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anti human angiostatin k1  (NSJ Bioreagents)


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    Structured Review

    NSJ Bioreagents anti human angiostatin k1
    Assessment of <t>angiostatin</t> levels along proximal colon segments. (A) The angiostatin level remained steady throughout the entire proximal segments in the nCT group, while up-regulated angiostatin levels were present in all segments in the nCRT group, with a gradual decrease along the proximal segments. (B) The levels of angiostatin in the P1 through P4 segments in the nCRT group were much higher than the average level of angiostatin in the nCT group (all P < 0.05). No significance was observed at site P5 compared with the nCT group ( P = 0.102). (C) Confirmation of angiostatin levels in different proximal colon segments by Western blotting (antibody from RayBiotech). (D) Representative figures showing the distribution patterns of angiostatin in different proximal segments in the two groups. In the nCRT group, strong angiostatin staining and dispersion in both the submucosal and mucosal layers were observed in P1 segments, with slightly weaker staining but no diffusion patterns found in P3 segments (100×). Compared with the P6 segments in the nCRT group, the entire proximal segment in the nCT group was mainly positive for angiostatin staining inside the vessels of the submucosa. (E) The proportion of diffusion patterns of angiostatin gradually decreased in proximal segments in the nCRT group. A diffusion pattern of angiostatin was observed only in 8% (1/13) of P1 segments in the nCT group, while the other proximal segments showed a non-diffuse pattern (normal pattern). (F) A significantly higher proportion of the non-diffuse pattern of angiostatin was observed at the proximal margins in the n-CRT-E group compared with the nCRT-C group (55% vs 87%, P = 0.039), whereas no significant difference was observed at the distal margins between the two groups (33% vs 27%, P = 0.742).
    Anti Human Angiostatin K1, supplied by NSJ Bioreagents, used in various techniques. Bioz Stars score: 90/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Images

    1) Product Images from "To what extent should the intestinal be resected proximally after radiotherapy: hint from a pathological view"

    Article Title: To what extent should the intestinal be resected proximally after radiotherapy: hint from a pathological view

    Journal: Gastroenterology Report

    doi: 10.1093/gastro/goz047

    Assessment of angiostatin levels along proximal colon segments. (A) The angiostatin level remained steady throughout the entire proximal segments in the nCT group, while up-regulated angiostatin levels were present in all segments in the nCRT group, with a gradual decrease along the proximal segments. (B) The levels of angiostatin in the P1 through P4 segments in the nCRT group were much higher than the average level of angiostatin in the nCT group (all P < 0.05). No significance was observed at site P5 compared with the nCT group ( P = 0.102). (C) Confirmation of angiostatin levels in different proximal colon segments by Western blotting (antibody from RayBiotech). (D) Representative figures showing the distribution patterns of angiostatin in different proximal segments in the two groups. In the nCRT group, strong angiostatin staining and dispersion in both the submucosal and mucosal layers were observed in P1 segments, with slightly weaker staining but no diffusion patterns found in P3 segments (100×). Compared with the P6 segments in the nCRT group, the entire proximal segment in the nCT group was mainly positive for angiostatin staining inside the vessels of the submucosa. (E) The proportion of diffusion patterns of angiostatin gradually decreased in proximal segments in the nCRT group. A diffusion pattern of angiostatin was observed only in 8% (1/13) of P1 segments in the nCT group, while the other proximal segments showed a non-diffuse pattern (normal pattern). (F) A significantly higher proportion of the non-diffuse pattern of angiostatin was observed at the proximal margins in the n-CRT-E group compared with the nCRT-C group (55% vs 87%, P = 0.039), whereas no significant difference was observed at the distal margins between the two groups (33% vs 27%, P = 0.742).
    Figure Legend Snippet: Assessment of angiostatin levels along proximal colon segments. (A) The angiostatin level remained steady throughout the entire proximal segments in the nCT group, while up-regulated angiostatin levels were present in all segments in the nCRT group, with a gradual decrease along the proximal segments. (B) The levels of angiostatin in the P1 through P4 segments in the nCRT group were much higher than the average level of angiostatin in the nCT group (all P < 0.05). No significance was observed at site P5 compared with the nCT group ( P = 0.102). (C) Confirmation of angiostatin levels in different proximal colon segments by Western blotting (antibody from RayBiotech). (D) Representative figures showing the distribution patterns of angiostatin in different proximal segments in the two groups. In the nCRT group, strong angiostatin staining and dispersion in both the submucosal and mucosal layers were observed in P1 segments, with slightly weaker staining but no diffusion patterns found in P3 segments (100×). Compared with the P6 segments in the nCRT group, the entire proximal segment in the nCT group was mainly positive for angiostatin staining inside the vessels of the submucosa. (E) The proportion of diffusion patterns of angiostatin gradually decreased in proximal segments in the nCRT group. A diffusion pattern of angiostatin was observed only in 8% (1/13) of P1 segments in the nCT group, while the other proximal segments showed a non-diffuse pattern (normal pattern). (F) A significantly higher proportion of the non-diffuse pattern of angiostatin was observed at the proximal margins in the n-CRT-E group compared with the nCRT-C group (55% vs 87%, P = 0.039), whereas no significant difference was observed at the distal margins between the two groups (33% vs 27%, P = 0.742).

    Techniques Used: Western Blot, Staining, Diffusion-based Assay

    Subgroup comparison of radiation injury at P6 segments
    Figure Legend Snippet: Subgroup comparison of radiation injury at P6 segments

    Techniques Used:



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    NSJ Bioreagents anti human angiostatin k1
    Assessment of <t>angiostatin</t> levels along proximal colon segments. (A) The angiostatin level remained steady throughout the entire proximal segments in the nCT group, while up-regulated angiostatin levels were present in all segments in the nCRT group, with a gradual decrease along the proximal segments. (B) The levels of angiostatin in the P1 through P4 segments in the nCRT group were much higher than the average level of angiostatin in the nCT group (all P < 0.05). No significance was observed at site P5 compared with the nCT group ( P = 0.102). (C) Confirmation of angiostatin levels in different proximal colon segments by Western blotting (antibody from RayBiotech). (D) Representative figures showing the distribution patterns of angiostatin in different proximal segments in the two groups. In the nCRT group, strong angiostatin staining and dispersion in both the submucosal and mucosal layers were observed in P1 segments, with slightly weaker staining but no diffusion patterns found in P3 segments (100×). Compared with the P6 segments in the nCRT group, the entire proximal segment in the nCT group was mainly positive for angiostatin staining inside the vessels of the submucosa. (E) The proportion of diffusion patterns of angiostatin gradually decreased in proximal segments in the nCRT group. A diffusion pattern of angiostatin was observed only in 8% (1/13) of P1 segments in the nCT group, while the other proximal segments showed a non-diffuse pattern (normal pattern). (F) A significantly higher proportion of the non-diffuse pattern of angiostatin was observed at the proximal margins in the n-CRT-E group compared with the nCRT-C group (55% vs 87%, P = 0.039), whereas no significant difference was observed at the distal margins between the two groups (33% vs 27%, P = 0.742).
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    Assessment of <t>angiostatin</t> levels along proximal colon segments. (A) The angiostatin level remained steady throughout the entire proximal segments in the nCT group, while up-regulated angiostatin levels were present in all segments in the nCRT group, with a gradual decrease along the proximal segments. (B) The levels of angiostatin in the P1 through P4 segments in the nCRT group were much higher than the average level of angiostatin in the nCT group (all P < 0.05). No significance was observed at site P5 compared with the nCT group ( P = 0.102). (C) Confirmation of angiostatin levels in different proximal colon segments by Western blotting (antibody from RayBiotech). (D) Representative figures showing the distribution patterns of angiostatin in different proximal segments in the two groups. In the nCRT group, strong angiostatin staining and dispersion in both the submucosal and mucosal layers were observed in P1 segments, with slightly weaker staining but no diffusion patterns found in P3 segments (100×). Compared with the P6 segments in the nCRT group, the entire proximal segment in the nCT group was mainly positive for angiostatin staining inside the vessels of the submucosa. (E) The proportion of diffusion patterns of angiostatin gradually decreased in proximal segments in the nCRT group. A diffusion pattern of angiostatin was observed only in 8% (1/13) of P1 segments in the nCT group, while the other proximal segments showed a non-diffuse pattern (normal pattern). (F) A significantly higher proportion of the non-diffuse pattern of angiostatin was observed at the proximal margins in the n-CRT-E group compared with the nCRT-C group (55% vs 87%, P = 0.039), whereas no significant difference was observed at the distal margins between the two groups (33% vs 27%, P = 0.742).
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    Assessment of <t>angiostatin</t> levels along proximal colon segments. (A) The angiostatin level remained steady throughout the entire proximal segments in the nCT group, while up-regulated angiostatin levels were present in all segments in the nCRT group, with a gradual decrease along the proximal segments. (B) The levels of angiostatin in the P1 through P4 segments in the nCRT group were much higher than the average level of angiostatin in the nCT group (all P < 0.05). No significance was observed at site P5 compared with the nCT group ( P = 0.102). (C) Confirmation of angiostatin levels in different proximal colon segments by Western blotting (antibody from RayBiotech). (D) Representative figures showing the distribution patterns of angiostatin in different proximal segments in the two groups. In the nCRT group, strong angiostatin staining and dispersion in both the submucosal and mucosal layers were observed in P1 segments, with slightly weaker staining but no diffusion patterns found in P3 segments (100×). Compared with the P6 segments in the nCRT group, the entire proximal segment in the nCT group was mainly positive for angiostatin staining inside the vessels of the submucosa. (E) The proportion of diffusion patterns of angiostatin gradually decreased in proximal segments in the nCRT group. A diffusion pattern of angiostatin was observed only in 8% (1/13) of P1 segments in the nCT group, while the other proximal segments showed a non-diffuse pattern (normal pattern). (F) A significantly higher proportion of the non-diffuse pattern of angiostatin was observed at the proximal margins in the n-CRT-E group compared with the nCRT-C group (55% vs 87%, P = 0.039), whereas no significant difference was observed at the distal margins between the two groups (33% vs 27%, P = 0.742).
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    Assessment of <t>angiostatin</t> levels along proximal colon segments. (A) The angiostatin level remained steady throughout the entire proximal segments in the nCT group, while up-regulated angiostatin levels were present in all segments in the nCRT group, with a gradual decrease along the proximal segments. (B) The levels of angiostatin in the P1 through P4 segments in the nCRT group were much higher than the average level of angiostatin in the nCT group (all P < 0.05). No significance was observed at site P5 compared with the nCT group ( P = 0.102). (C) Confirmation of angiostatin levels in different proximal colon segments by Western blotting (antibody from RayBiotech). (D) Representative figures showing the distribution patterns of angiostatin in different proximal segments in the two groups. In the nCRT group, strong angiostatin staining and dispersion in both the submucosal and mucosal layers were observed in P1 segments, with slightly weaker staining but no diffusion patterns found in P3 segments (100×). Compared with the P6 segments in the nCRT group, the entire proximal segment in the nCT group was mainly positive for angiostatin staining inside the vessels of the submucosa. (E) The proportion of diffusion patterns of angiostatin gradually decreased in proximal segments in the nCRT group. A diffusion pattern of angiostatin was observed only in 8% (1/13) of P1 segments in the nCT group, while the other proximal segments showed a non-diffuse pattern (normal pattern). (F) A significantly higher proportion of the non-diffuse pattern of angiostatin was observed at the proximal margins in the n-CRT-E group compared with the nCRT-C group (55% vs 87%, P = 0.039), whereas no significant difference was observed at the distal margins between the two groups (33% vs 27%, P = 0.742).
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    Image Search Results


    Assessment of angiostatin levels along proximal colon segments. (A) The angiostatin level remained steady throughout the entire proximal segments in the nCT group, while up-regulated angiostatin levels were present in all segments in the nCRT group, with a gradual decrease along the proximal segments. (B) The levels of angiostatin in the P1 through P4 segments in the nCRT group were much higher than the average level of angiostatin in the nCT group (all P < 0.05). No significance was observed at site P5 compared with the nCT group ( P = 0.102). (C) Confirmation of angiostatin levels in different proximal colon segments by Western blotting (antibody from RayBiotech). (D) Representative figures showing the distribution patterns of angiostatin in different proximal segments in the two groups. In the nCRT group, strong angiostatin staining and dispersion in both the submucosal and mucosal layers were observed in P1 segments, with slightly weaker staining but no diffusion patterns found in P3 segments (100×). Compared with the P6 segments in the nCRT group, the entire proximal segment in the nCT group was mainly positive for angiostatin staining inside the vessels of the submucosa. (E) The proportion of diffusion patterns of angiostatin gradually decreased in proximal segments in the nCRT group. A diffusion pattern of angiostatin was observed only in 8% (1/13) of P1 segments in the nCT group, while the other proximal segments showed a non-diffuse pattern (normal pattern). (F) A significantly higher proportion of the non-diffuse pattern of angiostatin was observed at the proximal margins in the n-CRT-E group compared with the nCRT-C group (55% vs 87%, P = 0.039), whereas no significant difference was observed at the distal margins between the two groups (33% vs 27%, P = 0.742).

    Journal: Gastroenterology Report

    Article Title: To what extent should the intestinal be resected proximally after radiotherapy: hint from a pathological view

    doi: 10.1093/gastro/goz047

    Figure Lengend Snippet: Assessment of angiostatin levels along proximal colon segments. (A) The angiostatin level remained steady throughout the entire proximal segments in the nCT group, while up-regulated angiostatin levels were present in all segments in the nCRT group, with a gradual decrease along the proximal segments. (B) The levels of angiostatin in the P1 through P4 segments in the nCRT group were much higher than the average level of angiostatin in the nCT group (all P < 0.05). No significance was observed at site P5 compared with the nCT group ( P = 0.102). (C) Confirmation of angiostatin levels in different proximal colon segments by Western blotting (antibody from RayBiotech). (D) Representative figures showing the distribution patterns of angiostatin in different proximal segments in the two groups. In the nCRT group, strong angiostatin staining and dispersion in both the submucosal and mucosal layers were observed in P1 segments, with slightly weaker staining but no diffusion patterns found in P3 segments (100×). Compared with the P6 segments in the nCRT group, the entire proximal segment in the nCT group was mainly positive for angiostatin staining inside the vessels of the submucosa. (E) The proportion of diffusion patterns of angiostatin gradually decreased in proximal segments in the nCRT group. A diffusion pattern of angiostatin was observed only in 8% (1/13) of P1 segments in the nCT group, while the other proximal segments showed a non-diffuse pattern (normal pattern). (F) A significantly higher proportion of the non-diffuse pattern of angiostatin was observed at the proximal margins in the n-CRT-E group compared with the nCRT-C group (55% vs 87%, P = 0.039), whereas no significant difference was observed at the distal margins between the two groups (33% vs 27%, P = 0.742).

    Article Snippet: Briefly, anti-human angiostatin K1-3 polyclonal primary antibody (R30129, NSJ Bioreagents, San Diego, CA, USA) was used to evaluate the distributed patterns of angiostatin.

    Techniques: Western Blot, Staining, Diffusion-based Assay

    Subgroup comparison of radiation injury at P6 segments

    Journal: Gastroenterology Report

    Article Title: To what extent should the intestinal be resected proximally after radiotherapy: hint from a pathological view

    doi: 10.1093/gastro/goz047

    Figure Lengend Snippet: Subgroup comparison of radiation injury at P6 segments

    Article Snippet: Briefly, anti-human angiostatin K1-3 polyclonal primary antibody (R30129, NSJ Bioreagents, San Diego, CA, USA) was used to evaluate the distributed patterns of angiostatin.

    Techniques: