Review





Similar Products

fgf23 monoclonal antibody  (MedChemExpress)
94
MedChemExpress fgf23 monoclonal antibody
Fgf23 Monoclonal Antibody, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/fgf23/pm41972542-48-1-7?v=MedChemExpress
Average 94 stars, based on 1 article reviews
fgf23 monoclonal antibody - by Bioz Stars, 2026-08
94/100 stars
  Buy from Supplier
anti fgf23 antibody  (Amgen)
86
Amgen anti fgf23 antibody
Schematic illustration of signaling pathways involved in BGJ398-mediated improvement in dysregulated phosphate metabolism in the kidneys of SCD mice. In SCD, anemia stimulates the kidneys to secrete erythropoietin (EPO), which travels to bone and triggers fibroblast growth factor 23 <t>(FGF23)</t> production. High level of FGF23 activate FGFR1/αKlotho signaling in the kidneys, leading to phosphaturia. Increased FGFR signaling in the kidneys also promotes inflammation. Inhibition of FGFR signaling with BGJ398 alleviates phosphate wasting and improves renal phosphate handling in SCD mice.
Anti Fgf23 Antibody, supplied by Amgen, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/fgf23/pmc13276783-275-12-19?v=Amgen
Average 86 stars, based on 1 article reviews
anti fgf23 antibody - by Bioz Stars, 2026-08
86/100 stars
  Buy from Supplier
c terminal fgf23  (Immutopics Inc)
86
Immutopics Inc c terminal fgf23
Schematic illustration of signaling pathways involved in BGJ398-mediated improvement in dysregulated phosphate metabolism in the kidneys of SCD mice. In SCD, anemia stimulates the kidneys to secrete erythropoietin (EPO), which travels to bone and triggers fibroblast growth factor 23 <t>(FGF23)</t> production. High level of FGF23 activate FGFR1/αKlotho signaling in the kidneys, leading to phosphaturia. Increased FGFR signaling in the kidneys also promotes inflammation. Inhibition of FGFR signaling with BGJ398 alleviates phosphate wasting and improves renal phosphate handling in SCD mice.
C Terminal Fgf23, supplied by Immutopics Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/fgf23/pm42082869-35-9-21?v=Immutopics+Inc
Average 86 stars, based on 1 article reviews
c terminal fgf23 - by Bioz Stars, 2026-08
86/100 stars
  Buy from Supplier
elisa kits  (Cusabio)
93
Cusabio elisa kits
Schematic illustration of signaling pathways involved in BGJ398-mediated improvement in dysregulated phosphate metabolism in the kidneys of SCD mice. In SCD, anemia stimulates the kidneys to secrete erythropoietin (EPO), which travels to bone and triggers fibroblast growth factor 23 <t>(FGF23)</t> production. High level of FGF23 activate FGFR1/αKlotho signaling in the kidneys, leading to phosphaturia. Increased FGFR signaling in the kidneys also promotes inflammation. Inhibition of FGFR signaling with BGJ398 alleviates phosphate wasting and improves renal phosphate handling in SCD mice.
Elisa Kits, supplied by Cusabio, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/fgf23/pmc12964290-220-6-8?v=Cusabio
Average 93 stars, based on 1 article reviews
elisa kits - by Bioz Stars, 2026-08
93/100 stars
  Buy from Supplier
fgf23 concentrations  (Cusabio)
93
Cusabio fgf23 concentrations
Schematic illustration of signaling pathways involved in BGJ398-mediated improvement in dysregulated phosphate metabolism in the kidneys of SCD mice. In SCD, anemia stimulates the kidneys to secrete erythropoietin (EPO), which travels to bone and triggers fibroblast growth factor 23 <t>(FGF23)</t> production. High level of FGF23 activate FGFR1/αKlotho signaling in the kidneys, leading to phosphaturia. Increased FGFR signaling in the kidneys also promotes inflammation. Inhibition of FGFR signaling with BGJ398 alleviates phosphate wasting and improves renal phosphate handling in SCD mice.
Fgf23 Concentrations, supplied by Cusabio, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/fgf23/pmc12964290-220-1-8?v=Cusabio
Average 93 stars, based on 1 article reviews
fgf23 concentrations - by Bioz Stars, 2026-08
93/100 stars
  Buy from Supplier
human fibroblast growth factor 23 elisa reagent  (Elabscience Biotechnology)
93
Elabscience Biotechnology human fibroblast growth factor 23 elisa reagent
Schematic illustration of signaling pathways involved in BGJ398-mediated improvement in dysregulated phosphate metabolism in the kidneys of SCD mice. In SCD, anemia stimulates the kidneys to secrete erythropoietin (EPO), which travels to bone and triggers fibroblast growth factor 23 <t>(FGF23)</t> production. High level of FGF23 activate FGFR1/αKlotho signaling in the kidneys, leading to phosphaturia. Increased FGFR signaling in the kidneys also promotes inflammation. Inhibition of FGFR signaling with BGJ398 alleviates phosphate wasting and improves renal phosphate handling in SCD mice.
Human Fibroblast Growth Factor 23 Elisa Reagent, supplied by Elabscience Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/fgf23/pmc13088700-280-6-15?v=Elabscience+Biotechnology
Average 93 stars, based on 1 article reviews
human fibroblast growth factor 23 elisa reagent - by Bioz Stars, 2026-08
93/100 stars
  Buy from Supplier
nephrology fgf23 levels  (Wolters Kluwer Health)
86
Wolters Kluwer Health nephrology fgf23 levels
Schematic illustration of signaling pathways involved in BGJ398-mediated improvement in dysregulated phosphate metabolism in the kidneys of SCD mice. In SCD, anemia stimulates the kidneys to secrete erythropoietin (EPO), which travels to bone and triggers fibroblast growth factor 23 <t>(FGF23)</t> production. High level of FGF23 activate FGFR1/αKlotho signaling in the kidneys, leading to phosphaturia. Increased FGFR signaling in the kidneys also promotes inflammation. Inhibition of FGFR signaling with BGJ398 alleviates phosphate wasting and improves renal phosphate handling in SCD mice.
Nephrology Fgf23 Levels, supplied by Wolters Kluwer Health, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/fgf23/10__34067_slash_kid__0000001179-71-13-2?v=Wolters+Kluwer+Health
Average 86 stars, based on 1 article reviews
nephrology fgf23 levels - by Bioz Stars, 2026-08
86/100 stars
  Buy from Supplier

Image Search Results


Schematic illustration of signaling pathways involved in BGJ398-mediated improvement in dysregulated phosphate metabolism in the kidneys of SCD mice. In SCD, anemia stimulates the kidneys to secrete erythropoietin (EPO), which travels to bone and triggers fibroblast growth factor 23 (FGF23) production. High level of FGF23 activate FGFR1/αKlotho signaling in the kidneys, leading to phosphaturia. Increased FGFR signaling in the kidneys also promotes inflammation. Inhibition of FGFR signaling with BGJ398 alleviates phosphate wasting and improves renal phosphate handling in SCD mice.

Journal: Biochemistry and Biophysics Reports

Article Title: Inhibition of FGFR signaling attenuates phosphaturia and early kidney injury in sickle cell disease mice

doi: 10.1016/j.bbrep.2026.102671

Figure Lengend Snippet: Schematic illustration of signaling pathways involved in BGJ398-mediated improvement in dysregulated phosphate metabolism in the kidneys of SCD mice. In SCD, anemia stimulates the kidneys to secrete erythropoietin (EPO), which travels to bone and triggers fibroblast growth factor 23 (FGF23) production. High level of FGF23 activate FGFR1/αKlotho signaling in the kidneys, leading to phosphaturia. Increased FGFR signaling in the kidneys also promotes inflammation. Inhibition of FGFR signaling with BGJ398 alleviates phosphate wasting and improves renal phosphate handling in SCD mice.

Article Snippet: The reagents used in this study, including the control IgG and the anti-FGF23 antibody (clone 58.5) kindly provided by Amgen Inc. (Thousand Oaks, CA).

Techniques: Protein-Protein interactions, Inhibition