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microarray data analysis gene ontology (go) analysis, pathway analysis, path-net, signal-net, and microrna-gene network  (Genminix Informatics Co Ltd)

 
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    Genminix Informatics Co Ltd microarray data analysis gene ontology (go) analysis, pathway analysis, path-net, signal-net, and microrna-gene network
    Microarray Data Analysis Gene Ontology (Go) Analysis, Pathway Analysis, Path Net, Signal Net, And Microrna Gene Network, supplied by Genminix Informatics Co Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/microarray+processing+and+analysis/pmc08120187-80-10-16?v=Genminix+Informatics+Co+Ltd
    Average 90 stars, based on 1 article reviews
    microarray data analysis gene ontology (go) analysis, pathway analysis, path-net, signal-net, and microrna-gene network - by Bioz Stars, 2026-07
    90/100 stars

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    Chronic normobaric hypoxia (CNH) exposure induces pulmonary arterial hypertension (PAH) and upregulates miR-335-3p expression in the lungs of mice. CNH treatment significantly increased RVSP (A) and RV/(LV+S) (B) (N=5-8 per group). (C) Heatmap and sample clustering analysis of the differential expressed miRs in the lungs of mice (N=4 per group). Each row represents a miR and each column represents a sample. (D) <t>Microarray</t> and (E) quantitative RT-PCR analysis of miR-335-3p expression in the lungs of mice. (F) Correlation analysis between miR-335-3p and RVSP, and (G) Correlation analysis between miR-335-3p and RV/(LV+S) (N=4 per group). Values shown are means±s.e.m. ** p <0.01, *** p <0.001 vs. Normoxia.
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    Chronic normobaric hypoxia (CNH) exposure induces pulmonary arterial hypertension (PAH) and upregulates miR-335-3p expression in the lungs of mice. CNH treatment significantly increased RVSP (A) and RV/(LV+S) (B) (N=5-8 per group). (C) Heatmap and sample clustering analysis of the differential expressed miRs in the lungs of mice (N=4 per group). Each row represents a miR and each column represents a sample. (D) Microarray and (E) quantitative RT-PCR analysis of miR-335-3p expression in the lungs of mice. (F) Correlation analysis between miR-335-3p and RVSP, and (G) Correlation analysis between miR-335-3p and RV/(LV+S) (N=4 per group). Values shown are means±s.e.m. ** p <0.01, *** p <0.001 vs. Normoxia.

    Journal: International Journal of Biological Sciences

    Article Title: Upregulation of miR-335-3p by NF-κB Transcriptional Regulation Contributes to the Induction of Pulmonary Arterial Hypertension via APJ during Hypoxia

    doi: 10.7150/ijbs.34517

    Figure Lengend Snippet: Chronic normobaric hypoxia (CNH) exposure induces pulmonary arterial hypertension (PAH) and upregulates miR-335-3p expression in the lungs of mice. CNH treatment significantly increased RVSP (A) and RV/(LV+S) (B) (N=5-8 per group). (C) Heatmap and sample clustering analysis of the differential expressed miRs in the lungs of mice (N=4 per group). Each row represents a miR and each column represents a sample. (D) Microarray and (E) quantitative RT-PCR analysis of miR-335-3p expression in the lungs of mice. (F) Correlation analysis between miR-335-3p and RVSP, and (G) Correlation analysis between miR-335-3p and RV/(LV+S) (N=4 per group). Values shown are means±s.e.m. ** p <0.01, *** p <0.001 vs. Normoxia.

    Article Snippet: Microarray processing and analysis for miRs was performed by the Ribobio Co., Ltd. (Guangzhou, China).

    Techniques: Expressing, Microarray, Quantitative RT-PCR