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tissue microarray  (Novus Biologicals)


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    Novus Biologicals tissue microarray
    Inhibition of SND1 enzymatic activity inhibits cell viability and AEG-1 function. A. Hep-pc-4 and Hep-AEG-1-14 cells were treated or not with 50, 100 and 200 μM pdTp for 1 to 4 days and cell viability was measured by standard MTT assay. B. Hep-pc-4, Hep-AEG-1-14, Hep3B and QGY-7703 cells were treated with 100 μM pdTp and colony formation assay was performed. The colonies were scored after two weeks. Data represent mean± SEM of three independent experiments. C. Analysis of SND1 expression in tissue <t>microarray</t> by immunohistochemistry. ˆ: p<0.05, #: p<0.02 and *: p<0.01 vs the corresponding data point in Hep-pc-4 cells.
    Tissue Microarray, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 93/100, based on 3 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/microarray/pmc03081619-71-0-11?v=Novus+Biologicals
    Average 93 stars, based on 3 article reviews
    tissue microarray - by Bioz Stars, 2026-08
    93/100 stars

    Images

    1) Product Images from "Increased RNA-Induced Silencing Complex (RISC) Activity Contributes to Hepatocellular Carcinoma"

    Article Title: Increased RNA-Induced Silencing Complex (RISC) Activity Contributes to Hepatocellular Carcinoma

    Journal:

    doi: 10.1002/hep.24216

    Inhibition of SND1 enzymatic activity inhibits cell viability and AEG-1 function. A. Hep-pc-4 and Hep-AEG-1-14 cells were treated or not with 50, 100 and 200 μM pdTp for 1 to 4 days and cell viability was measured by standard MTT assay. B. Hep-pc-4, Hep-AEG-1-14, Hep3B and QGY-7703 cells were treated with 100 μM pdTp and colony formation assay was performed. The colonies were scored after two weeks. Data represent mean± SEM of three independent experiments. C. Analysis of SND1 expression in tissue microarray by immunohistochemistry. ˆ: p<0.05, #: p<0.02 and *: p<0.01 vs the corresponding data point in Hep-pc-4 cells.
    Figure Legend Snippet: Inhibition of SND1 enzymatic activity inhibits cell viability and AEG-1 function. A. Hep-pc-4 and Hep-AEG-1-14 cells were treated or not with 50, 100 and 200 μM pdTp for 1 to 4 days and cell viability was measured by standard MTT assay. B. Hep-pc-4, Hep-AEG-1-14, Hep3B and QGY-7703 cells were treated with 100 μM pdTp and colony formation assay was performed. The colonies were scored after two weeks. Data represent mean± SEM of three independent experiments. C. Analysis of SND1 expression in tissue microarray by immunohistochemistry. ˆ: p<0.05, #: p<0.02 and *: p<0.01 vs the corresponding data point in Hep-pc-4 cells.

    Techniques Used: Inhibition, Activity Assay, MTT Assay, Colony Assay, Expressing, Microarray, Immunohistochemistry

    Immunoperoxidase staining of normal liver and different stages of HCC by tissue microarray using anti-SND1 antibody
    Figure Legend Snippet: Immunoperoxidase staining of normal liver and different stages of HCC by tissue microarray using anti-SND1 antibody

    Techniques Used: Immunoperoxidase Staining, Microarray, Staining



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    Image Search Results


    BNT351 shows comparable target affinity to 1-18 and no off-target binding (A) Left: the binding affinity of BNT351 and 1-18 to the stabilized HIV-1 envelope protein trimer (SOSIP BG505.664 ) by SPR (mean and individual values of triplicate measurements). Also see A. Right: binding of BNT351 and 1-18 to SOSIP BG505.664 by ELISA (mean ± standard deviation of triplicate measurements). (B) BNT351’s off-target binding was assessed by Retrogenix Cell Microarray Technology platform. Interactions identified during the library screen with ∼6,500 human proteins are shown. Also, see . Interaction with pepsinogen C (PGC) that appeared to be specific was re-tested by flow cytometry with HEK293 cells transfected with PGC. HEK293 cells transfected with FcγRIIIa and FcεRIg were used as a positive control. Duplicate measurements are shown for flow cytometry.

    Journal: iScience

    Article Title: Preclinical assessment of broadly neutralizing HIV-1 antibody BNT351 with optimized pharmacokinetics and potent antiviral activity

    doi: 10.1016/j.isci.2026.116022

    Figure Lengend Snippet: BNT351 shows comparable target affinity to 1-18 and no off-target binding (A) Left: the binding affinity of BNT351 and 1-18 to the stabilized HIV-1 envelope protein trimer (SOSIP BG505.664 ) by SPR (mean and individual values of triplicate measurements). Also see A. Right: binding of BNT351 and 1-18 to SOSIP BG505.664 by ELISA (mean ± standard deviation of triplicate measurements). (B) BNT351’s off-target binding was assessed by Retrogenix Cell Microarray Technology platform. Interactions identified during the library screen with ∼6,500 human proteins are shown. Also, see . Interaction with pepsinogen C (PGC) that appeared to be specific was re-tested by flow cytometry with HEK293 cells transfected with PGC. HEK293 cells transfected with FcγRIIIa and FcεRIg were used as a positive control. Duplicate measurements are shown for flow cytometry.

    Article Snippet: The Retrogenix cell microarray was performed by Charles River Laboratories and included 6,105 full-length human proteins (plasma membrane proteins, secreted or a cell surface-tethered secreted proteins) and 400 human heterodimers.

    Techniques: Binding Assay, Enzyme-linked Immunosorbent Assay, Standard Deviation, Microarray, Flow Cytometry, Transfection, Positive Control