gene expression profiling rna extraction, microarray hybridization and analysis (Arraystar inc)
90
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Arraystar inc
gene expression profiling rna extraction, microarray hybridization and analysis
Gene Expression Profiling Rna Extraction, Microarray Hybridization And Analysis, supplied by Arraystar inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rna+microarray+analysis/gene+expression+profiling+rna+extraction++microarray+hybridization+and+analysis/pm27835742-72-5-16
Average 90 stars, based on 1 article reviews
Gene Expression Profiling Rna Extraction, Microarray Hybridization And Analysis, supplied by Arraystar inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rna+microarray+analysis/gene+expression+profiling+rna+extraction++microarray+hybridization+and+analysis/pm27835742-72-5-16
Average 90 stars, based on 1 article reviews
gene expression profiling rna extraction, microarray hybridization and analysis - by Bioz Stars,
2026-09
90/100 stars
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Gene Expression:Article Title: Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis. Article Snippet: Accepted Manuscript Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis Miguel Hueso, Laura De Ramon, Estanislao Navarro, Elia Ripoll, Josep M. Cruzado, Josep M. Grinyo, Joan Torras PII: S0021-9150(16)31470-8 DOI: 10.1016/j.atherosclerosis.2016.11.002 Reference: ATH 14860 To appear in: Atherosclerosis Received Date: 24 July 2016 Revised Date: 6 October 2016 Accepted Date: 1 November 2016 Please cite this article as: Hueso M, De Ramon L, Navarro E, Ripoll E, Cruzado JM, Grinyo JM, Torras J, Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis, Atherosclerosis (2016), doi: 10.1016/j.atherosclerosis.2016.11.002.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. RNA Extraction:Article Title: Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis. Article Snippet: Accepted Manuscript Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis Miguel Hueso, Laura De Ramon, Estanislao Navarro, Elia Ripoll, Josep M. Cruzado, Josep M. Grinyo, Joan Torras PII: S0021-9150(16)31470-8 DOI: 10.1016/j.atherosclerosis.2016.11.002 Reference: ATH 14860 To appear in: Atherosclerosis Received Date: 24 July 2016 Revised Date: 6 October 2016 Accepted Date: 1 November 2016 Please cite this article as: Hueso M, De Ramon L, Navarro E, Ripoll E, Cruzado JM, Grinyo JM, Torras J, Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis, Atherosclerosis (2016), doi: 10.1016/j.atherosclerosis.2016.11.002.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. Microarray:Article Title: Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis. Article Snippet: Accepted Manuscript Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis Miguel Hueso, Laura De Ramon, Estanislao Navarro, Elia Ripoll, Josep M. Cruzado, Josep M. Grinyo, Joan Torras PII: S0021-9150(16)31470-8 DOI: 10.1016/j.atherosclerosis.2016.11.002 Reference: ATH 14860 To appear in: Atherosclerosis Received Date: 24 July 2016 Revised Date: 6 October 2016 Accepted Date: 1 November 2016 Please cite this article as: Hueso M, De Ramon L, Navarro E, Ripoll E, Cruzado JM, Grinyo JM, Torras J, Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis, Atherosclerosis (2016), doi: 10.1016/j.atherosclerosis.2016.11.002.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. Hybridization:Article Title: Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis. Article Snippet: Accepted Manuscript Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis Miguel Hueso, Laura De Ramon, Estanislao Navarro, Elia Ripoll, Josep M. Cruzado, Josep M. Grinyo, Joan Torras PII: S0021-9150(16)31470-8 DOI: 10.1016/j.atherosclerosis.2016.11.002 Reference: ATH 14860 To appear in: Atherosclerosis Received Date: 24 July 2016 Revised Date: 6 October 2016 Accepted Date: 1 November 2016 Please cite this article as: Hueso M, De Ramon L, Navarro E, Ripoll E, Cruzado JM, Grinyo JM, Torras J, Silencing of CD40 in vivo reduces progression of experimental atherogenesis through an NF-κB/miR-125b axis and reveals new potential mediators in the pathogenesis of atherosclerosis, Atherosclerosis (2016), doi: 10.1016/j.atherosclerosis.2016.11.002.. This is a PDF file of an unedited manuscript that has been accepted for publication.. As a service to our customers we are providing this early version of the manuscript. |
