microarray-based comparative genomic hybridization (Kohlhammer GmbH)
90
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Kohlhammer GmbH
microarray-based comparative genomic hybridization
Microarray Based Comparative Genomic Hybridization, supplied by Kohlhammer GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/microarray-based+assay/microarray+based+comparative+genomic+hybridization/pm22739938-183-24-4
Average 90 stars, based on 1 article reviews
Microarray Based Comparative Genomic Hybridization, supplied by Kohlhammer GmbH, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/microarray-based+assay/microarray+based+comparative+genomic+hybridization/pm22739938-183-24-4
Average 90 stars, based on 1 article reviews
microarray-based comparative genomic hybridization - by Bioz Stars,
2026-09
90/100 stars
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other:Article Title: Molecular cytogenetics in haematological malignancy: current technology and future prospects. Article Snippet: Cytogenetics has played a pivotal role in haematological malignancy, both as an aid to diagnosis and in identifying recurrent chromosomal rearrangements, an essential prerequisite to identifying genes involved in leukaemia and lymphoma pathogenesis.. In the late 1980s, a series of technologies based around fluorescence in situ hybridisation (FISH) revolutionised the field.. Interphase FISH, multiplex-FISH (M-FISH, SKY) and comparative genomic hybridisation (CGH) have emerged as the most significant of these. Article Title: Different breakage-prone regions on chromosome 1 detected in t(11;14)-positive mantle cell lymphoma cell lines and multiple myeloma cell lines are associated with different tumor progression-related mechanisms. Article Snippet: To better defi ne secondary aberrations that occur in addition to translocation t(11; 14)(q13;q32) in mantle cell lymphomas (MCL) and in multiple myelomas (MM), seven t(11; 14)-positive MCL cell lines and four t(11; 14)-positive MM cell lines were analysed by fl uorescence R-banding and spectral karyotyping (SKY).. Compared with published data obtained by G-banding, most chromosome aberrations were redefi ned or further specifi ed.. Furthermore, several additional chromosome aberrations were identifi ed. Article Title: Transcriptional profiling of Epstein–Barr virus (EBV) genes and host cellular genes in nasal NK/T-cell lymphoma and chronic active EBV infection Article Snippet: Matrix-based comparative genomic hybridisation (matrix-CGH) analysis demonstrated that TNFRSF10D is one of the pathologically relevant genes in Article Title: High-resolution oligonucleotide array-CGH pinpoints genes involved in cryptic losses in chronic lymphocytic leukemia. Article Snippet: mon chromosomal changes in CLL are trisomy 12 and deletions in 13q14, 11q22 ] q23 and 17p13, of which the 13q14 deletion is prognostically favorable and deletions in 11q22 ] q23 and 17p13 are unfavorable (Döhner et al., 2000).. While conventional cytogenetics of CLL was hampered by absence of mitoses that represent malignant cells (Knuutila et al., 1986), classic comparative genomic hybridization (cCGH) has been useful in analyzing genome-wide copy number alterations (Knuutila et al., 2000).. A technologically more advanced application, microarray-based CGH (array-CGH, aCGH) (Pinkel et al., 1998; Pollack et al., 1999), enables the detection of chromosomal alterations at higher resolution than cCGH and can be used for target gene screening. Microarray:Article Title: MiR-138 suppressed nasopharyngeal carcinoma growth and tumorigenesis by targeting the CCND1 oncogene. Article Snippet: .. Wessendorf S, Article Title: Tiling resolution array CGH of dic(7;9)(p11 approximately 13;p11 approximately 13) in B-cell precursor acute lymphoblastic leukemia reveals clustered breakpoints at 7p11.2 approximately 12.1 and 9p13.1. Article Snippet: a Department of Clinical Genetics and b Department of Oncology, Lund University Hospital, Lund c Lund Strategic Research Center for Stem Cell Biology and Cell Therapy, Lund University, Lund (Sweden) d Department of Medical Genetics and Molecular Medicine, Haukeland University Hospital, Helse-Bergen HF e Department of Medical Genetics, Rikshospitalet-Radiumhospitalet Medical Centre, Oslo f University of Oslo, Oslo (Norway) Article Title: Tiling resolution array comparative genomic hybridization, expression and methylation analyses of dup(1q) in Burkitt lymphomas and pediatric high hyperdiploid acute lymphoblastic leukemias reveal clustered near-centromeric breakpoints and overexpression of genes in 1q22-32.3. Article Snippet: Although gain of 1q occurs in 25% of Burkitt lymphomas (BLs) and 10% of pediatric high hyperdiploid acute lymphoblastic leukemias (ALLs), little is known about the origin, molecular genetic characteristics and functional outcome of dup(1q) in these disorders.. Ten dup(1q)-positive BLs/ALLs were investigated by tiling resolution (32k) array CGH analysis, which revealed that the proximal breakpoints in all cases were near-centromeric, in eight of them clustering within a 1.4 Mb segment in 1q12-21.1.. The 1q distal breakpoints were heterogeneous, being more distal in the ALLs than in the BLs. Article Title: Genetic profile of hepatocellular carcinoma revealed by array-based comparative genomic hybridization: identification of genetic indicators to predict patient outcome. Article Snippet: Pathology Division, National Cancer Center Research Institute, 5-1-1, Tsukiji, Chuo-ku, Tokyo 104-0045, Japan Department of Pathology, Graduate School of Medicine, University of Tokyo, Tokyo, Japan Division of Hepato-biliary and Pancreatic Surgery, National Cancer Center Hospital, Tokyo, Japan Cancer Proteomics Project, National Cancer Center Research Institute, Tokyo, Japan Department of Pathology, Keio University School of Medicine, Tokyo, Japan Cancer Genomics Project, National Cancer Center Research Institute, Tokyo, Japan Center for Medical Genetics, National Cancer Center Research Institute, Tokyo, Japan Department of Molecular Cytogenetics, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan Hybridization:Article Title: MiR-138 suppressed nasopharyngeal carcinoma growth and tumorigenesis by targeting the CCND1 oncogene. Article Snippet: .. Wessendorf S, Article Title: Tiling resolution array CGH of dic(7;9)(p11 approximately 13;p11 approximately 13) in B-cell precursor acute lymphoblastic leukemia reveals clustered breakpoints at 7p11.2 approximately 12.1 and 9p13.1. Article Snippet: a Department of Clinical Genetics and b Department of Oncology, Lund University Hospital, Lund c Lund Strategic Research Center for Stem Cell Biology and Cell Therapy, Lund University, Lund (Sweden) d Department of Medical Genetics and Molecular Medicine, Haukeland University Hospital, Helse-Bergen HF e Department of Medical Genetics, Rikshospitalet-Radiumhospitalet Medical Centre, Oslo f University of Oslo, Oslo (Norway) Article Title: Tiling resolution array comparative genomic hybridization, expression and methylation analyses of dup(1q) in Burkitt lymphomas and pediatric high hyperdiploid acute lymphoblastic leukemias reveal clustered near-centromeric breakpoints and overexpression of genes in 1q22-32.3. Article Snippet: Although gain of 1q occurs in 25% of Burkitt lymphomas (BLs) and 10% of pediatric high hyperdiploid acute lymphoblastic leukemias (ALLs), little is known about the origin, molecular genetic characteristics and functional outcome of dup(1q) in these disorders.. Ten dup(1q)-positive BLs/ALLs were investigated by tiling resolution (32k) array CGH analysis, which revealed that the proximal breakpoints in all cases were near-centromeric, in eight of them clustering within a 1.4 Mb segment in 1q12-21.1.. The 1q distal breakpoints were heterogeneous, being more distal in the ALLs than in the BLs. Article Title: Genetic profile of hepatocellular carcinoma revealed by array-based comparative genomic hybridization: identification of genetic indicators to predict patient outcome. Article Snippet: Pathology Division, National Cancer Center Research Institute, 5-1-1, Tsukiji, Chuo-ku, Tokyo 104-0045, Japan Department of Pathology, Graduate School of Medicine, University of Tokyo, Tokyo, Japan Division of Hepato-biliary and Pancreatic Surgery, National Cancer Center Hospital, Tokyo, Japan Cancer Proteomics Project, National Cancer Center Research Institute, Tokyo, Japan Department of Pathology, Keio University School of Medicine, Tokyo, Japan Cancer Genomics Project, National Cancer Center Research Institute, Tokyo, Japan Center for Medical Genetics, National Cancer Center Research Institute, Tokyo, Japan Department of Molecular Cytogenetics, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan |
