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GraphPad Prism Inc group wise comparison
Group Wise Comparison, supplied by GraphPad Prism Inc, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Article Title: De novo regulation of RD3 synthesis in residual neuroblastoma cells after intensive multi-modal clinical therapy harmonizes disease evolution
Article Snippet: .. Further, group-wise comparison with GraphPad PRISM revealed a consistent and significant (P < 0.001) loss of RD3 in cells derived after IMCT, compared with those derived prior to IMCT (Fig. ). .. The localization and expression levels observed with IF analysis corroborated near-identically with the IHC data.

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    GraphPad Prism Inc two way anova
    Localization and expressional modulation of metastamiRs target proteins in metastatic neuroblastoma. Representative image of the tissue micro array constructed with the replicates of non-metastatic xenografts controls and the manifold of metastatic tumors from spontaneous aggressive disease as well as the reproduced aggressive disease animals. Automated IHC stained panels showing the staining pattern and cellular localization of the metastamiRs’ protein targets (GRB10, MYC, SEMA3D, KRTAP1-1, MEGF10) in tumor samples. Histograms of Aperio-Spectrum image analysis and quantification of positivity for each target protein analyzed across the metastatic tumors of various animals presented with aggressive disease. The positivity values are compared to the non-metastatic xenograft controls using <t>ANOVA</t> with <t>Tukey’s</t> post-hoc correction using GraphPad PRISM
    Two Way Anova, supplied by GraphPad Prism Inc, used in various techniques. Bioz Stars score: 93/100, based on 18 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/two way anova/product/GraphPad Prism Inc
    Average 93 stars, based on 18 article reviews
    Price from $9.99 to $1999.99
    two way anova - by Bioz Stars, 2020-11
    93/100 stars
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    89
    GraphPad Prism Inc tukey s post hoc correction
    Localization and expressional modulation of metastamiRs target proteins in metastatic neuroblastoma. Representative image of the tissue micro array constructed with the replicates of non-metastatic xenografts controls and the manifold of metastatic tumors from spontaneous aggressive disease as well as the reproduced aggressive disease animals. Automated IHC stained panels showing the staining pattern and cellular localization of the metastamiRs’ protein targets (GRB10, MYC, SEMA3D, KRTAP1-1, MEGF10) in tumor samples. Histograms of Aperio-Spectrum image analysis and quantification of positivity for each target protein analyzed across the metastatic tumors of various animals presented with aggressive disease. The positivity values are compared to the non-metastatic xenograft controls using ANOVA with <t>Tukey’s</t> post-hoc correction using GraphPad PRISM
    Tukey S Post Hoc Correction, supplied by GraphPad Prism Inc, used in various techniques. Bioz Stars score: 89/100, based on 4 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/tukey s post hoc correction/product/GraphPad Prism Inc
    Average 89 stars, based on 4 article reviews
    Price from $9.99 to $1999.99
    tukey s post hoc correction - by Bioz Stars, 2020-11
    89/100 stars
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    Localization and expressional modulation of metastamiRs target proteins in metastatic neuroblastoma. Representative image of the tissue micro array constructed with the replicates of non-metastatic xenografts controls and the manifold of metastatic tumors from spontaneous aggressive disease as well as the reproduced aggressive disease animals. Automated IHC stained panels showing the staining pattern and cellular localization of the metastamiRs’ protein targets (GRB10, MYC, SEMA3D, KRTAP1-1, MEGF10) in tumor samples. Histograms of Aperio-Spectrum image analysis and quantification of positivity for each target protein analyzed across the metastatic tumors of various animals presented with aggressive disease. The positivity values are compared to the non-metastatic xenograft controls using ANOVA with Tukey’s post-hoc correction using GraphPad PRISM

    Journal: BMC Genomics

    Article Title: Reorganization of metastamiRs in the evolution of metastatic aggressive neuroblastoma cells

    doi: 10.1186/s12864-015-1642-x

    Figure Lengend Snippet: Localization and expressional modulation of metastamiRs target proteins in metastatic neuroblastoma. Representative image of the tissue micro array constructed with the replicates of non-metastatic xenografts controls and the manifold of metastatic tumors from spontaneous aggressive disease as well as the reproduced aggressive disease animals. Automated IHC stained panels showing the staining pattern and cellular localization of the metastamiRs’ protein targets (GRB10, MYC, SEMA3D, KRTAP1-1, MEGF10) in tumor samples. Histograms of Aperio-Spectrum image analysis and quantification of positivity for each target protein analyzed across the metastatic tumors of various animals presented with aggressive disease. The positivity values are compared to the non-metastatic xenograft controls using ANOVA with Tukey’s post-hoc correction using GraphPad PRISM

    Article Snippet: Core-specific metadata for the TMA was exported to excel and the group-wise comparisons were performed with two-way ANOVA with Tukey’s post-hoc correction (GraphPad Prism).

    Techniques: Microarray, Construct, Immunohistochemistry, Staining

    MetastamiRs dependent regulation of functional protein targets. a Operetta high-content confocal imaging showing immunofluorescence of ADAMTS-1, ASK-1, CREB, FOSB, GRB10, kRAS, MMP2, MMP3/10, p38, AKT, PTPN3, STAT3, TNFα and VEGFa in MSDACs. b Histograms of mean cell–Alexa Fluor intensity obtained from Columbus automated batch analysis showing alterations in the expression ( i ) GRB10, MMP2, p38, STAT3, TNFα and VEGF in cells with hsa-miR-125b mimic, ( ii ) EGFR FOSB, kRAS, p38, PTPN3 and VEGF in hsa-miR-27b mimic transfected cells, ( iii ) ASK1, CREB, MMP2, MMP3/10, PTPN3, STAT3and VEGF in MSDACs with hsa-miR-20a mimic and, ( iv ) MMP2, MMP3/10, PTPN3 and STAT3 with hsa-miR-93 mimic in MSDACs. c Histograms of mean cell–Alexa Fluor intensity showing alterations in the expression of ( i ) ADAMTS-1 and CREB with hsa-miR-1224-3p inhibition and, ( ii ) ADAMTS-1, ASK1, FOSB and AKT-1 with hsa-miR-1260 inhibition. Group-wise comparisons were performed with ANOVA with Tukey’s post-hoc correction

    Journal: BMC Genomics

    Article Title: Reorganization of metastamiRs in the evolution of metastatic aggressive neuroblastoma cells

    doi: 10.1186/s12864-015-1642-x

    Figure Lengend Snippet: MetastamiRs dependent regulation of functional protein targets. a Operetta high-content confocal imaging showing immunofluorescence of ADAMTS-1, ASK-1, CREB, FOSB, GRB10, kRAS, MMP2, MMP3/10, p38, AKT, PTPN3, STAT3, TNFα and VEGFa in MSDACs. b Histograms of mean cell–Alexa Fluor intensity obtained from Columbus automated batch analysis showing alterations in the expression ( i ) GRB10, MMP2, p38, STAT3, TNFα and VEGF in cells with hsa-miR-125b mimic, ( ii ) EGFR FOSB, kRAS, p38, PTPN3 and VEGF in hsa-miR-27b mimic transfected cells, ( iii ) ASK1, CREB, MMP2, MMP3/10, PTPN3, STAT3and VEGF in MSDACs with hsa-miR-20a mimic and, ( iv ) MMP2, MMP3/10, PTPN3 and STAT3 with hsa-miR-93 mimic in MSDACs. c Histograms of mean cell–Alexa Fluor intensity showing alterations in the expression of ( i ) ADAMTS-1 and CREB with hsa-miR-1224-3p inhibition and, ( ii ) ADAMTS-1, ASK1, FOSB and AKT-1 with hsa-miR-1260 inhibition. Group-wise comparisons were performed with ANOVA with Tukey’s post-hoc correction

    Article Snippet: Core-specific metadata for the TMA was exported to excel and the group-wise comparisons were performed with two-way ANOVA with Tukey’s post-hoc correction (GraphPad Prism).

    Techniques: Functional Assay, Imaging, Immunofluorescence, Expressing, Transfection, Inhibition

    A. Operetta high-content confocal immunofluorescence imaging showing localization of IFNγ, kRAS, CD8, CCND1, NF2, KI-67, FOXP2, MYCN, FOSL2, GRB10, NKX3.2, DCLK1, PhPT1 and MEGF10 in parental SH-SY5Y cells. B. Histograms of percent expression (mean and standard deviation) of target positivity (A/B*100, where A is the number of Alexa Fluor positive cells and B is the number of total (DAPI) cells per given field) obtained from Columbus analysis showing alterations in the expression of kRAS, FOXP2, FOSL2 and MEGF10 after hsa-miR-1206 inhibition; KI-67 after hsa-miR-600 inhibition; MYCN and DCLK1 (after hsa-miR-548a-5P inhibition; IFNγ, CD8, NF2, GRB10, kRAS after hsa-miR-513a-5P inhibition and, CCND1, NKX3.2, PhPT1, CD8, NF2 after hsa-miR-1908 inhibition. Group-wise comparisons were performed with ANOVA with Tukey's post-hoc correction.

    Journal: Oncotarget

    Article Title: Serum-circulating miRNAs predict neuroblastoma progression in mouse model of high-risk metastatic disease

    doi: 10.18632/oncotarget.7615

    Figure Lengend Snippet: A. Operetta high-content confocal immunofluorescence imaging showing localization of IFNγ, kRAS, CD8, CCND1, NF2, KI-67, FOXP2, MYCN, FOSL2, GRB10, NKX3.2, DCLK1, PhPT1 and MEGF10 in parental SH-SY5Y cells. B. Histograms of percent expression (mean and standard deviation) of target positivity (A/B*100, where A is the number of Alexa Fluor positive cells and B is the number of total (DAPI) cells per given field) obtained from Columbus analysis showing alterations in the expression of kRAS, FOXP2, FOSL2 and MEGF10 after hsa-miR-1206 inhibition; KI-67 after hsa-miR-600 inhibition; MYCN and DCLK1 (after hsa-miR-548a-5P inhibition; IFNγ, CD8, NF2, GRB10, kRAS after hsa-miR-513a-5P inhibition and, CCND1, NKX3.2, PhPT1, CD8, NF2 after hsa-miR-1908 inhibition. Group-wise comparisons were performed with ANOVA with Tukey's post-hoc correction.

    Article Snippet: Group-wise comparisons were performed with two-way ANOVA with Tukey's post-hoc correction (GraphPad Prism).

    Techniques: Immunofluorescence, Imaging, Expressing, Standard Deviation, Inhibition

    Histograms showing the expression levels of ( A ) JUND, ( B ) JUNC, ( C ) MYB, ( D ) FOSb, ( E ) STAT3, ( F ) TAL2, ( G ) MYCN, ( H ) cMYC, and ( I ) VEGF α in brain, liver, kidney, lung, spleen, and gut tissues from mice that were either mock-irradiated or exposed to whole body HZE particle radiation. Quantification of the corresponding cytoplasmic/nuclear/total positivity was performed using Columbus image data analysis from Operetta high content IF confocal imaging. The primary protein was tagged with secondary Ab tagged with Alexa Fluor-488 ® , while the cell membrane was marked with WGA-Alexa Fluor-594 ® and nuclear counterstained with DAPI. Group-wise comparisons were performed in GraphPad Prism using two-way ANOVA with Bonferroni’s post-hoc correction.

    Journal: Scientific Reports

    Article Title: High Energy Particle Radiation-associated Oncogenic Transformation in Normal Mice: Insight into the Connection between Activation of Oncotargets and Oncogene Addiction

    doi: 10.1038/srep37623

    Figure Lengend Snippet: Histograms showing the expression levels of ( A ) JUND, ( B ) JUNC, ( C ) MYB, ( D ) FOSb, ( E ) STAT3, ( F ) TAL2, ( G ) MYCN, ( H ) cMYC, and ( I ) VEGF α in brain, liver, kidney, lung, spleen, and gut tissues from mice that were either mock-irradiated or exposed to whole body HZE particle radiation. Quantification of the corresponding cytoplasmic/nuclear/total positivity was performed using Columbus image data analysis from Operetta high content IF confocal imaging. The primary protein was tagged with secondary Ab tagged with Alexa Fluor-488 ® , while the cell membrane was marked with WGA-Alexa Fluor-594 ® and nuclear counterstained with DAPI. Group-wise comparisons were performed in GraphPad Prism using two-way ANOVA with Bonferroni’s post-hoc correction.

    Article Snippet: Group-wise comparisons were performed using two-way ANOVA with Bonferroni’s post-hoc correction (GraphPad Prism).

    Techniques: Expressing, Mouse Assay, Irradiation, Imaging, Whole Genome Amplification

    Localization and expressional modulation of metastamiRs target proteins in metastatic neuroblastoma. Representative image of the tissue micro array constructed with the replicates of non-metastatic xenografts controls and the manifold of metastatic tumors from spontaneous aggressive disease as well as the reproduced aggressive disease animals. Automated IHC stained panels showing the staining pattern and cellular localization of the metastamiRs’ protein targets (GRB10, MYC, SEMA3D, KRTAP1-1, MEGF10) in tumor samples. Histograms of Aperio-Spectrum image analysis and quantification of positivity for each target protein analyzed across the metastatic tumors of various animals presented with aggressive disease. The positivity values are compared to the non-metastatic xenograft controls using ANOVA with Tukey’s post-hoc correction using GraphPad PRISM

    Journal: BMC Genomics

    Article Title: Reorganization of metastamiRs in the evolution of metastatic aggressive neuroblastoma cells

    doi: 10.1186/s12864-015-1642-x

    Figure Lengend Snippet: Localization and expressional modulation of metastamiRs target proteins in metastatic neuroblastoma. Representative image of the tissue micro array constructed with the replicates of non-metastatic xenografts controls and the manifold of metastatic tumors from spontaneous aggressive disease as well as the reproduced aggressive disease animals. Automated IHC stained panels showing the staining pattern and cellular localization of the metastamiRs’ protein targets (GRB10, MYC, SEMA3D, KRTAP1-1, MEGF10) in tumor samples. Histograms of Aperio-Spectrum image analysis and quantification of positivity for each target protein analyzed across the metastatic tumors of various animals presented with aggressive disease. The positivity values are compared to the non-metastatic xenograft controls using ANOVA with Tukey’s post-hoc correction using GraphPad PRISM

    Article Snippet: Group-wise comparisons were performed with ANOVA with Tukey’s post-hoc correction (GraphPad Prism).

    Techniques: Microarray, Construct, Immunohistochemistry, Staining

    MetastamiRs dependent regulation of functional protein targets. a Operetta high-content confocal imaging showing immunofluorescence of ADAMTS-1, ASK-1, CREB, FOSB, GRB10, kRAS, MMP2, MMP3/10, p38, AKT, PTPN3, STAT3, TNFα and VEGFa in MSDACs. b Histograms of mean cell–Alexa Fluor intensity obtained from Columbus automated batch analysis showing alterations in the expression ( i ) GRB10, MMP2, p38, STAT3, TNFα and VEGF in cells with hsa-miR-125b mimic, ( ii ) EGFR FOSB, kRAS, p38, PTPN3 and VEGF in hsa-miR-27b mimic transfected cells, ( iii ) ASK1, CREB, MMP2, MMP3/10, PTPN3, STAT3and VEGF in MSDACs with hsa-miR-20a mimic and, ( iv ) MMP2, MMP3/10, PTPN3 and STAT3 with hsa-miR-93 mimic in MSDACs. c Histograms of mean cell–Alexa Fluor intensity showing alterations in the expression of ( i ) ADAMTS-1 and CREB with hsa-miR-1224-3p inhibition and, ( ii ) ADAMTS-1, ASK1, FOSB and AKT-1 with hsa-miR-1260 inhibition. Group-wise comparisons were performed with ANOVA with Tukey’s post-hoc correction

    Journal: BMC Genomics

    Article Title: Reorganization of metastamiRs in the evolution of metastatic aggressive neuroblastoma cells

    doi: 10.1186/s12864-015-1642-x

    Figure Lengend Snippet: MetastamiRs dependent regulation of functional protein targets. a Operetta high-content confocal imaging showing immunofluorescence of ADAMTS-1, ASK-1, CREB, FOSB, GRB10, kRAS, MMP2, MMP3/10, p38, AKT, PTPN3, STAT3, TNFα and VEGFa in MSDACs. b Histograms of mean cell–Alexa Fluor intensity obtained from Columbus automated batch analysis showing alterations in the expression ( i ) GRB10, MMP2, p38, STAT3, TNFα and VEGF in cells with hsa-miR-125b mimic, ( ii ) EGFR FOSB, kRAS, p38, PTPN3 and VEGF in hsa-miR-27b mimic transfected cells, ( iii ) ASK1, CREB, MMP2, MMP3/10, PTPN3, STAT3and VEGF in MSDACs with hsa-miR-20a mimic and, ( iv ) MMP2, MMP3/10, PTPN3 and STAT3 with hsa-miR-93 mimic in MSDACs. c Histograms of mean cell–Alexa Fluor intensity showing alterations in the expression of ( i ) ADAMTS-1 and CREB with hsa-miR-1224-3p inhibition and, ( ii ) ADAMTS-1, ASK1, FOSB and AKT-1 with hsa-miR-1260 inhibition. Group-wise comparisons were performed with ANOVA with Tukey’s post-hoc correction

    Article Snippet: Group-wise comparisons were performed with ANOVA with Tukey’s post-hoc correction (GraphPad Prism).

    Techniques: Functional Assay, Imaging, Immunofluorescence, Expressing, Transfection, Inhibition