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dense snp microarray data  (23andMe)

 
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    23andMe dense snp microarray data
    Dense Snp Microarray Data, supplied by 23andMe, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/snp+microarray+data/dense+snp+microarray+data/pm33592389-34-4-41
    Average 90 stars, based on 1 article reviews
    dense snp microarray data - by Bioz Stars, 2026-09
    90/100 stars

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    Microarray:

    Article Title: Investigative genetic genealogy: Current methods, knowledge and practice.
    Article Snippet: .. While the use of dense SNP microarray data had already been studied in forensic contexts [7–11], such technology became readily available to the public in 2007 through direct-to-consumer testing companies (the ‘DTCs’) with the launch of tests from deCODE Genetics and 23andMe, costing nearly $1000 [12]. .. Early tests were based on the Illumina OmniExpress microarray, but the field is now dominated by the Illumina Infinium Global Screening Array (GSA), which currently has a core set of 654,027 SNPs and the ability to add up to 50,000 custom markers.1 As the cost of testing decreased and more companies entered the market, SNP databases began to grow exponentially.



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    Summary of major studies associating L-CH with lymphoid malignancy.

    Journal: Blood Cancer Journal

    Article Title: Lymphoid clonal hematopoiesis: implications for malignancy, immunity, and treatment

    doi: 10.1038/s41408-022-00773-8

    Figure Lengend Snippet: Summary of major studies associating L-CH with lymphoid malignancy.

    Article Snippet: Niroula et al. (2021) , Detection of SNVs/indels in WES data at 56 M-CH related genes and 235 genes recurrently mutated in lymphoid malignancy (VAF > 2%). mCAs were previously detected in a prior study. CH and mCAs were excluded if they were detected less than 6 months before a hematologic malignancy diagnosis. , 420,969 individuals with SNP microarray data and 55,383 individuals with WES from the UK Biobank and Mass General Brigham Biobank , CHIP occurs at both M-CHIP/M-mCA and L-CHIP/L-mCA loci and increases with age. M-CH and L-CH loci clearly segregate on the incidence myeloid and lymphoid malignancy. M-CHIP is dominated by DNMT3A, TET2, and ASXL1 somatic mutants, while L-CHIP is spread more evenly across a large number of genes. CLL was the dominant lymphoid malignancy occurring subsequent to L-CH. , [ ] .

    Techniques: Genome Wide, Sequencing, Mutagenesis, Clone Assay, Biomarker Discovery, Microarray, DNA Sequencing, Clinical Proteomics