 (AT 1 ) receptor (SAB3500209) [ 49 ], and mouse monoclonal anti-β-actin ( MA515739 ) [ 15 ] primary antibodies were purchased from Abgent Inc. (San Diego, CA), Sigma–Aldrich (St. Louis, MO), and Abcam (Cambridge, MA), respectively.

    Techniques: Control, Concentration Assay, Western Blot, Expressing, Isolation

    Ang II, AT1R, and AT2R expression in endometrium between fertile controls, RM and RIF. (A) Representative images of immunohistochemical staining of Ang II, AT1R, and AT2R among groups. LE, luminal epithelium; GE, glandular epithelium; ST, stroma cells. Scale bar=100 μm. (B) Corresponding quantitative analysis. Data are presented as mean ± SEM for AngII, AT1R, and AT2R and median (range) for stromal AT1R/AT2R expression ratios in all compartments of endometrium. *p < 0.05, ***p<0.001, NS, not significant.

    Journal: Frontiers in Endocrinology

    Article Title: Association of angiotensin II and receptors in peri-implantation endometrium with microvessel density and pregnancy outcomes of women with recurrent implantation failure after embryo transfer

    doi: 10.3389/fendo.2023.1206326

    Figure Lengend Snippet: Ang II, AT1R, and AT2R expression in endometrium between fertile controls, RM and RIF. (A) Representative images of immunohistochemical staining of Ang II, AT1R, and AT2R among groups. LE, luminal epithelium; GE, glandular epithelium; ST, stroma cells. Scale bar=100 μm. (B) Corresponding quantitative analysis. Data are presented as mean ± SEM for AngII, AT1R, and AT2R and median (range) for stromal AT1R/AT2R expression ratios in all compartments of endometrium. *p < 0.05, ***p<0.001, NS, not significant.

    Article Snippet: The slides were washed in PBS and incubated in blocking solution (5% goat serum albumin in PBS) for 1 h at room temperature, and incubated overnight at 4°C with primary antibody, either rabbit anti-human Ang II polyclonal antibody (PA5-33339, Thermo Fisher, US) at 1:400 dilution, rabbit anti-human AT1R polyclonal antibody (AAR-011, Alomone Labs, Israel) at 1:1500 dilution, rabbit anti-human AT2R antibody (ab19134, AbCam, UK) at 1:150 dilution, or rabbit polyclonal anti-human vWF antibody (A0082, DAKO, Japan) at 1:1000 dilution.

    Techniques: Expressing, Immunohistochemistry, Staining