monoclonal rat anti reticular fibroblast antibodies  (Novus Biologicals)

 
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    Name:
    Fibroblast Antibody ER TR7
    Description:
    The Fibroblast Antibody ER TR7 from Novus Biologicals is a rat monoclonal antibody to Fibroblast This antibody reacts with mouse The Fibroblast Antibody ER TR7 has been validated for the following applications Flow Cytometry Immunohistochemistry Immunocytochemistry Immunofluorescence Immunohistochemistry Frozen
    Catalog Number:
    nb100-64932
    Price:
    299
    Host:
    Rat
    Purity:
    Protein G purified
    Conjugate:
    Unconjugated
    Immunogen:
    Isolated C3H thymic stromal cells
    Size:
    0 125 mg
    Category:
    Primary Antibodies
    Isotype:
    IgG2a
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    Structured Review

    Novus Biologicals monoclonal rat anti reticular fibroblast antibodies
    Fibroblast Antibody ER TR7
    The Fibroblast Antibody ER TR7 from Novus Biologicals is a rat monoclonal antibody to Fibroblast This antibody reacts with mouse The Fibroblast Antibody ER TR7 has been validated for the following applications Flow Cytometry Immunohistochemistry Immunocytochemistry Immunofluorescence Immunohistochemistry Frozen
    https://www.bioz.com/result/monoclonal rat anti reticular fibroblast antibodies/product/Novus Biologicals
    Average 85 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    monoclonal rat anti reticular fibroblast antibodies - by Bioz Stars, 2020-08
    85/100 stars

    Images

    1) Product Images from "G-CSF/SCF reduces inducible arrhythmias in the infarcted heart potentially via increased connexin43 expression and arteriogenesis"

    Article Title: G-CSF/SCF reduces inducible arrhythmias in the infarcted heart potentially via increased connexin43 expression and arteriogenesis

    Journal: The Journal of Experimental Medicine

    doi: 10.1084/jem.20051151

    (A) Examples of monomorphic (top) and polymorphic (bottom) ventricular tachycardia induced by programmed stimulation using a single (top) or two (bottom) extra stimuli. Shown are three simultaneously recorded MAP recordings obtained from the right ventricular (MAP RV), left ventricular septal (MAP LVsept), and left ventricular free wall (MAP LVfree) epicardium. In the lower panel, the left ventricular free wall is recorded from the infarcted area and shows the typical alterations found in infarcted scar tissue: loss of the action potential dome and slow activation. Asterisks indicate stimulus artifacts in the right ventricular MAP recording. (B) Number of hearts with induced ventricular tachycardias ( > 10 consecutive premature ventricular activations) as a function of the number of extra stimuli (S2, first extra stimulus; S3, second extra stimulus) shown for hearts with G-CSF/SCF (black bars) and hearts from non-G-CSF/SCF control animals (gray bars). Ventricular tachycardia induction was less likely in G-CSF/SCF-treated hearts than in controls. (C) Left: Representative immunohistological sections of normal myocardium (top panels) and the border zone of the infarction (bottom panels) in untreated (left panels) and G-CSF/SCF-treated (right panels) hearts. Bright red immunofluorescence, connexin43; dark red, autofluorescence of myocytes; green, immunofluorescence detection of a fibrous tissue marker (rat anti-reticular fibroblast antibody; refer to Materials and methods, and see supplemental Materials and methods for a 3D visualization of connexin43 localization in the intercellular connections). Right (diagram): Mean connexin43 expression measured as connexin43 + area in relation to the whole area of myocardial tissue in the field of view. Connexin43 expression was markedly reduced in the border zone of the myocardial infarction. G-CSF/SCF increased connexin43 expression in the border zone without affecting connexin43 expression in normal myocardium.
    Figure Legend Snippet: (A) Examples of monomorphic (top) and polymorphic (bottom) ventricular tachycardia induced by programmed stimulation using a single (top) or two (bottom) extra stimuli. Shown are three simultaneously recorded MAP recordings obtained from the right ventricular (MAP RV), left ventricular septal (MAP LVsept), and left ventricular free wall (MAP LVfree) epicardium. In the lower panel, the left ventricular free wall is recorded from the infarcted area and shows the typical alterations found in infarcted scar tissue: loss of the action potential dome and slow activation. Asterisks indicate stimulus artifacts in the right ventricular MAP recording. (B) Number of hearts with induced ventricular tachycardias ( > 10 consecutive premature ventricular activations) as a function of the number of extra stimuli (S2, first extra stimulus; S3, second extra stimulus) shown for hearts with G-CSF/SCF (black bars) and hearts from non-G-CSF/SCF control animals (gray bars). Ventricular tachycardia induction was less likely in G-CSF/SCF-treated hearts than in controls. (C) Left: Representative immunohistological sections of normal myocardium (top panels) and the border zone of the infarction (bottom panels) in untreated (left panels) and G-CSF/SCF-treated (right panels) hearts. Bright red immunofluorescence, connexin43; dark red, autofluorescence of myocytes; green, immunofluorescence detection of a fibrous tissue marker (rat anti-reticular fibroblast antibody; refer to Materials and methods, and see supplemental Materials and methods for a 3D visualization of connexin43 localization in the intercellular connections). Right (diagram): Mean connexin43 expression measured as connexin43 + area in relation to the whole area of myocardial tissue in the field of view. Connexin43 expression was markedly reduced in the border zone of the myocardial infarction. G-CSF/SCF increased connexin43 expression in the border zone without affecting connexin43 expression in normal myocardium.

    Techniques Used: Activation Assay, Immunofluorescence, Marker, Expressing

    Related Articles

    Marker:

    Article Title: G-CSF/SCF reduces inducible arrhythmias in the infarcted heart potentially via increased connexin43 expression and arteriogenesis
    Article Snippet: .. The following primary antibodies were used: rat anti–mouse CD45 as a marker for blood cells (AB3088; Abcam Limited), monoclonal anti–troponin T as a marker of differentiated cardiomyocytes (cardiac isoform AB-1, clone 13-11; Lab Vision), rabbit anti–rat connexin43 polyclonal antibodies for the detection of gap junctions (71-0700; Zytomed GmbH), monoclonal rat anti-reticular fibroblast antibodies (clone ER-TR7; Novus Biologicals Inc.), rabbit smooth muscle anti-actin antibody for the detection of arteries and arterioles (RB-9010; Lab Vision Corporation), rabbit c-kit (SCF receptor) antibody (sc-168; Santa Cruz Biotechnology, Inc.), and rabbit anti–G-CSFR antibody (sc-694; Santa Cruz Biotechnology, Inc.). ..

    Article Title: Coronary and Aortic Endothelial Function Affected by Feedback between Adiponectin and TNF? in Type 2 Diabetic Mice
    Article Snippet: .. Primary antibodies for adiponectin (goat polyclonal, R & D, AF1119), and endothelial cell marker, von Willebrand factor (rabbit polyclonal, Abcam), or smooth muscle α-actin (rabbit polyclonal, Abcam) or fibroblast (rat monoclonal, Novus Biologicals) were used for sequential double immunofluorescence staining. .. Secondary fluorescent antibodies were either FITC or Texas Red conjugated.

    Double Immunofluorescence Staining:

    Article Title: Coronary and Aortic Endothelial Function Affected by Feedback between Adiponectin and TNF? in Type 2 Diabetic Mice
    Article Snippet: .. Primary antibodies for adiponectin (goat polyclonal, R & D, AF1119), and endothelial cell marker, von Willebrand factor (rabbit polyclonal, Abcam), or smooth muscle α-actin (rabbit polyclonal, Abcam) or fibroblast (rat monoclonal, Novus Biologicals) were used for sequential double immunofluorescence staining. .. Secondary fluorescent antibodies were either FITC or Texas Red conjugated.

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  • 85
    Novus Biologicals monoclonal rat anti reticular fibroblast antibodies
    (A) Examples of monomorphic (top) and polymorphic (bottom) ventricular tachycardia induced by programmed stimulation using a single (top) or two (bottom) extra stimuli. Shown are three simultaneously recorded MAP recordings obtained from the right ventricular (MAP RV), left ventricular septal (MAP LVsept), and left ventricular free wall (MAP LVfree) epicardium. In the lower panel, the left ventricular free wall is recorded from the infarcted area and shows the typical alterations found in infarcted scar tissue: loss of the action potential dome and slow activation. Asterisks indicate stimulus artifacts in the right ventricular MAP recording. (B) Number of hearts with induced ventricular tachycardias ( > 10 consecutive premature ventricular activations) as a function of the number of extra stimuli (S2, first extra stimulus; S3, second extra stimulus) shown for hearts with G-CSF/SCF (black bars) and hearts from non-G-CSF/SCF control animals (gray bars). Ventricular tachycardia induction was less likely in G-CSF/SCF-treated hearts than in controls. (C) Left: Representative immunohistological sections of normal myocardium (top panels) and the border zone of the infarction (bottom panels) in untreated (left panels) and G-CSF/SCF-treated (right panels) hearts. Bright red immunofluorescence, connexin43; dark red, autofluorescence of myocytes; green, immunofluorescence detection of a fibrous tissue marker <t>(rat</t> <t>anti-reticular</t> <t>fibroblast</t> antibody; refer to Materials and methods, and see supplemental Materials and methods for a 3D visualization of connexin43 localization in the intercellular connections). Right (diagram): Mean connexin43 expression measured as connexin43 + area in relation to the whole area of myocardial tissue in the field of view. Connexin43 expression was markedly reduced in the border zone of the myocardial infarction. G-CSF/SCF increased connexin43 expression in the border zone without affecting connexin43 expression in normal myocardium.
    Monoclonal Rat Anti Reticular Fibroblast Antibodies, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 85/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/monoclonal rat anti reticular fibroblast antibodies/product/Novus Biologicals
    Average 85 stars, based on 2 article reviews
    Price from $9.99 to $1999.99
    monoclonal rat anti reticular fibroblast antibodies - by Bioz Stars, 2020-08
    85/100 stars
      Buy from Supplier

    Image Search Results


    (A) Examples of monomorphic (top) and polymorphic (bottom) ventricular tachycardia induced by programmed stimulation using a single (top) or two (bottom) extra stimuli. Shown are three simultaneously recorded MAP recordings obtained from the right ventricular (MAP RV), left ventricular septal (MAP LVsept), and left ventricular free wall (MAP LVfree) epicardium. In the lower panel, the left ventricular free wall is recorded from the infarcted area and shows the typical alterations found in infarcted scar tissue: loss of the action potential dome and slow activation. Asterisks indicate stimulus artifacts in the right ventricular MAP recording. (B) Number of hearts with induced ventricular tachycardias ( > 10 consecutive premature ventricular activations) as a function of the number of extra stimuli (S2, first extra stimulus; S3, second extra stimulus) shown for hearts with G-CSF/SCF (black bars) and hearts from non-G-CSF/SCF control animals (gray bars). Ventricular tachycardia induction was less likely in G-CSF/SCF-treated hearts than in controls. (C) Left: Representative immunohistological sections of normal myocardium (top panels) and the border zone of the infarction (bottom panels) in untreated (left panels) and G-CSF/SCF-treated (right panels) hearts. Bright red immunofluorescence, connexin43; dark red, autofluorescence of myocytes; green, immunofluorescence detection of a fibrous tissue marker (rat anti-reticular fibroblast antibody; refer to Materials and methods, and see supplemental Materials and methods for a 3D visualization of connexin43 localization in the intercellular connections). Right (diagram): Mean connexin43 expression measured as connexin43 + area in relation to the whole area of myocardial tissue in the field of view. Connexin43 expression was markedly reduced in the border zone of the myocardial infarction. G-CSF/SCF increased connexin43 expression in the border zone without affecting connexin43 expression in normal myocardium.

    Journal: The Journal of Experimental Medicine

    Article Title: G-CSF/SCF reduces inducible arrhythmias in the infarcted heart potentially via increased connexin43 expression and arteriogenesis

    doi: 10.1084/jem.20051151

    Figure Lengend Snippet: (A) Examples of monomorphic (top) and polymorphic (bottom) ventricular tachycardia induced by programmed stimulation using a single (top) or two (bottom) extra stimuli. Shown are three simultaneously recorded MAP recordings obtained from the right ventricular (MAP RV), left ventricular septal (MAP LVsept), and left ventricular free wall (MAP LVfree) epicardium. In the lower panel, the left ventricular free wall is recorded from the infarcted area and shows the typical alterations found in infarcted scar tissue: loss of the action potential dome and slow activation. Asterisks indicate stimulus artifacts in the right ventricular MAP recording. (B) Number of hearts with induced ventricular tachycardias ( > 10 consecutive premature ventricular activations) as a function of the number of extra stimuli (S2, first extra stimulus; S3, second extra stimulus) shown for hearts with G-CSF/SCF (black bars) and hearts from non-G-CSF/SCF control animals (gray bars). Ventricular tachycardia induction was less likely in G-CSF/SCF-treated hearts than in controls. (C) Left: Representative immunohistological sections of normal myocardium (top panels) and the border zone of the infarction (bottom panels) in untreated (left panels) and G-CSF/SCF-treated (right panels) hearts. Bright red immunofluorescence, connexin43; dark red, autofluorescence of myocytes; green, immunofluorescence detection of a fibrous tissue marker (rat anti-reticular fibroblast antibody; refer to Materials and methods, and see supplemental Materials and methods for a 3D visualization of connexin43 localization in the intercellular connections). Right (diagram): Mean connexin43 expression measured as connexin43 + area in relation to the whole area of myocardial tissue in the field of view. Connexin43 expression was markedly reduced in the border zone of the myocardial infarction. G-CSF/SCF increased connexin43 expression in the border zone without affecting connexin43 expression in normal myocardium.

    Article Snippet: The following primary antibodies were used: rat anti–mouse CD45 as a marker for blood cells (AB3088; Abcam Limited), monoclonal anti–troponin T as a marker of differentiated cardiomyocytes (cardiac isoform AB-1, clone 13-11; Lab Vision), rabbit anti–rat connexin43 polyclonal antibodies for the detection of gap junctions (71-0700; Zytomed GmbH), monoclonal rat anti-reticular fibroblast antibodies (clone ER-TR7; Novus Biologicals Inc.), rabbit smooth muscle anti-actin antibody for the detection of arteries and arterioles (RB-9010; Lab Vision Corporation), rabbit c-kit (SCF receptor) antibody (sc-168; Santa Cruz Biotechnology, Inc.), and rabbit anti–G-CSFR antibody (sc-694; Santa Cruz Biotechnology, Inc.).

    Techniques: Activation Assay, Immunofluorescence, Marker, Expressing