antibody against trpv1  (Alomone Labs)


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    Structured Review

    Alomone Labs antibody against trpv1
    (a) Expression of extracellular neurotransmitters (A) HMGB1, (B) S100B), receptors (C) RAGE, (D) TLR2, (E) TLR4) and cytoplasmic inflammation signal molecules (F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse thalamus. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA) and <t>Trpv1</t> −/− groups. ∗ indicates statistical significance with P
    Antibody Against Trpv1, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/antibody against trpv1/product/Alomone Labs
    Average 95 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    antibody against trpv1 - by Bioz Stars, 2022-12
    95/100 stars

    Images

    1) Product Images from "Electroacupuncture Reduces Fibromyalgia Pain by Attenuating the HMGB1, S100B, and TRPV1 Signalling Pathways in the Mouse Brain"

    Article Title: Electroacupuncture Reduces Fibromyalgia Pain by Attenuating the HMGB1, S100B, and TRPV1 Signalling Pathways in the Mouse Brain

    Journal: Evidence-based Complementary and Alternative Medicine : eCAM

    doi: 10.1155/2022/2242074

    (a) Expression of extracellular neurotransmitters (A) HMGB1, (B) S100B), receptors (C) RAGE, (D) TLR2, (E) TLR4) and cytoplasmic inflammation signal molecules (F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse thalamus. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA) and Trpv1 −/− groups. ∗ indicates statistical significance with P
    Figure Legend Snippet: (a) Expression of extracellular neurotransmitters (A) HMGB1, (B) S100B), receptors (C) RAGE, (D) TLR2, (E) TLR4) and cytoplasmic inflammation signal molecules (F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse thalamus. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA) and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Techniques Used: Expressing, Western Blot

    (a) The expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse amygdala. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P
    Figure Legend Snippet: (a) The expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse amygdala. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Techniques Used: Expressing, Western Blot

    (a) Expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse PFC. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P
    Figure Legend Snippet: (a) Expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse PFC. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Techniques Used: Expressing, Western Blot

    (a) Expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse SSC. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P
    Figure Legend Snippet: (a) Expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse SSC. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Techniques Used: Expressing, Western Blot

    Comparative graph showing the hind paw withdrawal threshold and latency. Black: control group, red: ICS group, blue: ICS + electroacupuncture (EA) group, green: transient receptor vanilloid member one deletion ( Trpv1 −/− ) group. ∗ indicates statistical significance with p
    Figure Legend Snippet: Comparative graph showing the hind paw withdrawal threshold and latency. Black: control group, red: ICS group, blue: ICS + electroacupuncture (EA) group, green: transient receptor vanilloid member one deletion ( Trpv1 −/− ) group. ∗ indicates statistical significance with p

    Techniques Used:

    Schematic illustration of neuronal and non-neuronal mechanisms underlying EA-mediated analgesic effect on ICS-induced fibromyalgia pain. The summary diagram shows the importance of and mechanisms involving glial cells (astrocytes and microglia) and TRPV1 in fibromyalgia pain. EA inhibits HMGB1 and S100B release from non-neuronal cells or directly inhibits TRPV1 on the plasma membrane. Mice with a TRPV1 gene deletion ( Trpv1 −/− ) have the same phenotype than mice treated with EA.
    Figure Legend Snippet: Schematic illustration of neuronal and non-neuronal mechanisms underlying EA-mediated analgesic effect on ICS-induced fibromyalgia pain. The summary diagram shows the importance of and mechanisms involving glial cells (astrocytes and microglia) and TRPV1 in fibromyalgia pain. EA inhibits HMGB1 and S100B release from non-neuronal cells or directly inhibits TRPV1 on the plasma membrane. Mice with a TRPV1 gene deletion ( Trpv1 −/− ) have the same phenotype than mice treated with EA.

    Techniques Used: Mouse Assay

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    Alomone Labs antibody against trpv1
    (a) Expression of extracellular neurotransmitters (A) HMGB1, (B) S100B), receptors (C) RAGE, (D) TLR2, (E) TLR4) and cytoplasmic inflammation signal molecules (F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse thalamus. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA) and <t>Trpv1</t> −/− groups. ∗ indicates statistical significance with P
    Antibody Against Trpv1, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/antibody against trpv1/product/Alomone Labs
    Average 95 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    antibody against trpv1 - by Bioz Stars, 2022-12
    95/100 stars
      Buy from Supplier

    Image Search Results


    (a) Expression of extracellular neurotransmitters (A) HMGB1, (B) S100B), receptors (C) RAGE, (D) TLR2, (E) TLR4) and cytoplasmic inflammation signal molecules (F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse thalamus. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA) and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Journal: Evidence-based Complementary and Alternative Medicine : eCAM

    Article Title: Electroacupuncture Reduces Fibromyalgia Pain by Attenuating the HMGB1, S100B, and TRPV1 Signalling Pathways in the Mouse Brain

    doi: 10.1155/2022/2242074

    Figure Lengend Snippet: (a) Expression of extracellular neurotransmitters (A) HMGB1, (B) S100B), receptors (C) RAGE, (D) TLR2, (E) TLR4) and cytoplasmic inflammation signal molecules (F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse thalamus. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA) and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Article Snippet: The membrane was blocked with 5% nonfat milk in TBS-T buffer (10 mM Tris-HCl pH 7.5, 100 mM NaCl, 0.1% Tween-20) and then incubated with a primary antibody against TRPV1 (∼95 kDa, 1 : 1000; Alomone, Israel), HMGB1 (∼28 kDa, 1 : 1000; Alomone, Israel), S100B (∼10 kDa, 1 : 1000; Millipore, USA), TLR4 (∼35 kDa, 1 : 1000; Millipore, USA), RAGE (∼42 kDa, 1 : 1000; Millipore, USA), pPI3K (∼125 kDa, 1 : 1000; Millipore, USA), pAkt (∼60 kDa, 1 : 1000; Millipore, USA), pmTOR (∼60 kDa, 1 : 500; Millipore, USA), pERK1/2 (∼42–44 kDa, 1 : 1000; Millipore, USA), pp38 (∼42 kDa, 1 : 1000; Millipore, USA), pJNK (∼42 kDa, 1 : 1000; Millipore, USA), and pNF-κB (∼65 kDa, 1 : 1000; Millipore, USA) in TBS-T with 1% bovine serum albumin.

    Techniques: Expressing, Western Blot

    (a) The expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse amygdala. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Journal: Evidence-based Complementary and Alternative Medicine : eCAM

    Article Title: Electroacupuncture Reduces Fibromyalgia Pain by Attenuating the HMGB1, S100B, and TRPV1 Signalling Pathways in the Mouse Brain

    doi: 10.1155/2022/2242074

    Figure Lengend Snippet: (a) The expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse amygdala. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Article Snippet: The membrane was blocked with 5% nonfat milk in TBS-T buffer (10 mM Tris-HCl pH 7.5, 100 mM NaCl, 0.1% Tween-20) and then incubated with a primary antibody against TRPV1 (∼95 kDa, 1 : 1000; Alomone, Israel), HMGB1 (∼28 kDa, 1 : 1000; Alomone, Israel), S100B (∼10 kDa, 1 : 1000; Millipore, USA), TLR4 (∼35 kDa, 1 : 1000; Millipore, USA), RAGE (∼42 kDa, 1 : 1000; Millipore, USA), pPI3K (∼125 kDa, 1 : 1000; Millipore, USA), pAkt (∼60 kDa, 1 : 1000; Millipore, USA), pmTOR (∼60 kDa, 1 : 500; Millipore, USA), pERK1/2 (∼42–44 kDa, 1 : 1000; Millipore, USA), pp38 (∼42 kDa, 1 : 1000; Millipore, USA), pJNK (∼42 kDa, 1 : 1000; Millipore, USA), and pNF-κB (∼65 kDa, 1 : 1000; Millipore, USA) in TBS-T with 1% bovine serum albumin.

    Techniques: Expressing, Western Blot

    (a) Expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse PFC. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Journal: Evidence-based Complementary and Alternative Medicine : eCAM

    Article Title: Electroacupuncture Reduces Fibromyalgia Pain by Attenuating the HMGB1, S100B, and TRPV1 Signalling Pathways in the Mouse Brain

    doi: 10.1155/2022/2242074

    Figure Lengend Snippet: (a) Expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse PFC. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Article Snippet: The membrane was blocked with 5% nonfat milk in TBS-T buffer (10 mM Tris-HCl pH 7.5, 100 mM NaCl, 0.1% Tween-20) and then incubated with a primary antibody against TRPV1 (∼95 kDa, 1 : 1000; Alomone, Israel), HMGB1 (∼28 kDa, 1 : 1000; Alomone, Israel), S100B (∼10 kDa, 1 : 1000; Millipore, USA), TLR4 (∼35 kDa, 1 : 1000; Millipore, USA), RAGE (∼42 kDa, 1 : 1000; Millipore, USA), pPI3K (∼125 kDa, 1 : 1000; Millipore, USA), pAkt (∼60 kDa, 1 : 1000; Millipore, USA), pmTOR (∼60 kDa, 1 : 500; Millipore, USA), pERK1/2 (∼42–44 kDa, 1 : 1000; Millipore, USA), pp38 (∼42 kDa, 1 : 1000; Millipore, USA), pJNK (∼42 kDa, 1 : 1000; Millipore, USA), and pNF-κB (∼65 kDa, 1 : 1000; Millipore, USA) in TBS-T with 1% bovine serum albumin.

    Techniques: Expressing, Western Blot

    (a) Expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse SSC. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Journal: Evidence-based Complementary and Alternative Medicine : eCAM

    Article Title: Electroacupuncture Reduces Fibromyalgia Pain by Attenuating the HMGB1, S100B, and TRPV1 Signalling Pathways in the Mouse Brain

    doi: 10.1155/2022/2242074

    Figure Lengend Snippet: (a) Expression of extracellular neurotransmitters ((A) HMGB1, (B) S100B), receptors ((C) RAGE, (D) TLR2, (E) TLR4), and cytoplasmic inflammation signal molecules ((F) pPI3K, (G) pAkt, (H) pmTOR, (I) pERK, (J) pp38, (K) pJNK, (L) pNF- κ B) in the mouse SSC. The four lanes on the immunoblots correspond to the protein bands of, in order, control (Con), ICS-induced fibromyalgia (ICS), electroacupuncture (EA), and Trpv1 −/− groups. ∗ indicates statistical significance with P

    Article Snippet: The membrane was blocked with 5% nonfat milk in TBS-T buffer (10 mM Tris-HCl pH 7.5, 100 mM NaCl, 0.1% Tween-20) and then incubated with a primary antibody against TRPV1 (∼95 kDa, 1 : 1000; Alomone, Israel), HMGB1 (∼28 kDa, 1 : 1000; Alomone, Israel), S100B (∼10 kDa, 1 : 1000; Millipore, USA), TLR4 (∼35 kDa, 1 : 1000; Millipore, USA), RAGE (∼42 kDa, 1 : 1000; Millipore, USA), pPI3K (∼125 kDa, 1 : 1000; Millipore, USA), pAkt (∼60 kDa, 1 : 1000; Millipore, USA), pmTOR (∼60 kDa, 1 : 500; Millipore, USA), pERK1/2 (∼42–44 kDa, 1 : 1000; Millipore, USA), pp38 (∼42 kDa, 1 : 1000; Millipore, USA), pJNK (∼42 kDa, 1 : 1000; Millipore, USA), and pNF-κB (∼65 kDa, 1 : 1000; Millipore, USA) in TBS-T with 1% bovine serum albumin.

    Techniques: Expressing, Western Blot

    Comparative graph showing the hind paw withdrawal threshold and latency. Black: control group, red: ICS group, blue: ICS + electroacupuncture (EA) group, green: transient receptor vanilloid member one deletion ( Trpv1 −/− ) group. ∗ indicates statistical significance with p

    Journal: Evidence-based Complementary and Alternative Medicine : eCAM

    Article Title: Electroacupuncture Reduces Fibromyalgia Pain by Attenuating the HMGB1, S100B, and TRPV1 Signalling Pathways in the Mouse Brain

    doi: 10.1155/2022/2242074

    Figure Lengend Snippet: Comparative graph showing the hind paw withdrawal threshold and latency. Black: control group, red: ICS group, blue: ICS + electroacupuncture (EA) group, green: transient receptor vanilloid member one deletion ( Trpv1 −/− ) group. ∗ indicates statistical significance with p

    Article Snippet: The membrane was blocked with 5% nonfat milk in TBS-T buffer (10 mM Tris-HCl pH 7.5, 100 mM NaCl, 0.1% Tween-20) and then incubated with a primary antibody against TRPV1 (∼95 kDa, 1 : 1000; Alomone, Israel), HMGB1 (∼28 kDa, 1 : 1000; Alomone, Israel), S100B (∼10 kDa, 1 : 1000; Millipore, USA), TLR4 (∼35 kDa, 1 : 1000; Millipore, USA), RAGE (∼42 kDa, 1 : 1000; Millipore, USA), pPI3K (∼125 kDa, 1 : 1000; Millipore, USA), pAkt (∼60 kDa, 1 : 1000; Millipore, USA), pmTOR (∼60 kDa, 1 : 500; Millipore, USA), pERK1/2 (∼42–44 kDa, 1 : 1000; Millipore, USA), pp38 (∼42 kDa, 1 : 1000; Millipore, USA), pJNK (∼42 kDa, 1 : 1000; Millipore, USA), and pNF-κB (∼65 kDa, 1 : 1000; Millipore, USA) in TBS-T with 1% bovine serum albumin.

    Techniques:

    Schematic illustration of neuronal and non-neuronal mechanisms underlying EA-mediated analgesic effect on ICS-induced fibromyalgia pain. The summary diagram shows the importance of and mechanisms involving glial cells (astrocytes and microglia) and TRPV1 in fibromyalgia pain. EA inhibits HMGB1 and S100B release from non-neuronal cells or directly inhibits TRPV1 on the plasma membrane. Mice with a TRPV1 gene deletion ( Trpv1 −/− ) have the same phenotype than mice treated with EA.

    Journal: Evidence-based Complementary and Alternative Medicine : eCAM

    Article Title: Electroacupuncture Reduces Fibromyalgia Pain by Attenuating the HMGB1, S100B, and TRPV1 Signalling Pathways in the Mouse Brain

    doi: 10.1155/2022/2242074

    Figure Lengend Snippet: Schematic illustration of neuronal and non-neuronal mechanisms underlying EA-mediated analgesic effect on ICS-induced fibromyalgia pain. The summary diagram shows the importance of and mechanisms involving glial cells (astrocytes and microglia) and TRPV1 in fibromyalgia pain. EA inhibits HMGB1 and S100B release from non-neuronal cells or directly inhibits TRPV1 on the plasma membrane. Mice with a TRPV1 gene deletion ( Trpv1 −/− ) have the same phenotype than mice treated with EA.

    Article Snippet: The membrane was blocked with 5% nonfat milk in TBS-T buffer (10 mM Tris-HCl pH 7.5, 100 mM NaCl, 0.1% Tween-20) and then incubated with a primary antibody against TRPV1 (∼95 kDa, 1 : 1000; Alomone, Israel), HMGB1 (∼28 kDa, 1 : 1000; Alomone, Israel), S100B (∼10 kDa, 1 : 1000; Millipore, USA), TLR4 (∼35 kDa, 1 : 1000; Millipore, USA), RAGE (∼42 kDa, 1 : 1000; Millipore, USA), pPI3K (∼125 kDa, 1 : 1000; Millipore, USA), pAkt (∼60 kDa, 1 : 1000; Millipore, USA), pmTOR (∼60 kDa, 1 : 500; Millipore, USA), pERK1/2 (∼42–44 kDa, 1 : 1000; Millipore, USA), pp38 (∼42 kDa, 1 : 1000; Millipore, USA), pJNK (∼42 kDa, 1 : 1000; Millipore, USA), and pNF-κB (∼65 kDa, 1 : 1000; Millipore, USA) in TBS-T with 1% bovine serum albumin.

    Techniques: Mouse Assay