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primary antibody cgas  (Proteintech)


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    Proteintech primary antibody cgas
    Primary Antibody Cgas, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 202 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/primary antibody cgas/product/Proteintech
    Average 96 stars, based on 202 article reviews
    primary antibody cgas - by Bioz Stars, 2026-02
    96/100 stars

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    A) Scheme of treatment of <t>control</t> <t>myoblasts</t> with <t>cGAS</t> agonist during the myoblast differentiation process (n=3 independent treatments). B) Representative image of control myoblasts treated with lipofectamine (mock) or cGAS agonist after 3 days of differentiation. C) Quantification of the fusion index of control treated with lipofectamine (mock) or cGAS agonist after 3 days of differentiation. Means were tested using a one-way ANOVA test. *p<0.01. D) Maturation index of control myoblasts treated with lipofectamine (mock) or agonist after 3 days of differentiation. Means were tested using a one-way ANOVA test. n.s. not significant. E) Quantification of cGAS agonist-treated myotube area. Means were tested using a one-way ANOVA test. ****p<0.00001. F) Quantification of cGAS agonist-treated myotube width. Means were tested using a one-way ANOVA test. **p<0.001. G) Quantification of cGAS agonist-treated myotube length. Means were tested using a one-way ANOVA test. *p<0.01. H) qPCR for IFNB expression in cGAS agonist-treated myotubes. Means were tested using a one-way ANOVA test. *p<0.01. I) ELISA measurement of IFNB secretion by cGAS agonist-treated and mock myotubes. Means were tested using a one-way ANOVA test. n.s. not significant.
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    A) Scheme of treatment of <t>control</t> <t>myoblasts</t> with <t>cGAS</t> agonist during the myoblast differentiation process (n=3 independent treatments). B) Representative image of control myoblasts treated with lipofectamine (mock) or cGAS agonist after 3 days of differentiation. C) Quantification of the fusion index of control treated with lipofectamine (mock) or cGAS agonist after 3 days of differentiation. Means were tested using a one-way ANOVA test. *p<0.01. D) Maturation index of control myoblasts treated with lipofectamine (mock) or agonist after 3 days of differentiation. Means were tested using a one-way ANOVA test. n.s. not significant. E) Quantification of cGAS agonist-treated myotube area. Means were tested using a one-way ANOVA test. ****p<0.00001. F) Quantification of cGAS agonist-treated myotube width. Means were tested using a one-way ANOVA test. **p<0.001. G) Quantification of cGAS agonist-treated myotube length. Means were tested using a one-way ANOVA test. *p<0.01. H) qPCR for IFNB expression in cGAS agonist-treated myotubes. Means were tested using a one-way ANOVA test. *p<0.01. I) ELISA measurement of IFNB secretion by cGAS agonist-treated and mock myotubes. Means were tested using a one-way ANOVA test. n.s. not significant.
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    A) Scheme of treatment of <t>control</t> <t>myoblasts</t> with <t>cGAS</t> agonist during the myoblast differentiation process (n=3 independent treatments). B) Representative image of control myoblasts treated with lipofectamine (mock) or cGAS agonist after 3 days of differentiation. C) Quantification of the fusion index of control treated with lipofectamine (mock) or cGAS agonist after 3 days of differentiation. Means were tested using a one-way ANOVA test. *p<0.01. D) Maturation index of control myoblasts treated with lipofectamine (mock) or agonist after 3 days of differentiation. Means were tested using a one-way ANOVA test. n.s. not significant. E) Quantification of cGAS agonist-treated myotube area. Means were tested using a one-way ANOVA test. ****p<0.00001. F) Quantification of cGAS agonist-treated myotube width. Means were tested using a one-way ANOVA test. **p<0.001. G) Quantification of cGAS agonist-treated myotube length. Means were tested using a one-way ANOVA test. *p<0.01. H) qPCR for IFNB expression in cGAS agonist-treated myotubes. Means were tested using a one-way ANOVA test. *p<0.01. I) ELISA measurement of IFNB secretion by cGAS agonist-treated and mock myotubes. Means were tested using a one-way ANOVA test. n.s. not significant.
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    Image Search Results


    A) Scheme of treatment of control myoblasts with cGAS agonist during the myoblast differentiation process (n=3 independent treatments). B) Representative image of control myoblasts treated with lipofectamine (mock) or cGAS agonist after 3 days of differentiation. C) Quantification of the fusion index of control treated with lipofectamine (mock) or cGAS agonist after 3 days of differentiation. Means were tested using a one-way ANOVA test. *p<0.01. D) Maturation index of control myoblasts treated with lipofectamine (mock) or agonist after 3 days of differentiation. Means were tested using a one-way ANOVA test. n.s. not significant. E) Quantification of cGAS agonist-treated myotube area. Means were tested using a one-way ANOVA test. ****p<0.00001. F) Quantification of cGAS agonist-treated myotube width. Means were tested using a one-way ANOVA test. **p<0.001. G) Quantification of cGAS agonist-treated myotube length. Means were tested using a one-way ANOVA test. *p<0.01. H) qPCR for IFNB expression in cGAS agonist-treated myotubes. Means were tested using a one-way ANOVA test. *p<0.01. I) ELISA measurement of IFNB secretion by cGAS agonist-treated and mock myotubes. Means were tested using a one-way ANOVA test. n.s. not significant.

    Journal: bioRxiv

    Article Title: Genomic stress drives activation of interferon signaling and innate immune pathways during SMA Type II myoblasts differentiation

    doi: 10.64898/2025.12.23.696216

    Figure Lengend Snippet: A) Scheme of treatment of control myoblasts with cGAS agonist during the myoblast differentiation process (n=3 independent treatments). B) Representative image of control myoblasts treated with lipofectamine (mock) or cGAS agonist after 3 days of differentiation. C) Quantification of the fusion index of control treated with lipofectamine (mock) or cGAS agonist after 3 days of differentiation. Means were tested using a one-way ANOVA test. *p<0.01. D) Maturation index of control myoblasts treated with lipofectamine (mock) or agonist after 3 days of differentiation. Means were tested using a one-way ANOVA test. n.s. not significant. E) Quantification of cGAS agonist-treated myotube area. Means were tested using a one-way ANOVA test. ****p<0.00001. F) Quantification of cGAS agonist-treated myotube width. Means were tested using a one-way ANOVA test. **p<0.001. G) Quantification of cGAS agonist-treated myotube length. Means were tested using a one-way ANOVA test. *p<0.01. H) qPCR for IFNB expression in cGAS agonist-treated myotubes. Means were tested using a one-way ANOVA test. *p<0.01. I) ELISA measurement of IFNB secretion by cGAS agonist-treated and mock myotubes. Means were tested using a one-way ANOVA test. n.s. not significant.

    Article Snippet: Myoblasts were transfected at 80% confluence with 1mg/mL cGAS activator (2’3’-cGAMP Invivogen #tlrl-nacga23-02) using Lipofectamine 2000 (Invitrogen) in differentiation media according to the manufacturer’s instructions.

    Techniques: Control, Expressing, Enzyme-linked Immunosorbent Assay