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The effects of METH on neuron differentiation by immunofluorescence. The <t>NSE</t> positive cells decreased (A) <t>while</t> <t>GFAP</t> positive cells (B) increased in the striatum, hippocampus, and NAc after METH treatment.
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The effects of METH on neuron differentiation by immunofluorescence. The <t>NSE</t> positive cells decreased (A) <t>while</t> <t>GFAP</t> positive cells (B) increased in the striatum, hippocampus, and NAc after METH treatment.
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The effects of METH on neuron differentiation by immunofluorescence. The <t>NSE</t> positive cells decreased (A) <t>while</t> <t>GFAP</t> positive cells (B) increased in the striatum, hippocampus, and NAc after METH treatment.
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The effects of METH on neuron differentiation by immunofluorescence. The <t>NSE</t> positive cells decreased (A) <t>while</t> <t>GFAP</t> positive cells (B) increased in the striatum, hippocampus, and NAc after METH treatment.
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NKG2D ligands are induced in the lungs of mice infected with P. aeruginosa. (A) Western blot analysis of <t>RAE-1</t> in whole lungs of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 0, 8, and 24 h. The blot is representative of results obtained with four to six mice per group. (B to E) NKG2D ligand expression in lung epithelial cells and alveolar macrophages of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 24 h. The accumulation of NKG2D ligands in the lungs of CF1 mice was assessed by immunohistochemistry analysis using paraffin-embedded sections and a goat polyclonal antibody against <t>RAE-1γ.</t> (B) Large airway of PBS-treated mouse. (C) Large airway of P. aeruginosa-infected mouse. (D) Lung parenchyma of PBS-treated mouse. (E) Lung parenchyma of P. aeruginosa-infected mouse. The photomicrographs are representative of five mice per group. Original magnification, ×400.
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NKG2D ligands are induced in the lungs of mice infected with P. aeruginosa. (A) Western blot analysis of <t>RAE-1</t> in whole lungs of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 0, 8, and 24 h. The blot is representative of results obtained with four to six mice per group. (B to E) NKG2D ligand expression in lung epithelial cells and alveolar macrophages of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 24 h. The accumulation of NKG2D ligands in the lungs of CF1 mice was assessed by immunohistochemistry analysis using paraffin-embedded sections and a goat polyclonal antibody against <t>RAE-1γ.</t> (B) Large airway of PBS-treated mouse. (C) Large airway of P. aeruginosa-infected mouse. (D) Lung parenchyma of PBS-treated mouse. (E) Lung parenchyma of P. aeruginosa-infected mouse. The photomicrographs are representative of five mice per group. Original magnification, ×400.
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NKG2D ligands are induced in the lungs of mice infected with P. aeruginosa. (A) Western blot analysis of <t>RAE-1</t> in whole lungs of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 0, 8, and 24 h. The blot is representative of results obtained with four to six mice per group. (B to E) NKG2D ligand expression in lung epithelial cells and alveolar macrophages of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 24 h. The accumulation of NKG2D ligands in the lungs of CF1 mice was assessed by immunohistochemistry analysis using paraffin-embedded sections and a goat polyclonal antibody against <t>RAE-1γ.</t> (B) Large airway of PBS-treated mouse. (C) Large airway of P. aeruginosa-infected mouse. (D) Lung parenchyma of PBS-treated mouse. (E) Lung parenchyma of P. aeruginosa-infected mouse. The photomicrographs are representative of five mice per group. Original magnification, ×400.
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R&D Systems mouse il 12p40
NKG2D ligands are induced in the lungs of mice infected with P. aeruginosa. (A) Western blot analysis of <t>RAE-1</t> in whole lungs of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 0, 8, and 24 h. The blot is representative of results obtained with four to six mice per group. (B to E) NKG2D ligand expression in lung epithelial cells and alveolar macrophages of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 24 h. The accumulation of NKG2D ligands in the lungs of CF1 mice was assessed by immunohistochemistry analysis using paraffin-embedded sections and a goat polyclonal antibody against <t>RAE-1γ.</t> (B) Large airway of PBS-treated mouse. (C) Large airway of P. aeruginosa-infected mouse. (D) Lung parenchyma of PBS-treated mouse. (E) Lung parenchyma of P. aeruginosa-infected mouse. The photomicrographs are representative of five mice per group. Original magnification, ×400.
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R&D Systems pan jnk antibody
NKG2D ligands are induced in the lungs of mice infected with P. aeruginosa. (A) Western blot analysis of <t>RAE-1</t> in whole lungs of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 0, 8, and 24 h. The blot is representative of results obtained with four to six mice per group. (B to E) NKG2D ligand expression in lung epithelial cells and alveolar macrophages of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 24 h. The accumulation of NKG2D ligands in the lungs of CF1 mice was assessed by immunohistochemistry analysis using paraffin-embedded sections and a goat polyclonal antibody against <t>RAE-1γ.</t> (B) Large airway of PBS-treated mouse. (C) Large airway of P. aeruginosa-infected mouse. (D) Lung parenchyma of PBS-treated mouse. (E) Lung parenchyma of P. aeruginosa-infected mouse. The photomicrographs are representative of five mice per group. Original magnification, ×400.
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R&D Systems mouse il
NKG2D ligands are induced in the lungs of mice infected with P. aeruginosa. (A) Western blot analysis of <t>RAE-1</t> in whole lungs of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 0, 8, and 24 h. The blot is representative of results obtained with four to six mice per group. (B to E) NKG2D ligand expression in lung epithelial cells and alveolar macrophages of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 24 h. The accumulation of NKG2D ligands in the lungs of CF1 mice was assessed by immunohistochemistry analysis using paraffin-embedded sections and a goat polyclonal antibody against <t>RAE-1γ.</t> (B) Large airway of PBS-treated mouse. (C) Large airway of P. aeruginosa-infected mouse. (D) Lung parenchyma of PBS-treated mouse. (E) Lung parenchyma of P. aeruginosa-infected mouse. The photomicrographs are representative of five mice per group. Original magnification, ×400.
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R&D Systems anti mouse pan specific rae1
NKG2D ligands are induced in the lungs of mice infected with P. aeruginosa. (A) Western blot analysis of <t>RAE-1</t> in whole lungs of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 0, 8, and 24 h. The blot is representative of results obtained with four to six mice per group. (B to E) NKG2D ligand expression in lung epithelial cells and alveolar macrophages of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 24 h. The accumulation of NKG2D ligands in the lungs of CF1 mice was assessed by immunohistochemistry analysis using paraffin-embedded sections and a goat polyclonal antibody against <t>RAE-1γ.</t> (B) Large airway of PBS-treated mouse. (C) Large airway of P. aeruginosa-infected mouse. (D) Lung parenchyma of PBS-treated mouse. (E) Lung parenchyma of P. aeruginosa-infected mouse. The photomicrographs are representative of five mice per group. Original magnification, ×400.
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R&D Systems anti ciap
NKG2D ligands are induced in the lungs of mice infected with P. aeruginosa. (A) Western blot analysis of <t>RAE-1</t> in whole lungs of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 0, 8, and 24 h. The blot is representative of results obtained with four to six mice per group. (B to E) NKG2D ligand expression in lung epithelial cells and alveolar macrophages of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 24 h. The accumulation of NKG2D ligands in the lungs of CF1 mice was assessed by immunohistochemistry analysis using paraffin-embedded sections and a goat polyclonal antibody against <t>RAE-1γ.</t> (B) Large airway of PBS-treated mouse. (C) Large airway of P. aeruginosa-infected mouse. (D) Lung parenchyma of PBS-treated mouse. (E) Lung parenchyma of P. aeruginosa-infected mouse. The photomicrographs are representative of five mice per group. Original magnification, ×400.
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Image Search Results


The effects of METH on neuron differentiation by immunofluorescence. The NSE positive cells decreased (A) while GFAP positive cells (B) increased in the striatum, hippocampus, and NAc after METH treatment.

Journal: Pharmaceutical Biology

Article Title: Methamphetamine leads to the alterations of microRNA profiles in the nucleus accumbens of rats

doi: 10.1080/13880209.2020.1803366

Figure Lengend Snippet: The effects of METH on neuron differentiation by immunofluorescence. The NSE positive cells decreased (A) while GFAP positive cells (B) increased in the striatum, hippocampus, and NAc after METH treatment.

Article Snippet: The primary antibody of NSE (neuron specific enolase, catalog No. AF5169) and GFAP (glial fibrillary acidic protein, catalog No. AF2594) was obtained from the R&D system (Minneapolis, MN, USA).

Techniques: Immunofluorescence

NKG2D ligands are induced in the lungs of mice infected with P. aeruginosa. (A) Western blot analysis of RAE-1 in whole lungs of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 0, 8, and 24 h. The blot is representative of results obtained with four to six mice per group. (B to E) NKG2D ligand expression in lung epithelial cells and alveolar macrophages of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 24 h. The accumulation of NKG2D ligands in the lungs of CF1 mice was assessed by immunohistochemistry analysis using paraffin-embedded sections and a goat polyclonal antibody against RAE-1γ. (B) Large airway of PBS-treated mouse. (C) Large airway of P. aeruginosa-infected mouse. (D) Lung parenchyma of PBS-treated mouse. (E) Lung parenchyma of P. aeruginosa-infected mouse. The photomicrographs are representative of five mice per group. Original magnification, ×400.

Journal:

Article Title: The NKG2D-Activating Receptor Mediates Pulmonary Clearance of Pseudomonas aeruginosa

doi: 10.1128/IAI.74.5.2578-2586.2006

Figure Lengend Snippet: NKG2D ligands are induced in the lungs of mice infected with P. aeruginosa. (A) Western blot analysis of RAE-1 in whole lungs of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 0, 8, and 24 h. The blot is representative of results obtained with four to six mice per group. (B to E) NKG2D ligand expression in lung epithelial cells and alveolar macrophages of mice exposed to ∼1 × 106 CFU of P. aeruginosa for 24 h. The accumulation of NKG2D ligands in the lungs of CF1 mice was assessed by immunohistochemistry analysis using paraffin-embedded sections and a goat polyclonal antibody against RAE-1γ. (B) Large airway of PBS-treated mouse. (C) Large airway of P. aeruginosa-infected mouse. (D) Lung parenchyma of PBS-treated mouse. (E) Lung parenchyma of P. aeruginosa-infected mouse. The photomicrographs are representative of five mice per group. Original magnification, ×400.

Article Snippet: The membranes were incubated overnight with anti-RAE-1γ (1-μg/ml dilution; clone AF1136; R&D Systems, Minneapolis, MN).

Techniques: Infection, Western Blot, Expressing, Immunohistochemistry