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gold band plus 3 color regular range protein marker  (Yeasen Biotechnology)

 
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    Structured Review

    Yeasen Biotechnology gold band plus 3 color regular range protein marker
    Gold Band Plus 3 Color Regular Range Protein Marker, supplied by Yeasen Biotechnology, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/regularization/prestained+standard/pm42101653-66-0-13
    Average 86 stars, based on 1 article reviews
    gold band plus 3 color regular range protein marker - by Bioz Stars, 2026-09
    86/100 stars

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    Related Articles

    Marker:

    Article Title: Deubiquitinating enzyme USP42 promotes breast cancer progression by inhibiting JNK/p38-mediated apoptosis
    Article Snippet: .. The protein marker used in western blot was the GoldBand Plus 3-color Regular Range Protein Marker (8–180 kDa, Yeasen Biotechnology [Shanghai] Co., Ltd.). .. Specific information for the antibodies used in the experiment is as follows: USP42 (1:500, sc-390604, Santa Cruz Biotechnology, Inc., USA), JNK (9252, Cell Signaling Technology, 1:1000), p-JNK (9251, Cell Signaling Technology, 1:1000), p38 (9212, Cell Signaling Technology, 1:1000), p-p38 (9211, Cell Signaling Technology, 1:1000), β-actin (sc-47778, Santa Cruz Biotechnology, 1:1000).

    Article Title: Biofunctional ostrich tendon gelatin-based hot-melt type adhesive for enhanced wound hemostasis and healing
    Article Snippet: Gelatin-based tissue adhesives have significant potential as wound dressings in biomedical applications.. However, these adhesives often require modifications or chemical cross-linking to ensure adequate tissue adhesion and mechanical strength, which inevitably increases production complexity and costs.. Notably, few studies have reported the use of single-component, unmodified gelatin to prepare wound dressings.

    Article Title: Effects of Nutritional Disturbances on the Structure and Function of Mitochondria, Oxidative Stress Level, and Fat Deposition in Chicken Liver Cells
    Article Snippet: At last, the membrane was developed with the Chemiluminescence Kit (Vazyme, Nanjing, China) and imaged using fully automatic chemiluminescence/fluorescence image analysis system (Tanon, Shanghai, China). .. GoldBand 3-color prestaining protein marker (20351ES, Yeasen, Shanghai, China) was used to indicate protein molecular weight. .. The primary antibodies used in this study include MFN2 (sc-100560, Santa Cruz Biotechnology, Dallas, TX, USA), β-actin (AC026, ABclonal, Wuhan, China), GAPDH (10494-1-AP, Proteintech, Wuhan, China), SOD2 (24127-1-AP, Proteintech, Wuhan, China), LC3B (3868T, Cell Signaling Technology, Danvers, MA, USA), MT-ND1 (YA2089, MedChemExpress, Monmouth Junction, NJ, USA), CYTB (55090-1-AP, Proteintech, Wuhan, China).

    Article Title: Novel recombinant protein PK5-Gal-3C enhances the anti-tumor activity of T cells via binding with TRPV2.
    Article Snippet: Horseradish peroxidase (HRP)-labeled secondary antibodies were all purchased from Beyotime Biotechnology (Shanghai, China). .. Gold Band Plus 3-color Regular Range Protein Marker (8-180 kDa) was acquired from Yeasen Biotechnology (Shanghai, China). .. Human ELISA kits for TNF-α (RD-TNFα-Hu), for granzyme B (GzmB, CT211A), and for IFN-γ (RD-IFNγ-Hu) were all obtained from MabTech (Shanghai, China).

    Article Title: Anti-CLEC7A nanobody in situ engineering promotes amyloid-β oligomers clearance by CAR-microglia to alleviate Alzheimer's disease pathology in mice.
    Article Snippet: Chimeric antigen receptor microglia (CAR-M)-mediated amyloid-β oligomers (AβO) phagocytosis shows great promise in Alzheimer's disease (AD) treatment, however, the limited AβO degradation of CAR-M compromises their anti-AβO potency.. This work here reports an in situ engineered agonistic anti-C-type lectin domain containing 7 A (CLEC7A) nanobody to accelerate AβO degradation of CAR-M, augmenting their anti-AβO efficacy.. Specifically, with the intranasal-delivered microglia-targeting lipid nanoparticles (LNP), this work generates an AβO-specific degradation-potentiated CAR-M by introducing dual mRNAs encoding AβO-specific CAR and antiCLEC7A nanobody into the cerebral microglia.

    Article Title: Synthetic Disaggregators Enhance Central-Peripheral Amyloid-β Clearance in Alzheimer's Disease.
    Article Snippet: Pathogenic amyloid-β (Aβ) accumulation defines Alzheimer’s disease (AD), directly inflicting neuronal damage and driving chronic neuroinflammation.. While both central microglia and peripheral macrophages are critical for Aβ clearance, their functional impairment in AD inexorably leads to escalating Aβ burden and disease progression.. We here report an in situ engineered synthetic Aβ disaggregator (SAD) delivered to macrophages via neuroprotective DHA-based lipid nanoparticles (DLNPs).

    Article Title: Effects of Nutritional Disturbances on the Structure and Function of Mitochondria, Oxidative Stress Level, and Fat Deposition in Chicken Liver Cells.
    Article Snippet: At last, the membrane was developed with the Chemiluminescence Kit (Vazyme, Nanjing, China) and imaged using fully automatic chemiluminescence/fluorescence image analysis system (Tanon, Shanghai, China). .. GoldBand 3-color prestaining protein marker (20351ES, Yeasen, Shanghai, China) was used to indicate protein molecular weight. .. The primary antibodies used in this study include MFN2 (sc-100560, Santa Cruz Biotechnology, Dallas, TX, USA), β-actin (AC026, ABclonal, Wuhan, China), GAPDH (10494-1-AP, Proteintech, Wuhan, China), SOD2 (24127-1-AP, Proteintech, Wuhan, China), LC3B (3868T, Cell Signaling Technology, Danvers, MA, USA), MT-ND1 (YA2089, MedChemExpress, Monmouth Junction, NJ, USA), CYTB (55090-1-AP, Proteintech, Wuhan, China).

    Article Title: Ustiloxins induce kidney injury via the TLR2/MAPK/NF-κB pathway.
    Article Snippet: Ustiloxins levels are associated with abnormal renal function indicators, suggesting that ustiloxins may be risk factors for kidney damage.. Given limited evidence on the underlying mechanisms, this study investigated the mechanisms linking the TLR2/MAPK/NF-κB pathway to ustiloxin-induced kidney damage via transcriptomic and in vivo experiments.. Male mice were gavaged with ustiloxins at doses of 2, 5, and 12.5 mg/kg daily for three months.

    Western Blot:

    Article Title: Deubiquitinating enzyme USP42 promotes breast cancer progression by inhibiting JNK/p38-mediated apoptosis
    Article Snippet: .. The protein marker used in western blot was the GoldBand Plus 3-color Regular Range Protein Marker (8–180 kDa, Yeasen Biotechnology [Shanghai] Co., Ltd.). .. Specific information for the antibodies used in the experiment is as follows: USP42 (1:500, sc-390604, Santa Cruz Biotechnology, Inc., USA), JNK (9252, Cell Signaling Technology, 1:1000), p-JNK (9251, Cell Signaling Technology, 1:1000), p38 (9212, Cell Signaling Technology, 1:1000), p-p38 (9211, Cell Signaling Technology, 1:1000), β-actin (sc-47778, Santa Cruz Biotechnology, 1:1000).

    Article Title: Synthetic Disaggregators Enhance Central-Peripheral Amyloid-β Clearance in Alzheimer's Disease.
    Article Snippet: Pathogenic amyloid-β (Aβ) accumulation defines Alzheimer’s disease (AD), directly inflicting neuronal damage and driving chronic neuroinflammation.. While both central microglia and peripheral macrophages are critical for Aβ clearance, their functional impairment in AD inexorably leads to escalating Aβ burden and disease progression.. We here report an in situ engineered synthetic Aβ disaggregator (SAD) delivered to macrophages via neuroprotective DHA-based lipid nanoparticles (DLNPs).

    Article Title: Ustiloxins induce kidney injury via the TLR2/MAPK/NF-κB pathway.
    Article Snippet: Ustiloxins levels are associated with abnormal renal function indicators, suggesting that ustiloxins may be risk factors for kidney damage.. Given limited evidence on the underlying mechanisms, this study investigated the mechanisms linking the TLR2/MAPK/NF-κB pathway to ustiloxin-induced kidney damage via transcriptomic and in vivo experiments.. Male mice were gavaged with ustiloxins at doses of 2, 5, and 12.5 mg/kg daily for three months.

    Molecular Weight:

    Article Title: Effects of Nutritional Disturbances on the Structure and Function of Mitochondria, Oxidative Stress Level, and Fat Deposition in Chicken Liver Cells
    Article Snippet: At last, the membrane was developed with the Chemiluminescence Kit (Vazyme, Nanjing, China) and imaged using fully automatic chemiluminescence/fluorescence image analysis system (Tanon, Shanghai, China). .. GoldBand 3-color prestaining protein marker (20351ES, Yeasen, Shanghai, China) was used to indicate protein molecular weight. .. The primary antibodies used in this study include MFN2 (sc-100560, Santa Cruz Biotechnology, Dallas, TX, USA), β-actin (AC026, ABclonal, Wuhan, China), GAPDH (10494-1-AP, Proteintech, Wuhan, China), SOD2 (24127-1-AP, Proteintech, Wuhan, China), LC3B (3868T, Cell Signaling Technology, Danvers, MA, USA), MT-ND1 (YA2089, MedChemExpress, Monmouth Junction, NJ, USA), CYTB (55090-1-AP, Proteintech, Wuhan, China).

    Article Title: Effects of Nutritional Disturbances on the Structure and Function of Mitochondria, Oxidative Stress Level, and Fat Deposition in Chicken Liver Cells.
    Article Snippet: At last, the membrane was developed with the Chemiluminescence Kit (Vazyme, Nanjing, China) and imaged using fully automatic chemiluminescence/fluorescence image analysis system (Tanon, Shanghai, China). .. GoldBand 3-color prestaining protein marker (20351ES, Yeasen, Shanghai, China) was used to indicate protein molecular weight. .. The primary antibodies used in this study include MFN2 (sc-100560, Santa Cruz Biotechnology, Dallas, TX, USA), β-actin (AC026, ABclonal, Wuhan, China), GAPDH (10494-1-AP, Proteintech, Wuhan, China), SOD2 (24127-1-AP, Proteintech, Wuhan, China), LC3B (3868T, Cell Signaling Technology, Danvers, MA, USA), MT-ND1 (YA2089, MedChemExpress, Monmouth Junction, NJ, USA), CYTB (55090-1-AP, Proteintech, Wuhan, China).

    Membrane:

    Article Title: Anti-CLEC7A nanobody in situ engineering promotes amyloid-β oligomers clearance by CAR-microglia to alleviate Alzheimer's disease pathology in mice.
    Article Snippet: Chimeric antigen receptor microglia (CAR-M)-mediated amyloid-β oligomers (AβO) phagocytosis shows great promise in Alzheimer's disease (AD) treatment, however, the limited AβO degradation of CAR-M compromises their anti-AβO potency.. This work here reports an in situ engineered agonistic anti-C-type lectin domain containing 7 A (CLEC7A) nanobody to accelerate AβO degradation of CAR-M, augmenting their anti-AβO efficacy.. Specifically, with the intranasal-delivered microglia-targeting lipid nanoparticles (LNP), this work generates an AβO-specific degradation-potentiated CAR-M by introducing dual mRNAs encoding AβO-specific CAR and antiCLEC7A nanobody into the cerebral microglia.

    Bicinchoninic Acid Protein Assay:

    Article Title: Ustiloxins induce kidney injury via the TLR2/MAPK/NF-κB pathway.
    Article Snippet: Ustiloxins levels are associated with abnormal renal function indicators, suggesting that ustiloxins may be risk factors for kidney damage.. Given limited evidence on the underlying mechanisms, this study investigated the mechanisms linking the TLR2/MAPK/NF-κB pathway to ustiloxin-induced kidney damage via transcriptomic and in vivo experiments.. Male mice were gavaged with ustiloxins at doses of 2, 5, and 12.5 mg/kg daily for three months.

    Lysis:

    Article Title: Ustiloxins induce kidney injury via the TLR2/MAPK/NF-κB pathway.
    Article Snippet: Ustiloxins levels are associated with abnormal renal function indicators, suggesting that ustiloxins may be risk factors for kidney damage.. Given limited evidence on the underlying mechanisms, this study investigated the mechanisms linking the TLR2/MAPK/NF-κB pathway to ustiloxin-induced kidney damage via transcriptomic and in vivo experiments.. Male mice were gavaged with ustiloxins at doses of 2, 5, and 12.5 mg/kg daily for three months.

    Stripping Membranes:

    Article Title: Ustiloxins induce kidney injury via the TLR2/MAPK/NF-κB pathway.
    Article Snippet: Ustiloxins levels are associated with abnormal renal function indicators, suggesting that ustiloxins may be risk factors for kidney damage.. Given limited evidence on the underlying mechanisms, this study investigated the mechanisms linking the TLR2/MAPK/NF-κB pathway to ustiloxin-induced kidney damage via transcriptomic and in vivo experiments.. Male mice were gavaged with ustiloxins at doses of 2, 5, and 12.5 mg/kg daily for three months.



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