ãŽâ² actin  (Cell Signaling Technology Inc)


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    Structured Review

    Cell Signaling Technology Inc ãŽâ² actin
    CD1d deficiency protects mice from challenge with TLR ligands and pathogen infection. a ELISA of TNF, IL-6 and <t>IFN-β</t> in sera of Cd1d +/+ and Cd1d –/– mice ( n  = 5 per group) 2 h after intraperitoneal injection
    ãŽâ² Actin, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 91/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/ãŽâ² actin/product/Cell Signaling Technology Inc
    Average 91 stars, based on 2 article reviews
    Price from $9.99 to $1999.99
    ãŽâ² actin - by Bioz Stars, 2022-10
    91/100 stars

    Images

    1) Product Images from "Glycolipid iGb3 feedback amplifies innate immune responses via CD1d reverse signaling"

    Article Title: Glycolipid iGb3 feedback amplifies innate immune responses via CD1d reverse signaling

    Journal: Cell Research

    doi: 10.1038/s41422-018-0122-7

    CD1d deficiency protects mice from challenge with TLR ligands and pathogen infection. a ELISA of TNF, IL-6 and IFN-β in sera of Cd1d +/+ and Cd1d –/– mice ( n  = 5 per group) 2 h after intraperitoneal injection
    Figure Legend Snippet: CD1d deficiency protects mice from challenge with TLR ligands and pathogen infection. a ELISA of TNF, IL-6 and IFN-β in sera of Cd1d +/+ and Cd1d –/– mice ( n  = 5 per group) 2 h after intraperitoneal injection

    Techniques Used: Mouse Assay, Infection, Enzyme-linked Immunosorbent Assay, Injection

    2) Product Images from "CCR2 Signaling Selectively Regulates IFN-α: Role of β-Arrestin 2 in IFNAR1 Internalization"

    Article Title: CCR2 Signaling Selectively Regulates IFN-α: Role of β-Arrestin 2 in IFNAR1 Internalization

    Journal: Journal of immunology (Baltimore, Md. : 1950)

    doi: 10.4049/jimmunol.1800598

    β-Arrestin 2 Promotes IFNAR1 Internalization and Inhibits STAT1 Activation.
    Figure Legend Snippet: β-Arrestin 2 Promotes IFNAR1 Internalization and Inhibits STAT1 Activation.

    Techniques Used: Activation Assay

    β-Arrestin 2 is Required for CCL2-Mediated Decrease in IFN-α.
    Figure Legend Snippet: β-Arrestin 2 is Required for CCL2-Mediated Decrease in IFN-α.

    Techniques Used:

    β-Arrestin 2-Induced IFN Regulation is Not Restricted to Macrophages.
    Figure Legend Snippet: β-Arrestin 2-Induced IFN Regulation is Not Restricted to Macrophages.

    Techniques Used:

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    Cell Signaling Technology Inc ãŽâ² actin
    CD1d deficiency protects mice from challenge with TLR ligands and pathogen infection. a ELISA of TNF, IL-6 and <t>IFN-β</t> in sera of Cd1d +/+ and Cd1d –/– mice ( n  = 5 per group) 2 h after intraperitoneal injection
    ãŽâ² Actin, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/ãŽâ² actin/product/Cell Signaling Technology Inc
    Average 91 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    ãŽâ² actin - by Bioz Stars, 2022-10
    91/100 stars
      Buy from Supplier

    99
    Cell Signaling Technology Inc β actin
    Mito-TEMPO attenuates 5/6 nephrectomy-induced ER stress. Western blot analysis of CHOP, caspase-12, BiP, and GRP94 (a). Relative expression of BiP (b), GRP94 (c), CHOP (d), and caspase-12 (e) normalized to the expression of  β -actin. Data are presented as mean ± SD ( n  = 6).  ∗ P
    β Actin, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/β actin/product/Cell Signaling Technology Inc
    Average 99 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    β actin - by Bioz Stars, 2022-10
    99/100 stars
      Buy from Supplier

    Image Search Results


    CD1d deficiency protects mice from challenge with TLR ligands and pathogen infection. a ELISA of TNF, IL-6 and IFN-β in sera of Cd1d +/+ and Cd1d –/– mice ( n  = 5 per group) 2 h after intraperitoneal injection

    Journal: Cell Research

    Article Title: Glycolipid iGb3 feedback amplifies innate immune responses via CD1d reverse signaling

    doi: 10.1038/s41422-018-0122-7

    Figure Lengend Snippet: CD1d deficiency protects mice from challenge with TLR ligands and pathogen infection. a ELISA of TNF, IL-6 and IFN-β in sera of Cd1d +/+ and Cd1d –/– mice ( n  = 5 per group) 2 h after intraperitoneal injection

    Article Snippet: Antibodies to ERK phosphorylated at Thr202/Tyr204 (9106); JNK phosphorylated at Thr183/Tyr185 (9255); p38 phosphorylated at Thr180/Tyr182 (9211); NF-κB p65 phosphorylated at Ser536 (3033); IκBα phosphorylated at Ser32/36 (9246); IRF3 phosphorylated at Ser396 (4947); IKKα/β phosphorylated at Ser177/Ser181 (2697); TBK1 phosphorylated at Ser172 (5483); Pyk2 phosphorylated at Tyr402 (3291); Pyk2 (3292), IKKβ (8943), EEA1 (2411); Myc-tag (2272), hemagglutinin-tag (2367); and β-actin (3700) were obtained from Cell Signaling Technology.

    Techniques: Mouse Assay, Infection, Enzyme-linked Immunosorbent Assay, Injection

    β-Arrestin 2 Promotes IFNAR1 Internalization and Inhibits STAT1 Activation.

    Journal: Journal of immunology (Baltimore, Md. : 1950)

    Article Title: CCR2 Signaling Selectively Regulates IFN-α: Role of β-Arrestin 2 in IFNAR1 Internalization

    doi: 10.4049/jimmunol.1800598

    Figure Lengend Snippet: β-Arrestin 2 Promotes IFNAR1 Internalization and Inhibits STAT1 Activation.

    Article Snippet: Membranes were probed with antibodies to Phospho-Stat1 (Tyr701), Stat1, β-actin (Cell Signaling Technology), β-arrestin 1, β-arrestin 2, or GAPDH (Thermo Fisher Scientific).

    Techniques: Activation Assay

    β-Arrestin 2 is Required for CCL2-Mediated Decrease in IFN-α.

    Journal: Journal of immunology (Baltimore, Md. : 1950)

    Article Title: CCR2 Signaling Selectively Regulates IFN-α: Role of β-Arrestin 2 in IFNAR1 Internalization

    doi: 10.4049/jimmunol.1800598

    Figure Lengend Snippet: β-Arrestin 2 is Required for CCL2-Mediated Decrease in IFN-α.

    Article Snippet: Membranes were probed with antibodies to Phospho-Stat1 (Tyr701), Stat1, β-actin (Cell Signaling Technology), β-arrestin 1, β-arrestin 2, or GAPDH (Thermo Fisher Scientific).

    Techniques:

    β-Arrestin 2-Induced IFN Regulation is Not Restricted to Macrophages.

    Journal: Journal of immunology (Baltimore, Md. : 1950)

    Article Title: CCR2 Signaling Selectively Regulates IFN-α: Role of β-Arrestin 2 in IFNAR1 Internalization

    doi: 10.4049/jimmunol.1800598

    Figure Lengend Snippet: β-Arrestin 2-Induced IFN Regulation is Not Restricted to Macrophages.

    Article Snippet: Membranes were probed with antibodies to Phospho-Stat1 (Tyr701), Stat1, β-actin (Cell Signaling Technology), β-arrestin 1, β-arrestin 2, or GAPDH (Thermo Fisher Scientific).

    Techniques:

    Mito-TEMPO attenuates 5/6 nephrectomy-induced ER stress. Western blot analysis of CHOP, caspase-12, BiP, and GRP94 (a). Relative expression of BiP (b), GRP94 (c), CHOP (d), and caspase-12 (e) normalized to the expression of  β -actin. Data are presented as mean ± SD ( n  = 6).  ∗ P

    Journal: Oxidative Medicine and Cellular Longevity

    Article Title: Mito-TEMPO Alleviates Renal Fibrosis by Reducing Inflammation, Mitochondrial Dysfunction, and Endoplasmic Reticulum Stress

    doi: 10.1155/2018/5828120

    Figure Lengend Snippet: Mito-TEMPO attenuates 5/6 nephrectomy-induced ER stress. Western blot analysis of CHOP, caspase-12, BiP, and GRP94 (a). Relative expression of BiP (b), GRP94 (c), CHOP (d), and caspase-12 (e) normalized to the expression of β -actin. Data are presented as mean ± SD ( n = 6). ∗ P

    Article Snippet: Antibodies against CHOP, BiP, GRP94, β -actin, and HRP-conjugated secondary antibodies were purchased from Cell Signaling Technology (Beverly, MA, USA).

    Techniques: Western Blot, Expressing

    The effect of Mito-TEMPO on 5/6 nephrectomy-induced oxidative stress. The SOD2 activity (a) and MDA level (b) in isolated kidney mitochondria, serum T-SOD (c), and MDA (d) levels were measured following the manufacturer's protocol. Mitochondrial superoxide was detected by MitoSOX Red fluorogenic dye and images were taken with Nikon fluorescent microscope (e) (magnification, ×200), and the mean MitoSOX fluorescence intensity per image was calculated by Image Pro Plus software and normalized to the control group (f). Western blot analysis of Sirt3, SOD2, and AcSOD2 (g). Relative expression of AcSOD2 (h) normalized to the expression of SOD2 and Sirt3 (i) normalized to the expression of  β -actin. Data are presented as mean ± SD ( n  = 6).  ∗ P

    Journal: Oxidative Medicine and Cellular Longevity

    Article Title: Mito-TEMPO Alleviates Renal Fibrosis by Reducing Inflammation, Mitochondrial Dysfunction, and Endoplasmic Reticulum Stress

    doi: 10.1155/2018/5828120

    Figure Lengend Snippet: The effect of Mito-TEMPO on 5/6 nephrectomy-induced oxidative stress. The SOD2 activity (a) and MDA level (b) in isolated kidney mitochondria, serum T-SOD (c), and MDA (d) levels were measured following the manufacturer's protocol. Mitochondrial superoxide was detected by MitoSOX Red fluorogenic dye and images were taken with Nikon fluorescent microscope (e) (magnification, ×200), and the mean MitoSOX fluorescence intensity per image was calculated by Image Pro Plus software and normalized to the control group (f). Western blot analysis of Sirt3, SOD2, and AcSOD2 (g). Relative expression of AcSOD2 (h) normalized to the expression of SOD2 and Sirt3 (i) normalized to the expression of β -actin. Data are presented as mean ± SD ( n = 6). ∗ P

    Article Snippet: Antibodies against CHOP, BiP, GRP94, β -actin, and HRP-conjugated secondary antibodies were purchased from Cell Signaling Technology (Beverly, MA, USA).

    Techniques: Activity Assay, Multiple Displacement Amplification, Isolation, Microscopy, Fluorescence, Software, Western Blot, Expressing