polyclonal antibody Search Results


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  • 97
    Cell Signaling Technology Inc β actin
    β Actin, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/β actin/product/Cell Signaling Technology Inc
    Average 97 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    β actin - by Bioz Stars, 2022-10
    97/100 stars
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    80
    Cell Signaling Technology Inc akt
    Effect of tempol on the expression of <t>PKC/Nrf2</t> and <t>PI3K/Akt/Nrf2</t> pathways induced by ischemia/reperfusion. Data are expressed as mean ± SD from 5 independent animals (n=5). * p
    Akt, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 80/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/akt/product/Cell Signaling Technology Inc
    Average 80 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    akt - by Bioz Stars, 2022-10
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    99
    Cell Signaling Technology Inc anti β actin
    Time course effect of clindamycin on the phosphorylation of p70S6K in glioblastoma cell lines. Time course analysis of clindamycin (CLD) on p70S6K phosphorylation was determined in glioblastoma cell lines ( A , C , E , G ). Cells were assigned to the indicated times and cultured with CLD (440 μM) and subjected to Western blotting. The levels of phosphorylation and total p70S6K were quantified after standardizing the ratio of phospho/total p70S6K ( B , D , F , H ). <t>β-actin</t> was used as the internal control level in all cases. Data are shown as the mean ± SE, similar results were obtained from three independent experiments. Each group was compared to the 0 min level to determine the statistical significance (* p
    Anti β Actin, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/anti β actin/product/Cell Signaling Technology Inc
    Average 99 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    anti β actin - by Bioz Stars, 2022-10
    99/100 stars
      Buy from Supplier

    99
    Cell Signaling Technology Inc anti akt
    Time course effect of clindamycin on the phosphorylation of p70S6K in glioblastoma cell lines. Time course analysis of clindamycin (CLD) on p70S6K phosphorylation was determined in glioblastoma cell lines ( A , C , E , G ). Cells were assigned to the indicated times and cultured with CLD (440 μM) and subjected to Western blotting. The levels of phosphorylation and total p70S6K were quantified after standardizing the ratio of phospho/total p70S6K ( B , D , F , H ). <t>β-actin</t> was used as the internal control level in all cases. Data are shown as the mean ± SE, similar results were obtained from three independent experiments. Each group was compared to the 0 min level to determine the statistical significance (* p
    Anti Akt, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/anti akt/product/Cell Signaling Technology Inc
    Average 99 stars, based on 1 article reviews
    Price from $9.99 to $1999.99
    anti akt - by Bioz Stars, 2022-10
    99/100 stars
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    Image Search Results


    Effect of tempol on the expression of PKC/Nrf2 and PI3K/Akt/Nrf2 pathways induced by ischemia/reperfusion. Data are expressed as mean ± SD from 5 independent animals (n=5). * p

    Journal: Kidney & blood pressure research

    Article Title: Protective Effect of Tempol on Acute Kidney Injury Through PI3K/Akt/Nrf2 Signaling Pathway

    doi: 10.1159/000443414

    Figure Lengend Snippet: Effect of tempol on the expression of PKC/Nrf2 and PI3K/Akt/Nrf2 pathways induced by ischemia/reperfusion. Data are expressed as mean ± SD from 5 independent animals (n=5). * p

    Article Snippet: Proteins were transferred to an immobilon polyvinylidene difluoride membrane for 1h at 50 V. Membranes were blocked in 20 mM Tris-HCl (PH 7.4), 150 mM NaCl, and 0.1% Tween 20 (TBS-T) containing 5% fat-free milk powder for 1 h and immunodetected with antibodies to Nrf2 (polyclonal antibody; 1:2000), Heme oxygenase-1 (HO-1) (monoclonal antibody; 1:2000), PKC (polyclonal antibody; 1:2000), p-PKC (monoclonal antibody; 1:2000), β-actin (monoclonal antibody; 1:5000) (Abcam Cambridge, UK), Akt (polyclonal antibody; 1:2000), p-Akt (ser473 monoclonal antibody; 1:2000) (Cell Signaling Technology, Beverly, MA, USA) and lamin B1 (monoclonal antibody; 1:1000) (Santa Cruz Biotechnology, Santa Cruz, CA, USA).

    Techniques: Expressing

    Time course effect of clindamycin on the phosphorylation of p70S6K in glioblastoma cell lines. Time course analysis of clindamycin (CLD) on p70S6K phosphorylation was determined in glioblastoma cell lines ( A , C , E , G ). Cells were assigned to the indicated times and cultured with CLD (440 μM) and subjected to Western blotting. The levels of phosphorylation and total p70S6K were quantified after standardizing the ratio of phospho/total p70S6K ( B , D , F , H ). β-actin was used as the internal control level in all cases. Data are shown as the mean ± SE, similar results were obtained from three independent experiments. Each group was compared to the 0 min level to determine the statistical significance (* p

    Journal: Cancers

    Article Title: Novel Repositioning Therapy for Drug-Resistant Glioblastoma: In Vivo Validation Study of Clindamycin Treatment Targeting the mTOR Pathway and Combination Therapy with Temozolomide

    doi: 10.3390/cancers14030770

    Figure Lengend Snippet: Time course effect of clindamycin on the phosphorylation of p70S6K in glioblastoma cell lines. Time course analysis of clindamycin (CLD) on p70S6K phosphorylation was determined in glioblastoma cell lines ( A , C , E , G ). Cells were assigned to the indicated times and cultured with CLD (440 μM) and subjected to Western blotting. The levels of phosphorylation and total p70S6K were quantified after standardizing the ratio of phospho/total p70S6K ( B , D , F , H ). β-actin was used as the internal control level in all cases. Data are shown as the mean ± SE, similar results were obtained from three independent experiments. Each group was compared to the 0 min level to determine the statistical significance (* p

    Article Snippet: Western blotting was performed using anti-phospho-p70S6K (Thr389) (1:500), anti-phospho-S6 (Ser240/244) (1:1000), anti-S6 (1:4000), anti-phospho-4EBP1 (Thr37/46) (1:1000), and anti-β-actin (1:4000) polyclonal antibodies (Cell Signaling Technologies, Danvers, MA, USA).

    Techniques: Cell Culture, Western Blot

    Effect of clindamycin on the phosphorylation of p70S6K and S6 in glioblastoma cell lines. Dose responses of clindamycin (CLD) on the phosphorylation of p70S6K at Thr389 and S6 at Ser240/Ser244 in glioblastoma cell lines. Cells were treated with CLD (0, 110, 220, or 440 μM) for 72 h and subjected to Western blotting. The levels of phosphorylated and total p70S6K were indicated at the corresponding bands ( A , C , E , G ). Western blotting analysis was subjected to densitometric quantification after standardizing the ratio of phospho/total p70S6K (below panel). The phosphorylation and total S6 levels in glioblastoma cell lines were similarly analyzed via Western blotting ( B , D , F , H ). β-actin was used as the internal control level in all cases. Data are shown as the mean ± SE, similar results shown were representative of three independent experiments. Each group was compared to the 0 concentration level to determine statistical significance. (* p

    Journal: Cancers

    Article Title: Novel Repositioning Therapy for Drug-Resistant Glioblastoma: In Vivo Validation Study of Clindamycin Treatment Targeting the mTOR Pathway and Combination Therapy with Temozolomide

    doi: 10.3390/cancers14030770

    Figure Lengend Snippet: Effect of clindamycin on the phosphorylation of p70S6K and S6 in glioblastoma cell lines. Dose responses of clindamycin (CLD) on the phosphorylation of p70S6K at Thr389 and S6 at Ser240/Ser244 in glioblastoma cell lines. Cells were treated with CLD (0, 110, 220, or 440 μM) for 72 h and subjected to Western blotting. The levels of phosphorylated and total p70S6K were indicated at the corresponding bands ( A , C , E , G ). Western blotting analysis was subjected to densitometric quantification after standardizing the ratio of phospho/total p70S6K (below panel). The phosphorylation and total S6 levels in glioblastoma cell lines were similarly analyzed via Western blotting ( B , D , F , H ). β-actin was used as the internal control level in all cases. Data are shown as the mean ± SE, similar results shown were representative of three independent experiments. Each group was compared to the 0 concentration level to determine statistical significance. (* p

    Article Snippet: Western blotting was performed using anti-phospho-p70S6K (Thr389) (1:500), anti-phospho-S6 (Ser240/244) (1:1000), anti-S6 (1:4000), anti-phospho-4EBP1 (Thr37/46) (1:1000), and anti-β-actin (1:4000) polyclonal antibodies (Cell Signaling Technologies, Danvers, MA, USA).

    Techniques: Western Blot, Concentration Assay