Structured Review

Bruker Corporation gas chromatograph
Gas Chromatograph, supplied by Bruker Corporation, used in various techniques. Bioz Stars score: 92/100, based on 15 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/gas chromatograph/product/Bruker Corporation
Average 92 stars, based on 15 article reviews
Price from $9.99 to $1999.99
gas chromatograph - by Bioz Stars, 2020-09
92/100 stars

Images

Related Articles

Concentration Assay:

Article Title: Chlorophytum comosum-bacteria interactions for airborne benzene remediation: Effect of native endophytic Enterobacter sp. EN2 inoculation and blue-red LED light.
Article Snippet: .. This study was performed to determine the effect of plant-endophytic Enterobacter sp. EN2 interactions and blue-red LED light conditions on gaseous benzene removal by plants. .. This study was performed to determine the effect of plant-endophytic Enterobacter sp. EN2 interactions and blue-red LED light conditions on gaseous benzene removal by plants.

other:

Article Title: Influence of infrastructure on water quality and greenhouse gas dynamics in urban streams
Article Snippet: Concentrations of CO2 , CH4 , and N2 O were measured using a Bruker 450 (Bruker, Billerica, MA, USA) gas chromatograph equipped with a methanizer, flame ionization detector, and electron capture detector.

Similar Products

  • Logo
  • About
  • News
  • Press Release
  • Team
  • Advisors
  • Partners
  • Contact
  • Bioz Stars
  • Bioz vStars
  • 85
    Bruker Corporation brain glc ie
    Chemical pathways used for <t>Glc</t> metabolism modeling. The mathematical model considers metabolic steady-state conditions and is simplified in order to consider only the chemical pools with high concentration, such as <t>NAA</t> and Glyc, as well as the pools located
    Brain Glc Ie, supplied by Bruker Corporation, used in various techniques. Bioz Stars score: 85/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/brain glc ie/product/Bruker Corporation
    Average 85 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    brain glc ie - by Bioz Stars, 2020-09
    85/100 stars
      Buy from Supplier

    86
    Bruker Corporation mkp1 lko mice
    Enhanced hepatic MAPK phosphorylation in <t>MKP1-LKO</t> mice. (A) Liver lysates from chow-fed Mkp-1 fl / fl and MKP1-LKO mice were analyzed by immunoblotting with the indicated antibodies. (B) Immunoblots were quantitated by densitometry for the levels of phospho-p38
    Mkp1 Lko Mice, supplied by Bruker Corporation, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/mkp1 lko mice/product/Bruker Corporation
    Average 86 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    mkp1 lko mice - by Bioz Stars, 2020-09
    86/100 stars
      Buy from Supplier

    92
    Bruker Corporation nmrn
    Representative score plots of the Principal Component Analysis (PCA) from metabolomics and lipidomics analyses . Score plots of the metabolomics MS data acquired in the negative mode on the LC-MSn°2 platform (A), the LC-MSn°1 platform in the positive mode (B), the GC-MSn°1 platform (C), and the <t>NMRn°1</t> platform (D). Experimental conditions: control (open squares), docetaxel (plain triangles), lovastatin (open circles), and lovastatin + docetaxel (plain circles). Cells were treated for 36 h. The two dimensions of the analysis and their contribution to the variance are indicated. The data are representative of results from all platforms
    Nmrn, supplied by Bruker Corporation, used in various techniques. Bioz Stars score: 92/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/nmrn/product/Bruker Corporation
    Average 92 stars, based on 2 article reviews
    Price from $9.99 to $1999.99
    nmrn - by Bioz Stars, 2020-09
    92/100 stars
      Buy from Supplier

    89
    Bruker Corporation ischemia reperfusion
    The promoted mitochondrial biogenesis after cerebral <t>ischemia/reperfusion</t> injury in vivo by OPA1-v1 Δ S1 expression. (A–D) Representative immunoblots(A) and quantitative results of the levels of PGC-1α(B), NRF-1(C) and TFAM(D) in the cortex of different experimental groups; n = 4. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used, * p
    Ischemia Reperfusion, supplied by Bruker Corporation, used in various techniques. Bioz Stars score: 89/100, based on 0 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/ischemia reperfusion/product/Bruker Corporation
    Average 89 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    ischemia reperfusion - by Bioz Stars, 2020-09
    89/100 stars
      Buy from Supplier

    Image Search Results


    Chemical pathways used for Glc metabolism modeling. The mathematical model considers metabolic steady-state conditions and is simplified in order to consider only the chemical pools with high concentration, such as NAA and Glyc, as well as the pools located

    Journal: Neurochemistry international

    Article Title: Alteration of brain glycogen turnover in the conscious rat after 5 h of prolonged wakefulness

    doi: 10.1016/j.neuint.2009.02.023

    Figure Lengend Snippet: Chemical pathways used for Glc metabolism modeling. The mathematical model considers metabolic steady-state conditions and is simplified in order to consider only the chemical pools with high concentration, such as NAA and Glyc, as well as the pools located

    Article Snippet: Brain Glc IE, Glyc IE and NAA IE were measured in vitro by 1 H NMR as previously described , using a Bruker Avance-DRX 600 (14.1 T, 600 MHz) spectrometer (Bruker BioSpin SA, Fällanden, Switzerland).

    Techniques: Gas Chromatography, Concentration Assay

    Enhanced hepatic MAPK phosphorylation in MKP1-LKO mice. (A) Liver lysates from chow-fed Mkp-1 fl / fl and MKP1-LKO mice were analyzed by immunoblotting with the indicated antibodies. (B) Immunoblots were quantitated by densitometry for the levels of phospho-p38

    Journal: Molecular and Cellular Biology

    Article Title: Hepatic Mitogen-Activated Protein Kinase Phosphatase 1 Selectively Regulates Glucose Metabolism and Energy Homeostasis

    doi: 10.1128/MCB.00503-14

    Figure Lengend Snippet: Enhanced hepatic MAPK phosphorylation in MKP1-LKO mice. (A) Liver lysates from chow-fed Mkp-1 fl / fl and MKP1-LKO mice were analyzed by immunoblotting with the indicated antibodies. (B) Immunoblots were quantitated by densitometry for the levels of phospho-p38

    Article Snippet: Conscious male Mkp-1 fl /fl and MKP1-LKO mice were used to measure total body fat and lean mass using 1 H magnetic resonance spectroscopy (Bruker minispec analyzer; Echo Medical Systems, Houston, TX).

    Techniques: Mouse Assay, Western Blot

    Increased adiposity, hyperglycemia, and hyperinsulinemia in chow-fed MKP1-LKO mice. (A) Weight curves of chow-fed male Mkp-1 fl / fl and MKP1-LKO mice ( n = 10 to 12 per genotype). (B) Liver weights and liver-to-body-weight ratio of chow-fed Mkp-1 fl / fl and

    Journal: Molecular and Cellular Biology

    Article Title: Hepatic Mitogen-Activated Protein Kinase Phosphatase 1 Selectively Regulates Glucose Metabolism and Energy Homeostasis

    doi: 10.1128/MCB.00503-14

    Figure Lengend Snippet: Increased adiposity, hyperglycemia, and hyperinsulinemia in chow-fed MKP1-LKO mice. (A) Weight curves of chow-fed male Mkp-1 fl / fl and MKP1-LKO mice ( n = 10 to 12 per genotype). (B) Liver weights and liver-to-body-weight ratio of chow-fed Mkp-1 fl / fl and

    Article Snippet: Conscious male Mkp-1 fl /fl and MKP1-LKO mice were used to measure total body fat and lean mass using 1 H magnetic resonance spectroscopy (Bruker minispec analyzer; Echo Medical Systems, Houston, TX).

    Techniques: Mouse Assay

    Susceptibility to diet-induced obesity and protection from hepatosteatosis in MKP1-LKO mice. (A) Weight curves of HFD-fed male Mkp-1 fl / fl ( n = 9) and MKP1-LKO ( n = 10) mice for 20 to 24 weeks. (B and C) Spectroscopic analysis of total body lean mass (B)

    Journal: Molecular and Cellular Biology

    Article Title: Hepatic Mitogen-Activated Protein Kinase Phosphatase 1 Selectively Regulates Glucose Metabolism and Energy Homeostasis

    doi: 10.1128/MCB.00503-14

    Figure Lengend Snippet: Susceptibility to diet-induced obesity and protection from hepatosteatosis in MKP1-LKO mice. (A) Weight curves of HFD-fed male Mkp-1 fl / fl ( n = 9) and MKP1-LKO ( n = 10) mice for 20 to 24 weeks. (B and C) Spectroscopic analysis of total body lean mass (B)

    Article Snippet: Conscious male Mkp-1 fl /fl and MKP1-LKO mice were used to measure total body fat and lean mass using 1 H magnetic resonance spectroscopy (Bruker minispec analyzer; Echo Medical Systems, Houston, TX).

    Techniques: Mouse Assay

    Regulation of gluconeogenic genes by hepatic MKP-1. (A to C) mRNA expression of G6pc , Pck1 , and Pgc1α from livers of overnight-fasted chow-fed Mkp-1 fl / fl and MKP1-LKO mice ( n = 10 per genotype). Changes in relative luciferase activity in HepG2

    Journal: Molecular and Cellular Biology

    Article Title: Hepatic Mitogen-Activated Protein Kinase Phosphatase 1 Selectively Regulates Glucose Metabolism and Energy Homeostasis

    doi: 10.1128/MCB.00503-14

    Figure Lengend Snippet: Regulation of gluconeogenic genes by hepatic MKP-1. (A to C) mRNA expression of G6pc , Pck1 , and Pgc1α from livers of overnight-fasted chow-fed Mkp-1 fl / fl and MKP1-LKO mice ( n = 10 per genotype). Changes in relative luciferase activity in HepG2

    Article Snippet: Conscious male Mkp-1 fl /fl and MKP1-LKO mice were used to measure total body fat and lean mass using 1 H magnetic resonance spectroscopy (Bruker minispec analyzer; Echo Medical Systems, Houston, TX).

    Techniques: Expressing, Mouse Assay, Luciferase, Activity Assay

    Effects on hypothalamic neuropeptide expression in MKP1-LKO mice. RNA was isolated from the brain of chow-fed (A to C) and HFD-fed (D to F) Mkp-1 fl / fl and MKP1-LKO mice, and the expression of POMC, NPY, and AgRP was analyzed by quantitative PCR ( n = 5

    Journal: Molecular and Cellular Biology

    Article Title: Hepatic Mitogen-Activated Protein Kinase Phosphatase 1 Selectively Regulates Glucose Metabolism and Energy Homeostasis

    doi: 10.1128/MCB.00503-14

    Figure Lengend Snippet: Effects on hypothalamic neuropeptide expression in MKP1-LKO mice. RNA was isolated from the brain of chow-fed (A to C) and HFD-fed (D to F) Mkp-1 fl / fl and MKP1-LKO mice, and the expression of POMC, NPY, and AgRP was analyzed by quantitative PCR ( n = 5

    Article Snippet: Conscious male Mkp-1 fl /fl and MKP1-LKO mice were used to measure total body fat and lean mass using 1 H magnetic resonance spectroscopy (Bruker minispec analyzer; Echo Medical Systems, Houston, TX).

    Techniques: Expressing, Mouse Assay, Isolation, Real-time Polymerase Chain Reaction

    MKP1-LKO mice are glucose intolerant and exhibit hepatic insulin resistance. (A) Plasma glucose concentration during glucose tolerance tests in overnight fasted chow-fed (left panel) and HFD-fed (right panel) Mkp-1 fl / fl and MKP1-LKO mice ( n = 15 and

    Journal: Molecular and Cellular Biology

    Article Title: Hepatic Mitogen-Activated Protein Kinase Phosphatase 1 Selectively Regulates Glucose Metabolism and Energy Homeostasis

    doi: 10.1128/MCB.00503-14

    Figure Lengend Snippet: MKP1-LKO mice are glucose intolerant and exhibit hepatic insulin resistance. (A) Plasma glucose concentration during glucose tolerance tests in overnight fasted chow-fed (left panel) and HFD-fed (right panel) Mkp-1 fl / fl and MKP1-LKO mice ( n = 15 and

    Article Snippet: Conscious male Mkp-1 fl /fl and MKP1-LKO mice were used to measure total body fat and lean mass using 1 H magnetic resonance spectroscopy (Bruker minispec analyzer; Echo Medical Systems, Houston, TX).

    Techniques: Mouse Assay, Concentration Assay

    Hepatic MKP-1 regulates skeletal muscle mitochondrial respiration. (A) mRNA expression of PGC-1α from skeletal muscle of chow and HFD-fed Mkp-1 fl / fl and MKP1-LKO mice ( n = 5 per genotype). (B) Mitochondrial respiratory function in skeletal muscle

    Journal: Molecular and Cellular Biology

    Article Title: Hepatic Mitogen-Activated Protein Kinase Phosphatase 1 Selectively Regulates Glucose Metabolism and Energy Homeostasis

    doi: 10.1128/MCB.00503-14

    Figure Lengend Snippet: Hepatic MKP-1 regulates skeletal muscle mitochondrial respiration. (A) mRNA expression of PGC-1α from skeletal muscle of chow and HFD-fed Mkp-1 fl / fl and MKP1-LKO mice ( n = 5 per genotype). (B) Mitochondrial respiratory function in skeletal muscle

    Article Snippet: Conscious male Mkp-1 fl /fl and MKP1-LKO mice were used to measure total body fat and lean mass using 1 H magnetic resonance spectroscopy (Bruker minispec analyzer; Echo Medical Systems, Houston, TX).

    Techniques: Expressing, Pyrolysis Gas Chromatography, Mouse Assay

    Impaired STAT-3 phosphorylation and IL-6 expression in MKP1-LKO mice. (A and B) Liver lysates from chow-fed Mkp-1 fl / fl and MKP1-LKO mice were analyzed by immunoblotting with phospho-STAT-3 (Y705) (A) or phospho-STAT-3 (S727) (B); STAT-3 and JAK2 antibodies

    Journal: Molecular and Cellular Biology

    Article Title: Hepatic Mitogen-Activated Protein Kinase Phosphatase 1 Selectively Regulates Glucose Metabolism and Energy Homeostasis

    doi: 10.1128/MCB.00503-14

    Figure Lengend Snippet: Impaired STAT-3 phosphorylation and IL-6 expression in MKP1-LKO mice. (A and B) Liver lysates from chow-fed Mkp-1 fl / fl and MKP1-LKO mice were analyzed by immunoblotting with phospho-STAT-3 (Y705) (A) or phospho-STAT-3 (S727) (B); STAT-3 and JAK2 antibodies

    Article Snippet: Conscious male Mkp-1 fl /fl and MKP1-LKO mice were used to measure total body fat and lean mass using 1 H magnetic resonance spectroscopy (Bruker minispec analyzer; Echo Medical Systems, Houston, TX).

    Techniques: Expressing, Mouse Assay

    Hepatic and serum lipid profiles of chow-fed MKP1-LKO mice. (A to E) Hepatic cytosolic diacylglycerol (A), hepatic membrane diacylglycerol (B), total hepatic ceramide (C), hepatic long-chain acyl-CoA (D), and hepatic ceramide species (E) were measured

    Journal: Molecular and Cellular Biology

    Article Title: Hepatic Mitogen-Activated Protein Kinase Phosphatase 1 Selectively Regulates Glucose Metabolism and Energy Homeostasis

    doi: 10.1128/MCB.00503-14

    Figure Lengend Snippet: Hepatic and serum lipid profiles of chow-fed MKP1-LKO mice. (A to E) Hepatic cytosolic diacylglycerol (A), hepatic membrane diacylglycerol (B), total hepatic ceramide (C), hepatic long-chain acyl-CoA (D), and hepatic ceramide species (E) were measured

    Article Snippet: Conscious male Mkp-1 fl /fl and MKP1-LKO mice were used to measure total body fat and lean mass using 1 H magnetic resonance spectroscopy (Bruker minispec analyzer; Echo Medical Systems, Houston, TX).

    Techniques: Mouse Assay

    Reduced energy expenditure in MKP1-LKO mice. Chow-fed Mkp-1 fl / fl and MKP1-LKO mice were subjected to open-circuit calorimetry. (A) Energy expenditure; (B) oxygen consumption; (C) carbon dioxide production; (D) respiratory exchange ratio; (E) feeding;

    Journal: Molecular and Cellular Biology

    Article Title: Hepatic Mitogen-Activated Protein Kinase Phosphatase 1 Selectively Regulates Glucose Metabolism and Energy Homeostasis

    doi: 10.1128/MCB.00503-14

    Figure Lengend Snippet: Reduced energy expenditure in MKP1-LKO mice. Chow-fed Mkp-1 fl / fl and MKP1-LKO mice were subjected to open-circuit calorimetry. (A) Energy expenditure; (B) oxygen consumption; (C) carbon dioxide production; (D) respiratory exchange ratio; (E) feeding;

    Article Snippet: Conscious male Mkp-1 fl /fl and MKP1-LKO mice were used to measure total body fat and lean mass using 1 H magnetic resonance spectroscopy (Bruker minispec analyzer; Echo Medical Systems, Houston, TX).

    Techniques: Mouse Assay

    Representative score plots of the Principal Component Analysis (PCA) from metabolomics and lipidomics analyses . Score plots of the metabolomics MS data acquired in the negative mode on the LC-MSn°2 platform (A), the LC-MSn°1 platform in the positive mode (B), the GC-MSn°1 platform (C), and the NMRn°1 platform (D). Experimental conditions: control (open squares), docetaxel (plain triangles), lovastatin (open circles), and lovastatin + docetaxel (plain circles). Cells were treated for 36 h. The two dimensions of the analysis and their contribution to the variance are indicated. The data are representative of results from all platforms

    Journal: Cell Death & Disease

    Article Title: Consequences of blunting the mevalonate pathway in cancer identified by a pluri-omics approach

    doi: 10.1038/s41419-018-0761-0

    Figure Lengend Snippet: Representative score plots of the Principal Component Analysis (PCA) from metabolomics and lipidomics analyses . Score plots of the metabolomics MS data acquired in the negative mode on the LC-MSn°2 platform (A), the LC-MSn°1 platform in the positive mode (B), the GC-MSn°1 platform (C), and the NMRn°1 platform (D). Experimental conditions: control (open squares), docetaxel (plain triangles), lovastatin (open circles), and lovastatin + docetaxel (plain circles). Cells were treated for 36 h. The two dimensions of the analysis and their contribution to the variance are indicated. The data are representative of results from all platforms

    Article Snippet: Metabolomics and lipidomics analytical methods (see Supplementary information) Five instrumental platforms contributed to the analyses: NMRn°1 (1H-NMR, Bruker), LC-MSn°1 (UHPLC-LTQ-Orbitrap, Thermo), LC-MSn°2 (UHPLC-Exactive, Thermo), LC-MSn°3 (UPLC-HRMSe, Synapt Q-TOF G2, Waters), GC-MSn°1 (GC-MS, Agilent).

    Techniques: Mass Spectrometry

    The promoted mitochondrial biogenesis after cerebral ischemia/reperfusion injury in vivo by OPA1-v1 Δ S1 expression. (A–D) Representative immunoblots(A) and quantitative results of the levels of PGC-1α(B), NRF-1(C) and TFAM(D) in the cortex of different experimental groups; n = 4. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used, * p

    Journal: Redox Biology

    Article Title: Restoration of L-OPA1 alleviates acute ischemic stroke injury in rats via inhibiting neuronal apoptosis and preserving mitochondrial function

    doi: 10.1016/j.redox.2020.101503

    Figure Lengend Snippet: The promoted mitochondrial biogenesis after cerebral ischemia/reperfusion injury in vivo by OPA1-v1 Δ S1 expression. (A–D) Representative immunoblots(A) and quantitative results of the levels of PGC-1α(B), NRF-1(C) and TFAM(D) in the cortex of different experimental groups; n = 4. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used, * p

    Article Snippet: 2.5 Magnetic resonance imaging (MRI) Magnetic resonance imaging was conducted at 72 h after ischemia/reperfusion using a 7-T small animal MRI system (Bruker Medizintechnik, Germany) and the Paravision 6.0 software as previously described [ ] with minor modifications.

    Techniques: In Vivo, Expressing, Western Blot, Pyrolysis Gas Chromatography

    The restored mitochondrial cristae morphology and mitochondrial length after cerebral ischemia/reperfusion injury in vivo by OPA1-v1 Δ S1 expression. (A) Representative transmission electron microscopy (TEM) images of mitochondrial cristae number and matrix density and swelling in brain cortex. (B) Representative TEM images in the cortex of different experimental groups. (C) 50–60 mitochondria per experiment were scored in three categories: beyond four cristae (Class I), two or three cristae (Class II), no more than one cristae (Class III) per mitochondrion. (D) 50–60 mitochondria per experiment were scored based on matrix density and swelling: mitochondria with a dense matrix (Class A), swollen mitochondria with hypodense matrix (Class B). (E) Representative quantitative results of mitochondrial length with 50–60 mitochondria per experiment. n = 3, Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test, *** p

    Journal: Redox Biology

    Article Title: Restoration of L-OPA1 alleviates acute ischemic stroke injury in rats via inhibiting neuronal apoptosis and preserving mitochondrial function

    doi: 10.1016/j.redox.2020.101503

    Figure Lengend Snippet: The restored mitochondrial cristae morphology and mitochondrial length after cerebral ischemia/reperfusion injury in vivo by OPA1-v1 Δ S1 expression. (A) Representative transmission electron microscopy (TEM) images of mitochondrial cristae number and matrix density and swelling in brain cortex. (B) Representative TEM images in the cortex of different experimental groups. (C) 50–60 mitochondria per experiment were scored in three categories: beyond four cristae (Class I), two or three cristae (Class II), no more than one cristae (Class III) per mitochondrion. (D) 50–60 mitochondria per experiment were scored based on matrix density and swelling: mitochondria with a dense matrix (Class A), swollen mitochondria with hypodense matrix (Class B). (E) Representative quantitative results of mitochondrial length with 50–60 mitochondria per experiment. n = 3, Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test, *** p

    Article Snippet: 2.5 Magnetic resonance imaging (MRI) Magnetic resonance imaging was conducted at 72 h after ischemia/reperfusion using a 7-T small animal MRI system (Bruker Medizintechnik, Germany) and the Paravision 6.0 software as previously described [ ] with minor modifications.

    Techniques: In Vivo, Expressing, Transmission Assay, Electron Microscopy, Transmission Electron Microscopy

    Expression of OPA1-v1 Δ S1 preserves mitochondrial integrity after cerebral ischemia/reperfusion injury in vivo and vitro. (A, B) Representative flow cytometric analysis of JC-1 expression(A) and quantitation results of JC-1 aggregates (red uorescence) compare to JC-1 monomers (green uorescence) (B) in cultured primary neurons of different experimental groups; n = 3. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used, *** p

    Journal: Redox Biology

    Article Title: Restoration of L-OPA1 alleviates acute ischemic stroke injury in rats via inhibiting neuronal apoptosis and preserving mitochondrial function

    doi: 10.1016/j.redox.2020.101503

    Figure Lengend Snippet: Expression of OPA1-v1 Δ S1 preserves mitochondrial integrity after cerebral ischemia/reperfusion injury in vivo and vitro. (A, B) Representative flow cytometric analysis of JC-1 expression(A) and quantitation results of JC-1 aggregates (red uorescence) compare to JC-1 monomers (green uorescence) (B) in cultured primary neurons of different experimental groups; n = 3. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used, *** p

    Article Snippet: 2.5 Magnetic resonance imaging (MRI) Magnetic resonance imaging was conducted at 72 h after ischemia/reperfusion using a 7-T small animal MRI system (Bruker Medizintechnik, Germany) and the Paravision 6.0 software as previously described [ ] with minor modifications.

    Techniques: Expressing, In Vivo, Quantitation Assay, Cell Culture

    The attenuated cerebral ischemia/reperfusion-induced oxidative stress in vivo and in vitro by OPA1-v1 Δ S1 expression. (A–D) Quantitative results of the levels of MDA(A), SOD(B), GSH-Px(C) and GSH/GSSG ratio(D) in the cortex of different experimental groups; n = 5. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used,*** p

    Journal: Redox Biology

    Article Title: Restoration of L-OPA1 alleviates acute ischemic stroke injury in rats via inhibiting neuronal apoptosis and preserving mitochondrial function

    doi: 10.1016/j.redox.2020.101503

    Figure Lengend Snippet: The attenuated cerebral ischemia/reperfusion-induced oxidative stress in vivo and in vitro by OPA1-v1 Δ S1 expression. (A–D) Quantitative results of the levels of MDA(A), SOD(B), GSH-Px(C) and GSH/GSSG ratio(D) in the cortex of different experimental groups; n = 5. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used,*** p

    Article Snippet: 2.5 Magnetic resonance imaging (MRI) Magnetic resonance imaging was conducted at 72 h after ischemia/reperfusion using a 7-T small animal MRI system (Bruker Medizintechnik, Germany) and the Paravision 6.0 software as previously described [ ] with minor modifications.

    Techniques: In Vivo, In Vitro, Expressing, Multiple Displacement Amplification

    OPA1 cleavage in animal and cellular models of ischemia/reperfusion. (A, B) Representative immunoblots(A) and quantitative results of the levels of OPA1 (B) in the peri-ischemic cortex after tMCAO. n = 4. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used, * p

    Journal: Redox Biology

    Article Title: Restoration of L-OPA1 alleviates acute ischemic stroke injury in rats via inhibiting neuronal apoptosis and preserving mitochondrial function

    doi: 10.1016/j.redox.2020.101503

    Figure Lengend Snippet: OPA1 cleavage in animal and cellular models of ischemia/reperfusion. (A, B) Representative immunoblots(A) and quantitative results of the levels of OPA1 (B) in the peri-ischemic cortex after tMCAO. n = 4. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used, * p

    Article Snippet: 2.5 Magnetic resonance imaging (MRI) Magnetic resonance imaging was conducted at 72 h after ischemia/reperfusion using a 7-T small animal MRI system (Bruker Medizintechnik, Germany) and the Paravision 6.0 software as previously described [ ] with minor modifications.

    Techniques: Western Blot

    The inhibited cerebral ischemia/reperfusion injury-induced neuronal apoptosis in vivo and in vitro by OPA1-v1 Δ S1 expression. (A–D) Representative immunoblots (A) and quantitative results of the levels of Cleaved caspase-3(B), Caspase-3(C) and Bcl-2(D) and in the cortex of different experimental groups; n = 4. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used. *** p

    Journal: Redox Biology

    Article Title: Restoration of L-OPA1 alleviates acute ischemic stroke injury in rats via inhibiting neuronal apoptosis and preserving mitochondrial function

    doi: 10.1016/j.redox.2020.101503

    Figure Lengend Snippet: The inhibited cerebral ischemia/reperfusion injury-induced neuronal apoptosis in vivo and in vitro by OPA1-v1 Δ S1 expression. (A–D) Representative immunoblots (A) and quantitative results of the levels of Cleaved caspase-3(B), Caspase-3(C) and Bcl-2(D) and in the cortex of different experimental groups; n = 4. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used. *** p

    Article Snippet: 2.5 Magnetic resonance imaging (MRI) Magnetic resonance imaging was conducted at 72 h after ischemia/reperfusion using a 7-T small animal MRI system (Bruker Medizintechnik, Germany) and the Paravision 6.0 software as previously described [ ] with minor modifications.

    Techniques: In Vivo, In Vitro, Expressing, Western Blot

    The improved motor function recovery and decreased infarct volume by OPA1-v1 Δ S1 expression after cerebral ischemia/reperfusion injury in vivo. (A) Representative immunofluorescence image exhibiting the overexpression of OPA1-v1 Δ S1 using GFP-containing adeno-associated virus in brain cortex (magnification 1000 × ). n = 3. (B, C) Representative immunoblots(B) and quantitative analysis of the levels of OPA1(C) in the cortex of different experimental groups; n = 4. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used. * p

    Journal: Redox Biology

    Article Title: Restoration of L-OPA1 alleviates acute ischemic stroke injury in rats via inhibiting neuronal apoptosis and preserving mitochondrial function

    doi: 10.1016/j.redox.2020.101503

    Figure Lengend Snippet: The improved motor function recovery and decreased infarct volume by OPA1-v1 Δ S1 expression after cerebral ischemia/reperfusion injury in vivo. (A) Representative immunofluorescence image exhibiting the overexpression of OPA1-v1 Δ S1 using GFP-containing adeno-associated virus in brain cortex (magnification 1000 × ). n = 3. (B, C) Representative immunoblots(B) and quantitative analysis of the levels of OPA1(C) in the cortex of different experimental groups; n = 4. Data are expressed as mean ± SEM. One-way analysis of ANOVA with Bonferroni's post hoc test was used. * p

    Article Snippet: 2.5 Magnetic resonance imaging (MRI) Magnetic resonance imaging was conducted at 72 h after ischemia/reperfusion using a 7-T small animal MRI system (Bruker Medizintechnik, Germany) and the Paravision 6.0 software as previously described [ ] with minor modifications.

    Techniques: Expressing, In Vivo, Immunofluorescence, Over Expression, Western Blot