slc2a2 Search Results


89
Thermo Fisher gene exp slc2a2 hs01096908 m1
Gene Exp Slc2a2 Hs01096908 M1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 89/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 89 stars, based on 1 article reviews
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93
Thermo Fisher gene exp slc2a2 mm00446224 m1
Gene Exp Slc2a2 Mm00446224 M1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
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87
Thermo Fisher gene exp slc2a2 mm00446230 g1
Gene Exp Slc2a2 Mm00446230 G1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 87/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 87 stars, based on 1 article reviews
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95
Thermo Fisher gene exp slc2a2 hs01096905 m1
Gene Exp Slc2a2 Hs01096905 M1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 95 stars, based on 1 article reviews
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94
Thermo Fisher gene exp slc2a2 mm00446229 m1
Gene Exp Slc2a2 Mm00446229 M1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
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94
Proteintech anti human mouse glut2 polyclonal antibody
Metabolic profile assessment: (A) Fasting blood sugar (FBS) was assessed as indicated in materials and methods. <t>GLUT2</t> expressions were quantitated by (B) western blot and (C) RT-PCR methods. (D) Serum insulin C-peptide measured by ELISA. (E) Hepatic expression of inulin receptor (IR) was evaluated from liver sections by western blot (F) HOMA2 calculator calculated HOMA-IR. Paired and unpaired Student’s t -test and ANOVA were used for statistically significant differences. p value was compared between RD and HFD control groups or within RD or HFD groups. * p < 0.05; ** p < 0.01; *** p < 0.001; **** p < 0.0001.
Anti Human Mouse Glut2 Polyclonal Antibody, supplied by Proteintech, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
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94
Thermo Fisher gene exp slc2a2 rn00563565 m1
Figure 4. mRNA abundance of <t>Glut2,</t> Gck, Ins1, Ins2, Beta2 and Pdx1 in INS-1E cells treated with caffeic acid, naringenin or quercetin in presence of 11 or 25 mM glucose for 72 h were studied by real time RT-PCR using TaqMan® assays (ABI, Foster City, CA, USA). Duplicate samples were taken for each treatment and the samples were measured in triplicates. Gene expressions were normalized to 18 S ribosomal RNA. Difference in the mRNA abundance was calculated compared with their respective untreated controls. Open and closed bars represent the cells grown in control conditions or in presence of the compounds. *p < 0.05, **p < 0.01, ***p < 0.001.
Gene Exp Slc2a2 Rn00563565 M1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 94 stars, based on 1 article reviews
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94/100 stars
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92
Aviva Systems rabbit anti glut2
Figure 4. mRNA abundance of <t>Glut2,</t> Gck, Ins1, Ins2, Beta2 and Pdx1 in INS-1E cells treated with caffeic acid, naringenin or quercetin in presence of 11 or 25 mM glucose for 72 h were studied by real time RT-PCR using TaqMan® assays (ABI, Foster City, CA, USA). Duplicate samples were taken for each treatment and the samples were measured in triplicates. Gene expressions were normalized to 18 S ribosomal RNA. Difference in the mRNA abundance was calculated compared with their respective untreated controls. Open and closed bars represent the cells grown in control conditions or in presence of the compounds. *p < 0.05, **p < 0.01, ***p < 0.001.
Rabbit Anti Glut2, supplied by Aviva Systems, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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86
Thermo Fisher gene exp slc2a2 hs00165775 m1
Figure 4. mRNA abundance of <t>Glut2,</t> Gck, Ins1, Ins2, Beta2 and Pdx1 in INS-1E cells treated with caffeic acid, naringenin or quercetin in presence of 11 or 25 mM glucose for 72 h were studied by real time RT-PCR using TaqMan® assays (ABI, Foster City, CA, USA). Duplicate samples were taken for each treatment and the samples were measured in triplicates. Gene expressions were normalized to 18 S ribosomal RNA. Difference in the mRNA abundance was calculated compared with their respective untreated controls. Open and closed bars represent the cells grown in control conditions or in presence of the compounds. *p < 0.05, **p < 0.01, ***p < 0.001.
Gene Exp Slc2a2 Hs00165775 M1, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 86 stars, based on 1 article reviews
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Image Search Results


Metabolic profile assessment: (A) Fasting blood sugar (FBS) was assessed as indicated in materials and methods. GLUT2 expressions were quantitated by (B) western blot and (C) RT-PCR methods. (D) Serum insulin C-peptide measured by ELISA. (E) Hepatic expression of inulin receptor (IR) was evaluated from liver sections by western blot (F) HOMA2 calculator calculated HOMA-IR. Paired and unpaired Student’s t -test and ANOVA were used for statistically significant differences. p value was compared between RD and HFD control groups or within RD or HFD groups. * p < 0.05; ** p < 0.01; *** p < 0.001; **** p < 0.0001.

Journal: Frontiers in Nutrition

Article Title: High-fat diet mouse model receiving L-glucose supplementations propagates liver injury

doi: 10.3389/fnut.2024.1469952

Figure Lengend Snippet: Metabolic profile assessment: (A) Fasting blood sugar (FBS) was assessed as indicated in materials and methods. GLUT2 expressions were quantitated by (B) western blot and (C) RT-PCR methods. (D) Serum insulin C-peptide measured by ELISA. (E) Hepatic expression of inulin receptor (IR) was evaluated from liver sections by western blot (F) HOMA2 calculator calculated HOMA-IR. Paired and unpaired Student’s t -test and ANOVA were used for statistically significant differences. p value was compared between RD and HFD control groups or within RD or HFD groups. * p < 0.05; ** p < 0.01; *** p < 0.001; **** p < 0.0001.

Article Snippet: Rabbit anti-Human/Mouse GYS2 (Proteintech, 22371-1-AP), Rabbit anti-Human/Mouse Glycogen synthase [p Ser641] (Novus bio, NBP2-67315), rabbit anti-human/mouse PYGL antibody (Proteintech, 15851-1-AP), rabbit anti-human/mouse Glut2 polyclonal antibody (Proteintech, 20436-1-AP), rabbit anti-human/mouse ADRP/Perilipin 2 Polyclonal antibody (Proteintech, 15294-1-AP), rabbit anti-human/mouse Alpha Smooth Muscle antibody (Novus, NBP1-30894), mice anti-human/mouse AKT antibody (R&D, MAB 2055), mice anti-human/mouse phospho-AKT antibody (R&D, MAB 887), rabbit anti-human/mouse Insulin Receptor-beta antibody (Proteintech, 20433-1-AP), and rabbit anti-human/mouse beta Actin polyclonal antibody (Proteintech, 20536-1-AP).

Techniques: Western Blot, Reverse Transcription Polymerase Chain Reaction, Enzyme-linked Immunosorbent Assay, Expressing, Control

Figure 4. mRNA abundance of Glut2, Gck, Ins1, Ins2, Beta2 and Pdx1 in INS-1E cells treated with caffeic acid, naringenin or quercetin in presence of 11 or 25 mM glucose for 72 h were studied by real time RT-PCR using TaqMan® assays (ABI, Foster City, CA, USA). Duplicate samples were taken for each treatment and the samples were measured in triplicates. Gene expressions were normalized to 18 S ribosomal RNA. Difference in the mRNA abundance was calculated compared with their respective untreated controls. Open and closed bars represent the cells grown in control conditions or in presence of the compounds. *p < 0.05, **p < 0.01, ***p < 0.001.

Journal: Diabetes, obesity & metabolism

Article Title: Caffeic acid, naringenin and quercetin enhance glucose-stimulated insulin secretion and glucose sensitivity in INS-1E cells.

doi: 10.1111/dom.12236

Figure Lengend Snippet: Figure 4. mRNA abundance of Glut2, Gck, Ins1, Ins2, Beta2 and Pdx1 in INS-1E cells treated with caffeic acid, naringenin or quercetin in presence of 11 or 25 mM glucose for 72 h were studied by real time RT-PCR using TaqMan® assays (ABI, Foster City, CA, USA). Duplicate samples were taken for each treatment and the samples were measured in triplicates. Gene expressions were normalized to 18 S ribosomal RNA. Difference in the mRNA abundance was calculated compared with their respective untreated controls. Open and closed bars represent the cells grown in control conditions or in presence of the compounds. *p < 0.05, **p < 0.01, ***p < 0.001.

Article Snippet: The TaqMan assays used for the PCR were Glut2 (assay Rn00563565_m1), Ins1 (assay Rn02121433_g1), Ins2 (assay Rn01774648_g1), Akt1 (assay Rn00583646_m1), Akt2 (assay Rn00690900_m1), Irs1 (assay Rn02132493_s1), Pdx1 (assay Rn00755591_m1), Beta2 (assay Rn00824571_s1), Acc1 (assay Rn00573474_m1), Gck (assay Rn00561265_m1), Casp3 (assay Rn00563902_ m1), Bax (assay Rn01480160_g1), Bcl2 (assay Rn99999 125_m1), Hsp 70 (assay Rn04224718_u1) and Hsp 90 (assay Rn00822023_g1).

Techniques: Quantitative RT-PCR, Control