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diff-quick cell staining kit (Dade Behring)

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Dade Behring diff-quick cell staining kit
Diff Quick Cell Staining Kit, supplied by Dade Behring, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/diff+quick+kit/diff+quick+staining+kit/pm24759231-67-16-20
Average 90 stars, based on 1 article reviews

diff-quick cell staining kit - by Bioz Stars, 2026-11

90/100 stars

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Article Title: Semaphorin 3F inhibits breast cancer metastasis by regulating the Akt-mTOR and TGFβ signaling pathways via neuropilin-2.
Article Snippet: Cells that had migrated through the filters after 16 h at 37 °C were stained with the Diff-Quick cell staining kit (Dade Behring Inc.), and four fields were counted by phase microscopy.

Article Title: AZIN1 level is increased in medulloblastoma and correlates with c-Myc activity and tumor phenotype
Article Snippet: After 24 h, cells that had migrated through the filters after 20 h at 37 °C were stained with the Diff-Quick Cell Staining Kit (Dade Behring Inc.), and four fields were counted by phase microscopy.

Article Title: Semaphorin 3F inhibits breast cancer metastasis by regulating the Akt-mTOR and TGFβ signaling pathways via neuropilin-2
Article Snippet: Cells that had migrated through the filters after 16 h at 37 °C were stained with the Diff-Quick cell staining kit (Dade Behring Inc.), and four fields were counted by phase microscopy.

Article Title: Kinase function of TgTKL1 is essential for its role in Toxoplasma propagation and pathogenesis
Article Snippet: To visualize intact and lysed vacuoles, the cultures were stained using Diff-Quik (Dade-Behring) according to the manufacturer’s instructions.

Article Title: Integrative Molecular and Functional Analysis of Human Sperm Subpopulations to Identify New Biomarkers of Fertilization Potential.
Article Snippet: a Departamento de Biología de la Reproducción Dr. Carlos Gual Castro, Instituto Nacional de Ciencias Médicas y Nutrición Salvador Zubirán, Mexico City, Mexico b Fundación Tambre, Madrid, Spain c Center for Reproductive Sciences, Department of Obstetrics and Gynecology, University of California School of Medicine, San Francisco, CA, USA d Center for Neuroscience and Cell Biology, University of Coimbra, Coimbra, Portugal e Weill Institute for Neurosciences, Department of Neurological Surgery, University of California San Francisco School of Medicine, San Francisco, CA, USA f Department of Genetics, The McDonnell Genome Institute, Washington University School of Medicine, St. Louis, MO, USA g Department of Microbiology and Immunology, University of British Columbia, Vancouver, British Columbia, Canada h Department of Life Sciences, University of Coimbra, Coimbra, Portugal

Article Title: AZIN1 level is increased in medulloblastoma and correlates with c-Myc activity and tumor phenotype.
Article Snippet: After 24 h, cells that had migrated through the filters after 20 h at 37 °C were stained with the Diff-Quick Cell Staining Kit (Dade Behring Inc.), and four fields were counted by phase microscopy.

Membrane:

Article Title: IL-13Rα2 Is Involved in Resistance to Doxorubicin and Survival of Osteosarcoma Patients
Article Snippet: .. Cells that migrated or invaded to the lower side of the membrane were stained with Diff Quick stain solution (#468.037.1A, Diff-Quick, Dade Behring Inc., Newark, DE, USA). ..

Staining:

Diff-Quik:

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$RBM23 knockdown inhibits colon cancer cell growth and survival (A) Protein was extracted from the normal colon cell lines CCD-18Co and colon cancer cell lines (HCT116, HT29, LOVO, LS513, DLD-1, COLO205, HCT15) and quantified in equal amounts, and then the protein expression of RBM23 was detected by western blotting. (B) The mRNA expression level of RBM23 was measured by qRT-PCR in the normal colon cell line CCD-18Co and colon cancer cell lines (DLD-1, HCT15, HT29, HCT116). GAPDH was used as an internal control for normalization. (C) Western blot analysis was performed to measure the expression levels of c-Myc in HCT15 cells treated with control siRNA or RBM23 siRNA for 72 h. (D) HCT116 cells were treated with control siRNA or RBM23 siRNA #1, #2, #3 for 72 h. Protein expression levels of various factors were then analyzed by western blotting. (E) The effect of RBM23 deficiency on HCT116, a colon cancer cell line. HCT116 was transfected with 80 nM RBM23 siRNA and control siRNA, and cell viability was examined at 0 and 72 h of treatment. Cell viability was measured by CCK8 assay and optical density was measured by microplate reader at 450 nm. Data were normalized to the control siRNA group at 0 h (set as 100%). (F) The effect of RBM23 knockdown in colon cancer cells, HCT116, on cell proliferation. HCT116 cells were transfected with RBM23 siRNA (100 nM) and incubated for one week, and colonies were stained with Diff Quick solution. The colonies were counted and quantified. (G) Western blotting was performed to detect proteins extracted from HCT116 cells fractionated into nucleus and cytoplasm. Proteins from HCT116 cells transfected with RBM23 siRNA (100 nM) for 72 h were extracted and separated by NE-PER nuclear and cytoplasmic extraction kit into proteins extracted from the nucleus and cytoplasm. The proteins were then detected using western blotting. Student’s t -test: ***P < 0.001$

Journal: Cancer Cell International

Article Title: Inhibition of RBM23 induces ferroptosis in colon cancer cells via c-Myc regulation

doi: 10.1186/s12935-026-04262-x

Figure Lengend Snippet: RBM23 knockdown inhibits colon cancer cell growth and survival (A) Protein was extracted from the normal colon cell lines CCD-18Co and colon cancer cell lines (HCT116, HT29, LOVO, LS513, DLD-1, COLO205, HCT15) and quantified in equal amounts, and then the protein expression of RBM23 was detected by western blotting. (B) The mRNA expression level of RBM23 was measured by qRT-PCR in the normal colon cell line CCD-18Co and colon cancer cell lines (DLD-1, HCT15, HT29, HCT116). GAPDH was used as an internal control for normalization. (C) Western blot analysis was performed to measure the expression levels of c-Myc in HCT15 cells treated with control siRNA or RBM23 siRNA for 72 h. (D) HCT116 cells were treated with control siRNA or RBM23 siRNA #1, #2, #3 for 72 h. Protein expression levels of various factors were then analyzed by western blotting. (E) The effect of RBM23 deficiency on HCT116, a colon cancer cell line. HCT116 was transfected with 80 nM RBM23 siRNA and control siRNA, and cell viability was examined at 0 and 72 h of treatment. Cell viability was measured by CCK8 assay and optical density was measured by microplate reader at 450 nm. Data were normalized to the control siRNA group at 0 h (set as 100%). (F) The effect of RBM23 knockdown in colon cancer cells, HCT116, on cell proliferation. HCT116 cells were transfected with RBM23 siRNA (100 nM) and incubated for one week, and colonies were stained with Diff Quick solution. The colonies were counted and quantified. (G) Western blotting was performed to detect proteins extracted from HCT116 cells fractionated into nucleus and cytoplasm. Proteins from HCT116 cells transfected with RBM23 siRNA (100 nM) for 72 h were extracted and separated by NE-PER nuclear and cytoplasmic extraction kit into proteins extracted from the nucleus and cytoplasm. The proteins were then detected using western blotting. Student’s t -test: ***P < 0.001

Article Snippet: The plates were then incubated at 37 °C, 5% CO 2 for 1 week and the colonies were fixed and stained using Diff-Quick Kit (Sysmex Corporation, Kobe, Hyogo, Japan) and the number of colonies was counted.

Techniques: Knockdown, Expressing, Western Blot, Quantitative RT-PCR, Control, Transfection, CCK-8 Assay, Incubation, Staining, Diff-Quik, Extraction