software package version 2.0 (Broad Institute Inc)
90
Structured Review
Broad Institute Inc
software package version 2.0
Software Package Version 2.0, supplied by Broad Institute Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/java+desktop+software/java+desktop+software+version+2+0/pmc05344538-315-9-14
Average 90 stars, based on 1 article reviews
Software Package Version 2.0, supplied by Broad Institute Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/java+desktop+software/java+desktop+software+version+2+0/pmc05344538-315-9-14
Average 90 stars, based on 1 article reviews
software package version 2.0 - by Bioz Stars,
2026-09
90/100 stars
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Software:Article Title: Tumor Necrosis Factor-α Gene Signature Is Absent in Peripheral Blood Mononuclear Cells of Patients with Granulomatosis Polyangiitis Article Snippet: pISSN: 2093-940X, eISSN: 2233-4718 Copyright c 2015 by The Korean College of Rheumatology.. All rights reserved.. This is a Free Access article, which permits unrestricted non-commerical use, distribution, and reproduction in any medium, provided the original work is properly cited. Article Title: The transcriptome of HIV-1 infected intestinal CD4+ T cells exposed to enteric bacteria Article Snippet: Gene set enrichment analysis (GSEA) was performed using an open-source software package (version 2.0, Broad Institute http://www.broad.mit.edu/gsea ) [ ]. Article Title: Dysregulation of the causative genes for hereditary parkinsonism in the midbrain in Parkinson's disease. Article Snippet: Background and Object ives: Many hereditary movement disorders with complex phenotypes without a locus symbol prefix for familial PD present as parkinsonism; however, the dysregulation of genes associated with these phenotypes in the SNpc of PD patients has not been systematically studied.. Methods: Gene set enrichment analyses were performed using 10 previously published genome-wide expression datasets obtained by laser-captured microdissection of pigmented neurons in the SNpc.. A custom-curated gene set for hereditary parkinsonism consisting of causative genes (n 5 78) related to disorders with a parkinsonism phenotype, but not necessarily idiopathic or monogenic PD, was constructed from the Online Mendelian Inheritance in Man database. Standard Deviation:Article Title: Tumor Necrosis Factor-α Gene Signature Is Absent in Peripheral Blood Mononuclear Cells of Patients with Granulomatosis Polyangiitis Article Snippet: pISSN: 2093-940X, eISSN: 2233-4718 Copyright c 2015 by The Korean College of Rheumatology.. All rights reserved.. This is a Free Access article, which permits unrestricted non-commerical use, distribution, and reproduction in any medium, provided the original work is properly cited. Article Title: The transcriptome of HIV-1 infected intestinal CD4+ T cells exposed to enteric bacteria Article Snippet: Gene set enrichment analysis (GSEA) was performed using an open-source software package (version 2.0, Broad Institute http://www.broad.mit.edu/gsea ) [ ]. Article Title: Dysregulation of the causative genes for hereditary parkinsonism in the midbrain in Parkinson's disease. Article Snippet: Background and Object ives: Many hereditary movement disorders with complex phenotypes without a locus symbol prefix for familial PD present as parkinsonism; however, the dysregulation of genes associated with these phenotypes in the SNpc of PD patients has not been systematically studied.. Methods: Gene set enrichment analyses were performed using 10 previously published genome-wide expression datasets obtained by laser-captured microdissection of pigmented neurons in the SNpc.. A custom-curated gene set for hereditary parkinsonism consisting of causative genes (n 5 78) related to disorders with a parkinsonism phenotype, but not necessarily idiopathic or monogenic PD, was constructed from the Online Mendelian Inheritance in Man database. |
![Unbiased gene set enrichment analysis <t>(GSEA)</t> and muscular nicotinamide adenine dinucleotide (NAD + ) levels. (A) Volcano plot summarizing the results of unbiased GSEA with the normalized enrichment score (NES) and nominal P-value [−log 10 (P-val)]. Pink dots indicates statistically significantly altered gene sets. (B) Bubble plot highlighting representative gene sets linked to lactate metabolism. It indicates that for each gene set, the depth of the purple color indicates the nominal P-value, and the size of the node indicates the size. (C) Representative enrichment plot generated by GSEA related with lactate metabolism. NES, nominal (Nom) P-values, and false discovery rate (FDR) Q values are indicated. (D) Heatmap displaying representative genes of three genes, lactate metabolism, Mitochondrial electron transport NADH to ubiquinone, and Inner mitochondrial membrane protein complex. (E) NAD + levels in gastrocnemius. All values are represented as mean ± standard deviation, and the P-values were determined by one-way ANOVA followed by Tukey’s test. P-values of < 0.01 (**), and < 0.0001 (****) were considered statistically significant.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_5566/pmc10315566/pmc10315566__bmb-56-6-353-f4.jpg)
