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plexon inc linear electrode arrays
Linear Electrode Arrays, supplied by plexon inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/linear+electrode+array/linear+electrode+array/pm40198709-207-18-21
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Article Title: Multiplexing of cognitive encoding by oculomotor networks leads to incidental gaze shifts.
Article Snippet: 122 No. 15 e2422331122 https://doi.org/10.1073/pnas.2422331122 9 of 11 Recordings were conducted from single tungsten electrodes (FHC Inc.) or linear electrode arrays (Plexon; interelectrode spacing ≥ 50 μm, 16 to 32 channels), lowered through a grid (Crist Instruments or NAN instruments) in dura- piercing guide tubes via a microdrive system (NAN Instruments, David Kopf Instruments).


Article Title: A neural mechanism for conserved value computations integrating information and rewards.
Article Snippet: Electrophysiological recordings were performed using multicontact electrodes (Plexon V-probes, 32 channels, 50-μm spacing) inserted through a stainless steel guide tube and advanced by an oil-driven micromanipulator (MO-97A, Narishige).

Article Title: Functional architecture of executive control and associated event-related potentials in macaques
Article Snippet: Electroencephalogram (EEG) was recorded over the MFC with leads placed on the cranial surface beside the chamber while a linear electrode array (Plexon, 24 channels, 150 μm spacing) was inserted in SEF (Fig. ).

Article Title: Model-based characterization of the selectivity of neurons in primary visual cortex.
Article Snippet: In one animal, we implanted a 96-electrode planar Utah array (Blackrock Microsystems; 400 μm spacing), in the other, a linear electrode array (V-probe, Plexon; 24 contacts, 50μm spacing) was passed through the dura at the start of each day.

Magnetic Resonance Imaging:

Article Title: Decision-related activity and movement selection in primate visual cortex
Article Snippet: .. Recordings were performed with a linear electrode array (Plexon V-Probe) that was positioned so as to record from area MT, which was identified based on anatomical MRI scans, electrode depth, and the prevalence of strong, direction-selective visual responses. ..

Article Title: Decision-related activity and movement selection in primate visual cortex.
Article Snippet: .. Recordings were performed with a linear electrode array (Plexon V-Probe) that was positioned so as to record from area MT, which was identi ed based on anatomical magnetic resonance imaging scans, electrode depth, and the prevalence of strong, direction-selective visual responses. ..

Activity Assay:

Article Title: A context-dependent switch from sensing to feeling in the primate amygdala
Article Snippet: .. Single-unit spiking activity was recorded using linear electrode arrays (V-probes, Plexon Inc, Dallas, TX) with 16 (16 sessions) or 32 (32 sessions) equidistant contacts at 400 μm (16 contacts) or 200 μm (32 contacts) separation along a 260 μm diameter shaft. .. Data were collected using Plexon OmniPlex data acquisition hardware and software (RRID:SCR_014803).



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SWA propagation axis are controlled by excitable populations in vivo (A) Schematic representation of the in vivo recording setup. (B) Representative example of the SWA activity recorded. Each color represents and individual shank, placed as shown in (A). Shaded traces were recorded in the cortex and used for further analysis. (C) Example of the detection of Up states, shown as faint red bars, based on the calculation of the log MUA of the recorded trace, shown in orange and blue respectively. (D) Relative delay of each shank along the Up states of one recordings session. Negative values are shown in the shanks on which the Up state was recorded first. Note how after NE + Cch are released, the more caudal shank (orange) shifts from positive to negative delays, while the contrary occurs for the rest of the shanks, as the wave becomes predominantly caudorostral. (E) Time lag matrix showing all the cortical <t>electrodes</t> and Up states recorded in a session. Note how following the release the caudal shank (orange) shifts from positive to negative delays, meaning that it becomes the first shank on which the Ups are detected. (F) Change in the ratio of rostrocaudal Up states following the release in all the recorded sessions. There is a significant ( p = 0.039) decrease in the amount of rostrocaudal Up states. Control recordings, on which saline was released as a substitute of the NE + CCh cocktail, do not show a significant change in the directionality of the SWA. p values calculated using Wilcoxon signed-rank test ( p value <0.05).
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SWA propagation axis are controlled by excitable populations in vivo (A) Schematic representation of the in vivo recording setup. (B) Representative example of the SWA activity recorded. Each color represents and individual shank, placed as shown in (A). Shaded traces were recorded in the cortex and used for further analysis. (C) Example of the detection of Up states, shown as faint red bars, based on the calculation of the log MUA of the recorded trace, shown in orange and blue respectively. (D) Relative delay of each shank along the Up states of one recordings session. Negative values are shown in the shanks on which the Up state was recorded first. Note how after NE + Cch are released, the more caudal shank (orange) shifts from positive to negative delays, while the contrary occurs for the rest of the shanks, as the wave becomes predominantly caudorostral. (E) Time lag matrix showing all the cortical <t>electrodes</t> and Up states recorded in a session. Note how following the release the caudal shank (orange) shifts from positive to negative delays, meaning that it becomes the first shank on which the Ups are detected. (F) Change in the ratio of rostrocaudal Up states following the release in all the recorded sessions. There is a significant ( p = 0.039) decrease in the amount of rostrocaudal Up states. Control recordings, on which saline was released as a substitute of the NE + CCh cocktail, do not show a significant change in the directionality of the SWA. p values calculated using Wilcoxon signed-rank test ( p value <0.05).
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SWA propagation axis are controlled by excitable populations in vivo (A) Schematic representation of the in vivo recording setup. (B) Representative example of the SWA activity recorded. Each color represents and individual shank, placed as shown in (A). Shaded traces were recorded in the cortex and used for further analysis. (C) Example of the detection of Up states, shown as faint red bars, based on the calculation of the log MUA of the recorded trace, shown in orange and blue respectively. (D) Relative delay of each shank along the Up states of one recordings session. Negative values are shown in the shanks on which the Up state was recorded first. Note how after NE + Cch are released, the more caudal shank (orange) shifts from positive to negative delays, while the contrary occurs for the rest of the shanks, as the wave becomes predominantly caudorostral. (E) Time lag matrix showing all the cortical <t>electrodes</t> and Up states recorded in a session. Note how following the release the caudal shank (orange) shifts from positive to negative delays, meaning that it becomes the first shank on which the Ups are detected. (F) Change in the ratio of rostrocaudal Up states following the release in all the recorded sessions. There is a significant ( p = 0.039) decrease in the amount of rostrocaudal Up states. Control recordings, on which saline was released as a substitute of the NE + CCh cocktail, do not show a significant change in the directionality of the SWA. p values calculated using Wilcoxon signed-rank test ( p value <0.05).
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SWA propagation axis are controlled by excitable populations in vivo (A) Schematic representation of the in vivo recording setup. (B) Representative example of the SWA activity recorded. Each color represents and individual shank, placed as shown in (A). Shaded traces were recorded in the cortex and used for further analysis. (C) Example of the detection of Up states, shown as faint red bars, based on the calculation of the log MUA of the recorded trace, shown in orange and blue respectively. (D) Relative delay of each shank along the Up states of one recordings session. Negative values are shown in the shanks on which the Up state was recorded first. Note how after NE + Cch are released, the more caudal shank (orange) shifts from positive to negative delays, while the contrary occurs for the rest of the shanks, as the wave becomes predominantly caudorostral. (E) Time lag matrix showing all the cortical <t>electrodes</t> and Up states recorded in a session. Note how following the release the caudal shank (orange) shifts from positive to negative delays, meaning that it becomes the first shank on which the Ups are detected. (F) Change in the ratio of rostrocaudal Up states following the release in all the recorded sessions. There is a significant ( p = 0.039) decrease in the amount of rostrocaudal Up states. Control recordings, on which saline was released as a substitute of the NE + CCh cocktail, do not show a significant change in the directionality of the SWA. p values calculated using Wilcoxon signed-rank test ( p value <0.05).
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SWA propagation axis are controlled by excitable populations in vivo (A) Schematic representation of the in vivo recording setup. (B) Representative example of the SWA activity recorded. Each color represents and individual shank, placed as shown in (A). Shaded traces were recorded in the cortex and used for further analysis. (C) Example of the detection of Up states, shown as faint red bars, based on the calculation of the log MUA of the recorded trace, shown in orange and blue respectively. (D) Relative delay of each shank along the Up states of one recordings session. Negative values are shown in the shanks on which the Up state was recorded first. Note how after NE + Cch are released, the more caudal shank (orange) shifts from positive to negative delays, while the contrary occurs for the rest of the shanks, as the wave becomes predominantly caudorostral. (E) Time lag matrix showing all the cortical <t>electrodes</t> and Up states recorded in a session. Note how following the release the caudal shank (orange) shifts from positive to negative delays, meaning that it becomes the first shank on which the Ups are detected. (F) Change in the ratio of rostrocaudal Up states following the release in all the recorded sessions. There is a significant ( p = 0.039) decrease in the amount of rostrocaudal Up states. Control recordings, on which saline was released as a substitute of the NE + CCh cocktail, do not show a significant change in the directionality of the SWA. p values calculated using Wilcoxon signed-rank test ( p value <0.05).
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SWA propagation axis are controlled by excitable populations in vivo (A) Schematic representation of the in vivo recording setup. (B) Representative example of the SWA activity recorded. Each color represents and individual shank, placed as shown in (A). Shaded traces were recorded in the cortex and used for further analysis. (C) Example of the detection of Up states, shown as faint red bars, based on the calculation of the log MUA of the recorded trace, shown in orange and blue respectively. (D) Relative delay of each shank along the Up states of one recordings session. Negative values are shown in the shanks on which the Up state was recorded first. Note how after NE + Cch are released, the more caudal shank (orange) shifts from positive to negative delays, while the contrary occurs for the rest of the shanks, as the wave becomes predominantly caudorostral. (E) Time lag matrix showing all the cortical <t>electrodes</t> and Up states recorded in a session. Note how following the release the caudal shank (orange) shifts from positive to negative delays, meaning that it becomes the first shank on which the Ups are detected. (F) Change in the ratio of rostrocaudal Up states following the release in all the recorded sessions. There is a significant ( p = 0.039) decrease in the amount of rostrocaudal Up states. Control recordings, on which saline was released as a substitute of the NE + CCh cocktail, do not show a significant change in the directionality of the SWA. p values calculated using Wilcoxon signed-rank test ( p value <0.05).
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SWA propagation axis are controlled by excitable populations in vivo (A) Schematic representation of the in vivo recording setup. (B) Representative example of the SWA activity recorded. Each color represents and individual shank, placed as shown in (A). Shaded traces were recorded in the cortex and used for further analysis. (C) Example of the detection of Up states, shown as faint red bars, based on the calculation of the log MUA of the recorded trace, shown in orange and blue respectively. (D) Relative delay of each shank along the Up states of one recordings session. Negative values are shown in the shanks on which the Up state was recorded first. Note how after NE + Cch are released, the more caudal shank (orange) shifts from positive to negative delays, while the contrary occurs for the rest of the shanks, as the wave becomes predominantly caudorostral. (E) Time lag matrix showing all the cortical electrodes and Up states recorded in a session. Note how following the release the caudal shank (orange) shifts from positive to negative delays, meaning that it becomes the first shank on which the Ups are detected. (F) Change in the ratio of rostrocaudal Up states following the release in all the recorded sessions. There is a significant ( p = 0.039) decrease in the amount of rostrocaudal Up states. Control recordings, on which saline was released as a substitute of the NE + CCh cocktail, do not show a significant change in the directionality of the SWA. p values calculated using Wilcoxon signed-rank test ( p value <0.05).

Journal: iScience

Article Title: Global and local nature of cortical slow waves

doi: 10.1016/j.isci.2025.113213

Figure Lengend Snippet: SWA propagation axis are controlled by excitable populations in vivo (A) Schematic representation of the in vivo recording setup. (B) Representative example of the SWA activity recorded. Each color represents and individual shank, placed as shown in (A). Shaded traces were recorded in the cortex and used for further analysis. (C) Example of the detection of Up states, shown as faint red bars, based on the calculation of the log MUA of the recorded trace, shown in orange and blue respectively. (D) Relative delay of each shank along the Up states of one recordings session. Negative values are shown in the shanks on which the Up state was recorded first. Note how after NE + Cch are released, the more caudal shank (orange) shifts from positive to negative delays, while the contrary occurs for the rest of the shanks, as the wave becomes predominantly caudorostral. (E) Time lag matrix showing all the cortical electrodes and Up states recorded in a session. Note how following the release the caudal shank (orange) shifts from positive to negative delays, meaning that it becomes the first shank on which the Ups are detected. (F) Change in the ratio of rostrocaudal Up states following the release in all the recorded sessions. There is a significant ( p = 0.039) decrease in the amount of rostrocaudal Up states. Control recordings, on which saline was released as a substitute of the NE + CCh cocktail, do not show a significant change in the directionality of the SWA. p values calculated using Wilcoxon signed-rank test ( p value <0.05).

Article Snippet: One-third of the original induction dose was injected intramuscularly to maintain anesthesia once paw reflex could be evoked or the LFP trace exhibited signs of awakening, approximately every 2 h. Extracellular activity was then recorded by two linear silicon probe electrodes (NeuroNexus Technologies, Ann Arbor, MI).

Techniques: In Vivo, Activity Assay, Control, Saline