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multi-analyte elisarray kit qiagen mem-009a  (Qiagen)


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    Qiagen multi-analyte elisarray kit qiagen mem-009a
    Multi Analyte Elisarray Kit Qiagen Mem 009a, supplied by Qiagen, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/multi-analyte+elisarray+kit/human+common+chemokines+multi+analyte+elisarray+kit/pm39384746-239-7-10
    Average 90 stars, based on 1 article reviews
    multi-analyte elisarray kit qiagen mem-009a - by Bioz Stars, 2026-11
    90/100 stars

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    Related Articles

    other:

    Article Title: Annexin A2 promotes proliferative vitreoretinopathy in response to a macrophage inflammatory signal in mice.
    Article Snippet: Cyto/chemokine screening profiles were performed using a Multi-Analyte ELISArray kit (Qiagen MEM-004A and MEM-009A).

    Article Title: Doxorubicin‐induced senescence promotes stemness and tumorigenicity in EpCAM−/CD133− nonstem cell population in hepatocellular carcinoma cell line, HuH‐7
    Article Snippet: ELISA experiments were performed using Human Common Chemokines Multi‐Analyte ELISArray Kit (Qiagen MEH‐009A) and Human Inflammatory Cytokines Multi‐Analyte ELISArrayTM Kit (Qiagen MEH‐004A), respectively, according to manufacturer protocol.

    Article Title: A Mast-Cell-Specific Receptor Mediates Neurogenic Inflammation and Pain
    Article Snippet: Live versus Dead cells were stained using Live/Dead Fixable Aqua Dead Cell Stain Kit (Thermo Fisher).

    Article Title: A Mast-Cell-Specific Receptor Mediates Neurogenic Inflammation and Pain
    Article Snippet: Cytokine and Chemokine ELISAs Cytokines and chemokines were measured using Human Common Cyokines Multi-Analyte ELISArray Kit and Human Common Chemokines Multi-Analyte ELISArray Kit (QIAGEN).

    Article Title: Doxorubicin-induced senescence promotes stemness and tumorigenicity in EpCAM-/CD133- nonstem cell population in hepatocellular carcinoma cell line, HuH-7.
    Article Snippet: ELISA experiments were performed using Human Common Chemokines Multi-Analyte ELISArray Kit (Qiagen MEH-009A) and Human Inflammatory Cytokines Multi-Analyte ELISArrayTM Kit (Qiagen MEH-004A), respectively, according to manufacturer protocol.

    Article Title: Human Papillomavirus E6 and E7 oncoproteins affect the cell microenvironment by classical secretion and extracellular vesicles delivery of inflammatory mediators.
    Article Snippet: The connection between chronic inflammation and risk of cancer has been supported by several studies.. The development of cancer might be a process driven by the presence of a specific combination of inflammatory mediators, including cytokines, chemokines and enzymes, in the tumor microenvironment.. Virus-induced tumors, like HPV-induced Squamous Cell Carcinomas, represent a paradigmatic example of the interplay between inflammation, as integral part of the innate antiviral response, and malignant transformation.

    Article Title: A Mast-Cell-Specific Receptor Mediates Neurogenic Inflammation and Pain
    Article Snippet: Cytokines and chemokines were measured using Human Common Cyokines Multi-Analyte ELISArray Kit and Human Common Chemokines Multi-Analyte ELISArray Kit (QIAGEN).

    Enzyme-linked Immunosorbent Assay:

    Article Title: Adventitial Cell Atlas of wt (Wild Type) and ApoE (Apolipoprotein E)-Deficient Mice Defined by Single-Cell RNA Sequencing
    Article Snippet: .. Mouse CCL2 ELISA (R&D Systems; MJE00) and chemokine array (Qiagen; MEM- 009A) for supernatant of adventitial cells were performed with manufacturer’s protocol. ..



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    The effect of carbon black (CB), urban dust (UD), and nanoparticulate carbon black (NPCB) on cell membrane damage (( A ); lactate dehydrogenase (LDH) release), oxidative stress ( B ), DNA damage ( C ), and phagocytosis ( D ) in human monocytes (M), and monocyte-derived macrophages (MDM). The inflammatory response (TNFα; ( E )) to lipopolysaccharide (100 ng/mL for 24 h) is also included ( F ). All PMs were used at 100 µg/mL and the incubation time was 24 h. Cell transition was induced by phorbol-12-myristate 13-acetate (PMA; 100 nM) applied for 72 h. LDH was measured with CyQUANT LDH Cytotoxicity Assay kit, DNA ploidy was quantified using propidium iodide staining, intracellular oxidative stress was measured using 5-(and-6)-Carboxy-2′,7′-dichlorodihydrofluorescein diacetate (carboxy-H2DCFDA) while phagocytosis was assessed with fluorescent beads (Cayman Phagocytosis kit). Fluorescence was quantified with flow cytometry. TNFα was measured in the culture medium using a Multi-Analyte Inflammatory <t>Cytokine</t> <t>ELISArray</t> Kit (Quiagen, Wroclaw, Poland). Data are means ± SD of 6–10 assays. * p < 0.05; ** p < 0.01 for comparisons with the corresponding control cells; ^^ p < 0.01 for comparisons with corresponding naive cells; # p < 0.05 for comparisons with LPS-treated cells.
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    The effect of carbon black (CB), urban dust (UD), and nanoparticulate carbon black (NPCB) on cell membrane damage (( A ); lactate dehydrogenase (LDH) release), oxidative stress ( B ), DNA damage ( C ), and phagocytosis ( D ) in human monocytes (M), and monocyte-derived macrophages (MDM). The inflammatory response (TNFα; ( E )) to lipopolysaccharide (100 ng/mL for 24 h) is also included ( F ). All PMs were used at 100 µg/mL and the incubation time was 24 h. Cell transition was induced by phorbol-12-myristate 13-acetate (PMA; 100 nM) applied for 72 h. LDH was measured with CyQUANT LDH Cytotoxicity Assay kit, DNA ploidy was quantified using propidium iodide staining, intracellular oxidative stress was measured using 5-(and-6)-Carboxy-2′,7′-dichlorodihydrofluorescein diacetate (carboxy-H2DCFDA) while phagocytosis was assessed with fluorescent beads (Cayman Phagocytosis kit). Fluorescence was quantified with flow cytometry. TNFα was measured in the culture medium using a Multi-Analyte Inflammatory Cytokine ELISArray Kit (Quiagen, Wroclaw, Poland). Data are means ± SD of 6–10 assays. * p < 0.05; ** p < 0.01 for comparisons with the corresponding control cells; ^^ p < 0.01 for comparisons with corresponding naive cells; # p < 0.05 for comparisons with LPS-treated cells.

    Journal: Cells

    Article Title: Signalling Pathways of Inflammation and Cancer in Human Mononuclear Cells: Effect of Nanoparticle Air Pollutants

    doi: 10.3390/cells13161367

    Figure Lengend Snippet: The effect of carbon black (CB), urban dust (UD), and nanoparticulate carbon black (NPCB) on cell membrane damage (( A ); lactate dehydrogenase (LDH) release), oxidative stress ( B ), DNA damage ( C ), and phagocytosis ( D ) in human monocytes (M), and monocyte-derived macrophages (MDM). The inflammatory response (TNFα; ( E )) to lipopolysaccharide (100 ng/mL for 24 h) is also included ( F ). All PMs were used at 100 µg/mL and the incubation time was 24 h. Cell transition was induced by phorbol-12-myristate 13-acetate (PMA; 100 nM) applied for 72 h. LDH was measured with CyQUANT LDH Cytotoxicity Assay kit, DNA ploidy was quantified using propidium iodide staining, intracellular oxidative stress was measured using 5-(and-6)-Carboxy-2′,7′-dichlorodihydrofluorescein diacetate (carboxy-H2DCFDA) while phagocytosis was assessed with fluorescent beads (Cayman Phagocytosis kit). Fluorescence was quantified with flow cytometry. TNFα was measured in the culture medium using a Multi-Analyte Inflammatory Cytokine ELISArray Kit (Quiagen, Wroclaw, Poland). Data are means ± SD of 6–10 assays. * p < 0.05; ** p < 0.01 for comparisons with the corresponding control cells; ^^ p < 0.01 for comparisons with corresponding naive cells; # p < 0.05 for comparisons with LPS-treated cells.

    Article Snippet: TNFα was quantified in the culture medium of M and MDM with the Multi-Analyte Inflammatory Cytokine ELISArray Kit (Qiagen, Manchester, UK).

    Techniques: Membrane, Derivative Assay, Incubation, CyQUANT Assay, LDH Cytotoxicity Assay, Staining, Fluorescence, Flow Cytometry, Control