icilin  (Alomone Labs)


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    Structured Review

    Alomone Labs icilin
    Icilin, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/icilin/product/Alomone Labs
    Average 92 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    icilin - by Bioz Stars, 2022-12
    92/100 stars

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  • 95
    Alomone Labs p akt
    The <t>AKT</t> agonist <t>740Y-P</t> inhibited the effect of sFlt1. The PWMT of the 740 Y-P- and sFlt1-treated mice at different time points ( n = 6). The paw withdrawal threshold was significantly reduced on 3rd to 14th day after SNI (red curve) when compared to sham group, sFlt1 ameliorate the reduced PWMT from 5th to 14th day (green curve); 740Y-P reversed the sFlt1 improved PWMT (blue curve).The data are shown as the mean ± SEM ( n = 3). The results were analyzed by One-way analysis of variance ANOVA and by multiple comparisons, 740 Y-P versus sFlt1. ⁎ p
    P Akt, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    93
    Alomone Labs penitrem a
    The identification of K + channels in setting HCASMC resting membrane potential. (A–F) The change in the fractional fluorescence of DiBAC 4 (3) treated with 10 µM glibenclamide (A), 200 nM <t>penitrem</t> A (B), 1 µM tram34 (C), 200 nM apamin (D), 25 µM BaCl 2 (E), and 10 µM XE991 (F). (G) A summary of percentchange in fractional fluorescence of DiBAC 4 (3) after application of K + channel inhibitors: glibenclamide ( p
    Penitrem A, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Alomone Labs p q channel
    The identification of K + channels in setting HCASMC resting membrane potential. (A–F) The change in the fractional fluorescence of DiBAC 4 (3) treated with 10 µM glibenclamide (A), 200 nM <t>penitrem</t> A (B), 1 µM tram34 (C), 200 nM apamin (D), 25 µM BaCl 2 (E), and 10 µM XE991 (F). (G) A summary of percentchange in fractional fluorescence of DiBAC 4 (3) after application of K + channel inhibitors: glibenclamide ( p
    P Q Channel, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    94
    Alomone Labs rabbit anti cav 2 1 antibody
    The identification of K + channels in setting HCASMC resting membrane potential. (A–F) The change in the fractional fluorescence of DiBAC 4 (3) treated with 10 µM glibenclamide (A), 200 nM <t>penitrem</t> A (B), 1 µM tram34 (C), 200 nM apamin (D), 25 µM BaCl 2 (E), and 10 µM XE991 (F). (G) A summary of percentchange in fractional fluorescence of DiBAC 4 (3) after application of K + channel inhibitors: glibenclamide ( p
    Rabbit Anti Cav 2 1 Antibody, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    The AKT agonist 740Y-P inhibited the effect of sFlt1. The PWMT of the 740 Y-P- and sFlt1-treated mice at different time points ( n = 6). The paw withdrawal threshold was significantly reduced on 3rd to 14th day after SNI (red curve) when compared to sham group, sFlt1 ameliorate the reduced PWMT from 5th to 14th day (green curve); 740Y-P reversed the sFlt1 improved PWMT (blue curve).The data are shown as the mean ± SEM ( n = 3). The results were analyzed by One-way analysis of variance ANOVA and by multiple comparisons, 740 Y-P versus sFlt1. ⁎ p

    Journal: Molecular Pain

    Article Title: Targeting Vascular endothelial growth factor A with soluble vascular endothelial growth factor receptor 1 ameliorates nerve injury-induced neuropathic pain

    doi: 10.1177/17448069221094528

    Figure Lengend Snippet: The AKT agonist 740Y-P inhibited the effect of sFlt1. The PWMT of the 740 Y-P- and sFlt1-treated mice at different time points ( n = 6). The paw withdrawal threshold was significantly reduced on 3rd to 14th day after SNI (red curve) when compared to sham group, sFlt1 ameliorate the reduced PWMT from 5th to 14th day (green curve); 740Y-P reversed the sFlt1 improved PWMT (blue curve).The data are shown as the mean ± SEM ( n = 3). The results were analyzed by One-way analysis of variance ANOVA and by multiple comparisons, 740 Y-P versus sFlt1. ⁎ p

    Article Snippet: Multiple-immunofluorescence (IF) staining was conducted according to the standard protocol., In brief, sections were blocked with commercial blocking buffer and 0.3% Triton-100 for 60 min at room temperature, then incubated with the following primary antibodies overnight at 4°C: VEGFA (1:100, rabbit; Proteintech), TRPV1 (1:100, rabbit; Alomone Labs), and p-AKT (1:200, rabbit; Affinity).

    Techniques: Mouse Assay

    Immunofluorescence analyses the expression of VEGFA, p-AKT, and TRPV1 in the spinal cord. (a–d). VEGFA (green), p-AKT (red), and TRPV1(red) are mainly expressed in the superficial dorsal horn region (laminae I-II) of the spinal cord. The expression of VEGFA, p-AKT, and TRPV1 was robustly increased in SNI mice, but sFlt1 inhibited this increase (green, b and d). The average optical densities (AODs) in the images were quantified by ImageJ. The data are shown as the mean ± SEM ( n = 3). The results were analyzed by One-way ANOVA and by multiple comparisons. ⁎ p

    Journal: Molecular Pain

    Article Title: Targeting Vascular endothelial growth factor A with soluble vascular endothelial growth factor receptor 1 ameliorates nerve injury-induced neuropathic pain

    doi: 10.1177/17448069221094528

    Figure Lengend Snippet: Immunofluorescence analyses the expression of VEGFA, p-AKT, and TRPV1 in the spinal cord. (a–d). VEGFA (green), p-AKT (red), and TRPV1(red) are mainly expressed in the superficial dorsal horn region (laminae I-II) of the spinal cord. The expression of VEGFA, p-AKT, and TRPV1 was robustly increased in SNI mice, but sFlt1 inhibited this increase (green, b and d). The average optical densities (AODs) in the images were quantified by ImageJ. The data are shown as the mean ± SEM ( n = 3). The results were analyzed by One-way ANOVA and by multiple comparisons. ⁎ p

    Article Snippet: Multiple-immunofluorescence (IF) staining was conducted according to the standard protocol., In brief, sections were blocked with commercial blocking buffer and 0.3% Triton-100 for 60 min at room temperature, then incubated with the following primary antibodies overnight at 4°C: VEGFA (1:100, rabbit; Proteintech), TRPV1 (1:100, rabbit; Alomone Labs), and p-AKT (1:200, rabbit; Affinity).

    Techniques: Immunofluorescence, Expressing, Mouse Assay

    Differential spinal cord VEGFA pathway expression in each group as determined by Western blot. (a–d) Molecular expression and quantification in each group. The expression of VEGFR1 (a, b) and AKT (c, d) were not different in each group, while that of p-p38 was increased after SNI surgery compared with the SHAM group, but did not different from that in the sFlt1+SNI and in rpVEGFA+sFlt1+ SNI group (c, d). The increased expression of VEGFA, VEGFR2, p-AKT, and TRPV1 was ameliorated by sFIt1 treatment (green bar, B and D), and rpVEGFA inhibited the sFIt1-induced downregulation (blue bar, B and D). The data are shown as the mean ± SEM ( n = 3). The results were analyzed by One-way analysis of variance ANOVA and by multiple comparisons. ⁎ p

    Journal: Molecular Pain

    Article Title: Targeting Vascular endothelial growth factor A with soluble vascular endothelial growth factor receptor 1 ameliorates nerve injury-induced neuropathic pain

    doi: 10.1177/17448069221094528

    Figure Lengend Snippet: Differential spinal cord VEGFA pathway expression in each group as determined by Western blot. (a–d) Molecular expression and quantification in each group. The expression of VEGFR1 (a, b) and AKT (c, d) were not different in each group, while that of p-p38 was increased after SNI surgery compared with the SHAM group, but did not different from that in the sFlt1+SNI and in rpVEGFA+sFlt1+ SNI group (c, d). The increased expression of VEGFA, VEGFR2, p-AKT, and TRPV1 was ameliorated by sFIt1 treatment (green bar, B and D), and rpVEGFA inhibited the sFIt1-induced downregulation (blue bar, B and D). The data are shown as the mean ± SEM ( n = 3). The results were analyzed by One-way analysis of variance ANOVA and by multiple comparisons. ⁎ p

    Article Snippet: Multiple-immunofluorescence (IF) staining was conducted according to the standard protocol., In brief, sections were blocked with commercial blocking buffer and 0.3% Triton-100 for 60 min at room temperature, then incubated with the following primary antibodies overnight at 4°C: VEGFA (1:100, rabbit; Proteintech), TRPV1 (1:100, rabbit; Alomone Labs), and p-AKT (1:200, rabbit; Affinity).

    Techniques: Expressing, Western Blot

    The identification of K + channels in setting HCASMC resting membrane potential. (A–F) The change in the fractional fluorescence of DiBAC 4 (3) treated with 10 µM glibenclamide (A), 200 nM penitrem A (B), 1 µM tram34 (C), 200 nM apamin (D), 25 µM BaCl 2 (E), and 10 µM XE991 (F). (G) A summary of percentchange in fractional fluorescence of DiBAC 4 (3) after application of K + channel inhibitors: glibenclamide ( p

    Journal: PeerJ

    Article Title: Hypoxia and metabolic inhibitors alter the intracellular ATP:ADP ratio and membrane potential in human coronary artery smooth muscle cells

    doi: 10.7717/peerj.10344

    Figure Lengend Snippet: The identification of K + channels in setting HCASMC resting membrane potential. (A–F) The change in the fractional fluorescence of DiBAC 4 (3) treated with 10 µM glibenclamide (A), 200 nM penitrem A (B), 1 µM tram34 (C), 200 nM apamin (D), 25 µM BaCl 2 (E), and 10 µM XE991 (F). (G) A summary of percentchange in fractional fluorescence of DiBAC 4 (3) after application of K + channel inhibitors: glibenclamide ( p

    Article Snippet: However, exposure to hypoxia resulted in significant decrease in DiBAC4 (3) fluorescence when in the presence of 10 µM pinacidil (n = 5), 10 µM glibenclamide (n = 36), 25 µM BaCl2 (n = 14), 100 nM IbTX (n = 17), 200 nM penitrem A (n = 14), and 10 µM glibenclamide plus 25 µM BaCl2 (n = 25) ( ).

    Techniques: Fluorescence