α conotoxin imi  (Alomone Labs)


Bioz Verified Symbol Alomone Labs is a verified supplier
Bioz Manufacturer Symbol Alomone Labs manufactures this product  
  • Logo
  • About
  • News
  • Press Release
  • Team
  • Advisors
  • Partners
  • Contact
  • Bioz Stars
  • Bioz vStars
    • 93
    Name:
    alpha Conotoxin ImI
    Description:
    A Potent Antagonist of Mammalian α3 β2 Neuronal nAChR
    Catalog Number:
    C-290
    Price:
    98.0
    Category:
    Toxin
    Source:
    Synthetic peptide
    Applications:
    0
    Purity:
    >99% (HPLC)
    Size:
    0 1 mg
    Format:
    Lyophilized powder.
    Formula:
    C52H78N20O15S4
    Molecular Weight:
    1351 Da.
    Molecule Name:
    alpha-Conotoxin ImI, alpha-CTx ImI
    		Buy from Supplier

    Structured Review

    Alomone Labs α conotoxin imi
    alpha Conotoxin ImI
    A Potent Antagonist of Mammalian α3 β2 Neuronal nAChR
    https://www.bioz.com/result/α conotoxin imi/product/Alomone Labs
    Average 93 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    α conotoxin imi - by Bioz Stars, 2021-09
    93/100 stars

    Images

    1) Product Images from "Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors. Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors"

    Article Title: Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors. Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors

    Journal: British Journal of Pharmacology

    doi: 10.1111/bph.15270

    Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist α‐conotoxin ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P
    Figure Legend Snippet: Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist α‐conotoxin ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P

    Techniques Used:

    Related Articles

    other:

    Article Title: Nicotinic Acetylcholine Receptors Containing the α7-Like Subunit Mediate Contractions of Muscles Responsible for Space Positioning of the Snail, Helix pomatia L. Tentacle
    Article Snippet: The α-bungarotoxin (αBgTx), α-conotoxin ImI (α-CTx IMI) and αA-conotoxin PIVA (αA-CTx PIVA) were purchased from Alomone Labs (Jerusalem, Israel).

    Article Title: Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors. Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors
    Article Snippet: The α‐conotoxin ImI was ordered from Alomone Labs (Jerusalem, Israel).

    Analytical Sample Preparation:

    Article Title: Epimerization-free access to C-terminal cysteine peptide acids, carboxamides, secondary amides, and esters via complimentary strategies complimentary strategies †Electronic supplementary information (ESI) available. See DOI: 10.1039/c7sc03553e
    Article Snippet: .. We thank Alomone Labs for providing an analytical sample of α-conotoxin ImI free of charge. ..

    Similar Products

  • Logo
  • About
  • News
  • Press Release
  • Team
  • Advisors
  • Partners
  • Contact
  • Bioz Stars
  • Bioz vStars
    • α conotoxin imi  (Alomone Labs)
    93
    Alomone Labs α conotoxin imi
    Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist <t>α‐conotoxin</t> ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P
    α Conotoxin Imi, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/α conotoxin imi/product/Alomone Labs
    Average 93 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    α conotoxin imi - by Bioz Stars, 2021-09
    93/100 stars
    		  Buy from Supplier

    Image Search Results


    Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist α‐conotoxin ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P

    Journal: British Journal of Pharmacology

    Article Title: Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors. Nicotine stimulates ion transport via metabotropic β4 subunit containing nicotinic ACh receptors

    doi: 10.1111/bph.15270

    Figure Lengend Snippet: Effect of nicotinic receptor agonists and antagonists on transepithelial ion transport of mouse tracheal epithelium. (a) The α7 nicotinic ACh receptor (nAChR) antagonist α‐bungarotoxin (αBTX, 100 nmol·L −1 , n = 5, apical) or the α7 nAChR antagonist α‐conotoxin ImI (C. ImI, 4 μmol·L −1 , apical) or the α9α10 nAChR antagonist ACV‐1 (100 nmol·L −1 , apical) did not influence the nicotine effect (100 μmol·L −1 , apical, ΔI SC ; ns, not significant). (b) The epibatidine‐induced (α4β2, α3β2, α4β4 and α3β4 nAChR agonist) current peak (1 μmol·L −1 ) was significant compared to baseline current ( n = 5, * P

    Article Snippet: The α‐conotoxin ImI was ordered from Alomone Labs (Jerusalem, Israel).

    Techniques: