rv 1a  (ATCC)


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    ATCC rv 1a
    Expression level of full-length ACE2 and the truncated variant dACE2 in mock-treated airway epithelial organoids following RV infection <t>(HRV-01A).</t> Related to . (A) Organoids were mock infected or infected with RV. Some RV-infected cultures were pretreated for 18 h with 6 μM BX795 before infection with RV. (B) RT-qPCR was performed to quantify transcripts for full-length ACE2 (blue circles) or dACE2 (white triangles). Plot shows compiled data from three independent experiments, each with three to five biological replicates per condition. Individual values are shown (10–13 per condition). Numbers indicate P values of the Mann–Whitney test for significant differences between conditions. The primers used for full-length ACE2 target the junction of exons 8 and 9 are unique to the full-length ACE2 , and the primers used for dACE2 target the junction of exons 1 and 2 are unique to dACE2 , as previously described by .
    Rv 1a, supplied by ATCC, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rv 1a/product/ATCC
    Average 93 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rv 1a - by Bioz Stars, 2023-12
    93/100 stars

    Images

    1) Product Images from "Dynamic innate immune response determines susceptibility to SARS-CoV-2 infection and early replication kinetics"

    Article Title: Dynamic innate immune response determines susceptibility to SARS-CoV-2 infection and early replication kinetics

    Journal: The Journal of Experimental Medicine

    doi: 10.1084/jem.20210583

    Expression level of full-length ACE2 and the truncated variant dACE2 in mock-treated airway epithelial organoids following RV infection (HRV-01A). Related to . (A) Organoids were mock infected or infected with RV. Some RV-infected cultures were pretreated for 18 h with 6 μM BX795 before infection with RV. (B) RT-qPCR was performed to quantify transcripts for full-length ACE2 (blue circles) or dACE2 (white triangles). Plot shows compiled data from three independent experiments, each with three to five biological replicates per condition. Individual values are shown (10–13 per condition). Numbers indicate P values of the Mann–Whitney test for significant differences between conditions. The primers used for full-length ACE2 target the junction of exons 8 and 9 are unique to the full-length ACE2 , and the primers used for dACE2 target the junction of exons 1 and 2 are unique to dACE2 , as previously described by .
    Figure Legend Snippet: Expression level of full-length ACE2 and the truncated variant dACE2 in mock-treated airway epithelial organoids following RV infection (HRV-01A). Related to . (A) Organoids were mock infected or infected with RV. Some RV-infected cultures were pretreated for 18 h with 6 μM BX795 before infection with RV. (B) RT-qPCR was performed to quantify transcripts for full-length ACE2 (blue circles) or dACE2 (white triangles). Plot shows compiled data from three independent experiments, each with three to five biological replicates per condition. Individual values are shown (10–13 per condition). Numbers indicate P values of the Mann–Whitney test for significant differences between conditions. The primers used for full-length ACE2 target the junction of exons 8 and 9 are unique to the full-length ACE2 , and the primers used for dACE2 target the junction of exons 1 and 2 are unique to dACE2 , as previously described by .

    Techniques Used: Expressing, Variant Assay, Infection, Quantitative RT-PCR, MANN-WHITNEY

    Effects of RV1A replication in differentiated primary human bronchial epithelial cultures. Related to . (A–E) HRV-01A replication in bronchial epithelial organoids and host response over 5 d. (A–D) Viral RNA level and ISG mRNA levels are graphed as FC from ISG level at t = 1 h (postinoculation time point). Open symbols represent ISG levels in mock-infected cultures at day 5. Each point shows mean and SD of four biological replicates per condition. Results are representative of two independent experiments. Significant difference between mock and infected at day 5 is indicated by asterisks (**, P = 0.0286, Mann–Whitney test). (E) Protein level of IFNλ1 in the basolateral media at day 5 for mock- and RV-infected cultures. Graphs show mean and SD of four biological replicates per condition (A–E). Significant difference between mock and infected at day 5 is indicated by asterisks (**, P = 0.0286, Mann–Whitney test). (F) Cell type–specific RV infection in human bronchial epithelial organoid cultures 5 d after infection with RV (HRV-01A) or mock infection. Top panel shows cell types present, and bottom panel shows cell type–specific distribution of 70 cells containing at least one viral read. (G) Expression level of entry receptors for HRV-01A (LDLR, VLDLR, and LRP5) in RV-infected (top) or in mock-treated cultures (bottom panels). (H) Cellular composition of mock- or RV-1A–infected organoids at day 5 after infection.
    Figure Legend Snippet: Effects of RV1A replication in differentiated primary human bronchial epithelial cultures. Related to . (A–E) HRV-01A replication in bronchial epithelial organoids and host response over 5 d. (A–D) Viral RNA level and ISG mRNA levels are graphed as FC from ISG level at t = 1 h (postinoculation time point). Open symbols represent ISG levels in mock-infected cultures at day 5. Each point shows mean and SD of four biological replicates per condition. Results are representative of two independent experiments. Significant difference between mock and infected at day 5 is indicated by asterisks (**, P = 0.0286, Mann–Whitney test). (E) Protein level of IFNλ1 in the basolateral media at day 5 for mock- and RV-infected cultures. Graphs show mean and SD of four biological replicates per condition (A–E). Significant difference between mock and infected at day 5 is indicated by asterisks (**, P = 0.0286, Mann–Whitney test). (F) Cell type–specific RV infection in human bronchial epithelial organoid cultures 5 d after infection with RV (HRV-01A) or mock infection. Top panel shows cell types present, and bottom panel shows cell type–specific distribution of 70 cells containing at least one viral read. (G) Expression level of entry receptors for HRV-01A (LDLR, VLDLR, and LRP5) in RV-infected (top) or in mock-treated cultures (bottom panels). (H) Cellular composition of mock- or RV-1A–infected organoids at day 5 after infection.

    Techniques Used: Infection, MANN-WHITNEY, Expressing

    Effect of pretreatment with BX795 during sequential RV, SARS-CoV-2 infection, and low-MOI SARS-COV-2 infection. (A) Organoid cultures were pretreated with or without BX795 for 18 h, mock infected or infected with HRV-01A, incubated for 3 d, and then infected with SARS-CoV-2 MOI 0.5. BX795 was present throughout the experiment. (B) Effect of BX795 pretreatment on ISG induction 3 d after RV infection. Bars show FC in ISG mRNA level in RV infected cultures compared with mock without (left) or with (right) BX795 pretreatment. Bars shown mean and SEM of four replicates per condition and are representative of three independent experiments using primary cells from different donors. Significant difference in ISG level by Mann–Whitney is indicated ( # , P = 0.0303). (C) SARS-CoV-2 viral RNA level relative to the limit of detection in organoid cultures 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. (D) HRV-01A viral RNA level relative to the limit of detection in organoid cultures 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. This graph also includes cultures infected with RV, but not subsequently infected with SARS-CoV-2. (E) Cultures were pretreated with 6 µM BX795 or medium only for 18 h and then inoculated with SARS-CoV-2, MOI 0.05, at t = 0 (F–H). (F) SARS-CoV-2 viral RNA level relative to the limit of detection in organoid cultures 72 h after infection, with and without BX795 pretreatment (black or white bars, respectively. (G) SARS-CoV-2 viral RNA level relative to the limit of detection in apical wash 72 h after infection, with and without BX795 pretreatment (black or white bars, respectively). (H) Doubling time calculations for SARS-CoV-2 in organoids with and without BX795 pretreatment, assuming exponential growth between 1 and 72 h. Exponential growth curves were compared by the extra sum-of-squares F-test and found to be significantly different ( ### , P = 0.0011). For C, D, F, and G, bars show mean and SEM of four to six biological replicates per condition. P values indicate significant differences in ISG or viral RNA by Mann–Whitney test (*, P = 0.0449; ## , P = 0.0286; **, P = 0.0095; ***, P = 0.0043; ****, P < 0.0001). Data are representative of two independent experiments using primary cells from different donors.
    Figure Legend Snippet: Effect of pretreatment with BX795 during sequential RV, SARS-CoV-2 infection, and low-MOI SARS-COV-2 infection. (A) Organoid cultures were pretreated with or without BX795 for 18 h, mock infected or infected with HRV-01A, incubated for 3 d, and then infected with SARS-CoV-2 MOI 0.5. BX795 was present throughout the experiment. (B) Effect of BX795 pretreatment on ISG induction 3 d after RV infection. Bars show FC in ISG mRNA level in RV infected cultures compared with mock without (left) or with (right) BX795 pretreatment. Bars shown mean and SEM of four replicates per condition and are representative of three independent experiments using primary cells from different donors. Significant difference in ISG level by Mann–Whitney is indicated ( # , P = 0.0303). (C) SARS-CoV-2 viral RNA level relative to the limit of detection in organoid cultures 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. (D) HRV-01A viral RNA level relative to the limit of detection in organoid cultures 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. This graph also includes cultures infected with RV, but not subsequently infected with SARS-CoV-2. (E) Cultures were pretreated with 6 µM BX795 or medium only for 18 h and then inoculated with SARS-CoV-2, MOI 0.05, at t = 0 (F–H). (F) SARS-CoV-2 viral RNA level relative to the limit of detection in organoid cultures 72 h after infection, with and without BX795 pretreatment (black or white bars, respectively. (G) SARS-CoV-2 viral RNA level relative to the limit of detection in apical wash 72 h after infection, with and without BX795 pretreatment (black or white bars, respectively). (H) Doubling time calculations for SARS-CoV-2 in organoids with and without BX795 pretreatment, assuming exponential growth between 1 and 72 h. Exponential growth curves were compared by the extra sum-of-squares F-test and found to be significantly different ( ### , P = 0.0011). For C, D, F, and G, bars show mean and SEM of four to six biological replicates per condition. P values indicate significant differences in ISG or viral RNA by Mann–Whitney test (*, P = 0.0449; ## , P = 0.0286; **, P = 0.0095; ***, P = 0.0043; ****, P < 0.0001). Data are representative of two independent experiments using primary cells from different donors.

    Techniques Used: Infection, Incubation, MANN-WHITNEY

    SARS-CoV-2 viral load in apical wash, and ISG expression in organoids following pretreatment with BX795 in sequential RV, SARS-CoV-2 infection. Related to . (A–C) Organoid cultures were pretreated with or without BX795 for 18 h, mock infected or infected with HRV-01A, incubated for 3 d, and then infected with SARS-CoV-2, MOI 0.5 (from the experiment shown in ). SARS-CoV-2 viral RNA level relative to the limit of detection in RNA isolated from the apical wash collected 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. ISG mRNA levels relative to levels in mock-treated cultures. Bars show mean, and symbols show individual replicates. Bars show mean and SEM of five or six replicates per condition. Symbols indicate significant difference by Mann–Whitney test (***, P = 0.0043; ## , P = 0.0079; **, P = 0.0095; *, P = 0.0159). Data are representative of two independent experiments each with at four to six biological replicates per condition.
    Figure Legend Snippet: SARS-CoV-2 viral load in apical wash, and ISG expression in organoids following pretreatment with BX795 in sequential RV, SARS-CoV-2 infection. Related to . (A–C) Organoid cultures were pretreated with or without BX795 for 18 h, mock infected or infected with HRV-01A, incubated for 3 d, and then infected with SARS-CoV-2, MOI 0.5 (from the experiment shown in ). SARS-CoV-2 viral RNA level relative to the limit of detection in RNA isolated from the apical wash collected 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. ISG mRNA levels relative to levels in mock-treated cultures. Bars show mean, and symbols show individual replicates. Bars show mean and SEM of five or six replicates per condition. Symbols indicate significant difference by Mann–Whitney test (***, P = 0.0043; ## , P = 0.0079; **, P = 0.0095; *, P = 0.0159). Data are representative of two independent experiments each with at four to six biological replicates per condition.

    Techniques Used: Expressing, Infection, Incubation, Isolation, MANN-WHITNEY

    rv 1a  (ATCC)


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    ATCC rv 1a
    Expression level of full-length ACE2 and the truncated variant dACE2 in mock-treated airway epithelial organoids following RV infection <t>(HRV-01A).</t> Related to . (A) Organoids were mock infected or infected with RV. Some RV-infected cultures were pretreated for 18 h with 6 μM BX795 before infection with RV. (B) RT-qPCR was performed to quantify transcripts for full-length ACE2 (blue circles) or dACE2 (white triangles). Plot shows compiled data from three independent experiments, each with three to five biological replicates per condition. Individual values are shown (10–13 per condition). Numbers indicate P values of the Mann–Whitney test for significant differences between conditions. The primers used for full-length ACE2 target the junction of exons 8 and 9 are unique to the full-length ACE2 , and the primers used for dACE2 target the junction of exons 1 and 2 are unique to dACE2 , as previously described by .
    Rv 1a, supplied by ATCC, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rv 1a/product/ATCC
    Average 93 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rv 1a - by Bioz Stars, 2023-12
    93/100 stars

    Images

    1) Product Images from "Dynamic innate immune response determines susceptibility to SARS-CoV-2 infection and early replication kinetics"

    Article Title: Dynamic innate immune response determines susceptibility to SARS-CoV-2 infection and early replication kinetics

    Journal: The Journal of Experimental Medicine

    doi: 10.1084/jem.20210583

    Expression level of full-length ACE2 and the truncated variant dACE2 in mock-treated airway epithelial organoids following RV infection (HRV-01A). Related to . (A) Organoids were mock infected or infected with RV. Some RV-infected cultures were pretreated for 18 h with 6 μM BX795 before infection with RV. (B) RT-qPCR was performed to quantify transcripts for full-length ACE2 (blue circles) or dACE2 (white triangles). Plot shows compiled data from three independent experiments, each with three to five biological replicates per condition. Individual values are shown (10–13 per condition). Numbers indicate P values of the Mann–Whitney test for significant differences between conditions. The primers used for full-length ACE2 target the junction of exons 8 and 9 are unique to the full-length ACE2 , and the primers used for dACE2 target the junction of exons 1 and 2 are unique to dACE2 , as previously described by .
    Figure Legend Snippet: Expression level of full-length ACE2 and the truncated variant dACE2 in mock-treated airway epithelial organoids following RV infection (HRV-01A). Related to . (A) Organoids were mock infected or infected with RV. Some RV-infected cultures were pretreated for 18 h with 6 μM BX795 before infection with RV. (B) RT-qPCR was performed to quantify transcripts for full-length ACE2 (blue circles) or dACE2 (white triangles). Plot shows compiled data from three independent experiments, each with three to five biological replicates per condition. Individual values are shown (10–13 per condition). Numbers indicate P values of the Mann–Whitney test for significant differences between conditions. The primers used for full-length ACE2 target the junction of exons 8 and 9 are unique to the full-length ACE2 , and the primers used for dACE2 target the junction of exons 1 and 2 are unique to dACE2 , as previously described by .

    Techniques Used: Expressing, Variant Assay, Infection, Quantitative RT-PCR, MANN-WHITNEY

    Effects of RV1A replication in differentiated primary human bronchial epithelial cultures. Related to . (A–E) HRV-01A replication in bronchial epithelial organoids and host response over 5 d. (A–D) Viral RNA level and ISG mRNA levels are graphed as FC from ISG level at t = 1 h (postinoculation time point). Open symbols represent ISG levels in mock-infected cultures at day 5. Each point shows mean and SD of four biological replicates per condition. Results are representative of two independent experiments. Significant difference between mock and infected at day 5 is indicated by asterisks (**, P = 0.0286, Mann–Whitney test). (E) Protein level of IFNλ1 in the basolateral media at day 5 for mock- and RV-infected cultures. Graphs show mean and SD of four biological replicates per condition (A–E). Significant difference between mock and infected at day 5 is indicated by asterisks (**, P = 0.0286, Mann–Whitney test). (F) Cell type–specific RV infection in human bronchial epithelial organoid cultures 5 d after infection with RV (HRV-01A) or mock infection. Top panel shows cell types present, and bottom panel shows cell type–specific distribution of 70 cells containing at least one viral read. (G) Expression level of entry receptors for HRV-01A (LDLR, VLDLR, and LRP5) in RV-infected (top) or in mock-treated cultures (bottom panels). (H) Cellular composition of mock- or RV-1A–infected organoids at day 5 after infection.
    Figure Legend Snippet: Effects of RV1A replication in differentiated primary human bronchial epithelial cultures. Related to . (A–E) HRV-01A replication in bronchial epithelial organoids and host response over 5 d. (A–D) Viral RNA level and ISG mRNA levels are graphed as FC from ISG level at t = 1 h (postinoculation time point). Open symbols represent ISG levels in mock-infected cultures at day 5. Each point shows mean and SD of four biological replicates per condition. Results are representative of two independent experiments. Significant difference between mock and infected at day 5 is indicated by asterisks (**, P = 0.0286, Mann–Whitney test). (E) Protein level of IFNλ1 in the basolateral media at day 5 for mock- and RV-infected cultures. Graphs show mean and SD of four biological replicates per condition (A–E). Significant difference between mock and infected at day 5 is indicated by asterisks (**, P = 0.0286, Mann–Whitney test). (F) Cell type–specific RV infection in human bronchial epithelial organoid cultures 5 d after infection with RV (HRV-01A) or mock infection. Top panel shows cell types present, and bottom panel shows cell type–specific distribution of 70 cells containing at least one viral read. (G) Expression level of entry receptors for HRV-01A (LDLR, VLDLR, and LRP5) in RV-infected (top) or in mock-treated cultures (bottom panels). (H) Cellular composition of mock- or RV-1A–infected organoids at day 5 after infection.

    Techniques Used: Infection, MANN-WHITNEY, Expressing

    Effect of pretreatment with BX795 during sequential RV, SARS-CoV-2 infection, and low-MOI SARS-COV-2 infection. (A) Organoid cultures were pretreated with or without BX795 for 18 h, mock infected or infected with HRV-01A, incubated for 3 d, and then infected with SARS-CoV-2 MOI 0.5. BX795 was present throughout the experiment. (B) Effect of BX795 pretreatment on ISG induction 3 d after RV infection. Bars show FC in ISG mRNA level in RV infected cultures compared with mock without (left) or with (right) BX795 pretreatment. Bars shown mean and SEM of four replicates per condition and are representative of three independent experiments using primary cells from different donors. Significant difference in ISG level by Mann–Whitney is indicated ( # , P = 0.0303). (C) SARS-CoV-2 viral RNA level relative to the limit of detection in organoid cultures 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. (D) HRV-01A viral RNA level relative to the limit of detection in organoid cultures 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. This graph also includes cultures infected with RV, but not subsequently infected with SARS-CoV-2. (E) Cultures were pretreated with 6 µM BX795 or medium only for 18 h and then inoculated with SARS-CoV-2, MOI 0.05, at t = 0 (F–H). (F) SARS-CoV-2 viral RNA level relative to the limit of detection in organoid cultures 72 h after infection, with and without BX795 pretreatment (black or white bars, respectively. (G) SARS-CoV-2 viral RNA level relative to the limit of detection in apical wash 72 h after infection, with and without BX795 pretreatment (black or white bars, respectively). (H) Doubling time calculations for SARS-CoV-2 in organoids with and without BX795 pretreatment, assuming exponential growth between 1 and 72 h. Exponential growth curves were compared by the extra sum-of-squares F-test and found to be significantly different ( ### , P = 0.0011). For C, D, F, and G, bars show mean and SEM of four to six biological replicates per condition. P values indicate significant differences in ISG or viral RNA by Mann–Whitney test (*, P = 0.0449; ## , P = 0.0286; **, P = 0.0095; ***, P = 0.0043; ****, P < 0.0001). Data are representative of two independent experiments using primary cells from different donors.
    Figure Legend Snippet: Effect of pretreatment with BX795 during sequential RV, SARS-CoV-2 infection, and low-MOI SARS-COV-2 infection. (A) Organoid cultures were pretreated with or without BX795 for 18 h, mock infected or infected with HRV-01A, incubated for 3 d, and then infected with SARS-CoV-2 MOI 0.5. BX795 was present throughout the experiment. (B) Effect of BX795 pretreatment on ISG induction 3 d after RV infection. Bars show FC in ISG mRNA level in RV infected cultures compared with mock without (left) or with (right) BX795 pretreatment. Bars shown mean and SEM of four replicates per condition and are representative of three independent experiments using primary cells from different donors. Significant difference in ISG level by Mann–Whitney is indicated ( # , P = 0.0303). (C) SARS-CoV-2 viral RNA level relative to the limit of detection in organoid cultures 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. (D) HRV-01A viral RNA level relative to the limit of detection in organoid cultures 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. This graph also includes cultures infected with RV, but not subsequently infected with SARS-CoV-2. (E) Cultures were pretreated with 6 µM BX795 or medium only for 18 h and then inoculated with SARS-CoV-2, MOI 0.05, at t = 0 (F–H). (F) SARS-CoV-2 viral RNA level relative to the limit of detection in organoid cultures 72 h after infection, with and without BX795 pretreatment (black or white bars, respectively. (G) SARS-CoV-2 viral RNA level relative to the limit of detection in apical wash 72 h after infection, with and without BX795 pretreatment (black or white bars, respectively). (H) Doubling time calculations for SARS-CoV-2 in organoids with and without BX795 pretreatment, assuming exponential growth between 1 and 72 h. Exponential growth curves were compared by the extra sum-of-squares F-test and found to be significantly different ( ### , P = 0.0011). For C, D, F, and G, bars show mean and SEM of four to six biological replicates per condition. P values indicate significant differences in ISG or viral RNA by Mann–Whitney test (*, P = 0.0449; ## , P = 0.0286; **, P = 0.0095; ***, P = 0.0043; ****, P < 0.0001). Data are representative of two independent experiments using primary cells from different donors.

    Techniques Used: Infection, Incubation, MANN-WHITNEY

    SARS-CoV-2 viral load in apical wash, and ISG expression in organoids following pretreatment with BX795 in sequential RV, SARS-CoV-2 infection. Related to . (A–C) Organoid cultures were pretreated with or without BX795 for 18 h, mock infected or infected with HRV-01A, incubated for 3 d, and then infected with SARS-CoV-2, MOI 0.5 (from the experiment shown in ). SARS-CoV-2 viral RNA level relative to the limit of detection in RNA isolated from the apical wash collected 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. ISG mRNA levels relative to levels in mock-treated cultures. Bars show mean, and symbols show individual replicates. Bars show mean and SEM of five or six replicates per condition. Symbols indicate significant difference by Mann–Whitney test (***, P = 0.0043; ## , P = 0.0079; **, P = 0.0095; *, P = 0.0159). Data are representative of two independent experiments each with at four to six biological replicates per condition.
    Figure Legend Snippet: SARS-CoV-2 viral load in apical wash, and ISG expression in organoids following pretreatment with BX795 in sequential RV, SARS-CoV-2 infection. Related to . (A–C) Organoid cultures were pretreated with or without BX795 for 18 h, mock infected or infected with HRV-01A, incubated for 3 d, and then infected with SARS-CoV-2, MOI 0.5 (from the experiment shown in ). SARS-CoV-2 viral RNA level relative to the limit of detection in RNA isolated from the apical wash collected 72 h after SARS-CoV-2 infection, with and without BX795 and/or RV pretreatment. ISG mRNA levels relative to levels in mock-treated cultures. Bars show mean, and symbols show individual replicates. Bars show mean and SEM of five or six replicates per condition. Symbols indicate significant difference by Mann–Whitney test (***, P = 0.0043; ## , P = 0.0079; **, P = 0.0095; *, P = 0.0159). Data are representative of two independent experiments each with at four to six biological replicates per condition.

    Techniques Used: Expressing, Infection, Incubation, Isolation, MANN-WHITNEY

    rhinovirus 1a  (ATCC)


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    ATCC rhinovirus 1a
    KEY RESOURCES TABLE
    Rhinovirus 1a, supplied by ATCC, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rhinovirus 1a/product/ATCC
    Average 93 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rhinovirus 1a - by Bioz Stars, 2023-12
    93/100 stars

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    1) Product Images from "Magnitude and timing of the antiviral response determine SARS-CoV-2 replication early in infection"

    Article Title: Magnitude and timing of the antiviral response determine SARS-CoV-2 replication early in infection

    Journal: medRxiv

    doi: 10.1101/2021.01.22.21249812

    KEY RESOURCES TABLE
    Figure Legend Snippet: KEY RESOURCES TABLE

    Techniques Used: Recombinant, SYBR Green Assay, Isolation, Enzyme-linked Immunosorbent Assay, Software

    rhinovirus 1a  (ATCC)


    Bioz Verified Symbol ATCC is a verified supplier
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    Structured Review

    ATCC rhinovirus 1a
    Rhinovirus 1a, supplied by ATCC, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/rhinovirus 1a/product/ATCC
    Average 93 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    rhinovirus 1a - by Bioz Stars, 2023-12
    93/100 stars

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