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microarray printer  (CapitalBio Corporation)

 
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    Structured Review

    CapitalBio Corporation microarray printer
    Microarray Printer, supplied by CapitalBio Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/smartarray+tm+-48+microarrayer/microarray/pmc08628518-184-15-19
    Average 90 stars, based on 1 article reviews
    microarray printer - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    Microarray:

    Article Title: Laboratory tests and analysis of drug resistance in non-tuberculous mycobacteria
    Article Snippet: .. Microarray technology (Capitalbio Technology Co., Ltd., Chengdu, China) was used for strain identification. .. Gene microarray microdrop technology was used to detect fluorescently labeled DNA fragments, followed by the analysis of Mycobacterium species by comparing the arrangement of specific positions on the microarray.

    Article Title: Recent Advances in Exosomal Protein Detection Via Liquid Biopsy Biosensors for Cancer Screening, Diagnosis, and Prognosis
    Article Snippet: In order to realize multiplexed sensing, Zhu et al . combined SPR imaging (SPRi) with antibody microarray to simultaneously quantify multiple exosome transmembrane proteins ( 39 ). .. As shown in , antibodies against exosomal proteins were printed on the gold surface by SmartArrayer 48 microarray printer (capitalBio, China). .. The capture of exosomes by antibody microarray affected the refractive index, changed the reflected laser intensities, and allowed the measurement of exosomal proteins by SPRi (PlexAray HT, Plexera Bioscience).

    Article Title: Hydrocortisone alleviates sepsis-induced acute kidney injury through HSF-1-mediated transcriptional suppression of XPO1.
    Article Snippet: Background: Hydrocortisone (HYD) has been suggested as a drug for anti-sepsis treatment, but its clinical efficacy remains controversial.. This study aims to evaluate the function of HYD in sepsis-induced acute kidney injury (AKI) and the molecules involved.. Methods: A mouse model of sepsis was induced by cecal ligation and puncture (CLP) and treated with HYD.

    Article Title: Next‐generation sequencing identifies rare pathogenic and novel candidate variants in a cohort of Chinese patients with syndromic or nonsyndromic hearing loss
    Article Snippet: Blood samples were obtained from the family members and genomic DNA was extracted from the whole blood, according to standard procedures, using a Qiagen DNA Blood Midi/Mini Kit (Qiagen). .. Prescreening of hotspot mutations in GJB2 (c.35delG, c.176_191del16, c.235delC, c.299‐300delAT), SLC26A4 (c.919‐2A>G, c.1174A>T, c.1226G>A, c.1229C>T, c.1707+5G>A, c.1975G>C, c.2027T>A, c.2168A>G), MT ‐ RNR1 (m.1494C>T, m.1555A>G), and GJB3 (c.538C>T) was performed in all probands by microarray (CapitalBio, China; Xiang et al., ). ..

    Article Title: MYC-mediated silencing of miR-181a-5p promotes pathogenic Th17 responses by modulating AKT3-FOXO3 signaling.
    Article Snippet: The absorbance was measured at 450 nm with wavelength correction set to 540 nm. miRNA microarray RNA samples were extracted from CD4+ T cells of naı̈ve or EAU mice using TRIzol reagent (Thermo Fisher Scientific) and subjected to miRNA array analysis by 8 3 60 K miR Microarray (Agilent Technologies, Santa Clara, CA, USA). .. The microarray was performed by CapitalBio Corporation (Beijing, China), and microarray data were analyzed using the GeneSpring GX software (Agilent Technologies). .. Firstly, the wild-type (WT) Akt3 30 UTR fragment containing the putative miR-181a-5p binding site or mutant (mut) Akt3 30 UTR fragment was amplified and cloned into pMIR-REPORT Vector (Promega, Madison, WI, USA) between the Hind III and Spe I site as we described previously (Wei et al., 2019).

    other:

    Article Title: Integration of transcriptomic data identifies key hallmark genes in hypertrophic cardiomyopathy
    Article Snippet: E-GEOD-68316 , Human heart tissue , mRNA , Microarray , CapitalBio GPL20113 , 2015 , 7/5.

    Article Title: The toxicity of perfluorodecanoic acid is mainly manifested as a deflected immune function.
    Article Snippet: Background Perfluorodecanoic acid (PFDA) is a type of perfluoroalkyl acid (PFAA).. PFDA has toxicity similar to dioxin; its effect on the body is not through a single target or a single pathway.. However, the mechanism at the global level is still unclear.

    Article Title: Upregulated long noncoding RNA ENST00000470447.1 inhibits cell migration and invasion and predicts better disease-free survival of oral cancer.
    Article Snippet: Department of Epidemiology and Health Statistics, Fujian Provincial Key Laboratory of Environment Factors and Cancer, School of Public Health, Fujian Medical University, Fujian, China Key Laboratory of Ministry of Education for Gastrointestinal Cancer, Fujian Key Laboratory of Tumor Microbiology, Fujian Medical University, Fujian, China Laboratory Center, The Major Subject of Environment and Health of Fujian Key Universities, School of Public Health, Fujian Medical University, Fujian, China Department of Oral and Maxillofacial Surgery, the First Affiliated Hospital of Fujian Medical University, Fujian, China

    Amplification:

    Article Title: Hydrocortisone alleviates sepsis-induced acute kidney injury through HSF-1-mediated transcriptional suppression of XPO1.
    Article Snippet: Background: Hydrocortisone (HYD) has been suggested as a drug for anti-sepsis treatment, but its clinical efficacy remains controversial.. This study aims to evaluate the function of HYD in sepsis-induced acute kidney injury (AKI) and the molecules involved.. Methods: A mouse model of sepsis was induced by cecal ligation and puncture (CLP) and treated with HYD.

    Labeling:

    Article Title: Hydrocortisone alleviates sepsis-induced acute kidney injury through HSF-1-mediated transcriptional suppression of XPO1.
    Article Snippet: Background: Hydrocortisone (HYD) has been suggested as a drug for anti-sepsis treatment, but its clinical efficacy remains controversial.. This study aims to evaluate the function of HYD in sepsis-induced acute kidney injury (AKI) and the molecules involved.. Methods: A mouse model of sepsis was induced by cecal ligation and puncture (CLP) and treated with HYD.

    Hybridization:

    Article Title: Hydrocortisone alleviates sepsis-induced acute kidney injury through HSF-1-mediated transcriptional suppression of XPO1.
    Article Snippet: Background: Hydrocortisone (HYD) has been suggested as a drug for anti-sepsis treatment, but its clinical efficacy remains controversial.. This study aims to evaluate the function of HYD in sepsis-induced acute kidney injury (AKI) and the molecules involved.. Methods: A mouse model of sepsis was induced by cecal ligation and puncture (CLP) and treated with HYD.

    Imaging:

    Article Title: Hydrocortisone alleviates sepsis-induced acute kidney injury through HSF-1-mediated transcriptional suppression of XPO1.
    Article Snippet: Background: Hydrocortisone (HYD) has been suggested as a drug for anti-sepsis treatment, but its clinical efficacy remains controversial.. This study aims to evaluate the function of HYD in sepsis-induced acute kidney injury (AKI) and the molecules involved.. Methods: A mouse model of sepsis was induced by cecal ligation and puncture (CLP) and treated with HYD.

    Software:

    Article Title: MYC-mediated silencing of miR-181a-5p promotes pathogenic Th17 responses by modulating AKT3-FOXO3 signaling.
    Article Snippet: The absorbance was measured at 450 nm with wavelength correction set to 540 nm. miRNA microarray RNA samples were extracted from CD4+ T cells of naı̈ve or EAU mice using TRIzol reagent (Thermo Fisher Scientific) and subjected to miRNA array analysis by 8 3 60 K miR Microarray (Agilent Technologies, Santa Clara, CA, USA). .. The microarray was performed by CapitalBio Corporation (Beijing, China), and microarray data were analyzed using the GeneSpring GX software (Agilent Technologies). .. Firstly, the wild-type (WT) Akt3 30 UTR fragment containing the putative miR-181a-5p binding site or mutant (mut) Akt3 30 UTR fragment was amplified and cloned into pMIR-REPORT Vector (Promega, Madison, WI, USA) between the Hind III and Spe I site as we described previously (Wei et al., 2019).



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