bluescript sk1 vector (Promega)
90
Structured Review
Promega
bluescript sk1 vector
Bluescript Sk1 Vector, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/bluescript+vector/bluescript+sk1+vector/pm09374111-140-32-35
Average 90 stars, based on 1 article reviews
Bluescript Sk1 Vector, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/bluescript+vector/bluescript+sk1+vector/pm09374111-140-32-35
Average 90 stars, based on 1 article reviews
bluescript sk1 vector - by Bioz Stars,
2026-09
90/100 stars
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Clone Assay:Article Title: Transforming growth factor-beta1 is a potent inhibitor of glutathione synthesis in the lung epithelial cell line A549: transcriptional effect on the GSH rate-limiting enzyme gamma-glutamylcysteine synthetase. Article Snippet: Glutathione (GSH) is an essential antioxidant tripeptide that protects mammalian cells against oxidants and xenobiotics.. Patients with fibrotic lung disorders have very low levels of GSH in their alveolar epithelial lining fluid (ELF), whereas transforming growth factor (TGF)b is overexpressed in their alveolar epithelial cells.. We observed that TGFb 1 increased susceptibility of the human alveolar epithelial cell line A549 to H 2 O 2 -mediated cytotoxicity ( P , 0.05), decreased the activities of the antioxidant enzymes glutathione reductase and catalase by 31%, and markedly decreased GSH content in A549 cells ( P , 0.01). Plasmid Preparation:Article Title: Transforming growth factor-beta1 is a potent inhibitor of glutathione synthesis in the lung epithelial cell line A549: transcriptional effect on the GSH rate-limiting enzyme gamma-glutamylcysteine synthetase. Article Snippet: Glutathione (GSH) is an essential antioxidant tripeptide that protects mammalian cells against oxidants and xenobiotics.. Patients with fibrotic lung disorders have very low levels of GSH in their alveolar epithelial lining fluid (ELF), whereas transforming growth factor (TGF)b is overexpressed in their alveolar epithelial cells.. We observed that TGFb 1 increased susceptibility of the human alveolar epithelial cell line A549 to H 2 O 2 -mediated cytotoxicity ( P , 0.05), decreased the activities of the antioxidant enzymes glutathione reductase and catalase by 31%, and markedly decreased GSH content in A549 cells ( P , 0.01). Hybridization:Article Title: Transforming growth factor-beta1 is a potent inhibitor of glutathione synthesis in the lung epithelial cell line A549: transcriptional effect on the GSH rate-limiting enzyme gamma-glutamylcysteine synthetase. Article Snippet: Glutathione (GSH) is an essential antioxidant tripeptide that protects mammalian cells against oxidants and xenobiotics.. Patients with fibrotic lung disorders have very low levels of GSH in their alveolar epithelial lining fluid (ELF), whereas transforming growth factor (TGF)b is overexpressed in their alveolar epithelial cells.. We observed that TGFb 1 increased susceptibility of the human alveolar epithelial cell line A549 to H 2 O 2 -mediated cytotoxicity ( P , 0.05), decreased the activities of the antioxidant enzymes glutathione reductase and catalase by 31%, and markedly decreased GSH content in A549 cells ( P , 0.01). Control:Article Title: Transforming growth factor-beta1 is a potent inhibitor of glutathione synthesis in the lung epithelial cell line A549: transcriptional effect on the GSH rate-limiting enzyme gamma-glutamylcysteine synthetase. Article Snippet: Glutathione (GSH) is an essential antioxidant tripeptide that protects mammalian cells against oxidants and xenobiotics.. Patients with fibrotic lung disorders have very low levels of GSH in their alveolar epithelial lining fluid (ELF), whereas transforming growth factor (TGF)b is overexpressed in their alveolar epithelial cells.. We observed that TGFb 1 increased susceptibility of the human alveolar epithelial cell line A549 to H 2 O 2 -mediated cytotoxicity ( P , 0.05), decreased the activities of the antioxidant enzymes glutathione reductase and catalase by 31%, and markedly decreased GSH content in A549 cells ( P , 0.01). |