computer-based morphometric analysis image 1 (universal imaging inc)
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universal imaging inc
computer-based morphometric analysis image 1
Computer Based Morphometric Analysis Image 1, supplied by universal imaging inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/computer-based+morphometric+analysis/computer+assisted+morphometry+image+1/pmc08668279-62-9-12
Average 90 stars, based on 1 article reviews
Computer Based Morphometric Analysis Image 1, supplied by universal imaging inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/computer-based+morphometric+analysis/computer+assisted+morphometry+image+1/pmc08668279-62-9-12
Average 90 stars, based on 1 article reviews
computer-based morphometric analysis image 1 - by Bioz Stars,
2026-09
90/100 stars
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other:Article Title: Glycyrrhizin Attenuates Toll Like Receptor-2, -4 and Experimental Vasospasm in a Rat Model Article Snippet: Measurement of the BA cross-sectional area was made using an automatic Article Title: Valproic acid attenuates intercellular adhesion molecule-1 and E-selectin through a chemokine ligand 5 dependent mechanism and subarachnoid hemorrhage induced vasospasm in a rat model Article Snippet: Automated measurements of the luminal cross-sectional area were made using Article Title: Attenuation in Proinflammatory Factors and Reduction in Neuronal Cell Apoptosis and Cerebral Vasospasm by Minocycline during Early Phase after Subarachnoid Hemorrhage in the Rat Article Snippet: The arterial Article Title: Purine anti-metabolite attenuates nuclear factor κB and related pro-inflammatory cytokines in experimental vasospasm. Article Snippet: Background Increased nuclear factor κB (NF-κB) bioexpression, as well as TNF-α, IL-1β and IL-6 levels, were observed after aneurysmal subarachnoid hemorrhage (SAH).. It is of interest to investigate the effect of 6-mercaptopurine (6-mp) on cytokines/NF-κB in this SAH model. Materials and methods A rodent double-hemorrhage SAH model was employed.. Serum and cerebrospinal fluid (CSF) samples were collected to examine IL-1, IL-6 and TNF-α levels. Article Title: Attenuation of experimental subarachnoid hemorrhage--induced cerebral vasospasm by the adenosine A2A receptor agonist CGS 21680. Article Snippet: J. Neurosurg.. / Volume 106 / March, 2007 DENOSINE is a ubiquitous molecule that plays a pivotal role in a number of physiological and pathophysiological conditions in mammals.49 One of the primary roles of adenosine within the cardiovascular system appears to be the direct regulation of both cardiac and vascular function.. Effects of adenosine are mediated by four G protein–coupled receptors: A1, A2A, A2B, and A3. Article Title: 17Beta-estradiol activates adenosine A(2a) receptor after subarachnoid hemorrhage. Article Snippet: *Department of Neurosurgery, Kaohsiung Medical University Hospital, Kaohsiung, Taiwan; †Faculty of Medicine, Graduate Institute of Medicine, College of Medicine, Kaohsiung Medical University, Kaohsiung, Taiwan; ‡Departments of Neuroscience and Neurological Surgery, University of Virginia Health System, Charlottesville, Virginia; §Division of Plastic and Reconstructive Surgery, Department of Surgery, Kaohsiung Medical University Hospital, Kaohsiung, Taiwan; kGraduate Institute of Biochemistry, Kaohsiung Medical University, Kaohsiung, Taiwan; and {Department of Clinical Research, Kaohsiung Medical University Hospital, Kaohsiung, Taiwan, ROC Article Title: Attenuation of cerebral vasospasm and secondary injury by 17beta-estradiol following experimental subarachnoid hemorrhage. Article Snippet: 457 Although cerebral vasospasm after aneurysmal SAH has been recognized for over half of a century, it remains a major complication in patients suffering from SAH.3 As no consistently effective therapy for vasospasm exits, the pathophysiological mechanisms contributing to this problem must be elucidated.. Unlike other forms of stroke, aneurysmal SAH occurs more frequently in women than in men.5 However, the notion of a sex difference in SAH outcome is controversial, and the influence of the female sex hormone is unclear.8 Estrogens have been found to induce vasodilation in the cardiovascular system via both genomic and nongenomic mechanisms through the generation of NO, cyclic guanosine monophosphate, cyclic adenosine monophosphate, adenosine, and prostacycline or alterations in ion channel activity.4,6 Our previous studies have shown that E2 can attenuate experimental SAH–induced cerebral vasospasm,11 inhibit endothelin-1 production,10 and prevent the augmentation of inducible NO synthase expression by interfering with nuclear factor–kappa B transactivation11,19 while also preserving normal endothelial NO synthase expression after SAH.11 Moreover, estrogens appear to have neuroprotective properties that reduce neuronal apoptosis and secondary injury following cerebral ischemia.2,13,20,23 Our previous work has also demonstrated that the Attenuation of cerebral vasospasm and secondary injury by 17β-estradiol following experimental subarachnoid hemorrhage |