α conotoxin imi  (Alomone Labs)


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    Alomone Labs α conotoxin imi
    Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist <t>α‐conotoxin</t> ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P
    α Conotoxin Imi, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/α conotoxin imi/product/Alomone Labs
    Average 92 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    α conotoxin imi - by Bioz Stars, 2022-07
    92/100 stars

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    1) Product Images from "Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors. Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors"

    Article Title: Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors. Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors

    Journal: British Journal of Pharmacology

    doi: 10.1111/bph.15270

    Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist α‐conotoxin ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P
    Figure Legend Snippet: Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist α‐conotoxin ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P

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    Alomone Labs α conotoxin imi
    Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist <t>α‐conotoxin</t> ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P
    α Conotoxin Imi, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/α conotoxin imi/product/Alomone Labs
    Average 92 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    α conotoxin imi - by Bioz Stars, 2022-07
    92/100 stars
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    Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist α‐conotoxin ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P

    Journal: British Journal of Pharmacology

    Article Title: Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors. Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors

    doi: 10.1111/bph.15270

    Figure Lengend Snippet: Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist α‐conotoxin ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P

    Article Snippet: The α‐conotoxin ImI was ordered from Alomone Labs (Jerusalem, Israel).

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