cdna microarray genechip human genome hg-u133a (Thermo Fisher)
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cdna microarray genechip human genome hg-u133a
Cdna Microarray Genechip Human Genome Hg U133a, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+cdna+microarray/seebluetm+plus2+pre+stained+protein+standard/pmc12070984-99-0-7
Average 90 stars, based on 1 article reviews
Cdna Microarray Genechip Human Genome Hg U133a, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+cdna+microarray/seebluetm+plus2+pre+stained+protein+standard/pmc12070984-99-0-7
Average 90 stars, based on 1 article reviews
cdna microarray genechip human genome hg-u133a - by Bioz Stars,
2026-11
90/100 stars
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Molecular Weight:Article Title: A Quantitative High-Throughput Screen Identifies Compounds that Upregulate the p53 Isoform Δ133p53α and Inhibit Cellular Senescence Article Snippet: Δ133p53α, a human/primate-specific p53 protein isoform, delays or inhibits the induction of the cellular senescence and associated secretory phenotype (SASP) in various types of human cells, including astrocytes from neurodegenerative diseases, Hutchinson−Gilford progeria syndrome (HGPS) fibroblasts, and exhausted CD8+ T-cells.. A major regulatory mechanism of Δ133p53α expression in these human cells is protein degradation via chaperone-assisted selective autophagy (CASA).. 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The estimation of bands was performed using a Marker:Article Title: A Quantitative High-Throughput Screen Identifies Compounds that Upregulate the p53 Isoform Δ133p53α and Inhibit Cellular Senescence Article Snippet: Δ133p53α, a human/primate-specific p53 protein isoform, delays or inhibits the induction of the cellular senescence and associated secretory phenotype (SASP) in various types of human cells, including astrocytes from neurodegenerative diseases, Hutchinson−Gilford progeria syndrome (HGPS) fibroblasts, and exhausted CD8+ T-cells.. A major regulatory mechanism of Δ133p53α expression in these human cells is protein degradation via chaperone-assisted selective autophagy (CASA).. 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