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Santa Cruz Biotechnology mouse monoclonal anti vegf igg
Immunohistochemical localisation of <t>VEGF‐C</t> in paraffin‐embedded uterine sections of day 4 ( a – f ) and ( g – k ) of gestation. The growth factor strongly expressed in endometrial surface epithelium (ESE) and glandular epithelium (GE), and moderately in stromal cells in control females ( a – c ) on day 4 of gestation. The myometrium (M) and perimetrium (P) in control females showed moderate and strong expression of VEGF‐C, respectively. CBE administration ( d – f ) caused diffused expression in ESE, in stromal cells, and in the endometrial glands. Control females’ uteri on day 5 ( g – i ) of gestation showed strong expression in ESE, GE, certain regions of sub‐epithelial stromal (S) cells and blood vessels (BV). Administration of CBE caused structural aberration in uteri of day 5 gestation ( j – k ). Expression of VEGF‐C was diffused in the desquamated endometrial surface epithelium (DESE) and degenerated glandular epithelium (DGE). Expression was lesser in the vacuolated (V) epithelial cells. The expression of growth factor was indicated by arrow ( thick arrow ). The negative control (L) was immunostained with normal <t>IgG.</t> Original magnification ( a – l ) ×40
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Article Title: Crude bark extract of Dysozylum alliarium induces alteration in histological structures and VEGF‐C expression in uterus during days 4–7 of gestation in albino rat

Journal: Reproductive Medicine and Biology

doi: 10.1007/s12522-012-0143-8

Immunohistochemical localisation of VEGF‐C in paraffin‐embedded uterine sections of day 4 ( a – f ) and ( g – k ) of gestation. The growth factor strongly expressed in endometrial surface epithelium (ESE) and glandular epithelium (GE), and moderately in stromal cells in control females ( a – c ) on day 4 of gestation. The myometrium (M) and perimetrium (P) in control females showed moderate and strong expression of VEGF‐C, respectively. CBE administration ( d – f ) caused diffused expression in ESE, in stromal cells, and in the endometrial glands. Control females’ uteri on day 5 ( g – i ) of gestation showed strong expression in ESE, GE, certain regions of sub‐epithelial stromal (S) cells and blood vessels (BV). Administration of CBE caused structural aberration in uteri of day 5 gestation ( j – k ). Expression of VEGF‐C was diffused in the desquamated endometrial surface epithelium (DESE) and degenerated glandular epithelium (DGE). Expression was lesser in the vacuolated (V) epithelial cells. The expression of growth factor was indicated by arrow ( thick arrow ). The negative control (L) was immunostained with normal IgG. Original magnification ( a – l ) ×40
Figure Legend Snippet: Immunohistochemical localisation of VEGF‐C in paraffin‐embedded uterine sections of day 4 ( a – f ) and ( g – k ) of gestation. The growth factor strongly expressed in endometrial surface epithelium (ESE) and glandular epithelium (GE), and moderately in stromal cells in control females ( a – c ) on day 4 of gestation. The myometrium (M) and perimetrium (P) in control females showed moderate and strong expression of VEGF‐C, respectively. CBE administration ( d – f ) caused diffused expression in ESE, in stromal cells, and in the endometrial glands. Control females’ uteri on day 5 ( g – i ) of gestation showed strong expression in ESE, GE, certain regions of sub‐epithelial stromal (S) cells and blood vessels (BV). Administration of CBE caused structural aberration in uteri of day 5 gestation ( j – k ). Expression of VEGF‐C was diffused in the desquamated endometrial surface epithelium (DESE) and degenerated glandular epithelium (DGE). Expression was lesser in the vacuolated (V) epithelial cells. The expression of growth factor was indicated by arrow ( thick arrow ). The negative control (L) was immunostained with normal IgG. Original magnification ( a – l ) ×40

Techniques Used: Immunohistochemistry, Expressing, Negative Control

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Article Title: Crude bark extract of Dysozylum alliarium induces alteration in histological structures and VEGF‐C expression in uterus during days 4–7 of gestation in albino rat
Article Snippet: .. Cat No.Optitran BA‐S 85, 200 × 3 mm) were blocked for one and half hour in 3 % BSA in Tris‐buffered saline (Santa Cruz Biotechnology) and then incubated with mouse monoclonal anti VEGF IgG at 1.5 μg/ml (Santa Cruz Biotechnology, Santa Cruz, USA Cat No. sc‐7269) concentration in TBS–BSA overnight at 4 °C. .. Following primary antibody incubation, membranes were rinsed with Tris‐buffered saline with tween‐20 (pH 7.4) and then incubated with HRP (horseradish peroxidase) conjugated secondary antibody (goat anti‐mouse IgG, Santa Cruz Biotech, USA Cat No. sc‐2005) in 1:1,000 dilution for 2 h at room temperature.

Incubation:

Article Title: Crude bark extract of Dysozylum alliarium induces alteration in histological structures and VEGF‐C expression in uterus during days 4–7 of gestation in albino rat
Article Snippet: .. Cat No.Optitran BA‐S 85, 200 × 3 mm) were blocked for one and half hour in 3 % BSA in Tris‐buffered saline (Santa Cruz Biotechnology) and then incubated with mouse monoclonal anti VEGF IgG at 1.5 μg/ml (Santa Cruz Biotechnology, Santa Cruz, USA Cat No. sc‐7269) concentration in TBS–BSA overnight at 4 °C. .. Following primary antibody incubation, membranes were rinsed with Tris‐buffered saline with tween‐20 (pH 7.4) and then incubated with HRP (horseradish peroxidase) conjugated secondary antibody (goat anti‐mouse IgG, Santa Cruz Biotech, USA Cat No. sc‐2005) in 1:1,000 dilution for 2 h at room temperature.

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  • 85
    Santa Cruz Biotechnology mouse monoclonal anti vegf igg
    Immunohistochemical localisation of <t>VEGF‐C</t> in paraffin‐embedded uterine sections of day 4 ( a – f ) and ( g – k ) of gestation. The growth factor strongly expressed in endometrial surface epithelium (ESE) and glandular epithelium (GE), and moderately in stromal cells in control females ( a – c ) on day 4 of gestation. The myometrium (M) and perimetrium (P) in control females showed moderate and strong expression of VEGF‐C, respectively. CBE administration ( d – f ) caused diffused expression in ESE, in stromal cells, and in the endometrial glands. Control females’ uteri on day 5 ( g – i ) of gestation showed strong expression in ESE, GE, certain regions of sub‐epithelial stromal (S) cells and blood vessels (BV). Administration of CBE caused structural aberration in uteri of day 5 gestation ( j – k ). Expression of VEGF‐C was diffused in the desquamated endometrial surface epithelium (DESE) and degenerated glandular epithelium (DGE). Expression was lesser in the vacuolated (V) epithelial cells. The expression of growth factor was indicated by arrow ( thick arrow ). The negative control (L) was immunostained with normal <t>IgG.</t> Original magnification ( a – l ) ×40
    Mouse Monoclonal Anti Vegf Igg, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 85/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/mouse monoclonal anti vegf igg/product/Santa Cruz Biotechnology
    Average 85 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    mouse monoclonal anti vegf igg - by Bioz Stars, 2020-09
    85/100 stars
      Buy from Supplier

    93
    Santa Cruz Biotechnology anti vegf
    (a) <t>VEGF</t> immunostaining in the proliferative zone of the growth plate (a–c: tibia, fetus, 23 weeks after gestation). In the proliferative zone immunostaining for VEGF was negative. (b) Within the zone of hypertrophic cartilage immunolabelling for VEGF was positive. (c) Immunostaining for the VEGF receptor FLT-1 in the growth plate. FLT-1 immunostaining was restricted to vascular endothelial cells, smooth muscle cells and perivascular fibroblasts. FLT-1 was absent in the proliferative and hypertrophic zone. (d) Immunstaining for VEGF in epiphyseal cartilage (d,e: tibial epiphysis in fetus, week 23 of gestation). Chondrocytes around cartilage channels (cc) were VEGF positive. (e) Immunostaining for the VEGF receptor KDR in epiphyseal cartilage. KDR could be <t>immunostained</t> on invading blood vessels (V) but also on single chondrocytes. Scale bars = 65 μm.
    Anti Vegf, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 21 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/anti vegf/product/Santa Cruz Biotechnology
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    85
    Santa Cruz Biotechnology mouse monoclonal anti recombinant vegf a igg
    (a) <t>VEGF</t> immunostaining in the proliferative zone of the growth plate (a–c: tibia, fetus, 23 weeks after gestation). In the proliferative zone immunostaining for VEGF was negative. (b) Within the zone of hypertrophic cartilage immunolabelling for VEGF was positive. (c) Immunostaining for the VEGF receptor FLT-1 in the growth plate. FLT-1 immunostaining was restricted to vascular endothelial cells, smooth muscle cells and perivascular fibroblasts. FLT-1 was absent in the proliferative and hypertrophic zone. (d) Immunstaining for VEGF in epiphyseal cartilage (d,e: tibial epiphysis in fetus, week 23 of gestation). Chondrocytes around cartilage channels (cc) were VEGF positive. (e) Immunostaining for the VEGF receptor KDR in epiphyseal cartilage. KDR could be <t>immunostained</t> on invading blood vessels (V) but also on single chondrocytes. Scale bars = 65 μm.
    Mouse Monoclonal Anti Recombinant Vegf A Igg, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 85/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/mouse monoclonal anti recombinant vegf a igg/product/Santa Cruz Biotechnology
    Average 85 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    mouse monoclonal anti recombinant vegf a igg - by Bioz Stars, 2020-09
    85/100 stars
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    Immunohistochemical localisation of VEGF‐C in paraffin‐embedded uterine sections of day 4 ( a – f ) and ( g – k ) of gestation. The growth factor strongly expressed in endometrial surface epithelium (ESE) and glandular epithelium (GE), and moderately in stromal cells in control females ( a – c ) on day 4 of gestation. The myometrium (M) and perimetrium (P) in control females showed moderate and strong expression of VEGF‐C, respectively. CBE administration ( d – f ) caused diffused expression in ESE, in stromal cells, and in the endometrial glands. Control females’ uteri on day 5 ( g – i ) of gestation showed strong expression in ESE, GE, certain regions of sub‐epithelial stromal (S) cells and blood vessels (BV). Administration of CBE caused structural aberration in uteri of day 5 gestation ( j – k ). Expression of VEGF‐C was diffused in the desquamated endometrial surface epithelium (DESE) and degenerated glandular epithelium (DGE). Expression was lesser in the vacuolated (V) epithelial cells. The expression of growth factor was indicated by arrow ( thick arrow ). The negative control (L) was immunostained with normal IgG. Original magnification ( a – l ) ×40

    Journal: Reproductive Medicine and Biology

    Article Title: Crude bark extract of Dysozylum alliarium induces alteration in histological structures and VEGF‐C expression in uterus during days 4–7 of gestation in albino rat

    doi: 10.1007/s12522-012-0143-8

    Figure Lengend Snippet: Immunohistochemical localisation of VEGF‐C in paraffin‐embedded uterine sections of day 4 ( a – f ) and ( g – k ) of gestation. The growth factor strongly expressed in endometrial surface epithelium (ESE) and glandular epithelium (GE), and moderately in stromal cells in control females ( a – c ) on day 4 of gestation. The myometrium (M) and perimetrium (P) in control females showed moderate and strong expression of VEGF‐C, respectively. CBE administration ( d – f ) caused diffused expression in ESE, in stromal cells, and in the endometrial glands. Control females’ uteri on day 5 ( g – i ) of gestation showed strong expression in ESE, GE, certain regions of sub‐epithelial stromal (S) cells and blood vessels (BV). Administration of CBE caused structural aberration in uteri of day 5 gestation ( j – k ). Expression of VEGF‐C was diffused in the desquamated endometrial surface epithelium (DESE) and degenerated glandular epithelium (DGE). Expression was lesser in the vacuolated (V) epithelial cells. The expression of growth factor was indicated by arrow ( thick arrow ). The negative control (L) was immunostained with normal IgG. Original magnification ( a – l ) ×40

    Article Snippet: Cat No.Optitran BA‐S 85, 200 × 3 mm) were blocked for one and half hour in 3 % BSA in Tris‐buffered saline (Santa Cruz Biotechnology) and then incubated with mouse monoclonal anti VEGF IgG at 1.5 μg/ml (Santa Cruz Biotechnology, Santa Cruz, USA Cat No. sc‐7269) concentration in TBS–BSA overnight at 4 °C.

    Techniques: Immunohistochemistry, Expressing, Negative Control

    (a) VEGF immunostaining in the proliferative zone of the growth plate (a–c: tibia, fetus, 23 weeks after gestation). In the proliferative zone immunostaining for VEGF was negative. (b) Within the zone of hypertrophic cartilage immunolabelling for VEGF was positive. (c) Immunostaining for the VEGF receptor FLT-1 in the growth plate. FLT-1 immunostaining was restricted to vascular endothelial cells, smooth muscle cells and perivascular fibroblasts. FLT-1 was absent in the proliferative and hypertrophic zone. (d) Immunstaining for VEGF in epiphyseal cartilage (d,e: tibial epiphysis in fetus, week 23 of gestation). Chondrocytes around cartilage channels (cc) were VEGF positive. (e) Immunostaining for the VEGF receptor KDR in epiphyseal cartilage. KDR could be immunostained on invading blood vessels (V) but also on single chondrocytes. Scale bars = 65 μm.

    Journal: Journal of Anatomy

    Article Title: Expression of VEGF121 and VEFG165 in hypertrophic chondrocytes of the human growth plate and epiphyseal cartilage

    doi: 10.1046/j.1469-7580.2002.00085.x

    Figure Lengend Snippet: (a) VEGF immunostaining in the proliferative zone of the growth plate (a–c: tibia, fetus, 23 weeks after gestation). In the proliferative zone immunostaining for VEGF was negative. (b) Within the zone of hypertrophic cartilage immunolabelling for VEGF was positive. (c) Immunostaining for the VEGF receptor FLT-1 in the growth plate. FLT-1 immunostaining was restricted to vascular endothelial cells, smooth muscle cells and perivascular fibroblasts. FLT-1 was absent in the proliferative and hypertrophic zone. (d) Immunstaining for VEGF in epiphyseal cartilage (d,e: tibial epiphysis in fetus, week 23 of gestation). Chondrocytes around cartilage channels (cc) were VEGF positive. (e) Immunostaining for the VEGF receptor KDR in epiphyseal cartilage. KDR could be immunostained on invading blood vessels (V) but also on single chondrocytes. Scale bars = 65 μm.

    Article Snippet: For immunohistochemistry, sections were dewaxed, incubated with testicular hyaluronidase (2 mg mL−1 in PBS, pH 5.0, for 30 min at room temperature) and pronase (1 mg mL−1 in PBS, pH 7.4, 30 min at room temperature) and with H2 O2 (3% for 10 min) for quenching of the endogenous peroxidase, immunostained with anti-VEGF (5 μg mL−1 in tris-buffered saline, 60 min, mouse monoclonal IgG, Santa Cruz Biotechnology, CA, USA), anti-KDR (5 µg mL−1 in tris-buffered saline, 60 min; mouse monoclonal IgG, Santa Cruz Biotechnology), or anti-FLT-1 (5 μg mL−1 in tris-buffered saline, 60 min; rabbit affinity-purified polyclonal antibody, Santa Cruz Biotechnology) followed by biotinylated secondary antibodies and a peroxidase-labelled streptavidin-biotin staining technique; nuclei were counterstained with haemalaun.

    Techniques: Immunostaining