µ conotoxin giiib  (Alomone Labs)


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    Alomone Labs µ conotoxin giiib
    µ Conotoxin Giiib, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    conotoxin giiib c 270  (Alomone Labs)


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    Alomone Labs conotoxin giiib c 270
    Conotoxin Giiib C 270, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    µ conotoxin giiib  (Alomone Labs)


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    Alomone Labs µ conotoxin giiib
    µ Conotoxin Giiib, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    µ conotoxin giiib  (Alomone Labs)


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    Alomone Labs µ conotoxin giiib
    In (a) we show the percentage of singly- and polyinnervated NMJ after 4 applications over the LAL surface (one application every day between P5–P8 (observation at P9) of one of the following VGCC inhibitor substances: nitrendipine (NT 1 μM, an L-type channel blocker), <t>ω-conotoxin-GVIA</t> (ω-CON 1 μM, N-type channel blocker), and ω-agatoxin-IVA (ω-AGA 100 nM, P/Q-type blocker). Also, the L activator Bay-K8644 (5 μM), the P/Q- and N-type activator GV-58 (20 μM), and the intracellular calcium chelator BAPTA-AM (5 μM). The histogram in ( b ) shows the percentage of S1-S4 clusters in the untreated control mice (PBS) and after the 4 applications of the aforesaid substances. Data were presented as percentages of NMJ ± SD. Fisher’s test: * p < 0.05, ** p < 0.01, *** p < 0.005. The confocal images in (c) show examples of representative NMJ areas with singly, dually, and innervated by three or more axons (the corresponding number of asterisks) from YFP muscles. At the left, the L-type channel blocker nitrendipine (NT) delays axon loss because many multi-innervated NMJs persist. By the contrary, at the right, the L activator Bay-K8644 increases the number of monoinnervated junctions. The bar indicates 10 μm
    µ Conotoxin Giiib, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 94 stars, based on 1 article reviews
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    µ conotoxin giiib - by Bioz Stars, 2023-01
    94/100 stars

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    1) Product Images from "Involvement of the Voltage-Gated Calcium Channels L- P/Q- and N-Types in Synapse Elimination During Neuromuscular Junction Development"

    Article Title: Involvement of the Voltage-Gated Calcium Channels L- P/Q- and N-Types in Synapse Elimination During Neuromuscular Junction Development

    Journal: Molecular Neurobiology

    doi: 10.1007/s12035-022-02818-2

    In (a) we show the percentage of singly- and polyinnervated NMJ after 4 applications over the LAL surface (one application every day between P5–P8 (observation at P9) of one of the following VGCC inhibitor substances: nitrendipine (NT 1 μM, an L-type channel blocker), ω-conotoxin-GVIA (ω-CON 1 μM, N-type channel blocker), and ω-agatoxin-IVA (ω-AGA 100 nM, P/Q-type blocker). Also, the L activator Bay-K8644 (5 μM), the P/Q- and N-type activator GV-58 (20 μM), and the intracellular calcium chelator BAPTA-AM (5 μM). The histogram in ( b ) shows the percentage of S1-S4 clusters in the untreated control mice (PBS) and after the 4 applications of the aforesaid substances. Data were presented as percentages of NMJ ± SD. Fisher’s test: * p < 0.05, ** p < 0.01, *** p < 0.005. The confocal images in (c) show examples of representative NMJ areas with singly, dually, and innervated by three or more axons (the corresponding number of asterisks) from YFP muscles. At the left, the L-type channel blocker nitrendipine (NT) delays axon loss because many multi-innervated NMJs persist. By the contrary, at the right, the L activator Bay-K8644 increases the number of monoinnervated junctions. The bar indicates 10 μm
    Figure Legend Snippet: In (a) we show the percentage of singly- and polyinnervated NMJ after 4 applications over the LAL surface (one application every day between P5–P8 (observation at P9) of one of the following VGCC inhibitor substances: nitrendipine (NT 1 μM, an L-type channel blocker), ω-conotoxin-GVIA (ω-CON 1 μM, N-type channel blocker), and ω-agatoxin-IVA (ω-AGA 100 nM, P/Q-type blocker). Also, the L activator Bay-K8644 (5 μM), the P/Q- and N-type activator GV-58 (20 μM), and the intracellular calcium chelator BAPTA-AM (5 μM). The histogram in ( b ) shows the percentage of S1-S4 clusters in the untreated control mice (PBS) and after the 4 applications of the aforesaid substances. Data were presented as percentages of NMJ ± SD. Fisher’s test: * p < 0.05, ** p < 0.01, *** p < 0.005. The confocal images in (c) show examples of representative NMJ areas with singly, dually, and innervated by three or more axons (the corresponding number of asterisks) from YFP muscles. At the left, the L-type channel blocker nitrendipine (NT) delays axon loss because many multi-innervated NMJs persist. By the contrary, at the right, the L activator Bay-K8644 increases the number of monoinnervated junctions. The bar indicates 10 μm

    Techniques Used:

    µ conotoxin giiib  (Alomone Labs)


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    Alomone Labs µ conotoxin giiib
    µ Conotoxin Giiib, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    l conotoxin giiib  (Alomone Labs)


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    Alomone Labs l conotoxin giiib
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    giiib  (Alomone Labs)


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    Alomone Labs giiib
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    m conotoxin giiib  (Alomone Labs)


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    Alomone Labs m conotoxin giiib
    M Conotoxin Giiib, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    1985 ω conotoxin giiib  (Alomone Labs)


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    Alomone Labs 1985 ω conotoxin giiib
    MEPP recordings in 3 μM <t>ω-conotoxin</t> GIVA (a) or 3 μM ω-conotoxin GIVA plus 1μM clenbuterol (b). EPPs recorded in 3 μM ω-conotoxin GIVA or 3 μM ω-conotoxin GIVA plus 1 μM clenbuterol followed the stimulation frequency, and no major differences in amplitude or kinetics were observed (c). The thin and thick arrows show the stimulation artefact and EPP, respectively.
    1985 ω Conotoxin Giiib, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    1) Product Images from "Sympathomimetics Regulate Neuromuscular Junction Transmission through TRPV1, P/Q- and N-type Ca 2+ Channels"

    Article Title: Sympathomimetics Regulate Neuromuscular Junction Transmission through TRPV1, P/Q- and N-type Ca 2+ Channels

    Journal: Molecular and cellular neurosciences

    doi: 10.1016/j.mcn.2019.01.007

    MEPP recordings in 3 μM ω-conotoxin GIVA (a) or 3 μM ω-conotoxin GIVA plus 1μM clenbuterol (b). EPPs recorded in 3 μM ω-conotoxin GIVA or 3 μM ω-conotoxin GIVA plus 1 μM clenbuterol followed the stimulation frequency, and no major differences in amplitude or kinetics were observed (c). The thin and thick arrows show the stimulation artefact and EPP, respectively.
    Figure Legend Snippet: MEPP recordings in 3 μM ω-conotoxin GIVA (a) or 3 μM ω-conotoxin GIVA plus 1μM clenbuterol (b). EPPs recorded in 3 μM ω-conotoxin GIVA or 3 μM ω-conotoxin GIVA plus 1 μM clenbuterol followed the stimulation frequency, and no major differences in amplitude or kinetics were observed (c). The thin and thick arrows show the stimulation artefact and EPP, respectively.

    Techniques Used:

     ω-conotoxin  GVIA prevents clenbuterol-enhanced neuromuscular transmission
    Figure Legend Snippet: ω-conotoxin GVIA prevents clenbuterol-enhanced neuromuscular transmission

    Techniques Used: Significance Assay

    1985 ω conotoxin giiib  (Alomone Labs)


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    Alomone Labs 1985 ω conotoxin giiib
    MEPP recordings in 3 μM <t>ω-conotoxin</t> GIVA (a) or 3 μM ω-conotoxin GIVA plus 1μM clenbuterol (b). EPPs recorded in 3 μM ω-conotoxin GIVA or 3 μM ω-conotoxin GIVA plus 1 μM clenbuterol followed the stimulation frequency, and no major differences in amplitude or kinetics were observed (c). The thin and thick arrows show the stimulation artefact and EPP, respectively.
    1985 ω Conotoxin Giiib, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Average 94 stars, based on 1 article reviews
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    1985 ω conotoxin giiib - by Bioz Stars, 2023-01
    94/100 stars

    Images

    1) Product Images from "Sympathomimetics Regulate Neuromuscular Junction Transmission through TRPV1, P/Q- and N-type Ca 2+ Channels"

    Article Title: Sympathomimetics Regulate Neuromuscular Junction Transmission through TRPV1, P/Q- and N-type Ca 2+ Channels

    Journal: Molecular and cellular neurosciences

    doi: 10.1016/j.mcn.2019.01.007

    MEPP recordings in 3 μM ω-conotoxin GIVA (a) or 3 μM ω-conotoxin GIVA plus 1μM clenbuterol (b). EPPs recorded in 3 μM ω-conotoxin GIVA or 3 μM ω-conotoxin GIVA plus 1 μM clenbuterol followed the stimulation frequency, and no major differences in amplitude or kinetics were observed (c). The thin and thick arrows show the stimulation artefact and EPP, respectively.
    Figure Legend Snippet: MEPP recordings in 3 μM ω-conotoxin GIVA (a) or 3 μM ω-conotoxin GIVA plus 1μM clenbuterol (b). EPPs recorded in 3 μM ω-conotoxin GIVA or 3 μM ω-conotoxin GIVA plus 1 μM clenbuterol followed the stimulation frequency, and no major differences in amplitude or kinetics were observed (c). The thin and thick arrows show the stimulation artefact and EPP, respectively.

    Techniques Used:

     ω-conotoxin  GVIA prevents clenbuterol-enhanced neuromuscular transmission
    Figure Legend Snippet: ω-conotoxin GVIA prevents clenbuterol-enhanced neuromuscular transmission

    Techniques Used: Significance Assay

    muscle contraction  (Alomone Labs)


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    Alomone Labs muscle contraction
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    Alomone Labs µ conotoxin giiib
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    Alomone Labs conotoxin giiib c 270
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    Alomone Labs l conotoxin giiib
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    Alomone Labs 1985 ω conotoxin giiib
    MEPP recordings in 3 μM <t>ω-conotoxin</t> GIVA (a) or 3 μM ω-conotoxin GIVA plus 1μM clenbuterol (b). EPPs recorded in 3 μM ω-conotoxin GIVA or 3 μM ω-conotoxin GIVA plus 1 μM clenbuterol followed the stimulation frequency, and no major differences in amplitude or kinetics were observed (c). The thin and thick arrows show the stimulation artefact and EPP, respectively.
    1985 ω Conotoxin Giiib, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/1985 ω conotoxin giiib/product/Alomone Labs
    Average 94 stars, based on 1 article reviews
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    Alomone Labs muscle contraction
    MEPP recordings in 3 μM <t>ω-conotoxin</t> GIVA (a) or 3 μM ω-conotoxin GIVA plus 1μM clenbuterol (b). EPPs recorded in 3 μM ω-conotoxin GIVA or 3 μM ω-conotoxin GIVA plus 1 μM clenbuterol followed the stimulation frequency, and no major differences in amplitude or kinetics were observed (c). The thin and thick arrows show the stimulation artefact and EPP, respectively.
    Muscle Contraction, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    MEPP recordings in 3 μM ω-conotoxin GIVA (a) or 3 μM ω-conotoxin GIVA plus 1μM clenbuterol (b). EPPs recorded in 3 μM ω-conotoxin GIVA or 3 μM ω-conotoxin GIVA plus 1 μM clenbuterol followed the stimulation frequency, and no major differences in amplitude or kinetics were observed (c). The thin and thick arrows show the stimulation artefact and EPP, respectively.

    Journal: Molecular and cellular neurosciences

    Article Title: Sympathomimetics Regulate Neuromuscular Junction Transmission through TRPV1, P/Q- and N-type Ca 2+ Channels

    doi: 10.1016/j.mcn.2019.01.007

    Figure Lengend Snippet: MEPP recordings in 3 μM ω-conotoxin GIVA (a) or 3 μM ω-conotoxin GIVA plus 1μM clenbuterol (b). EPPs recorded in 3 μM ω-conotoxin GIVA or 3 μM ω-conotoxin GIVA plus 1 μM clenbuterol followed the stimulation frequency, and no major differences in amplitude or kinetics were observed (c). The thin and thick arrows show the stimulation artefact and EPP, respectively.

    Article Snippet: Salbutamol and clenbuterol concentrations used in this study are within those reported in the literature for the treatment of asthmatic patients. ( Jacobson et al., 2003 ; Yamamoto et al., 1985 ) ω-conotoxin GIIIB (a.k.a. myotoxin II, geographutoxin II, GTx-II; Alomone, cat. C270), a selective blocker of skeletal muscle Nav1.4 channels, was used at a concentration of 1μM. ω-conotoxin GVIA (a.k.a.

    Techniques:

     ω-conotoxin  GVIA prevents clenbuterol-enhanced neuromuscular transmission

    Journal: Molecular and cellular neurosciences

    Article Title: Sympathomimetics Regulate Neuromuscular Junction Transmission through TRPV1, P/Q- and N-type Ca 2+ Channels

    doi: 10.1016/j.mcn.2019.01.007

    Figure Lengend Snippet: ω-conotoxin GVIA prevents clenbuterol-enhanced neuromuscular transmission

    Article Snippet: Salbutamol and clenbuterol concentrations used in this study are within those reported in the literature for the treatment of asthmatic patients. ( Jacobson et al., 2003 ; Yamamoto et al., 1985 ) ω-conotoxin GIIIB (a.k.a. myotoxin II, geographutoxin II, GTx-II; Alomone, cat. C270), a selective blocker of skeletal muscle Nav1.4 channels, was used at a concentration of 1μM. ω-conotoxin GVIA (a.k.a.

    Techniques: Significance Assay