5 hydroxy methyl furfural  (Millipore)


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  • 96
    Name:
    Furfural
    Description:

    Catalog Number:
    319910
    Price:
    None
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    Structured Review

    Millipore 5 hydroxy methyl furfural
    Furfural

    https://www.bioz.com/result/5 hydroxy methyl furfural/product/Millipore
    Average 96 stars, based on 2 article reviews
    Price from $9.99 to $1999.99
    5 hydroxy methyl furfural - by Bioz Stars, 2020-09
    96/100 stars

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    Related Articles

    Modification:

    Article Title: Conversion and assimilation of furfural and 5-(hydroxymethyl)furfural by Pseudomonas putida KT2440
    Article Snippet: .. Modified M9 medium was supplemented with 1g /L HMF (Sigma Aldrich, W501808), 1g /L furfural (Sigma Aldrich, 185914) or 1g /L each of HMF and furfural. .. The engineered strain was adapted to M9 medium supplemented with 1g /L HMF and 1g /L furfural via 10 rounds of serial dilution (1:10) and outgrowth (approximately 30 generations), prior to sample harvest and analysis (described below).

    Activation Assay:

    Article Title: Role of Support Oxygen Vacancies in the Gas Phase Hydrogenation of Furfural over Gold
    Article Snippet: .. Catalyst Testing Hydrogenation of furfural (Sigma-Aldrich, 99%) was carried out at atmospheric pressure and 413–523 K in situ after activation in a continuous flow fixed bed tubular reactor (15 mm i.d.). ..

    Flow Cytometry:

    Article Title: Role of Support Oxygen Vacancies in the Gas Phase Hydrogenation of Furfural over Gold
    Article Snippet: .. Catalyst Testing Hydrogenation of furfural (Sigma-Aldrich, 99%) was carried out at atmospheric pressure and 413–523 K in situ after activation in a continuous flow fixed bed tubular reactor (15 mm i.d.). ..

    other:

    Article Title: Efficient Electrocatalytic Reduction of Furfural to Furfuryl Alcohol in a Microchannel Flow Reactor
    Article Snippet: 1 General Information For the chemicals, furfural (99%), furfuryl alcohol (98%), 3-furancarboxaldehyde (97%), furan-3-methanol (99%), potassium hydroxide pellets (85%), potassium ethoxide (95%), sodium hydroxide pellets (98%), and tetrabutylammonium bromide (99%), were purchased from Sigma-Aldrich and used as received.

    Article Title: Inhibitory Effects of a Variety of Aldehydes on Amaranthus tricolor L. and Echinochloa crus-galli (L.) Beauv.
    Article Snippet: Chemicals Tween® 80, acetaldehyde ( 1 ), propionaldehyde ( 2 ), butyraldehyde ( 3 ), (E )-crotonaldehyde ( 5 ), (E )-cinnamaldehyde ( 7 ), o -tolualdehyde ( 8 ), cuminaldehyde ( 9 ), 2-(trifluoromethyl)benzaldehyde ( 10 ), 2-fluorobenzaldehyde ( 11 ), 2,4-dichlorobenzaldehyde ( 12 ), 3-bromobenzaldehyde ( 13 ), p -anisaldehyde ( 14 ), m -anisaldehyde ( 15 ), 2,4,6-trimethoxybenzaldehyde ( 16 ), vanillin ( 17 ), 3-nitrobenzaldehyde ( 20 ), 2-formylbenzoic acid ( 21 ), 4-formylbenzoic acid ( 22 ), 2-(2-formylphenoxy) acetic acid ( 23 ), 4-(dimethylamino)benzaldehyde ( 24 ), 4-((2-hydroxyethyl)(methyl)amino) benzaldehyde ( 25 ), 4-(bis (2-hydroxyethyl)amino)benzaldehyde ( 26 ), furfural ( 28 ), 1H -pyrrole- 2-carbaldehyde ( 29 ), thiophene-2-carbaldehyde ( 30 ), 1-methyl-1H -pyrrole-2-carbaldehyde ( 31 ), picolinaldehyde ( 32 ), 2-bromonicotinaldehyde ( 33 ), oxazole-4-carbaldehyde ( 34 ), thiazole-2-carbaldehyde ( 35 ), 1H -indole-3-carbaldehyde ( 36 ) and 1H -pyrrolo[2,3-b ]pyridine-2-carbaldehyde ( 37 ) were purchased from Sigma-Aldrich (Singapore).

    In Situ:

    Article Title: Role of Support Oxygen Vacancies in the Gas Phase Hydrogenation of Furfural over Gold
    Article Snippet: .. Catalyst Testing Hydrogenation of furfural (Sigma-Aldrich, 99%) was carried out at atmospheric pressure and 413–523 K in situ after activation in a continuous flow fixed bed tubular reactor (15 mm i.d.). ..

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  • 94
    Millipore carboxyatractyloside
    Mitochondrial ATP uptake and adenine nucleotide content. (A) Uptake of ATP by mitochondria isolated from YPL-grown cells with disruption or overexpression of SAL1 , in the presence or absence of Ca 2+ and <t>carboxyatractyloside</t> (CATR). The values represent
    Carboxyatractyloside, supplied by Millipore, used in various techniques. Bioz Stars score: 94/100, based on 11 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/carboxyatractyloside/product/Millipore
    Average 94 stars, based on 11 article reviews
    Price from $9.99 to $1999.99
    carboxyatractyloside - by Bioz Stars, 2020-09
    94/100 stars
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    90
    Millipore shikonin
    PKM2-dependent glycolysis is required for inflammasome activation. ( a ) LPS (500 ng ml − 1 , 3 h)-primed BMDMs and PMs were treated with inflammasome activators (ATP (5 mM, 30 min) or poly(dA:dT) (1 μg ml − 1 , 8 h)) in the absence or presence of <t>shikonin</t> (5 μM) or 2DG (2 mM). PEP and lactate levels were assayed using a commercial kit ( n =3, * P
    Shikonin, supplied by Millipore, used in various techniques. Bioz Stars score: 90/100, based on 26 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/shikonin/product/Millipore
    Average 90 stars, based on 26 article reviews
    Price from $9.99 to $1999.99
    shikonin - by Bioz Stars, 2020-09
    90/100 stars
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    99
    Millipore trolox
    FTY720 and TRAIL-mediated apoptosis is independent of ROS signaling in Caki cells (A) Caki cells were treated with 15 μM FTY720 for the indicated time periods and loaded with H 2 DCF-DA fluorescent dye. The H 2 DCF-DA fluorescence intensity was detected by flow cytometry. (B) Caki cells were pretreated with 200 μM <t>trolox</t> (Trol), 5 mM <t>NAC,</t> and 2 mM GEE for 30 min and then stimulated with 15 μM FTY720 for 24 h. The protein expression levels of DR5, Mcl-1 and actin were determined by western blotting. The level of actin was used as a loading control. (C) Caki cells were pretreated with 200 μM trolox (Trol), 5 mM NAC, and 2 mM GEE for 30 min, and then stimulated with 15 μM FTY720 plus 50 ng/ml TRAIL for 24 h. Apoptosis was analyzed in the sub-G1 population by FACS analysis. The protein expression levels of PARP and actin were determined by western blotting. The level of actin was used as the loading control. The values in (C) represent the mean ± SD from three independent samples.
    Trolox, supplied by Millipore, used in various techniques. Bioz Stars score: 99/100, based on 235 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/trolox/product/Millipore
    Average 99 stars, based on 235 article reviews
    Price from $9.99 to $1999.99
    trolox - by Bioz Stars, 2020-09
    99/100 stars
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    Image Search Results


    Mitochondrial ATP uptake and adenine nucleotide content. (A) Uptake of ATP by mitochondria isolated from YPL-grown cells with disruption or overexpression of SAL1 , in the presence or absence of Ca 2+ and carboxyatractyloside (CATR). The values represent

    Journal: Molecular genetics and genomics : MGG

    Article Title: Pleiotropic effects of the yeast Sal1 and Aac2 carriers on mitochondrial function via an activity distinct from adenine nucleotide transport

    doi: 10.1007/s00438-008-0342-5

    Figure Lengend Snippet: Mitochondrial ATP uptake and adenine nucleotide content. (A) Uptake of ATP by mitochondria isolated from YPL-grown cells with disruption or overexpression of SAL1 , in the presence or absence of Ca 2+ and carboxyatractyloside (CATR). The values represent

    Article Snippet: Mitochondria (0.4 mg of proteins) were added to 0.5 ml of incubation mixture containing 0.6 M mannitol, 2 mM K2 PO4 /KH2 PO4 , pH 6.8, 10 mM Tris-maleate, pH 6.8, 5 mM MgCl2 , 10 mM KCl and 0.1% ethanol, with or without 5 μM carboxyatractyloside (Calbiochem) and CaCl2 .

    Techniques: Isolation, Over Expression

    PKM2-dependent glycolysis is required for inflammasome activation. ( a ) LPS (500 ng ml − 1 , 3 h)-primed BMDMs and PMs were treated with inflammasome activators (ATP (5 mM, 30 min) or poly(dA:dT) (1 μg ml − 1 , 8 h)) in the absence or presence of shikonin (5 μM) or 2DG (2 mM). PEP and lactate levels were assayed using a commercial kit ( n =3, * P

    Journal: Nature Communications

    Article Title: PKM2-dependent glycolysis promotes NLRP3 and AIM2 inflammasome activation

    doi: 10.1038/ncomms13280

    Figure Lengend Snippet: PKM2-dependent glycolysis is required for inflammasome activation. ( a ) LPS (500 ng ml − 1 , 3 h)-primed BMDMs and PMs were treated with inflammasome activators (ATP (5 mM, 30 min) or poly(dA:dT) (1 μg ml − 1 , 8 h)) in the absence or presence of shikonin (5 μM) or 2DG (2 mM). PEP and lactate levels were assayed using a commercial kit ( n =3, * P

    Article Snippet: Shikonin (#CAS 517-89-5) was obtained from Millipore Corporation (Billerica, MA, USA).

    Techniques: Activation Assay

    PKM2-dependent glycolysis promotes EIF2AK2 phosphorylation. ( a ) LPS-primed BMDMs were treated with ATP (5 mM, 30 min) in the absence or presence of shikonin (1 and 5 μM). p-EIF2AK2 was assayed ( n =3, * P

    Journal: Nature Communications

    Article Title: PKM2-dependent glycolysis promotes NLRP3 and AIM2 inflammasome activation

    doi: 10.1038/ncomms13280

    Figure Lengend Snippet: PKM2-dependent glycolysis promotes EIF2AK2 phosphorylation. ( a ) LPS-primed BMDMs were treated with ATP (5 mM, 30 min) in the absence or presence of shikonin (1 and 5 μM). p-EIF2AK2 was assayed ( n =3, * P

    Article Snippet: Shikonin (#CAS 517-89-5) was obtained from Millipore Corporation (Billerica, MA, USA).

    Techniques:

    Pharmacological inhibition of the PKM2 pathway protects septic mice. ( a , b ) p-EIF2AK2, EIF2AK2 and caspase-1 activity were assayed in isolated PMs from mice during endotoxemia or polymicrobial sepsis in the absence or presence of shikonin (8 mg kg − 1 ) or C16 (50 μg kg − 1 ). In addition, the protein levels of p-EIF2AK2 and EIF2AK2 were assayed in PMs from mice with vehicle (no LPS) injection or sham operated for CLP. ( c ) Mice ( n =20 mice per group) were injected with a single dose of C16 (8 mg kg − 1 ), followed 30 min later by an infusion of endotoxin (LPS, 5 mg kg − 1 , intraperitoneally), and were then re-treated with C16 12 and 24 h later. The Kaplan–Meyer method was used to compare differences in survival rates between groups (* P

    Journal: Nature Communications

    Article Title: PKM2-dependent glycolysis promotes NLRP3 and AIM2 inflammasome activation

    doi: 10.1038/ncomms13280

    Figure Lengend Snippet: Pharmacological inhibition of the PKM2 pathway protects septic mice. ( a , b ) p-EIF2AK2, EIF2AK2 and caspase-1 activity were assayed in isolated PMs from mice during endotoxemia or polymicrobial sepsis in the absence or presence of shikonin (8 mg kg − 1 ) or C16 (50 μg kg − 1 ). In addition, the protein levels of p-EIF2AK2 and EIF2AK2 were assayed in PMs from mice with vehicle (no LPS) injection or sham operated for CLP. ( c ) Mice ( n =20 mice per group) were injected with a single dose of C16 (8 mg kg − 1 ), followed 30 min later by an infusion of endotoxin (LPS, 5 mg kg − 1 , intraperitoneally), and were then re-treated with C16 12 and 24 h later. The Kaplan–Meyer method was used to compare differences in survival rates between groups (* P

    Article Snippet: Shikonin (#CAS 517-89-5) was obtained from Millipore Corporation (Billerica, MA, USA).

    Techniques: Inhibition, Mouse Assay, Activity Assay, Isolation, Injection

    Pharmacological inhibition of PKM2 impairs inflammasome activation. LPS (500 ng ml − 1 , 3 h)-primed mouse BMDMs and human PMA-differentiated THP1 cells were respectively treated with inflammasome activators (ATP (5 mM, 30 min), poly(dA:dT) (1 μg ml − 1 , 8 h), MDP (200 ng ml − 1 , 8 h) or flagellin (200 ng ml − 1 , 8 h)) in the absence or presence of shikonin at the same time (1 and 5 μM). IL-1β, IL-18 and HMGB1 ( a , b ) in supernatants and caspase-1 activity ( c ) in whole-cell extract were assayed with ELISA or activity assay kit ( n =3, * P

    Journal: Nature Communications

    Article Title: PKM2-dependent glycolysis promotes NLRP3 and AIM2 inflammasome activation

    doi: 10.1038/ncomms13280

    Figure Lengend Snippet: Pharmacological inhibition of PKM2 impairs inflammasome activation. LPS (500 ng ml − 1 , 3 h)-primed mouse BMDMs and human PMA-differentiated THP1 cells were respectively treated with inflammasome activators (ATP (5 mM, 30 min), poly(dA:dT) (1 μg ml − 1 , 8 h), MDP (200 ng ml − 1 , 8 h) or flagellin (200 ng ml − 1 , 8 h)) in the absence or presence of shikonin at the same time (1 and 5 μM). IL-1β, IL-18 and HMGB1 ( a , b ) in supernatants and caspase-1 activity ( c ) in whole-cell extract were assayed with ELISA or activity assay kit ( n =3, * P

    Article Snippet: Shikonin (#CAS 517-89-5) was obtained from Millipore Corporation (Billerica, MA, USA).

    Techniques: Inhibition, Activation Assay, Activity Assay, Enzyme-linked Immunosorbent Assay

    FTY720 and TRAIL-mediated apoptosis is independent of ROS signaling in Caki cells (A) Caki cells were treated with 15 μM FTY720 for the indicated time periods and loaded with H 2 DCF-DA fluorescent dye. The H 2 DCF-DA fluorescence intensity was detected by flow cytometry. (B) Caki cells were pretreated with 200 μM trolox (Trol), 5 mM NAC, and 2 mM GEE for 30 min and then stimulated with 15 μM FTY720 for 24 h. The protein expression levels of DR5, Mcl-1 and actin were determined by western blotting. The level of actin was used as a loading control. (C) Caki cells were pretreated with 200 μM trolox (Trol), 5 mM NAC, and 2 mM GEE for 30 min, and then stimulated with 15 μM FTY720 plus 50 ng/ml TRAIL for 24 h. Apoptosis was analyzed in the sub-G1 population by FACS analysis. The protein expression levels of PARP and actin were determined by western blotting. The level of actin was used as the loading control. The values in (C) represent the mean ± SD from three independent samples.

    Journal: Oncotarget

    Article Title: FTY720 enhances TRAIL-mediated apoptosis by up-regulating DR5 and down-regulating Mcl-1 in cancer cells

    doi:

    Figure Lengend Snippet: FTY720 and TRAIL-mediated apoptosis is independent of ROS signaling in Caki cells (A) Caki cells were treated with 15 μM FTY720 for the indicated time periods and loaded with H 2 DCF-DA fluorescent dye. The H 2 DCF-DA fluorescence intensity was detected by flow cytometry. (B) Caki cells were pretreated with 200 μM trolox (Trol), 5 mM NAC, and 2 mM GEE for 30 min and then stimulated with 15 μM FTY720 for 24 h. The protein expression levels of DR5, Mcl-1 and actin were determined by western blotting. The level of actin was used as a loading control. (C) Caki cells were pretreated with 200 μM trolox (Trol), 5 mM NAC, and 2 mM GEE for 30 min, and then stimulated with 15 μM FTY720 plus 50 ng/ml TRAIL for 24 h. Apoptosis was analyzed in the sub-G1 population by FACS analysis. The protein expression levels of PARP and actin were determined by western blotting. The level of actin was used as the loading control. The values in (C) represent the mean ± SD from three independent samples.

    Article Snippet: The recombinant human TRAIL was purchased from KOMA Biotech (Seoul, Korea), and z-VAD-fmk, sphingosine kinase inhibitor (SKI), N-acetyl-L-cysteine (NAC) and Trolox was obtained from Calbiochem (San Diego, CA, USA).

    Techniques: Fluorescence, Flow Cytometry, Cytometry, Expressing, Western Blot, FACS

    Reactive oxygen species has a critical role in carboplatin plus thioridazine-mediated PSMA5 expression. ( a ) AMC-HN4 cells were treated with 200 nM carboplatin plus 10 μ M thioridazine for 6 h (left panel) or the indicated time periods (right panel), and the cells were then loaded with the H 2 DCF-DA fluorescent dye. The H 2 DCF-DA fluorescence intensity was detected by a fluorescence microscope (left panel) and flow cytometry (right panel). ( b ) AMC-HN4 cells were treated with 200 nM carboplatin plus 10 μ M thioridazine for the indicated time periods. The protein expression levels of Prx-SO3 and actin were determined by western blotting. The level of actin was used as a loading control. ( c ) AMC-HN4 cells were pretreated with 5 mM NAC, 2 mM GEE, and 200 μ M trolox for 30 min and were then treated with 200 nM carboplatin plus 10 μ M thioridazine for 24 h. After treatment, the nuclear extracts were analyzed for Nrf2 and Ref-1 by western blotting as described in the Materials and Methods. Ref-1 was used as a marker of the nuclear fraction. ( d ) AMC-HN4 cells were transfected with an ARE-luciferase construct for 24 h. The cells were pretreated with 5 mM NAC, 2 mM GEE, and 200 μ M trolox for 30 min and were then treated with 200 nM carboplatin plus 10 μ M thioridazine for 24 h. After treatment, the cells were lysed and assayed for luciferase activity. ( e ) AMC-HN4 cells were transiently transfected with a plasmid harboring the luciferase gene under the control of the PSMA5/-277 promoter. After transfection, the cells were pretreated with 5 mM NAC, 2 mM GEE, and 200 μ M trolox for 30 min and were then treated with 200 nM carboplatin plus 10 μ M thioridazine for 24 h. The luciferase activity was analyzed. ( f ) AMC-HN4 cells were pretreated with 5 mM NAC, 2 mM GEE, and 200 μ M trolox for 30 min and were then treated with 200 nM carboplatin plus 10 μ M thioridazine for 24 h. The sub-G1 fraction was measured by flow cytometry. The protein expression levels of PARP, PSMA5, c-FLIP, Mcl-1, and actin were determined by western blotting. The level of actin was used as a loading control. The values in a , d , e and f represent the mean±S.D. from three independent samples. * P

    Journal: Cell Death & Disease

    Article Title: Thioridazine enhances sensitivity to carboplatin in human head and neck cancer cells through downregulation of c-FLIP and Mcl-1 expression

    doi: 10.1038/cddis.2017.8

    Figure Lengend Snippet: Reactive oxygen species has a critical role in carboplatin plus thioridazine-mediated PSMA5 expression. ( a ) AMC-HN4 cells were treated with 200 nM carboplatin plus 10 μ M thioridazine for 6 h (left panel) or the indicated time periods (right panel), and the cells were then loaded with the H 2 DCF-DA fluorescent dye. The H 2 DCF-DA fluorescence intensity was detected by a fluorescence microscope (left panel) and flow cytometry (right panel). ( b ) AMC-HN4 cells were treated with 200 nM carboplatin plus 10 μ M thioridazine for the indicated time periods. The protein expression levels of Prx-SO3 and actin were determined by western blotting. The level of actin was used as a loading control. ( c ) AMC-HN4 cells were pretreated with 5 mM NAC, 2 mM GEE, and 200 μ M trolox for 30 min and were then treated with 200 nM carboplatin plus 10 μ M thioridazine for 24 h. After treatment, the nuclear extracts were analyzed for Nrf2 and Ref-1 by western blotting as described in the Materials and Methods. Ref-1 was used as a marker of the nuclear fraction. ( d ) AMC-HN4 cells were transfected with an ARE-luciferase construct for 24 h. The cells were pretreated with 5 mM NAC, 2 mM GEE, and 200 μ M trolox for 30 min and were then treated with 200 nM carboplatin plus 10 μ M thioridazine for 24 h. After treatment, the cells were lysed and assayed for luciferase activity. ( e ) AMC-HN4 cells were transiently transfected with a plasmid harboring the luciferase gene under the control of the PSMA5/-277 promoter. After transfection, the cells were pretreated with 5 mM NAC, 2 mM GEE, and 200 μ M trolox for 30 min and were then treated with 200 nM carboplatin plus 10 μ M thioridazine for 24 h. The luciferase activity was analyzed. ( f ) AMC-HN4 cells were pretreated with 5 mM NAC, 2 mM GEE, and 200 μ M trolox for 30 min and were then treated with 200 nM carboplatin plus 10 μ M thioridazine for 24 h. The sub-G1 fraction was measured by flow cytometry. The protein expression levels of PARP, PSMA5, c-FLIP, Mcl-1, and actin were determined by western blotting. The level of actin was used as a loading control. The values in a , d , e and f represent the mean±S.D. from three independent samples. * P

    Article Snippet: NAC and Trolox were obtained from Calbiochem (San Diego, CA, USA).

    Techniques: Expressing, Fluorescence, Microscopy, Flow Cytometry, Cytometry, Western Blot, Marker, Transfection, Luciferase, Construct, Activity Assay, Plasmid Preparation