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CapitalBio Corporation contact microarray printer smartarrayer 136
Contact Microarray Printer Smartarrayer 136, supplied by CapitalBio Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/microarray+printers/contact+microarray+printer+smartarrayer+136/pm40143425-153-18-23
Average 90 stars, based on 1 article reviews
contact microarray printer smartarrayer 136 - by Bioz Stars, 2026-09
90/100 stars

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Related Articles

Microarray:

Article Title: Synergetic improvements of sensitivity and specificity of nanowire field effect transistor gene chip by designing neutralized DNA as probe
Article Snippet: .. To immobilize the probes on the chip surface, 10 mM sodium phosphate buffer containing 1 μM probe DNA (regular, fully neutralized, or partially neutralized chimeric DNA) was prepared to spot on the NW by using a microarray spotter (SmartArrayer 136, CapitalBio Corporation, China) and incubated with the FET chip overnight. ..


Article Title: Allele-selective lowering of mutant HTT protein by HTT-LC3 linker compounds.
Article Snippet: .. Each compound was dissolved in DMSO at a concentration of 10 mM and printed in duplicates along vertical direction on homemade phenyl-isocyanate functionalized glass slides with a contact microarray printer (SmartArrayer 136, CapitalBio Corporation). ..

Article Title: Pharmacological boosting of cGAS activation sensitizes chemotherapy by enhancing antitumor immunity.
Article Snippet: .. Each compound was dissolved in DMSO to a concentration of 10 mM and printed in duplicate on home-made phenyl-isocyanate functionalized slide with a contact microarray printer (SmartArrayer 136, CapitalBio Corporation, Beijing, China).89 Biotin-BSA at a concentration of 7 600 nM in 13 phosphate-buffered saline (PBS) and biotin-(PEG)2-NH2 at a concentration of 5 mM in DMSOwere also printed as the inner and the outer borders of SMMs to serve as positionmarkers and positive controls. ..

Article Title: P53 upregulation by USP7-engaging molecular glues.
Article Snippet: .. Each compound was dissolved in dimethyl sulfoxide (DMSO) at a concentration of 10 mmol/L and printed in duplicate along a vertical direction on homemade phenyl-isocyanate functionalized glass slides with a contact microarray printer (SmartArrayer 136; CapitalBio Corporation, Beijing, China). ..

Article Title: Developing an Efficient and General Strategy for Immobilization of Small Molecules onto Microarrays Using Isocyanate Chemistry
Article Snippet: .. Five biotinylated compounds, biotin-(PEG) 2 -NH 2 , biotin4-nitrophenyl ester, biotin-NH-NH 2 , biotin-(PEG) 6 -OH, and D-biotin (with intrinsic COOH group), were printed on as-prepared isocyanate functionalized glass slides with a contact microarray printer (SmartArrayer 136 Microarray Spotter, CapitalBio Corporation, Beijing, China). ..

Article Title: R406 and its structural analogs reduce SNCA/α-synuclein levels via autophagic degradation.
Article Snippet: .. Each compound was printed twice in a vertical direction on homemade glass slides functionalized with phenyl-isocyanate using a contact microarray printer (SmartArrayer 136, CapitalBio Corporation, Beijing, China). ..

Incubation:

Article Title: Synergetic improvements of sensitivity and specificity of nanowire field effect transistor gene chip by designing neutralized DNA as probe
Article Snippet: .. To immobilize the probes on the chip surface, 10 mM sodium phosphate buffer containing 1 μM probe DNA (regular, fully neutralized, or partially neutralized chimeric DNA) was prepared to spot on the NW by using a microarray spotter (SmartArrayer 136, CapitalBio Corporation, China) and incubated with the FET chip overnight. ..

Concentration Assay:


Article Title: Allele-selective lowering of mutant HTT protein by HTT-LC3 linker compounds.
Article Snippet: .. Each compound was dissolved in DMSO at a concentration of 10 mM and printed in duplicates along vertical direction on homemade phenyl-isocyanate functionalized glass slides with a contact microarray printer (SmartArrayer 136, CapitalBio Corporation). ..

Article Title: Pharmacological boosting of cGAS activation sensitizes chemotherapy by enhancing antitumor immunity.
Article Snippet: .. Each compound was dissolved in DMSO to a concentration of 10 mM and printed in duplicate on home-made phenyl-isocyanate functionalized slide with a contact microarray printer (SmartArrayer 136, CapitalBio Corporation, Beijing, China).89 Biotin-BSA at a concentration of 7 600 nM in 13 phosphate-buffered saline (PBS) and biotin-(PEG)2-NH2 at a concentration of 5 mM in DMSOwere also printed as the inner and the outer borders of SMMs to serve as positionmarkers and positive controls. ..

Article Title: P53 upregulation by USP7-engaging molecular glues.
Article Snippet: .. Each compound was dissolved in dimethyl sulfoxide (DMSO) at a concentration of 10 mmol/L and printed in duplicate along a vertical direction on homemade phenyl-isocyanate functionalized glass slides with a contact microarray printer (SmartArrayer 136; CapitalBio Corporation, Beijing, China). ..

Saline:

Article Title: Pharmacological boosting of cGAS activation sensitizes chemotherapy by enhancing antitumor immunity.
Article Snippet: .. Each compound was dissolved in DMSO to a concentration of 10 mM and printed in duplicate on home-made phenyl-isocyanate functionalized slide with a contact microarray printer (SmartArrayer 136, CapitalBio Corporation, Beijing, China).89 Biotin-BSA at a concentration of 7 600 nM in 13 phosphate-buffered saline (PBS) and biotin-(PEG)2-NH2 at a concentration of 5 mM in DMSOwere also printed as the inner and the outer borders of SMMs to serve as positionmarkers and positive controls. ..

other:

Article Title: Bioinformatics and microarray analysis of microRNA expression profiles of murine embryonic stem cells, neural stem cells induced from ESCs and isolated from E8·5 mouse neural tube
Article Snippet: To better understand whether microRNAs (miRNAs) are involved in the self-renewal of stem cells and fate determination of neural stem cells and to identify the miRNA expression patterns of different neural stem cells (NSC) in vitro and in vivo, we examined miRNA expression profiles of murine embryonic stem cells (ESC), NSC induced from ESC and isolated from E8.5 mouse neural tube (E8.5-NSC) using microarray technique.. It was found that a total of 40 miRNAs had similar expression level in all the three cells [false discovery rate (FDR)50, fold change ,3.0].. Moreover, q-PCR showed that some members of miR-106b and miR-17–92 families were expressed in the ESC, NSC induced from ESC (ESC-NSC) and hematopoietic stem cells (HSC).



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Comprehensive <t>microarray</t> polymer profiling (CoMPP) from Arabidopsis thaliana wild type and ARAK1‐OE seedlings grown on 3 mM L‐Ara. Analysis of cell wall composition by carbohydrate profiling is shown. Alcohol‐insoluble residue (AIR) samples were isolated from roots of wild type (WT) and ARAK1‐OE mutants growing for 14 days on 0.5× MS‐agar plates supplemented with 3 mM L‐Ara. Cell wall components were extracted from AIR samples with diaminocyclo‐hexane‐tetra‐acetic acid (CDTA) and NaOH. Blue colors indicate increased intensity of respective antibody binding according to the legend.
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<t>Microarray</t> analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D
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<t>Microarray</t> analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D
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Image Search Results


Comprehensive microarray polymer profiling (CoMPP) from Arabidopsis thaliana wild type and ARAK1‐OE seedlings grown on 3 mM L‐Ara. Analysis of cell wall composition by carbohydrate profiling is shown. Alcohol‐insoluble residue (AIR) samples were isolated from roots of wild type (WT) and ARAK1‐OE mutants growing for 14 days on 0.5× MS‐agar plates supplemented with 3 mM L‐Ara. Cell wall components were extracted from AIR samples with diaminocyclo‐hexane‐tetra‐acetic acid (CDTA) and NaOH. Blue colors indicate increased intensity of respective antibody binding according to the legend.

Journal: Plant Direct

Article Title: Arabinokinase Limits the Flux of Arabinose Into Nucleotide Sugars to Prevent Toxicity

doi: 10.1002/pld3.70094

Figure Lengend Snippet: Comprehensive microarray polymer profiling (CoMPP) from Arabidopsis thaliana wild type and ARAK1‐OE seedlings grown on 3 mM L‐Ara. Analysis of cell wall composition by carbohydrate profiling is shown. Alcohol‐insoluble residue (AIR) samples were isolated from roots of wild type (WT) and ARAK1‐OE mutants growing for 14 days on 0.5× MS‐agar plates supplemented with 3 mM L‐Ara. Cell wall components were extracted from AIR samples with diaminocyclo‐hexane‐tetra‐acetic acid (CDTA) and NaOH. Blue colors indicate increased intensity of respective antibody binding according to the legend.

Article Snippet: Arrays were printed as distinct dots onto nitrocellulose membranes with an ArrayJet Sprint microarray printer (ArrayJet, Roslin, UK) and probed with ~40 cell wall probes, including antibodies and carbohydrate binding modules (Table ).

Techniques: Microarray, Polymer, Residue, Isolation, Binding Assay

Microarray analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D

Journal: Journal of Neuroinflammation

Article Title: Targeting glial fibrillary acidic protein in glaucoma: a monoclonal antibody approach to modulate glial reactivity and neuroinflammation for neuroprotection

doi: 10.1186/s12974-025-03482-8

Figure Lengend Snippet: Microarray analysis reveals the effects of GFAP mAb treatment on key inflammatory mediators in glaucomatous retina. ( A - C ) The expression of TLR4 and S100A8 was significantly downregulated by 25–50 µg GFAP mAb treatment compared to the vehicle. The marker for microglial activation, CD68, was significantly decreased in the 25 µg GFAP mAb treatment group. ( D - F ) Proteins of the inflammasome pathway, NLRP3, GSDMD and Caspase-1 were significantly downregulated in the 25 µg GFAP mAb treatment group. With the 50 µg dose, these protein expressions showed a similar downward trend, but without statistical significance. ( G ) Expression profiling of inflammation-associated mediators showed that 25 µg GFAP mAb significantly decreased pro-inflammatory factors (TNF-α, IL-1β, IL-8, MMP9, and IFN-γ), and increased the anti-inflammatory cytokine IL-10 compared to the vehicle group. In contrast, the 50 µg dose significantly reduced IFN-γ only. ( H ) Representative images of spots showing IL-1β levels in the subarrays for each group. Image analysis was conducted with Imagene software, using the median intensity from each spot to calculate the mean of the triplicate spots for each marker. For CD68 and TLR4, due to significant heterogeneity in SDs, statistical analysis was performed using Welch’s ANOVA followed by the Tamhane T2 post hoc test. All other data were analyzed using one-way ANOVA with Tukey’s post hoc test. Data are presented as the mean ± SD; n = 4 per group; * p < 0.05, ** p < 0.01, *** p < 0.001; ns = not significant. TLR4 – Toll-Like Receptor 4. S100A8 – S100 Calcium Binding Protein A8. NLRP3 – NLR Family Pyrin Domain Containing 3. GSDMD – Gasdermin D

Article Snippet: Briefly, the microarray was prepared using a noncontact microarray printer (SciFLEXARRAYER S3; Scienion, Berlin, Germany).

Techniques: Microarray, Expressing, Marker, Activation Assay, Software, Binding Assay