scc15 cells Search Results


tongue squamous cell carcinoma cell line scc-15  (JCRB Cell Bank)
90
JCRB Cell Bank tongue squamous cell carcinoma cell line scc-15
Tongue Squamous Cell Carcinoma Cell Line Scc 15, supplied by JCRB Cell Bank, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/scc15+cells/pmc07894009-21-3-17?v=JCRB+Cell+Bank
Average 90 stars, based on 1 article reviews
tongue squamous cell carcinoma cell line scc-15 - by Bioz Stars, 2026-07
90/100 stars
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scc-15-fluc cells  (HFK Bioscience)
90
HFK Bioscience scc-15-fluc cells
Scc 15 Fluc Cells, supplied by HFK Bioscience, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/scc15+cells/pmc07933933-103-4-18?v=HFK+Bioscience
Average 90 stars, based on 1 article reviews
scc-15-fluc cells - by Bioz Stars, 2026-07
90/100 stars
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scc15, cancer cells  (National Centre for Cell Science)
90
National Centre for Cell Science scc15, cancer cells
Scc15, Cancer Cells, supplied by National Centre for Cell Science, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/scc15+cells/10__4103_slash_jcrt__jcrt_263_18-51-10-15?v=National+Centre+for+Cell+Science
Average 90 stars, based on 1 article reviews
scc15, cancer cells - by Bioz Stars, 2026-07
90/100 stars
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scc15 cell line  (Ribobio co)
90
Ribobio co scc15 cell line
STAT3 contributes to invasion and migration of OSCC cells. (A) Western blot detection of STAT3, p-STAT3 (Tyr705), MMP2 and MMP9 expression. GAPDH was used as a loading control. (B) MTT analysis was used to determine the IC 50 values for both OSCC cell lines. (C) Targeting STAT3 significantly inhibited SCC25 and <t>SCC15</t> cell invasion and migration ability, as determined by Transwell assay (magnification, ×40). (D) Cell migration ability was measured by a wound-healing assay (magnification, ×40). *** P<0.001 vs. EV group; ### P<0.001 EV + Stattic group vs. STAT3 group. EV, empty vector; IC 50 , half maximal inhibitory concentration; MMP, matrix metalloproteinase; OSCC, oral squamous cell carcinoma; p-STAT3, phosphorylated-STAT3; STAT3, signal transducer and activator of transcription 3.
Scc15 Cell Line, supplied by Ribobio co, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/scc15+cells/pmc05843395-133-2-17?v=Ribobio+co
Average 90 stars, based on 1 article reviews
scc15 cell line - by Bioz Stars, 2026-07
90/100 stars
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scc15 cells transduced with luciferase lentivirus  (Promega)
90
Promega scc15 cells transduced with luciferase lentivirus
STAT3 contributes to invasion and migration of OSCC cells. (A) Western blot detection of STAT3, p-STAT3 (Tyr705), MMP2 and MMP9 expression. GAPDH was used as a loading control. (B) MTT analysis was used to determine the IC 50 values for both OSCC cell lines. (C) Targeting STAT3 significantly inhibited SCC25 and <t>SCC15</t> cell invasion and migration ability, as determined by Transwell assay (magnification, ×40). (D) Cell migration ability was measured by a wound-healing assay (magnification, ×40). *** P<0.001 vs. EV group; ### P<0.001 EV + Stattic group vs. STAT3 group. EV, empty vector; IC 50 , half maximal inhibitory concentration; MMP, matrix metalloproteinase; OSCC, oral squamous cell carcinoma; p-STAT3, phosphorylated-STAT3; STAT3, signal transducer and activator of transcription 3.
Scc15 Cells Transduced With Luciferase Lentivirus, supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/scc15+cells/pm37828451-97-16-18?v=Promega
Average 90 stars, based on 1 article reviews
scc15 cells transduced with luciferase lentivirus - by Bioz Stars, 2026-07
90/100 stars
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ut-scc-15  (SAS institute)
90
SAS institute ut-scc-15
STAT3 contributes to invasion and migration of OSCC cells. (A) Western blot detection of STAT3, p-STAT3 (Tyr705), MMP2 and MMP9 expression. GAPDH was used as a loading control. (B) MTT analysis was used to determine the IC 50 values for both OSCC cell lines. (C) Targeting STAT3 significantly inhibited SCC25 and <t>SCC15</t> cell invasion and migration ability, as determined by Transwell assay (magnification, ×40). (D) Cell migration ability was measured by a wound-healing assay (magnification, ×40). *** P<0.001 vs. EV group; ### P<0.001 EV + Stattic group vs. STAT3 group. EV, empty vector; IC 50 , half maximal inhibitory concentration; MMP, matrix metalloproteinase; OSCC, oral squamous cell carcinoma; p-STAT3, phosphorylated-STAT3; STAT3, signal transducer and activator of transcription 3.
Ut Scc 15, supplied by SAS institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/scc15+cells/pm21641070-47-8-11?v=SAS+institute
Average 90 stars, based on 1 article reviews
ut-scc-15 - by Bioz Stars, 2026-07
90/100 stars
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Image Search Results


STAT3 contributes to invasion and migration of OSCC cells. (A) Western blot detection of STAT3, p-STAT3 (Tyr705), MMP2 and MMP9 expression. GAPDH was used as a loading control. (B) MTT analysis was used to determine the IC 50 values for both OSCC cell lines. (C) Targeting STAT3 significantly inhibited SCC25 and SCC15 cell invasion and migration ability, as determined by Transwell assay (magnification, ×40). (D) Cell migration ability was measured by a wound-healing assay (magnification, ×40). *** P<0.001 vs. EV group; ### P<0.001 EV + Stattic group vs. STAT3 group. EV, empty vector; IC 50 , half maximal inhibitory concentration; MMP, matrix metalloproteinase; OSCC, oral squamous cell carcinoma; p-STAT3, phosphorylated-STAT3; STAT3, signal transducer and activator of transcription 3.

Journal: International Journal of Oncology

Article Title: Role of the EZH2/miR-200 axis in STAT3-mediated OSCC invasion

doi: 10.3892/ijo.2018.4293

Figure Lengend Snippet: STAT3 contributes to invasion and migration of OSCC cells. (A) Western blot detection of STAT3, p-STAT3 (Tyr705), MMP2 and MMP9 expression. GAPDH was used as a loading control. (B) MTT analysis was used to determine the IC 50 values for both OSCC cell lines. (C) Targeting STAT3 significantly inhibited SCC25 and SCC15 cell invasion and migration ability, as determined by Transwell assay (magnification, ×40). (D) Cell migration ability was measured by a wound-healing assay (magnification, ×40). *** P<0.001 vs. EV group; ### P<0.001 EV + Stattic group vs. STAT3 group. EV, empty vector; IC 50 , half maximal inhibitory concentration; MMP, matrix metalloproteinase; OSCC, oral squamous cell carcinoma; p-STAT3, phosphorylated-STAT3; STAT3, signal transducer and activator of transcription 3.

Article Snippet: SCC25 and SCC15 cell lines were transfected with NC siRNA or siRNA against EZH2 (20 nM; Guangzhou Ribobio Co., Ltd., Guangzhou, China), which were labeled as si-NC (5′-UUCUCCGAACGUGUCACGU-3′) or si-EZH2 (#1, 5′-GCUGGAAUCAAAGGAUACA-3′; #2, 5′-GTGCCCTTGTGTGATAGCACAA-3′; #3, 5′-GGCACT TTCATTGAAGAACTAA-3′), respectively, using Lipo-fectamine 2000 reagent, according to the manufacturer's protocol.

Techniques: Migration, Western Blot, Expressing, Control, Transwell Assay, Wound Healing Assay, Plasmid Preparation, Concentration Assay

EZH2/miR-200/b/a/429 axis regulates the invasiveness of OSCC cells in vitro . (A) Following transfection with EZH2 siRNAs (si#1 and si#2) for 3 days, the expression levels of EZH2 were detected using western blotting. (B) OSCC cells underwent western blot analysis using antibodies specific to STAT3, p-STAT3 (Tyr705), EZH2, H3K27me3, H3 and GAPDH. EZH2 siRNA markedly inhibited EZH2 and H3K27me3 expression. In addition, p-STAT3 (Tyr705), but not STAT3, was suppressed by EZH2 depletion. (C) EZH2 siRNA induced miR-200b/a/429 expression. (D) Results of a Transwell assay demonstrated that EZH2 knockdown markedly attenuated migration (without Matrigel) and invasion (with Matrigel) of SCC25 and SCC15 cells (scale bar, 100 μ m; magnification, ×40). (E) Scratch assay demonstrated that EZH2 silencing markedly delayed wound healing in SCC25 and SCC15 cells (magnification, ×40). *P<0.05 and *** P<0.001 vs. si-NC group. EZH2, enhancer of zeste homolog 3; H3, histone 3; H3K27me3, trimethylation of lysine 27 in H3; miR-200b/a/429, microRNA-200b, -200a and -429; NC, negative control; OSCC, oral squamous cell carcinoma; p-STAT3, phosphorylated-STAT3; siRNA/si, small interfering RNA; STAT3, signal transducer and activator of transcription 3.

Journal: International Journal of Oncology

Article Title: Role of the EZH2/miR-200 axis in STAT3-mediated OSCC invasion

doi: 10.3892/ijo.2018.4293

Figure Lengend Snippet: EZH2/miR-200/b/a/429 axis regulates the invasiveness of OSCC cells in vitro . (A) Following transfection with EZH2 siRNAs (si#1 and si#2) for 3 days, the expression levels of EZH2 were detected using western blotting. (B) OSCC cells underwent western blot analysis using antibodies specific to STAT3, p-STAT3 (Tyr705), EZH2, H3K27me3, H3 and GAPDH. EZH2 siRNA markedly inhibited EZH2 and H3K27me3 expression. In addition, p-STAT3 (Tyr705), but not STAT3, was suppressed by EZH2 depletion. (C) EZH2 siRNA induced miR-200b/a/429 expression. (D) Results of a Transwell assay demonstrated that EZH2 knockdown markedly attenuated migration (without Matrigel) and invasion (with Matrigel) of SCC25 and SCC15 cells (scale bar, 100 μ m; magnification, ×40). (E) Scratch assay demonstrated that EZH2 silencing markedly delayed wound healing in SCC25 and SCC15 cells (magnification, ×40). *P<0.05 and *** P<0.001 vs. si-NC group. EZH2, enhancer of zeste homolog 3; H3, histone 3; H3K27me3, trimethylation of lysine 27 in H3; miR-200b/a/429, microRNA-200b, -200a and -429; NC, negative control; OSCC, oral squamous cell carcinoma; p-STAT3, phosphorylated-STAT3; siRNA/si, small interfering RNA; STAT3, signal transducer and activator of transcription 3.

Article Snippet: SCC25 and SCC15 cell lines were transfected with NC siRNA or siRNA against EZH2 (20 nM; Guangzhou Ribobio Co., Ltd., Guangzhou, China), which were labeled as si-NC (5′-UUCUCCGAACGUGUCACGU-3′) or si-EZH2 (#1, 5′-GCUGGAAUCAAAGGAUACA-3′; #2, 5′-GTGCCCTTGTGTGATAGCACAA-3′; #3, 5′-GGCACT TTCATTGAAGAACTAA-3′), respectively, using Lipo-fectamine 2000 reagent, according to the manufacturer's protocol.

Techniques: In Vitro, Transfection, Expressing, Western Blot, Transwell Assay, Knockdown, Migration, Wound Healing Assay, Negative Control, Small Interfering RNA